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1 reduced food intake and body weight (mainly body fat mass).
2 bolished when values are normalised to whole-body fat mass.
3 GPRKO female mice is due to the reduction in body fat mass.
4 ks ingested lipids to energy expenditure and body fat mass.
5 ame HF diet, despite comparable increases of body fat mass.
6 decrease in circulating IGF-I independent of body fat mass.
7 ) when controlled for the1-y change in total body fat mass.
8 weeks AQP7 null mice had 3.7-fold increased body fat mass.
9 d an MLS >2.0 for percentage of fat mass and body fat mass.
10 sis, atherosclerosis, onset of diabetes, and body fat mass.
11 oncentration is highly correlated with total body fat mass.
12 % more than controls due mainly to increased body fat mass.
13 ntrol rats and had large reductions in their body fat mass.
14 elevated plasma leptin levels independent of body fat mass.
15 itional health issues related to their large body fat mass.
16 I -1.6 to -0.3, p = 0.0073), decreased whole-body fat mass (-1.8 kg, 95% CI -2.9 to -0.7, p = 0.0016)
17 hole-body lean mass (-1.0 +/- 0.2 kg), whole-body fat mass (-6.9 +/- 0.5 kg), appendicular lean mass
18 with an adjusted 1.3 points lower percentage body fat mass (95% CI: -2.2, -0.4; P = 0.005) and an adj
19 ent attenuated HFD-induced body weight gain, body fat mass accumulation, increased energy expenditure
21 In vivo, HSF1 antagonizes AMPK to control body fat mass and drive the lipogenic phenotype and grow
22 CCK-KO mice had reduced body weight gain and body fat mass and enlarged adipocytes, despite the same
25 d carbohydrate feeding on energy metabolism, body fat mass and muscle expression of fuel metabolism g
26 strength and bone mass, and increased total body fat mass and visceral fat depot, blood lipid concen
28 mass (right arm, right leg, trunk, and whole-body fat mass) and fat-free mass, and the risk of heart
29 In conclusion, leptin levels reflect total body fat mass, and although visceral fat is known to pre
30 (compliant model) indicated that age, whole-body fat mass, and bone resorption were common predictor
32 NX rats show increased food intake, enhanced body fat mass, and elevated plasma levels of triglycerid
33 as present on the native molecule, decreased body fat mass, and improved systemic insulin sensitivity
34 nd A reduced body weight, food intake, whole-body fat mass, and intramuscular triglycerides compared
35 ots, bioelectrical impedance measurements of body fat mass, and measurements of body weight and girth
36 associations between body mass index (BMI), body fat mass, and percent body fat, measured by dual-en
37 neral content (BMC), lean tissue mass (LTM), body fat mass, and percentage fat are presented as funct
40 bdominal fat areas and negatively with lower-body fat mass as a percentage of total-body fat, after c
43 but the precise amount of calorie intake or body fat mass associated with optimal health and maximum
44 cknesses cannot be used to assess changes in body fat mass because of age-related fat redistribution.
45 mice) and effects on liver weight and total body fat mass being essentially independent of mERalpha
46 order to determine the correlation of BMI or body fat mass (BFM) with blood pressure, fasting blood g
47 ring gained more body weight and had greater body fat mass compared to the control, and these differe
48 ight decreased by 10.2 +/- 5.5% (P < 0.001), body fat mass decreased by 18.7 +/- 11.3% (P < 0.001), a
51 e VF+ compared with the VF- group, but whole-body fat mass (determined using 3H2O) was not significan
52 ) [CI, -0.41 to 1.02 kg/m(2)]; P = 0.39), or body fat mass (difference, 0.39 kg [CI, -1.04 to 1.92 kg
55 different (P > 0.05) between machines, whole-body fat mass [DXAH-DXAL (DXAdiff) = 1152 +/- 1395 g], p
56 FHC and HF groups had increased body weight, body fat mass, fasting glucose, and were insulin-resista
57 ion patterns were observed for obesity risk, body fat mass, fat percentage, fat mass index, and waist
58 later (n = 236), using DXA to quantify whole-body fat mass (FM) and MRI for abdominal subcutaneous (S
59 y expenditure (AEE), energy intake (EI), and body fat mass (FM) were measured longitudinally in 10 wo
62 ean change from baseline to week 48 in total body fat mass (FM); secondary and exploratory end points
63 ith the rs373863828 A allele had lower whole-body fat mass [FM 1.4 (0.7) vs. 1.7 (0.7) kg, aMD -0.4,
64 we reported that S6K1(-/-) mice have reduced body fat mass, have elevated rates of lipolysis, have se
65 waist circumference, skinfold thickness, and body fat mass in 1,301 children from six European birth
66 se dependently reduced body weight and whole-body fat mass in DIO mice due to marked increases in tot
67 is known to be associated with lower BMI and body fat mass in Northern Europeans and T haplogroup whi
68 eased the energy expenditure and lowered the body fat mass in two diet-induced obesity mouse models,
69 entified homeostat for body weight regulates body fat mass independently of fat-derived leptin, revea
70 playing a critical role in the regulation of body fat mass, lipid metabolism, and insulin resistance.
71 icant differences in estimated percentage of body fat mass loss between treatment groups, and the fra
75 lue < 0.001, 0.01 < eta(2) < 0.14) for whole body fat mass percentage and index of low muscle mass.
78 and increased production from expanded total body fat mass, rather than alterations in leptin clearan
79 resented with a reduced total BW and overall body fat mass, smaller adipocytes, and reduced leptin le
80 obese individuals are proportional to whole-body fat mass, suggesting a compensatory down-regulation
81 ternate-day fasting less effectively reduces body fat mass than a matched degree of daily energy rest
82 rum leptin levels are lowered by the loss of body fat mass that would accompany starvation and malnut
83 uglycemic clamp was increased, whereas total body fat mass, VAT, SSAT, and hemoglobin A1c were reduce
84 glucose tolerance test and minimal modeling, body fat mass was assessed by dual-energy X-ray absorpti
88 ; p = 0.01) following the confinement, whole body fat mass was only reduced in the Exercise group (-1
90 ntrast, at age 4 weeks, adipocyte volume and body fat mass were comparable in wild type and AQP7 null
91 body lean mass and handgrip strength versus body fat mass with atherogenic traits measured in young
92 increases nutrient oxidation, and decreases body fat mass without altering food intake, lean body ma