ライフサイエンスコーパス: blood pressure

1 lower BMI reducing risk of diabetes and high blood pressure.

2 6 mm Hg (95% CI (-1.39, -0.13)) in diastolic blood pressure.

3 ther with systolic (SBP) and diastolic (DBP) blood pressure.

4 lated to changes in systemic oxygenation and blood pressure.

5 intensive care unit (ICU) patients to raise blood pressure.

6 s with coronary atherosclerosis and systolic blood pressure.

7 ow in isolated hearts and increased systemic blood pressure.

8 production, vascular relaxation, and raised blood pressure.

9 iovascular risk attributable to the elevated blood pressure.

10 lular neurons and was sufficient to increase blood pressure.

11 ic peptide, a cardiac hormone that regulates blood pressure.

12 the regulation of vascular contractility and blood pressure.

13 uctural adaptations are related to very high blood pressure.

14 ively associated with younger age and normal blood pressure.

15 nd elevation in central, but not peripheral, blood pressure.

16 erioles and a sustained increase in systemic blood pressure.

17 mediates vascular responsiveness to regulate blood pressure.

18 ed to a significant reduction in mean aortic blood pressure.

19 SNA), left cardiac vagus (VNA), and arterial blood pressure.

20 Low-salt diet did not achieve reductions of blood pressure.

21 modest elevations in resting heart rate and blood pressure.

22 lar filtration rate (eGFR), proteinuria, and blood pressure.

23 did not significantly change heart rates or blood pressures.

24 re, 2.4% (95% CI: 0.6, 4.3) higher diastolic blood pressure, 2.1% (95% CI: 0.5, 3.8) higher waist cir

25 had 1.5% (95% CI: 0.1, 2.9) higher systolic blood pressure, 2.4% (95% CI: 0.6, 4.3) higher diastolic

26 Terlipressin increased mean systemic blood pressure (28 +/- 5 mm Hg; p < 0.0001) and systemic

27 ntage, 45%), body mass index (40%), systolic blood pressure (29%), insulin (20%), physical activity (

28 the rates among those with elevated systolic blood pressure (3.78 [95% CI, 2.76-4.81]), high total ch

29 essure from the International Consortium for Blood Pressure (74 064 individuals) using MR.

30 of the beta3-AR pathway inevitably increases blood pressure, a significant risk factor for cardiovasc

31 Real arterial blood pressure (ABP) measurements from 34 traumatic brai

32 er the methods identify the optimal arterial blood pressure (ABPopt) and lower limit of autoregulatio

33 g had 1-2 mmHg higher systolic and diastolic blood pressure across the life course, but lower measure

34 s showed that while angiotensin II increased blood pressure acutely in all animals, VEGFCc156s-treate

35 thood blood pressure; reduction of childhood blood pressure; adverse effects of treatments.

36 prostane, and measures of renal function and blood pressure among children with CKD.

37 , and the Confusion, Urea, Respiratory rate, Blood pressure and age >= 65 (CURB-65) score was calcula

38 tant role in homeostatic control of arterial blood pressure and brain blood flow.

39 g salt to health, with a particular focus on blood pressure and cardiovascular disease, as well as th

40 on the association between lifetime systolic blood pressure and cognitive function in a community-bas

41 ction for traits, including height, systolic blood pressure and college education, and that the impac

42 djusting for age, sex, body mass index, mean blood pressure and comorbidity (i.e. hypertension, diabe

43 pecies also experience effects of gravity on blood pressure and distribution.

44 strictor firing while measuring beat-by-beat blood pressure and forearm vascular conductance.

45 he association between postdonation systolic blood pressure and graft failure.

46 ar control circuits and mediate increases in blood pressure and heart rate induced by falls in brain

47 low frequency oscillations typically seen in blood pressure and heart rate variability.

48 ntrol circuits to increase systemic arterial blood pressure and heart rate with the purpose of mainta

49 aerosols in human subjects led to increased blood pressure and heart rate, similar to traditional ci

50 ormal levels of protein metabolism products, blood pressure and hematocrit.

51 nitrate-independent effects of BRJ to lower blood pressure and improve vascular function in endothel

52 e physical activity for people with elevated blood pressure and lipid levels were effective in reduci

53 dverse health outcomes, including changes in blood pressure and lung function.

54 lpha) has emerged as a critical modulator of blood pressure and neural plasticity; however, the mecha

55 The causal effect of blood pressure and obesity on lifespan was further suppo

56 mference (WC), waist-to-height ratio (WtHR), blood pressure and plasma lipids.

