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1 ment of sacral and tail vertebrae (secondary body formation).
2 o the posterior skeleton and tail (secondary body formation).
3 ied two domains of NP that control inclusion body formation.
4 iral transcription occurs prior to inclusion body formation.
5 end of the dimer for paraspeckle subnuclear body formation.
6 also to investigate the biology of inclusion body formation.
7 m sorted single cells, and enhanced embryoid body formation.
8 egetative cells that contributed to fruiting body formation.
9 omosome inactivation (MSCI), and leads to XY body formation.
10 ordered assembly can play a role in nuclear body formation.
11 yclodextrin perturbed TRIM5alpha cytoplasmic body formation.
12 y results in an inherited disorder of ketone body formation.
13 the SN in PD and it is associated with Lewy body formation.
14 budding, and ESCRT-catalyzed multivesicular body formation.
15 RRK2-induced neuronal toxicity and inclusion body formation.
16 s involving vegetative swarming and fruiting body formation.
17 nd cheating behavior during asexual fruiting-body formation.
18 tion of UPS is not specific to PD or to Lewy body formation.
19 to ensure appropriate timing during fruiting body formation.
20 revents the switch and disrupts the embryoid body formation.
21 the efficient encapsidation and cytoplasmic body formation.
22 at includes swarming, predation and fruiting body formation.
23 , and the processes of primary and secondary body formation.
24 cell volume, DNA degradation, and apoptotic body formation.
25 single cell reversals, swarming and fruiting body formation.
26 ses and the therapeutic benefit of inclusion body formation.
27 required for directed motility and fruiting body formation.
28 s gliding motility for swarming and fruiting body formation.
29 ep-up in prey availability inhibits fruiting body formation.
30 rentiation capacity as indicated by embryoid body formation.
31 cogen leads to aberrant branching and Lafora body formation.
32 es about the mechanisms involved in fruiting body formation.
33 eta-d-allose-mediated inhibition of fruiting body formation.
34 events during the initial stages of fruiting body formation.
35 that reproduces the early stages of fruiting body formation.
36 al rates during the early stages of fruiting body formation.
37 ential for social (S-) motility and fruiting body formation.
38 ontraction, membrane blebbing, and apoptotic body formation.
39 tissue-specific roles in centriole and basal body formation.
40 levels persisted through 5 days of embryoid body formation.
41 meiotic sex chromosome inactivation, and XY body formation.
42 yte infiltration, and eosinophilic inclusion body formation.
43 anslational repressors and facilitators of P body formation.
44 both SfaD and GpgA are required for fruiting body formation.
45 ns were defective in S-motility and fruiting body formation.
46 B permitted all events except ring and polar body formation.
47 inoic acid, or IFN-gamma induced PML-nuclear body formation.
48 including protein aggregation and inclusion body formation.
49 ring center, respectively, inhibiting polar body formation.
50 racellular VWF trafficking and Weibel-Palade body formation.
51 rane blebbing, cell shrinkage, and apoptotic body formation.
52 at includes vegetative swarming and fruiting-body formation.
53 n in ligase activity, thereby promoting Lewy body formation.
54 ation of ES cells in the absence of embryoid body formation.
55 he initiation of asexual and sexual fruiting body formation.
56 its motility during aggregation and fruiting body formation.
57 d toxicity, lead accumulation, and inclusion body formation.
58 to other targets and may be linked to Heinz body formation.
59 disease, leads to a defect in Weibel--Palade body formation.
60 ly expressed in the DCT, is critical for WNK body formation.
61 (shiitake mushroom) associated with fruiting-body formation.
62 al for both vegetative swarming and fruiting body formation.
63 e cycle that includes multicellular fruiting body formation.
64 developmental process of M. xanthus fruiting body formation.
65 ese strains rescued sporulation and fruiting body formation.
66 mation, cell surface blebbing, and apoptotic body formation.
67 terization of a mutant defective in fruiting body formation.
68 a complex life cycle that includes fruiting body formation.
69 d for developmental aggregation and fruiting body formation.
