ライフサイエンスコーパス: blood pH

1 nt amounts of protons and to maintain stable blood pH.

2 epithelia, contributing to the buffering of blood pH.

3 t longer than 2 wks and lower precannulation blood pH.

4 roblems related to fluids, electrolytes, and blood pH.

5 well as generating new HCO3- for regulating blood pH.

6 and thereby contributes to the regulation of blood pH.

7 onate administration significantly increased blood pH.

8 calcium-phosphorus product, or to changes in blood pH.

9 ing again being dependent on zinc and normal blood pH.

10 lays an important role in maintaining normal blood pH.

11 e blood glucose levels and prevent a fall in blood pH.

12 Blood pH 7.18 was inversely associated with favourable n

13 Decreases in blood pH activate NHE3, the proximal tubular apical memb

14 model to understand the factors influencing blood pH after bicarbonate removal.

15 The average blood pH, after mixing with the replacement solution, wa

16 acidification) accompanied by an increase in blood pH and a decrease in pCO(2) compared to WT litterm

17 IHCA since its effects may be influenced by blood pH and administration timing.

18 y resuscitation (CPR) would be influenced by blood pH and administration timing.

19 on model to provide continuous monitoring of blood pH and arterial CO(2) in between gaps of blood dra

20 er (KME) supplement have been shown to lower blood pH and arterial CO(2), which are important regulat

21 in vasopressor requirements, improvement in blood pH and in parameters of intracranial pressure (ICP

22 d that optimal binding takes place at normal blood pH and is markedly reduced when pH is adjusted wit

23 ) of O(2) and CO(2) were measured as well as blood pH and lactate.

24 the potential implications of fish's unique blood pH and plasma composition on the distribution (D)

25 Rats in Lac + Hyd group had similar blood pH and ROS levels when compared to the M-Post grou

26 mately 50%, and delayed recovery of arterial blood pH and standard [HCO(3) (-)] from their initial de

27 +) excretion, and early recovery of arterial blood pH and standard [HCO(3) (-)].

28 atient trajectories as a function of lactate blood pH and volumes, among other relevant physiological

29 h, five-minute Apgar score, umbilical-artery blood pH, and morbidity due to prematurity for all singl

30 mpaired neurodevelopmental reflexes, altered blood pH, and reduced bodyweight.

31 l systemic roles in renal acid excretion and blood pH balance, male fertility, bone remodeling, synap

32 10His/p.Gln913Arg) in an individual with low blood pH, blindness and neurological signs that resemble

33 Nbce1b/c-null mice have normal blood pH, but exhibit increased mortality, growth retard

34 Respiratory acidosis, a decrease in blood pH caused by a rise in [CO(2)], rapidly triggers a

35 Regulation of blood pH-critical for virtually every facet of life-requ

36 In the pH-uncorrected group, arterial blood pH decreased from 7.41 +/- 0.03 at normocapnia to

37 es of JHCO3 as well as the normal defense of blood pH during whole-body acidosis.

38 ed between SB use and blood pH (Non-SB use x blood pH > 7.18; OR 1.56; 95% CI 1.01-2.41; p value = 0.

39 95% CI 0.45 to 0.94), and pre-ECMO arterial blood pH >7.17 (OR 0.50, 95% CI 0.30 to 0.84) were assoc

40 -) and H(+) production->decreased muscle and blood pH->with increased H(+) buffered by blood [HCO(3)

41 6 +/- 9 torr (7.9 +/- 1.2 kPa) (p < .01) and blood pH had decreased from 7.317 +/- 0.041 to 7.151 +/-

42 e hydronephrosis, low plasma potassium, high blood pH, hypercalciuria, and proteinuria.

43 All changes in blood pH, in health and in disease, occur through change

44 ressant and anti-seizure drugs, fructose and blood pH-in undiluted plasma.

45 HCl-feeding significantly lowered blood pH, increased urea excretion, and increased the ab

46 Blood pH is tightly maintained between 7.35 and 7.45, an

47 -term ( 37 weeks) infant, and umbilical cord blood pH less than 7.0.

48 ) had lactic acidosis defined as an arterial blood pH less than 7.35 and a lactate concentration grea

49 new vision deficits, metabolic acidosis with blood pH </= 7.15, persistent metabolic acidosis despite

50 (OR 3.06, 95% CI 1.42 to 6.58), and arterial blood pH <7.2 (OR 2.23, 95% CI 1.23 to 4.06) were associ

51 eaths per minute (OR, 1.6; 95% CI, 1.1-2.3), blood pH <7.35 (OR, 3.2; 95% CI, 1.8-5.7), blood urea ni

52 t interactions were noted between SB use and blood pH (Non-SB use x blood pH > 7.18; OR 1.56; 95% CI

53 , bicarbonate should be given at an arterial blood pH of < or =7.0.

54 nutes, neonatal seizure, an umbilical-artery blood pH of 7.05 or less with a base deficit of 12 mmol

55 Lithium effects on blood pH or ionized calcium did not correlate with effec

56 lin, glucose and lactate concentrations, and blood pH, PCO2 and HCO3(-) concentration.

57 Arterial blood pH, PCO2, cardiac output, intracranial pressure, a

58 rium caused a transient decrease in arterial blood pH (pHa) and a short but intense burst of afferent

59 so elicited pronounced decreases in arterial blood pH, pO(2) and sO(2) accompanied by pronounced incr

60 ter controlling for recipient factors, donor blood pH positively predicted lung graft survival (OR 1.

61 Experiments in selective breeding for blood pH, sexual drive, body weight, sexual dimorphism i

62 intravenously as a bolus to correct arterial blood pH toward a target arterial pH of 7.40 (dose calcu

63 e risk in term infants with umbilical-artery blood pH values of 7.0 or less (180; 95 percent confiden

64 gar scores of 3 or less and umbilical-artery blood pH values of 7.0 or less was approximately doubled

65 Paired Apgar scores and umbilical-artery blood pH values were determined for 145,627 infants to a

66 In the pH-corrected group, arterial blood pH was 7.42 +/- 0.02 at normocapnia and was mainta

67 n the pH-uncorrected group (n = 6), arterial blood pH was allowed to decreased without treatment.

68 When blood pH was increased from 7.40 to 7.74 by breathing lo

69 Arterial blood pH was measured to determine the level of acidosis

70 A preliminary study of arterial blood pH was performed to confirm acidosis in our model.

71 Arterial blood pH was significantly lower and Paco2 was significa

72 Arterial blood pH with 35% CO2 decreased to the same levels in bo

73 (-) and H(+) production decreased muscle and blood pH with increased H(+) buffered by blood [HCO(3)(-

74 Increasing blood pH with intravenous bicarbonate substantially impr

75 be significantly attenuated by correction of blood pH with the administration of sodium bicarbonate,