57 Diastolic blood pressure and pulse pressure are causally associate

58 uence over 5 years by recording beat-to-beat blood pressure and R-R intervals over 10 min.

59 human processes, including the regulation of blood pressure and respiration, as well as neurodegenera

60 The association between systolic blood pressure and structural progression was comparable

61 steepened the relationship between diastolic blood pressure and sympathetic discharge frequency, sugg

62 ogical processes, partially independent from blood pressure and totally independent from glucose leve

63 vestigate the relationship between premorbid blood pressure and vasopressor duration for patients wit

64 ardiovascular physiology (eg, heart rate and blood pressure) and pathophysiology (eg, onset of advers

65 reductions in weight, inflammatory markers, blood pressure, and AF type, and the beneficial effects

66 e were significant differences in body size, blood pressure, and baseline pulmonary capillary wedge p

67 entral obesity, insulin resistance, elevated blood pressure, and dyslipidemia.

68 ial and ventricular remodeling, sleep apnea, blood pressure, and improved glycemic control, all of wh

69 osteopontin, lowered plasma endothelin-1 and blood pressure, and improved mouse survival without affe

70 ecreases in adiposity, body composition, and blood pressure, and improved the lipid profile.

71 tion, inflammatory and neurotrophic markers, blood pressure, and lipid concentrations.

72 uded body mass index (BMI), glucose control, blood pressure, and lipids, assessed at 12 months after

73 namic parameters (heart rate, stroke volume, blood pressure, and peripheral blood flow) and electroca

74 ss index, systolic blood pressure, diastolic blood pressure, and pulse pressure in the UK Biobank, we

75 opment and function, audition, regulation of blood pressure, and renal function.

76 ognitive function, adolescent/adult smoking, blood pressure, and serum total cholesterol level.

77 27% of the 30-50% estimated heritability of blood pressure, and the effect of each SNP on the blood

78 1 (insulin-like growth factor 1) in systolic blood pressure, and the strong causal association of lip

79 and hip measurements, systolic and diastolic blood pressure, and triglycerides were higher in the UK

80 e growth factor 1, lipids, glucose, insulin, blood pressure, and weight.

81 ight, axial length, intraocular and systemic blood pressure, and within-person clustering, to provide

82 th increases in leptin, LDL cholesterol, and blood pressure; and increases in butyrate with increases

83 ent refined diagnostic related groups codes, blood pressure, anesthetics and narcotics administered,

84 dothelial function but impacts on ambulatory blood pressure appear to be variable in individuals with

85 gger for administering fluids and changes in blood pressure are commonly used for tracking changes in

86 cations for patients with premorbid low/high blood pressure are unknown.Objectives: To investigate th

87 Including blood pressure as a covariate did not alter these CBF fi

88 of contractile VSMCs and increased systemic blood pressure as well as right ventricular systolic pre

89 he primary outcome was reduction in systolic blood pressure at 24 months.

90 An impaired decline in blood pressure at rest is typical in people with diabete

91 Existing cerebrovascular blood pressure autoregulation metrics have not been tran

92 ively associated with systolic and diastolic blood pressure (beta = -0.194; 95% CI: -0.153, -0.013; a

93 Covariates included age, gender, ethnicity, blood pressure, body mass index, and spherical equivalen

94 iovascular control relevant to regulation of blood pressure, body temperature, and cerebral blood flo

95 in arterial stiffness and neural control of blood pressure (BP) among older adults.

96 nflicting results on the association between blood pressure (BP) and dementia traits.

97 The DASH diet has been found to lower blood pressure (BP) and low-density lipoprotein choleste

98 ic acid (GABA) influences daytime ambulatory blood pressure (BP) and other cardiometabolic risk facto

99 hese are previously established variants for blood pressure (BP) and the FTO variant has also been as

100 Whereas blood pressure (BP) control in the population and subseq

101 isms responsible for maintenance of arterial blood pressure (BP) during haemorrhage in humans.

102 of antihypertensive medications, global mean blood pressure (BP) has remained constant or has decreas

103 a incidence, stunting in the child, and high blood pressure (BP) in the older adult woman.