70 ion through to the terminal stage of Hassall body formation.
71 bition of translation or inhibition of the P body formation.
72 the episodic nature of lunar crust or parent body formation.
73 UBL domain of Dsk2 is critical for inclusion body formation.
74 tional motility during swarming and fruiting body formation.
75 for a short time before noticeable inclusion body formation.
76 of misfolded proteins by promoting inclusion body formation.
77 ly from the presomitic mesoderm (PSM) during body formation.
78 e sensitive to Lfng dosage than is secondary body formation.
79 y to promote TTP expression and subsequent P-body formation.
80 overlapped with the changes during embryoid body formation.
81 mad signaling, as Smad3 deletion abrogated P-body formation.
82 al motility, predatory rippling and fruiting body formation.
83 specific roles of family members in protein body formation.
84 est that the HSR does not mitigate inclusion body formation.
85 and Fas I expression during locust mushroom body formation.
86 formation, DNA fragmentation, and apoptotic body formation.
87 5, which is essential for processing body (P-body) formation.
88 30-fold during the first 3 days of embryoid body formation, a culture system model of early embryoge
89 regation of misfolded proteins and inclusion body formation, a hallmark of neurodegenerative disease.
90 iparum, which results in reduced osmiophilic body formation, a marked decrease in female fitness, and
92 on mutant exhibited a 24 h delay in fruiting body formation, accumulated less glycogen in the station
93 ve conditions is often hampered by inclusion-body formation after overexpression from T7 promoter-bas
94 h encode proteins that affect multivesicular body formation, also showed altered vacuolar morphology
96 g correlation between the level of inclusion body formation and aggregation propensity, thus demonstr
98 t correlated with impaired in insecta hyphal body formation and altered host immune prophenol oxidase
99 show strong defects in mRNA decapping and P body formation and are blocked in translational repressi
100 ia formation through p62-dependent inclusion body formation and blockage of Bardet-Biedl syndrome 4 (
102 rable, the rate-limiting steps for inclusion body formation and death can be traced to different conf
105 sphorylation is required for heterochromatin body formation and DNA elimination, whereas it is dispen
106 (2+)](i), cell membrane integrity, apoptotic body formation and DNA fragmentation in cultured HepG2 c
108 from the cytoplasm to the membrane, fruiting body formation and EPS production were restored to the l
110 ns and their assembly dynamics for new basal body formation and function are not well understood.
112 ephrogenic epithelia from after the s-shaped body formation and in the developing collecting ducts re
113 ype seems to preclude lead-induced inclusion body formation and increases lead toxicity at the organ
114 ic traits, namely endoplasmic reticulum (ER) body formation and induction of indole glucosinolate (IG
115 ar basis and biological function of both sex body formation and meiotic sex chromosome inactivation (
116 complex is cytoplasmic and is involved in P-body formation and mRNA decay by promoting decapping.
118 nal regulation of key regulators NAI1 for ER body formation and MYB51/122 for IGs biosynthesis).
