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1 nt a critical barrier for a bacterium in the bloodstream.
2 elease of tumor-specific biomarkers into the bloodstream.
3 anslocation from the lamina propria into the bloodstream.
4 s can circulate for at least 48 hours in the bloodstream.
5 urface proteins (e.g., CA-125) shed into the bloodstream.
6 iversal property of HIV-1 circulating in the bloodstream.
7 sure in hydrodynamic situations, such as the bloodstream.
8 levels and the glucagon/insulin ratio in the bloodstream.
9 , where they secrete their products into the bloodstream.
10 mensal bacteria and can be absorbed into the bloodstream.
11 d for full S. marcescens pathogenesis in the bloodstream.
12 ect clonal transmission of probiotics to the bloodstream.
13 ly-labeled circulating cells directly in the bloodstream.
14 ion through the endothelium barrier into the bloodstream.
15 ital tract before virus is detectable in the bloodstream.
16 irst 2 weeks after virus was detected in the bloodstream.
17 e transmissible "stumpy forms" in their host bloodstream.
18 and efferent lymphatic vessels to enter the bloodstream.
19 ion and transmission potential in the mammal bloodstream.
20 ic sites including the respiratory tract and bloodstream.
21 a stochastic translocation pattern into the bloodstream.
22 eractions under dynamic flow that mimics the bloodstream.
23 poietic progenitors that migrate through the bloodstream.
24 ap that can actively recruit cancer cells in bloodstream.
25 e used to deliver intravenous therapy to the bloodstream.
26 microsensors that circulate directly in the bloodstream.
27 arbor complex viral populations within their bloodstreams.
30 movement of interstitial chemokines into the bloodstream, a response that supported abluminal-to-lumi
31 solates and compared these isolates with 293 bloodstream and 83 veterinary surveillance ESBL-E coli i
32 ks protein synthesis, can spread through the bloodstream and affect organs, such as the heart and kid
33 thus enabling Y. pestis to reproduce in the bloodstream and be transmitted to new hosts through flea
36 gen that has been associated with nosocomial bloodstream and deep wound infections causing a high mor
37 tal stimuli that Babesia spp. utilize in the bloodstream and for transmission to the tick, with an em
40 pe that provides a survival advantage in the bloodstream and promotes their ability to establish over
41 numbers of B. burgdorferi spirochetes in the bloodstream and, ultimately, consistently reduced spiroc
44 ith circulating tumor cells (CTC) within the bloodstream, and their involvement in the establishment
45 d motility migrate and are launched into the bloodstream as single circulating tumor cells (CTC) or m
46 arp photoacoustic resonances directly in the bloodstream as the basis for new super-resolution photoa
47 eonates, a surge of AVP is released into the bloodstream at levels exceeding release during life-thre
48 on in the airway and oral application in the bloodstream: BK dysfunction recovered acutely and over t
49 radiation, may promote an IGFBP-4 release in bloodstream both in mice irradiated with 100 mGy X-ray a
50 sitive S. aureus cultures using a hierarchy (bloodstream [BSI], surgical site [SSI], and all other ty
51 only effective to kill microfilariae in the bloodstream, but is often ineffective to kill adult fila
52 ss to the circulation are plucked out of the bloodstream by the intravascular macrophages of the live
53 d sample for enhanced molecular diagnosis of bloodstream Candida infection and especially compared it
55 rt of entry to the lymphatic drainage and/or bloodstream, causing dissemination of life-threatening i
56 tory factors are intensely released into the bloodstream, causing the so-called "cytokine storm".
59 kinetics and intensity of Ara h 6 passage in bloodstream depend on both individual and food matrix.
61 scle, and sustained elevation of MG53 in the bloodstream does not have a deleterious impact on db/db
62 cting part of the hydrodynamic forces of the bloodstream encountered by the classical spherical shape
63 as both a survival niche and conduit to the bloodstream for S. pyogenes, explaining the phenomenon o
65 lar axonemal inner-arm dynein complex in the bloodstream form and investigated its mechanistic role i
67 developed permeabilized cell system for the bloodstream form of T. brucei, we show that down-regulat
71 on of the specialized subtelomeric PTUs, the Bloodstream-form Expression-Sites (BESs), which house th
72 dited CYb mRNA is downregulated in mammalian bloodstream forms (BSF) at the level of editing initiati
73 hat show nanomolar potency against T. brucei bloodstream forms, Leishmania and Trypanosoma cruzi.