104 Blood pressure (BP) is a cardiovascular parameter applie

105 High blood pressure (BP) is a known risk factor for mobility

106 High blood pressure (BP) is a risk factor for cardiovascular

107 assessed the efficacy of dietary patterns on blood pressure (BP) lowering but their findings are larg

108 Blood pressure (BP) management is a crucial part of crit

109 P value = 4.69 x 10-10), automated systolic blood pressure (BP) measurement (beta 0.11, 95% CI, 0.03

110 The purpose of this study was to assess blood pressure (BP) reduction and event rates after RDN

111 A greater blood pressure (BP) response to contraction was observed

112 Genetic studies of blood pressure (BP) to date have mainly analyzed common

113 Large blood pressure (BP) variability may contribute to stroke

114 Blood pressure (BP) was inconsistently associated with m

115 Blood pressure (BP) was measured with an automatic BP mo

116 nd vegetables is associated with lowering of blood pressure (BP), but the nutrient(s) responsible for

117 t circumference (WC), systolic and diastolic blood pressure (BP), fasting blood glucose, glycated hem

118 on, arterial stiffness, central and systemic blood pressure (BP), insulin sensitivity (1/fasting insu

119 tcomes were change in systolic and diastolic blood pressure (BP), Short Physical Performance Battery

120 scle Na/K ATPase modulates vascular tone and blood pressure (BP), the role of its accessory protein p

121 Although intensive blood pressure (BP)-lowering treatment reduces risk for

122 relationships between urinary compounds and blood pressure (BP).

123 S is a risk factor for hypertension and high blood pressure (BP).

124 phy, 12 paired recordings), and beat-to-beat blood pressure (BP; photoplethysmography) during the pre

125 ian populations, evidence suggests that 24-h blood pressures (BP) are more predictive of long-term ca

126 of the association between lifetime systolic blood pressure burden and midlife cognitive function was

127 ong been appreciated as a major regulator of blood pressure, but has more recently been recognized as

128 ion, hearing, proprioception, and control of blood pressure, but identifying the molecules underlying

129 ased systolic and diastolic central (aortic) blood pressure by 4 mm Hg (95% CI: 2.8 to 5.5 mm Hg) and

130 encoding uromodulin has been shown to affect blood pressure by influencing sodium homeostasis, and th

131 BFM prevented the increase in systolic blood pressure, cardiac weight, and renal damage induced

132 Every blood pressure category had a continuum of renin-indepen

133 and MR supports a causal role for education, blood pressure, cholesterol levels, smoking, and diabete

134 ng age, gender, race, hemoglobin A1C levels, blood pressure, cholesterol levels, use of blood thinner

135 2 mRNA and protein levels, and (c) increased blood pressure compared with TG mice containing WT intro

136 nces in effects of nicotine on heart rate or blood pressure, confirmed comparable dosing exposure acr

137 are, was noninferior with regard to systolic blood pressure control at 12 weeks.

138 procedural complications within 30 days and blood pressure control at 6 and 12 months.

139 Similarly, a strategy of intensive blood pressure control did not result in a significant c

140 The PCORnet Blood Pressure Control Laboratory is a platform designed

141 The PCORnet Blood Pressure Control Laboratory is designed to be a re

142 drugs are widely available, in many patients blood pressure control to guideline-recommended target v

143 is not associated with substantial change in blood pressure control, although further research is nee

144 n important role in fluid volume balance and blood pressure control.

145 of early treatment for SCI for all cases was blood pressure control.

146 Blood-pressure control (<140/90 mm Hg) was achieved in 5

147 antation did not further affect body weight, blood pressure, creatinine, or hematocrit in either grou

148 The systolic blood pressure (SBP), diastolic blood pressure (DBP) and mean arterial pressure (MAP) we

149 helial function and systolic (SBP)/diastolic blood pressure (DBP) in individuals with prehypertension

150 patterns on systolic (SBP) and/or diastolic blood pressure (DBP) levels.

151 tional data suggest that excessive diastolic blood pressure (DBP) lowering might increase the risk of

152 h fasting blood glucose, HbA1c and diastolic blood pressure (DBP), and positively correlated with age

153 mid-adulthood: systolic (SBP) and diastolic blood pressure (DBP), high-density-lipoprotein cholester

154 rescues endothelial dysfunction and impaired blood pressure decline during inactivity despite obesity

155 es endothelial dysfunction and impairment in blood pressure decline during inactivity.