119 ynuclein ubiquitination and its role in Lewy body formation and neurodegeneration remain poorly under
121 required for translational repression and P-body formation and plays an indirect role in mRNA decapp
122 ssion was necessary for TGF-beta-dependent P-body formation and promoted growth inhibition by TGF-bet
123 etion of fdgA resulted in defective fruiting body formation and reduced sporulation efficiency (1% th
124 es, increasing ATZ-BiP binding and inclusion body formation and reducing ATZ interactions with compon
125 n regulating mRNA degradation and processing body formation and reinforces the influence of phosphory
127 We also identify 13 new activators of TORC1-body formation and show that seven of these proteins reg
131 is study that IgG or IL-5 also induces lipid body formation and subsequent leukotriene C4 production
132 stress and invasive bacterial pathogens in U body formation and suggest that this process contributes
134 scillatory Lfng during primary and secondary body formation and that posterior development is less se
135 importance of the Golgi network in lamellar body formation and the necessity of acidification of the
136 otent stem cells (hiPSC), bypassing embryoid body formation and the use of exogenous molecules, coati
138 rmal and mesodermal lineages during embryoid body formation and under inductive conditions using reti
139 provide a molecular explanation for Balbiani body formation and, surprisingly, show that viable ferti
143 ion for the assembly of decapping factors, P-body formation, and constitutive decay of instable mRNAs
146 f the pdu operon are required for polyhedral body formation, and immunoelectron microscopy shows that
149 des co-ordinated group movement and fruiting body formation, and requires directed motility and contr
150 alize into basal bodies, thus blocking basal body formation, and the microtubule ribbon was completel
151 ever, the mechanisms that underlie inclusion body formation, and the precise relationship between inc
152 down, disappeared at the time of first polar body formation, and then reappeared as larger clusters i
153 nal accumulation of alpha-synuclein and Lewy body formation are characteristic to many neurodegenerat
155 data indicate that aggregation and inclusion body formation are mechanistically and temporally distin
156 rthermore, endocytic traffic and brefeldin A body formation are perturbed in attrappc11/rog2, suggest
158 ique GlcCer species, and defects in lamellar body formation associated with decreased expression of t
159 amma-zeins could initiate and target protein body formation at specific regions of the rough endoplas
160 whereas VipD interferes with multivesicular body formation at the late endosome and endoplasmic reti
161 inhibition of infection was independent of P-body formation because expression of GFP-Dcp1a mutants t
162 thoracic and lumbar axial skeleton (primary body formation), but are largely dispensable for the dev
163 active Gtr1/2 is a potent inhibitor of TORC1-body formation, but cells missing Gtr1/2 still form TORC
164 elf-organization is thought to drive nuclear body formation, but whether this occurs stochastically o
165 s in other species, suggesting that Balbiani body formation by amyloid-like assembly could be a conse
166 CDMP-1 in the notochord inhibited vertebral body formation by blocking migration of sclerotome cells
167 ies suggest that FL1 participates in protein body formation by facilitating the localization of 22-kD
168 g microtubules may be required for inclusion body formation by HtEx1 and that inclusion body formatio
169 , IbpA/B or ClpA does not suppress inclusion body formation by the aggregation-prone protein preS2-S'
173 ed whether-protein aggregation and inclusion body formation cause neurotoxicity are presently unknown
177 formation of the anterior skeleton (primary body formation), compared to the posterior skeleton and
178 virus, we showed that the onset of inclusion body formation corresponds to the onset of viral genome
179 cellular processes, including multivesicular body formation, cytokinesis, and viral budding, require
180 nd that an Arabidopsis mutant defective in p-body formation (Decapping 5; dcp5-1) showed reduced fitn
181 ribution of Buc in oocytes precedes Balbiani body formation, defining Buc as the earliest marker of o
183 maH2AX with sites of DNA damage, reduced sex body formation, diminished Rad51 foci and absence of Mlh
189 in vitro differentiation protocols (embryoid body formation, endodermal induction, directed different
190 ressed in M. xanthus, NafA restored fruiting body formation, EPS production, and S-motility to difA m
191 d for the hES-ATII cells, including lamellar body formation, expression of surfactant proteins A, B,
192 es mature neurofibrillary inclusion and Lewy body formation found in Alzheimer's and Parkinson's dise
194 ncover a DNA under replication-53BP1 nuclear body formation-G1 arrest axis as an unanticipated outcom
195 r key factors proposed to be essential for P body formation, GW182, Edc3, and Edc4, were unaffected b
196 ondrogenesis by human ESCs required embryoid body formation; however, embryoid body formation often r
200 vels and structural modeling indicate that P-body formation in cells with the mutation in CCT3 is lin
204 se data uncover a new mechanism of apoptotic body formation in monocytes and also compounds that can
206 required for translational repression and P-body formation in pat1Delta strains under these conditio
210 among deadenylation, mRNP remodeling, and P-body formation in selective decay of mammalian mRNA.
212 sion, budded virus production, and occlusion body formation in SF-21 cells but not in TN-5B1-4 cells.
