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1 ectly regulating actin assembly in the human blood platelet.
2 ific proteins in the unique environment of a blood platelet.
3 egakaryocyte proliferation and production of blood platelets.
4 rough binding to c-Mpl receptor expressed on blood platelets.
5 assembly and extension of filopodia in human blood platelets.
6  endothelial cells and in the secretome from blood platelets.
7 eir organelles, and give an example in human blood platelets.
8 described mechanism explaining the origin of blood platelets.
9 (eQTLs) in derived MKs and 1830 cis-eQTLs in blood platelets.
10 nal stems primarily from red blood cells and blood platelets.
11 pe 2B von Willebrand factor (vWF) binding to blood platelets.
12 ine receptor that controls the production of blood platelets.
13 is while revealing fundamental details about blood platelets.
14 ycoprotein-Ib (GPIb) receptors to cluster on blood platelets.
15 n brain and, outside the CNS, in circulating blood platelets.
16 bules maintains the discoid shape of resting blood platelets.
17 g, branched proplatelets, which remodel into blood platelets.
18 r genetic profiling of highly purified human blood platelets.
19 ase in megakaryocytes without an increase in blood platelets.
20 ng proplatelets, the immediate precursors of blood platelets.
21 DA is primarily stored in and transported by blood platelets.
22 6 associates with CD9 and integrins on human blood platelets.
23  highly expressed in brain, spinal cord, and blood platelets.
24 atelets, the microtubule-based precursors of blood platelets.
25 eceptors were similar to those described for blood platelets.
26 marrow cells that in turn break down to form blood platelets.
27                            Maternal and cord blood platelet 5-HT levels were obtained from all partic
28 patients secondary to transfusion with whole blood platelets (72 reactions), apheresis platelets (2),
29 tion observed, for instance, at the onset of blood platelet activation.
30 ight microscopy was used to visualize canine blood platelet adhesion and aggregation in a parallel pl
31                            The inhibition of blood platelet aggregation and secretion was studied usi
32 this inhibition was borne out in whole human blood platelet aggregation assays.
33  Willebrand factor Ag and ADP-induced, whole-blood platelet aggregation normalized by platelet count
34                                        Whole blood platelet aggregation was enhanced, and plasma fibr
35  a fasting blood sample was taken, and whole-blood platelet aggregation was measured.Measurements and
36 in, activated GPIIb,IIIa and GPIb-IX), whole-blood platelet aggregation, platelet count and hematocri
37 erreactive platelets, as determined by whole blood platelet aggregometry and flow cytometric analysis
38                                        Whole blood platelet aggregometry revealed a significant reduc
39 inhibition based on flow cytometry and whole blood platelet aggregometry.
40  in plasma SPM concentrations and peripheral blood platelet and leukocyte responses.
41 as well as hematologic toxicity (assessed by blood platelet and neutrophil count nadir) (P < 0.05).
42               Myocardial ischaemia activates blood platelets and cardiac sympathetic afferents, which
43          Anaplasma platys infects peripheral blood platelets and causes infectious cyclic thrombocyto
44  its ability to directly regulate binding of blood platelets and cells to extracellular substrata.
45 pression of ephrins and Eph kinases on human blood platelets and explore their role in the formation
46 red in vivo induce spontaneous activation of blood platelets and formation of aggregates, even in the
47                                         Both blood platelets and genetics contribute to the developme
48 a(2)beta(1) expression on megakaryocytes and blood platelets and must thereby modulate collagen-relat
49 acterized by a lack of alpha-granules within blood platelets and progressive bone marrow fibrosis.