156 risk, we collected, laboratory test results, blood pressure, demographic, cognitive, motor, olfactory

157 as at least 3 coexistent morbidities of high blood pressure, diabetes mellitus, osteoporosis, non-AID

158 dium intake), metabolic factors (ie, lipids, blood pressure, diabetes, obesity), socioeconomic and ps

159 When applied to body mass index, systolic blood pressure, diastolic blood pressure, and pulse pres

160 nd combined with genetic effects on systolic blood pressure, diastolic blood pressure, mean arterial

161 ein level, history of hypertension, systolic blood pressure, diastolic blood pressure, tobacco use, s

162 Intensive treatment to lower systolic blood pressure did not result in a clinically relevant d

163 vasorelaxation, microvascular perfusion, and blood pressure during acid-base disturbances and altered

164 scle blood flow and exaggerated increases in blood pressure during exercise.

165 lockade reduces these cells and prevents the blood pressure elevation and renal inflammation on reexp

166 greater dissociative symptoms and transient blood pressure elevations with the higher doses.

167 erences were found in IHL, body composition, blood pressure, energy metabolism, physical performance,

168 spitalization rates, including demographics, blood pressure, estimated glomerular filtration rate (eG

169 Hospitalizations and low blood pressure events did not differ significantly betwe

170 independent of changes in end-tidal PCO2 and blood pressure External carotid artery blood flow increa

171 ively associated with systolic and diastolic blood pressure, faecal SCFAs, Bacteroides plebeius and B

172 associated with worse triglycerides-, HDL-, blood pressure-, fasting glucose- and hemoglobin A1C val

173 At 24 months, the mean systolic blood pressure fell by 9.0 mm Hg in the intervention gro

174 transduction (sNVT), accompanied changes in blood pressure following acute intermittent hypercapnic

175 autonomic reflexes to change heart rate and blood pressure for cardiovascular homeostasis.

176 Mean systolic blood pressure from baseline to 12 months decreased from

177 Review, we appraise the current knowledge of blood pressure genomics, explore the causal pathways for

178 Weight, blood pressure, glucose, F(2)-isoprostanes, NOX2 (NADPH

179 decrease in both the noninvasive arm-to-leg blood pressure gradient (41.2+/-18.7 to 5.6+/-9.6 mm Hg)

180 e >=102 cm for males or >=88 cm for females, blood pressure >=130 mmHg for systolic or >=85 mmHg for

181 0 to <500 versus <150 mg/g), higher systolic blood pressure (>=140 versus 120 to <130 mmHg), diabetes

182 Intraarterial blood pressure, heart rate, and simultaneous brachial ar

183 between childhood hypertension and abnormal blood pressure in adulthood (odds ratios, 1.1-4.5; risk

184 rials demonstrate that salt reduction lowers blood pressure in both individuals who are hypertensive

185 of benefits and harms of screening for high blood pressure in children and adolescents.

186 vations in sympathetic vasomotor outflow and blood pressure in healthy humans.

187 In conclusion, Mb significantly decreased blood pressure in hypertensive models.

188 s ex vivo and in vivo and increased systemic blood pressure in mice and rats.

189 EC-specific PKD2 knockout elevated blood pressure in mice without altering cardiac function

190 l AKAP150-TRPV4 signaling, vasodilation, and blood pressure in obesity.

191 tential neurovascular mechanism for elevated blood pressure in obstructive sleep apnoea.

192 er, offering new opportunities to manipulate blood pressure in the pulmonary circulation.

193 profile of increased systolic and diastolic blood pressure, increased C-reactive protein (CRP), redu

194 e of tachycardia via temporal summation, and blood pressure increases as a function of the degree of

195 ls displayed a delayed systemic reduction in blood pressure independent of alterations in angiotensin

196 PVN TNFR1 prevented the increase in systolic blood pressure induced by AngII.

197 TNFR1 silencing in the PVN inhibits elevated blood pressure induced by AngII.

198 9361 participants in the randomized Systolic Blood Pressure Intervention Trial, which compared intens

199 Although postdonation systolic blood pressure is associated with graft failure, the rep

200 ity of production was associated with higher blood pressure, kaliuresis, and lower serum potassium le

201 ake, cheese consumption and average systolic blood pressure, largely disregarding the impact of a hea

202 nce of uncontrolled blood pressure (systolic blood pressure level >140 mm Hg or diastolic blood press

203 blood pressure level >140 mm Hg or diastolic blood pressure level >90 mm Hg), uncontrolled diabetes (

204 by autonomic-induced elevated and sustained blood pressure levels and abnormal body core temperature