214 r negatively controls multicellular fruiting body formation in the bacterium Myxococcus xanthus, inhi
215 es have demonstrated synuclein-positive Lewy body formation in the brains of individuals with parkins
216 ch as phosphorylation can regulate inclusion body formation in the context of alpha-synuclein and syn
217 causing mitotic abnormalities, 53BP1 nuclear body formation in the ensuing G1 phase, and G1 arrest.
219 rexpression construct to restore prolamellar body formation in the porB-1 porC-1 double mutant backgr
222 tiple hematopoietic lineages during embryoid body formation in vitro, but to date, an ES-derived hema
223 usion: SAMe treatment prevented Mallory Denk body formation in vivo and in vitro by preventing the ex
224 PML this motif is essential for PML nuclear body formation in vivo and PML-homo and hetero interacti
228 of beclin (a protein required for autophagic body formation) inhibited caspase-independent macrophage
229 dependently (0.01-100 nM) elicited new lipid body formation, intracellular LTC(4) formation at lipid
232 We determined that loss of Weibel-Palade body formation is associated with markedly reduced secre
235 The findings provide evidence that inclusion-body formation is in itself a sufficient cause of neurod
236 levels are reduced, suggesting that primary body formation is more sensitive to Lfng dosage than is
241 ld be further attributed to reduced lamellar body formation, loss of apical polarization of LB secret
243 n body formation by HtEx1 and that inclusion body formation may have evolved as a cellular mechanism
246 As in other organisms a dynamic shift in P-body formation occurs in response to diverse physiologic
248 ins and drives relocalization of HIRA to PML bodies, formation of SAHF and senescence, likely through
252 d embryoid body formation; however, embryoid body formation often results in heterogeneous differenti
255 ition occurred contemporaneously with parent body formation or shortly after it probably within 100 M
256 ls may not require yellow cells for fruiting body formation or starvation-induced sporulation of tan
260 on the cell surface, is delayed in fruiting body formation, produces fewer spores, is delayed in ger
262 xploited to tackle the problems of inclusion body formation, proteolytic degradation and disulfide bo
263 extracellular signals required for fruiting body formation rather than in the sensing of such extrac
265 on of the cell cycle and impaired processing-body formation, reminiscent of Nanos2-knockout phenotype
269 ts in high CO2 but not later steps (fruiting body formation, sporulation), indicating a major role fo
270 embryonic stem (hES) cells rely on embryoid body formation, stromal feeder co-culture or selective s
272 odegenerative process in disorders with Lewy body formation, such as Parkinson's disease and dementia
274 inuous simulation all the stages of fruiting-body formation that have been experimentally observed: n
275 ull mutant had negligible effects on protein body formation, the betaRNAi and gammaRNAi alone only ca
276 EGG-5, we observe defects in meiosis, polar body formation, the block to polyspermy, F-actin dynamic
277 EBP gene nla4 affects the timing of fruiting body formation, the morphology of mature fruiting bodies
280 olonized D. discoideum spores after fruiting body formation; this observation, together with the abil
281 newly isolated mutants defective in fruiting body formation to determine a possible relationship betw
284 w that infection with MeV triggers inclusion body formation via liquid-liquid phase separation (LLPS)
285 ation has been reported to be required for P body formation, viral inhibition of deadenylation, throu
291 -regulation of laforin cannot explain Lafora body formation, we conclude that malin functions to main
292 e precise role of calcium signaling in polar body formation, we used live-cell imaging coupled with t
293 lock in chromatin condensation and apoptotic body formation when nuclei from HeLa cells expressing la
294 Stress granule assembly is dependent on P-body formation, whereas P-body assembly is independent o
295 tion was accompanied by decreased processing body formation, which contributed to the inhibition of N
296 results indicate that DCP5 is required for P-body formation, which likely facilitates efficient decap
297 ogenesis, S-motility, rippling, and fruiting body formation, while orfL is dispensable for these proc
298 ate the Gtr1/2-dependent repression of TORC1-body formation, while the remaining proteins drive the s
299 t showed substantia nigra cell loss and Lewy body formation, with small numbers of cortical Lewy bodi