50 l time (O(2)(.-)) release in activated human blood platelets and skeletal rat limb muscles following
51 n (GP) IIb-IIIa activation characteristic of blood platelets and that these cultured MKs would be cap
52 y of megakaryocytes, the marrow precursor of blood platelets, and from confusion surrounding the cyto
53 ved that bone marrow CD34(+), CD61(+) cells, blood platelets, and megakaryocytic leukemia cell lines
54 ed by formation of tumor cell complexes with blood platelets, and that metastasis can be attenuated b
55 , the results of the present study show that blood platelets are a rich source of both glycosyltransf
56                                              Blood platelets are anuclear cell fragments that are ess
57                                              Blood platelets are anucleate cell fragments that play a
58                                        Human blood platelets are anucleate cells whose response to ex
59                                              Blood platelets are critical for hemostasis and thrombos
60                                              Blood platelets are crucial in hemostasis, thrombosis, a
61 crotubule coils similar to those observed in blood platelets are detected only at the ends of proplat
62                                              Blood platelets are formed by fragmentation of long memb
63                                              Blood platelets are functional anucleate cells generated
64                                              Blood platelets are involved in primary and secondary he
65                                              Blood platelets are produced by large bone marrow (BM) p
66                                              Blood platelets are produced by megakaryocytes (MKs), th
67                                  Circulating blood platelets are responsible for maintaining hemostas
68                                  Circulating blood platelets are specialized cells that prevent bleed
69 VWF), including its binding interaction with blood platelets, are regulated by the magnitude of appli
70 -1-P), may act as an autocrine stimulator of blood platelets, as it is abundantly stored in platelets
71 ld-type control donor hearts flushed free of blood/platelets before preservation/transplantation.
72          Liver regeneration is stimulated by blood platelets, but the molecular mechanisms involved a
73                                Activation of blood platelets by physiological stimuli (e.g. thrombin,
74                                   Cooling of blood platelets clusters the von Willebrand factor recep
75                                              Blood platelets contain 60-100-mer polymers of phosphate
76                                              Blood platelets contain polyphosphate, and the secretion
77                               Aggregation of blood platelets contributes to the arrest of bleeding at
78 atelets per each transfusion to maintain the blood platelet count >10 x 10/L in stable nonbleeding pa
79                                        Whole-blood platelet count and hematocrit increased by 15% and
80 n, with each phase regulating the peripheral blood platelet count.
81 ere compared in PRP (turbidimetry) and whole blood (platelet counting with an Ultra-Flo 100 Platelet
82 oplanin-positive tumors had lower peripheral blood platelet counts (P < .001) and higher D-dimer leve
83 notyped and the latter were assessed by cord blood platelet counts and cerebral ultrasound scans.
84  A hallmark of Kawasaki disease is increased blood platelet counts and platelet activation, which is
85  clodronate-encapsulated liposomes increases blood platelet counts in FlnA(loxP) PF4-Cre mice and rev
86                To assess critically both the blood platelet counts that prompt a platelet transfusion
87 ate the bone marrow, completely reconstitute blood platelet counts, and contribute to multiple haemat
88                                   Peripheral blood platelet counts, following rhTPO and G-CSF, were i
89 elets daily to maintain normal physiological blood platelet counts.
90 tions are instituted for obesity and reduced blood platelet counts.
91 ring the late stages of megakaryopoiesis and blood platelet development.
92 ed by the heart during hypoxic stress and by blood platelets during thrombus formation.
93                                    The human blood platelet fibrinogen receptor, integrin alphaIIbbet
94 n of megakaryocytes (MKs)--the precursors of blood platelets--from human pluripotent stem cells (hPSC
95                   Specifically, we evaluated blood platelet function, nervous system development, and
96 n, a 100-kDa protein on the surface of human blood platelets has been identified as an ADP receptor.