205 Blood pressure, lipid panel, oxidized (GSH) & reduced gl

206 on all known loci, 3 mediating trait-based (blood pressure, lipids, and body mass index) subscores,

207 hanges in obesity-related metabolic markers (blood pressure, low- and high-density lipoproteins [LDL

208 effective in reducing cardiovascular events, blood pressure, low-density lipoproteins, and adiposity-

209 The primary exposure was premorbid blood pressure: low (systolic <100); normal (systolic 10

210 The primary outcome was systolic blood pressure lower than 150 mm Hg at 12-week follow-up

211 , were aged 80 years and older, had systolic blood pressure lower than 150 mm Hg, and were receiving

212 er population, higher prevalence of elevated blood pressure, lower smoking rates, and lower prevalenc

213 The effect of blood pressure lowering might not be evident in specific

214 Blood pressure lowering with antihypertensive agents com

215 meta-analysis of randomized clinical trials, blood pressure lowering with antihypertensive agents com

216 le in blood (t(0.5) ~ 18 h), has significant blood-pressure lowering effect, and shows fast recovery

217 Hypertension was defined as prescription of blood pressure-lowering drugs as obtained from the natio

218 ave provided proof-of-principle data for its blood pressure-lowering efficacy.

219 A polypill comprising statins, multiple blood-pressure-lowering drugs, and aspirin has been prop

220 (CA) is often expressed by the mean arterial blood pressure (MAP)-cerebral blood flow (CBF) relations

221 here was no effect of either intervention on blood pressure, markers of inflammation, or cartilage tu

222 ffects on systolic blood pressure, diastolic blood pressure, mean arterial pressure, and pulse pressu

223 The main limitation was use of blood pressure measurement on a single occasion to diagn

224 hesion, shear stress-induced cell alignment, blood pressure measurements, and flow-induced vasodilata

225 rphometric assessments, expression analysis, blood pressure measurements, and single molecule fluores

226 and a specificity of 0.70 for 2 office-based blood pressure measurements.

227 ssure, use of any AHM with efficacy to lower blood pressure might reduce the risk for dementia.

228 ted health, fatigue, care satisfaction, home blood pressure monitoring, and falls.

229 ents included anthropometry, 24-h ambulatory blood pressure monitoring, and insulin sensitivity.

230 sed approaches have been introduced to lower blood pressure; most of these strategies aim to modulate

231 orrelate with a significant elevation in the blood pressure of patients with hypertension.

232 ndomization to examine the causal effects of blood pressure on AAA.

233 Apples had no effect on blood pressure or other CVD markers.

234 a (including race or ethnicity, age, sex and blood pressure) or the combination of both data types (i

235 c fat, intramyocellular lipids, fecal SCFAs, blood pressure, or 24-h heart rate variability.

236 simulations show that chemotherapy-increased blood pressure, or reduction of interstitial hydraulic c

237 Heart rate and blood pressure oscillate in phase with respiratory activ

238 siological parameters, including heart rate, blood pressure, oxygen saturation, and ventilation param

239 al model included respiratory rate, systolic blood pressure, oxygenation, retractions, capillary refi

240 With the exception of IL16 and diastolic blood pressure (P=0.58), these relationships were valida

241 studies (GWAS) as being associated with the blood pressure phenotype explain only approximately 27%

242 pressure, and the effect of each SNP on the blood pressure phenotype is small.

243 body composition, resting energy metabolism, blood pressure, plasma markers, physical performance, qu

244 .92 to -2.44 mm Hg]; p = 0.003) and systolic blood pressure (point estimate, -12.36 mm Hg [-23.52 to

245 citation, these patients had lower diastolic blood pressure (point estimate, -6.68 mm Hg [-10.92 to -

246 Systolic and diastolic blood pressures provide information about cardiovascular

247 replacement therapy, hypertension (systolic blood pressure ranging from 140 to 190 mm Hg), anemia re

248 orter 2) inhibitors have been shown to lower blood pressure, reduce weight, have salutary effects on

249 urgent treatment of mass effect, aggressive blood pressure reduction and correction of contributing

250 association between childhood and adulthood blood pressure; reduction of childhood blood pressure; a

251 otensins known to possess important roles in blood pressure regulation and electrolyte balance.

252 They play an important role in blood pressure regulation and reducing the risk of heart

253 le of endothelial AKAP150-TRPV4 signaling in blood pressure regulation under normal and obese conditi

254 d facilitate physiological function, such as blood pressure regulation.

