ライフサイエンスコーパス: blood pressure elevation

1 ney, which promotes vascular dysfunction and blood pressure elevation.

2 of essential hypertension that may antedate blood pressure elevation.

3 treatments affected Ang II-induced systolic blood pressure elevation.

4 p is associated with a substantial transient blood pressure elevation.

5 ation and thereby mitigates T-cell-dependent blood pressure elevation.

6 ructure and function in response to systolic blood pressure elevation.

7 ) tissue infiltration, organ dysfunction and blood pressure elevation.

8 ture and function that worsens with systolic blood pressure elevation.

9 ibuted significantly to vasoconstriction and blood pressure elevation.

10 , podocyte injury and apoptosis, but without blood pressure elevation.

11 brain, to effects in the skin that attenuate blood pressure elevation.

12 ssure but amplifies hyperventilation-induced blood pressure elevations.

13 rican American = 1, Smoking = 1 and Systolic blood pressure elevation = 1, Creatinine elevation = 1;

14 dy of young patients (n=130) with persistent blood pressure elevation above the 90th percentile was c

15 The difference in blood pressure elevation accruing from DC activation was

16 To what extent blood pressure elevation affects left ventricular (LV) s

17 ciation remained strong after adjustment for blood pressure elevation after age 35 years and other co

18 e were observed during phenylephrine-induced blood pressure elevations: an increase (n = 38), a decre

19 of 20-HETE-dependent hypertension prevented blood pressure elevation and 20-HETE-mediated increases

20 of this gene in the RVLM results in chronic blood pressure elevation and cardiac hypertrophy in norm

21 eus of the solitary tract (NST) activated by blood pressure elevation and confirmed their barosensiti

22 long-lived T(EM) cells in the development of blood pressure elevation and end-organ damage that occur

23 eart rate responses to phenylephrine-induced blood pressure elevation and IKACh sensitivity to carbac

24 oods, and dietary patterns and their role in blood pressure elevation and in the treatment of pediatr

25 ion that suggests alternative mechanisms for blood pressure elevation and maintenance.

26 lockade reduces these cells and prevents the blood pressure elevation and renal inflammation on reexp

27 onal hyperlipidemia predisposed offspring to blood pressure elevation and sustained increases in lept

28 ng B7 ligands, angiotensin II caused minimal blood pressure elevation and vascular inflammation, and

29 s a slowly developing chemosensory-dependent blood pressure elevation ascribed in part to NMDA recept

30 se groups in APC showed significant systolic blood pressure elevations at 1 and 3 years (200 mg twice

31 VH groups treated with L-NAME alone produced blood pressure elevations but were spared of renal funct

32 In rats, DARA 7 reduced blood pressure elevations caused by intravenous infusion

33 scues cognitive function without attenuating blood pressure elevation, circulating IL-17 or brain ang

34 antigens then produce cytokines that promote blood pressure elevation, consistent with the idea that

35 minology: "mild" and "moderate") in terms of blood pressure elevation does not explain all outcomes o

36 cular response to exercise and the sustained blood pressure elevation during PECO was similar in pati

37 ences of hypoxic, hypercapnic, and transient blood pressure elevation episodes that may damage or alt

38 assification schemes based on the pattern of blood pressure elevation, extent of damage to target org

39 Although systolic blood-pressure elevation had a greater effect on outcome

40 elationships between vascular stiffening and blood pressure elevation have not been fully delineated.

41 ice are hypertensive ( approximately 14 mmHg blood pressure elevation), hypertension does not appear

42 e importance of recognition and treatment of blood pressure elevation in children and adolescents.

43 effects in ameliorating end-organ damage and blood pressure elevation in experimental hypertension.

44 ) can block O(2)(-) production and attenuate blood pressure elevation in mice.

45 T cells are required for significant blood pressure elevation in mouse models of hypertension

46 ction in cell culture and in vivo and blunts blood pressure elevation in response to a high-fat diet.

47 nsfer of T cells to RAG-1(-/-) mice restored blood pressure elevation in response to stress.

48 pressure measurements to wild type, however, blood pressure elevations in response to ang II infusion

49 t restored PGC-1alpha recovery and prevented blood pressure elevation induced by DOCA-salt.

50 tial for worsening CVD risk factors (such as blood pressure elevation, insulin resistance, and lipid

51 sue of the Journal that maternal gestational blood pressure elevation is associated with reduced reti

52 forward mechanism in which modest degrees of blood pressure elevation lead to T-cell activation, whic

53 the purpose of addressing which comes first: blood pressure elevation or arterial stiffening.

54 ased on stenosis severity, level of systolic blood pressure elevation, or according to the magnitude

55 < 0.05 versus VH + L-NAME, +NaCl) as well as blood pressure elevations (P < 0.01 versus VH + L-NAME a

56 Mice lacking T lymphocytes are resistant to blood pressure elevation, suggesting a key contribution

57 (0.1 nmol i.c.v.); however, the magnitude of blood pressure elevation to hypocretin did not equal tha

58 nd physical assessment, with confirmation of blood pressure elevation using an appropriate-size blood

59 ble to central influences or are mediated by blood pressure elevation, we administered hydralazine (2

60 d proliferation of VSMCs were observed after blood-pressure elevation, which were prevented by geneti

61 how brain pericytes constrict in response to blood pressure elevation with in vivo two-photon microsc

62 greater dissociative symptoms and transient blood pressure elevations with the higher doses.