74 ivity against Trypanosoma brucei rhodesiense bloodstream forms, which is another important aetiologic
75 r ferroportin to control iron entry into the bloodstream from dietary sources, iron recycling macroph
77 ent of circulating proteins, which enter the bloodstream from inflamed tissues, also offers insight i
78 ulating tumor cells (CTCs) are shed into the bloodstream from primary tumors, but only a small subset
79 apted clade, of which 23 were represented in bloodstream, hospital sewage, and municipal wastewater i
84 ital, a retrospective cohort of adult KPC-KP bloodstream infection (BSI) cases (January 2014 to Decem
85 tibiotic treatment for lower UTI and risk of bloodstream infection (BSI) in adults aged >=65 years in
86 icenter prospective study of all episodes of bloodstream infection (BSI) in high-risk FN patients (20
88 prediction models have been shown to predict bloodstream infection (BSI) likelihood in this populatio
89 t of vancomycin-resistant Enterococcus (VRE) bloodstream infection (BSI) on outcomes of allogeneic he
90 007-2017) of hospital-based Salmonella Typhi bloodstream infection (BSI) surveillance in the Democrat
91 ed a DOOR endpoint for Staphylococcus aureus bloodstream infection (BSI) through a survey to infectio
92 Little is known of the long-term risks of bloodstream infection (BSI) with extended spectrum beta-
96 of Community acquired Staphylococcus aureus bloodstream infection (CA-SABSI) with myocardial infarct
97 luate differences in central line-associated bloodstream infection (CLABSI) rates by how central line
99 hospital-onset multidrug-resistant organism bloodstream infection (MDRO-BSI) and Clostridium diffici
100 sed association with central-line associated bloodstream infection (odds ratio, 0.505; 95% CI, 0.336-
101 1,000 line days) and central-line associated bloodstream infection (peripherally inserted central cat
102 esistant Enterococcus (VRE) and ICU-acquired bloodstream infection (UABSIs) were analysed from 1,189,
103 Salmonella Typhi was the leading cause of bloodstream infection among infants and young children <
104 from 16 sites in ten countries: 174 with CSE bloodstream infection and 123 with CRE bloodstream infec
105 um samples from 30 children with a bacterial bloodstream infection and 35 children with Plasmodium fa
106 % (35 of 174 patients) for patients with CSE bloodstream infection and 35% (43 of 123 patients) for p
107 ous infections usually arise from an initial bloodstream infection and are frequently recalcitrant to
108 roorganisms associated with catheter-related bloodstream infection and colonization was significantly
109 Salmonella (NTS) isolated from persons with bloodstream infection and diarrheal disease from 2007 th
110 d Salmonella Typhimurium are major causes of bloodstream infection and diarrheal disease in East Afri
111 ne prophylaxis was associated with decreased bloodstream infection and intestinal colonization by gra
112 nomic analysis of E. faecium associated with bloodstream infection and isolated from wastewater.
113 epidemic, using temporal trends in S. Typhi bloodstream infection and perforated abdominal viscus at
114 with higher rates of central-line associated bloodstream infection and venous thromboembolism than ce
115 o be associated with central-line associated bloodstream infection and venous thromboembolism were in
116 wastewater with 187 isolates associated with bloodstream infection at five hospitals in the East of E
117 ics, previous antibiotic exposure, and index bloodstream infection caused by either rGNB or Candida s
118 biotics, previous antibiotic exposure, index bloodstream infection caused by either rGNB or Candida s
120 d its impact on the outcome of patients with bloodstream infection due to Enterobacteriaceae (BSI-E).
122 with an increased risk for catheter-related bloodstream infection due to nonfermenting Gram-negative
123 swabs of their environment, together with 1 bloodstream infection during the study and 4 others over
126 with daptomycin plus a beta-lactam for MRSA bloodstream infection had lower odds of composite clinic
127 ous catheters (CVCs) reduce catheter-related bloodstream infection in adults and children receiving i
129 uring intestinal colonization and subsequent bloodstream infection in immunocompromised pediatric pat
140 am isolate showed that the subject's E. coli bloodstream infection likely originated from the intesti
141 ted in a strain that was acutely virulent in bloodstream infection models in mice and in ex vivo mode
144 n the post-Directive central line-associated bloodstream infection rates associated with a unit incre
146 ano-mupirocin in a murine model of S. aureus bloodstream infection resulted in improved antibiotic di
147 teria was used as predictor of gram-negative bloodstream infection using Cox proportional hazards mod
148 Candidemia is a common healthcare-associated bloodstream infection with high morbidity and mortality.
152 ound three loci with a suggestive linkage to bloodstream infection, all on chromosome 4, at 46.6 cent
154 mia, one of the most common causes of fungal bloodstream infection, leads to mortality rates up to 40
155 ong central venous catheter catheter-related bloodstream infection, nonfermenting Gram-negative bacil
156 the early host response to S. aureus during bloodstream infection, promoting enhanced responses by b
157 al domination was associated with subsequent bloodstream infection, which was observed overall and in
159 d genetic intermixing between wastewater and bloodstream infection, with highly related isolates shar
175 lity, we analysed 1691 patients with candida bloodstream infection; 776 (45.9%) who had an infectious
176 ital, a retrospective cohort of adult KPC-KP bloodstream infections (BSI) cases (January 2014 to Dece
177 in etiologies and susceptibility patterns of bloodstream infections (BSI) in hospitalized children in
182 ), yet the prevalence of IE in patients with bloodstream infections (BSIs) caused by different strept
184 as notified by hospital A of 3 patients with bloodstream infections (BSIs) with a rapidly growing non
185 d cultures, the gold standard for diagnosing bloodstream infections (BSIs), are insensitive and limit
186 critically needed for Staphylococcus aureus bloodstream infections (BSIs), particularly for methicil
190 fections (CAUTI) and central line-associated bloodstream infections (CLABSI) per 1000 device days.