97 standing of thrombopoiesis--the formation of blood platelets--has improved greatly in the last decade
98                      The crucial function of blood platelets in hemostasis is to prevent blood loss b
99                          Isolated from human blood platelets in octyl glucoside, the alphaIIbbeta3 co
100 acellular ligand binding to alphaIIbbeta3 on blood platelets induces a transmembrane conformational c
101                            Here we show that blood platelets interact with lung cancer cells and that
102                          The discoid form of blood platelets is important to their function in hemost
103 ents are unclear as BDNF released from human blood platelets is the main contributor to the serum lev
104 nalysis of megakaryocytes, the precursors of blood platelets, is limited by their rarity in vivo, and
105 und that T(-)GR(-) F2 mice displayed similar blood platelet levels as that seen in T(-) mice, indicat
106 taPP is enriched in brain and in circulating blood platelets, little is understood of its physiologic
107 novel eye drops from placental tissues: cord blood platelet lysate (CBED) and amniotic membrane extra
108 akaryocytes were identified as the origin of blood platelets; marrow-derived megakaryocytic progenito
109                                 As activated blood platelets mediate the primary response to vascular
110                                    Activated blood platelets mediate the primary response to vascular
111                                   To produce blood platelets, megakaryocytes elaborate proplatelets,
112 about platelet production and whether mature blood platelets might emerge via different mechanisms.
113 phoresis of the samples of solubilized human blood platelets modified by WR-K and then reduced by tri
114 morrhagic necrosis and a precipitous drop in blood platelets observed at 6 h.
115                                              Blood platelets play an essential role in ischemic heart
116      Megakaryocytes, the progenitor cells of blood platelets, play a crucial role in hemostasis by re
117                           Native circulating blood platelets present with a discoid flat morphology m
118  Factor (VWF), which mediates coagulation of blood platelets (primary hemostasis) especially when it
119                    Our results indicate that blood platelets provide a valuable platform for pan-canc
120                                  Circulating blood platelets regulate the initial phase of the hemost
121         In conclusion, this study shows that blood platelets require 2 unique alpha- and beta-tubulin
122 ch could be observed and quantified in whole blood, platelet-rich and platelet-free plasma and in leu
123                                              Blood platelet RNA-sequencing is increasingly used among
124 ell lines (platelet precursor cells) and 290 blood platelet samples from these subjects.
125             Measurement of maternal and cord blood platelet serotonin (5-HT) provides an index of inh
126              Validation with RNA-seq data of blood platelets shows that DDR achieves the superior per
127                               Tumor-educated blood platelets (TEPs) are implicated as central players
128 ed liquid biopsies, including tumor-educated blood platelets (TEPs), have emerged as promising biomar
129 ors are present in both blood leukocytes and blood platelets, the relative levels of P2X1 receptor ex
130 ession levels of the integrin alpha2beta1 on blood platelets through a mechanism that is probably med
131             Mammalian megakaryocytes release blood platelets through a remarkable process of cytoplas
132 l cells and activation-dependent adhesion of blood platelets through direct interaction with integrin
133 Willebrand factor (VWF) mediates adhesion of blood platelets to fibrillar collagen types I, II, and I
134     Various agonists but also chilling cause blood platelets to increase cytosolic calcium, polymeriz
135  plays a fundamental role in the adhesion of blood platelets to the extracellular matrix.
136 ransplant candidates often leads to multiple blood/platelet transfusions and subsequent development o
137 and FAK proteins in megakaryocytic cells and blood platelets was observed.
138 variation in alpha2beta1 expression by human blood platelets was previously described.
139    Because the major source of Sph1P is from blood platelets, we speculate that Edg-mediated Sph1P ne
140  oxygenase activity levels in umbilical cord blood platelets were higher, whereas biliverdin reductas
141 horylation/dephosphorylation events in human blood platelets were investigated during their adhesion
142 n dysregulation of 5-HT, we examined whether blood platelets, which have 5-HT and 14-3-3 signaling pa
143               Myocardial ischaemia activates blood platelets, which in turn stimulate cardiac sympath
144 cessed, resulting in spontaneous adhesion of blood platelets, which presents as severe, life-threaten
145 s 1a and 1b induced the aggregation of human blood platelets with a potency similar to type I collage

 
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