255 ributes to electrolyte/fluid-homeostasis and blood pressure regulation.

256 ates vascular responsiveness contributing to blood pressure regulation.

257 Compared to heart rate, body temperature and blood pressure, respiratory rate is the vital sign that

258 Non-blood biomarkers were systolic blood pressure, resting heart rate and body mass index.

259 history of hypertension and office systolic blood pressure (SBP) with major adverse cardiovascular e

260 The systolic blood pressure (SBP), diastolic blood pressure (DBP) and

261 es in eGFR, serum creatinine (SCr), systolic blood pressure (SBP), renal hypoxia, and renal vein leve

262 age, gender, total-cholesterol and systolic blood pressure (SBP).

263 erload that is out of proportion to systolic blood pressure (SBP).

264 Anthropometric measures, blood pressure, serum 25-hydroxyvitamin D [25(OH)D], tot

265 However, interventions such as lowering blood pressure, smoking cessation, and lifestyle optimis

266 D risk factors (raised blood glucose, raised blood pressure, smoking, overweight, and obesity) at the

267 In the high blood pressure stratum (n=15 537), those using any AHM h

268 ncreased risks of heart failure and elevated blood pressure, subsequent studies found a link between

269 f the RVLM increases sympathetic outflow and blood pressure substantially, providing a mechanism by w

270 onductance (FVC; regional sNVT) or diastolic blood pressure (systemic sNVT).

271 Prevalence of uncontrolled blood pressure (systolic blood pressure level >140 mm Hg

272 t evidence suggested potential harm with low blood pressure targets in patients with peripheral arter

273 terval -6.2 to -1.6) and for office systolic blood pressure the difference was -6.5 mm Hg (-9.6 to -3

274 ine in several key areas: the measurement of blood pressure, the definition of hypertension, target t

275 Finally, genetic risks for smoking and high blood pressure, the two main clinical risk factors, play

276 or older with known hypertension or elevated blood pressure, those with dyslipidemia, or those who ha

277 rtension, systolic blood pressure, diastolic blood pressure, tobacco use, statin use, body mass index

278 tension as determined by the requirement for blood pressure treatment early postdonation did not port

279 Recent clinical guidelines support intensive blood pressure treatment targets.

280 so they are independent of calibration with blood pressure, unlike the proprietary measures), and we

281 indices: doppler mean COA gradient, systolic blood pressure, upper-to-lower-extremity SBP gradient, a

282 Among people with hypertensive levels of blood pressure, use of any AHM with efficacy to lower bl

283 identified novel biomarkers associated with blood pressure using MR.

284 Cells in vascular walls are exposed to blood pressure variability (BPV)-induced cycle-by-cycle

285 causal mediation pathway for this effect by blood pressure, vasopressor therapy, or potassium levels

286 use trajectories and natural log-transformed blood pressure, waist circumference, triglycerides, fast

287 nce between the two groups for 24-h systolic blood pressure was -3.9 mm Hg (Bayesian 95% credible int

288 ticipants was 20 kg/m2 and the mean systolic blood pressure was 115 mm Hg.

289 The mean reduction in diastolic blood pressure was 2.8 mm Hg greater in the intervention

290 Mean change in systolic blood pressure was 3.4 mm Hg (95% CI, 1.1 to 5.8 mm Hg)

291 The LPS-induced drop in blood pressure was higher in SHR than in Wistar rats.

292 proximately 19 mg per deciliter and systolic blood pressure was lower by approximately 5.8 mm Hg with

293 Reduced systolic blood pressure was observed with IF (-4.9 mm Hg; -7.2, -

294 Systemic diastolic blood pressure was reduced (0 W, 40%; P < 0.05), but sys

295 sham controls, at one week post-SAC systolic blood pressure was significantly elevated and left ventr

296 Optogenetic excitation robustly elevated blood pressure, water intake, and sodium intake, while o

297 Novel circulating proteins associated with blood pressure were also identified.

298 PRS for increased education and diastolic blood pressure were associated with reduced risk for AD.

299 In the pre-eclamptic group, blood pressure were correlated with the proportion of ox

300 ference, blood lipids, glucose, insulin, and blood pressure were measured.

301 n between flavan-3-ol biomarker and systolic blood pressure when compared to normotensive participant

302 nt neurogenic and inflammatory influences on blood pressure, yet the role played by each of these pro