192 he incidence of VTE, central line-associated bloodstream infections (CLABSIs), and catheter malfuncti
195 or equal to median was associated with fewer bloodstream infections (OR, 0.67 [95% CI, 0.45-0.98).
196 Clinical trials for Staphylococcus aureus bloodstream infections (SAB) are broadly grouped into 2
197 events were febrile neutropenia (22 [66%]), bloodstream infections (six [16%]), and invasive fungal
198 sistant Enterococcus (VRE), and ICU-acquired bloodstream infections (UABSIs) for 1 189 142 patients f
200 of antimicrobial therapy targeted toward CPE bloodstream infections and assist infection control and
201 s, with hypercapsule mutants associated with bloodstream infections and capsule-deficient mutants ass
202 anemia, intermittent proteinuria, recurrent bloodstream infections and chronic pulmonary disease.
203 enous catheter and arterial catheter-related bloodstream infections and colonization according to the
211 exidine (CHG) bathing decreases incidence of bloodstream infections at intensive care units, but its
212 al center in New York City who had S. aureus bloodstream infections between 1 January 2007 and 31 Dec
217 can identify patients at risk for subsequent bloodstream infections caused by resistant bacteria.
219 aches to identify patients at risk of fungal bloodstream infections for pre-emptive therapeutic inter
220 eillance of Salmonella Typhi and Paratyphi A bloodstream infections from 5 October 2015 through 4 Oct
221 es transitioning patients with gram-negative bloodstream infections from intravenous to oral therapy
223 nd 2018, there was an associated rise in VRE bloodstream infections in hospitals where contact precau
224 ine (CHG) bathing decreases the incidence of bloodstream infections in intensive care units, but its
230 lso been developed detecting the presence of bloodstream infections including electrochemical, potent
231 We hypothesized that septic patients with bloodstream infections may transition across states char
232 on profiles of 9,215 P. vivax parasites from bloodstream infections of Aotus and Saimiri monkeys.
234 next-generation sequencing-based analyses of bloodstream infections provide a valuable diagnostic pla
239 cal blood culture samples from patients with bloodstream infections were incubated for 1 h with the "
240 spp. causing illness, describe non-Brucella bloodstream infections, and identify risk factors for br
241 dida albicans is a leading cause of systemic bloodstream infections, and synthesis of the phospholipi
243 both in terms of intestinal colonization and bloodstream infections, compared with non-prophylaxed pa
244 lently for 30 isolates derived from clinical bloodstream infections, confirming system optimization f
245 nesis has primarily focused on pneumonia and bloodstream infections, even though one in five A. bauma
246 obiota is connected to risk of gram-negative bloodstream infections, expanding on our prior work in t
247 iated with male sex, central line-associated bloodstream infections, long-term acute care hospitals,
248 ate earlier optimization of the treatment of bloodstream infections, particularly in conjunction with
250 rapid identification of Escherichia coli in bloodstream infections, we employed an existing colorime
251 f 4967 unique patients with Enterobacterales bloodstream infections, we sought to answer the question
263 li strains across time points as well as the bloodstream isolate showed that the subject's E. coli bl
264 he virulence of 100 individual P. aeruginosa bloodstream isolates and performed whole-genome sequenci
265 strains comprised of both oral isolates and bloodstream isolates from patients diagnosed with IE.
273 rom respiratory pathogens are present in the bloodstream of most CF patients, which could potentially
274 rge from the intestinal lumen and invade the bloodstream of vulnerable patients, causing disseminated
275 ocation to first microbiologically confirmed bloodstream or cerebrospinal fluid (CSF) infection betwe
277 The time from random allocation to first bloodstream or CSF infection was similar between the two
279 d blood volume and the rarity of CTCs in the bloodstream preclude longitudinal, in-depth studies of t
280 conclude that EPS protects hosts from acute bloodstream S. aureus infection not only by inducing mac
283 that depending on the environment, local or bloodstream, the consequences of the interactions betwee
284 aemoparasites that survive in the vertebrate bloodstream through antigenic variation of their Variant
288 ntracellular content, DNA included, into the bloodstream to form neutrophil extracellular traps (NETs
290 agocyte, they are rapidly recruited from the bloodstream to sites of infection or injury to internali
291 crucial for spread of P. aeruginosa from the bloodstream to the feces during bacteremia, a process th
292 show that P. aeruginosa can spread from the bloodstream to the gallbladder, where it replicates to e
295 mice with sustained elevation of MG53 in the bloodstream (tPA-MG53) have a healthier and longer life-
296 chronic osteomyelitis was passed through the bloodstream using a bacteriemia mouse model and derivati
299 ntially deliver the drugs efficiently to the bloodstream, where the microfilariae reside, while also
300 able therapeutic daptomycin treatment in the bloodstream, while preventing transmissible resistance e