ライフサイエンスコーパス: body temperature

1 is their ability to adapt to mammalian core body temperature.

2 altered sleep is not secondary to decreased body temperature.

3 s such as heart rate, blood oxygenation, and body temperature.

4 ion of butyrate led to a significant drop in body temperature.

5 on temperature and is the highest around the body temperature.

6 ble liquid at room temperature and gels near body temperature.

7 ncrease in all three variables as well as in body temperature.

8 temperature and while forming an hydrogel at body temperature.

9 Food restriction triggers a lowering in body temperature.

10 ation of locomotor activity, sleep, and core body temperature.

11 adian rhythms in both locomotor activity and body temperature.

12 tion, with the main symptom a sudden drop in body temperature.

13 t 10 degrees C that resulted in 15 degrees C body temperature.

14 liquid at room temperature to a hydrogel at body temperature.

15 ncluding heart rate, arterial pressures, and body temperature.

16 lesion could produce two opposite effects on body temperature.

17 mice to regulate brown fat and maintain core body temperature.

18 logy, qPCR, HPLC, LC/MS and measures of core body temperature.

19 the central circadian clock to help regulate body temperature.

20 to use preference behaviour to regulate its body temperature.

21 controlling for confounding factors such as body temperature.

22 nto mice to form a collagen-fibril matrix at body temperature.

23 l signature in the range equivalent to human body temperature.

24 hypothalamic nuclei involved in controlling body temperature.

25 ient rash and a short-lived elevation of the body temperature.

26 such as basal metabolic rate, body size, or body temperature.

27 wer from physiological fluids and operate at body temperature.

28 rotected mice from cold-induced decreases in body temperature.

29 temic anaphylaxis was measured by changes in body temperature.

30 wakefulness at baseline and at elevated core body temperature.

31 rience substantial and rapid fluctuations in body temperature.

32 he feeding of tissues and the maintenance of body temperature.

33 er arterial blood pressure and have a stable body temperature.

34 hibited a temporary decrease in their winter body temperature.

35 matching the surrounding tissue mechanics at body temperature.

36 ties to use metabolic heat to maintain their body temperature.

37 uralistic seizures induced by increased core body temperature.

38 imental conditions that maintain normal core body temperature.

39 ndence of physiologic processes, focusing on body temperature.

40 se cells are sufficient for acute changes in body temperature.

41 is less important for baseline regulation of body temperature.

42 not have any significant effect on sleep or body temperature.

43 for avoiding thermal extremes and regulating body temperature.

44 wild-type, and this defect is amplified near body-temperature.

45 centrations were lower in lizards with lower body temperatures.

46 usceptible persons may develop lethally high body temperatures.

47 ater electrodermal activity, and even higher body temperatures.

48 sleep's restorative effects depend on lower body temperature [23, 24].

49 We also showed that at body temperatures 3,4-DGE is formed from 3-DG and that c

50 em was used to continuously record activity, body temperature (3 min intervals) as well as the positi

51 itions and condensation is enhanced at human body temperatures (33 degrees C and 37 degrees C) and re

52 from in vitro kinetics drug release at human body temperature (37 degrees C) and hyperthermic tempera

53 Warming to normal body temperature (37 degrees C) does not restore normal

54 fully recovered its original shape at human body temperature (37 degrees C), which indicated the gre

55 n pathogen is exquisitely responsive to host body temperature (37 degrees C), which induces a major m

56 This substantive and continuing shift in body temperature-a marker for metabolic rate-provides a

57 induce lipolysis and, as a result, increase body temperature after refeeding.

58 phase feeding accelerated adaptation of core body temperature and activity rhythms, however, did not

59 tions exhibited a pronounced daily rhythm in body temperature and activity, with low values at sunris

60 There was no correlation between body temperature and baseline plasma corticosterone in f

61 and locomotor activity, along with increased body temperature and BAT gene expression, specifically C

62 ss1r KO mice have significant alterations in body temperature and BAT thermogenic gene expression, pe

63 as observed briefly, but only after the core body temperature and blood pressure began to decrease as

64 Compared to heart rate, body temperature and blood pressure, respiratory rate is

65 gy expenditure and food intake, which lowers body temperature and body weight.

66 was accompanied by an inability to maintain body temperature and by hyperglycaemia.

67 ation in 3-month-old LP offspring normalizes body temperature and causes postprandial hyperglycemia.

68 hermogenic programs, leading to reduced core body temperature and cold intolerance.

69 ical energy into heat to maintain a constant body temperature and counteract metabolic disease.

70 The results show that llamas adjusted their body temperature and daily energy expenditure according

71 as have not lost the ability to adjust their body temperature and daily energy expenditure under adve

72 tion rates due to a 6 degrees C reduction in body temperature and decreased protease activity; and (i

73 penic 3-month-old LP progeny shows increased body temperature and energy dissipation in association w

74 ABSTRACT: We sought to determine if body temperature and energy expenditure are influenced b

75 ency, some species can cease to defend their body temperature and enter a hypothermic and hypometabol

76 oes not result in arrhythmicity because core body temperature and exploratory activity rhythms persis

77 xperiments in rabbit ventricular myocytes at body temperature and found that 1) at a low pacing frequ

78 primary data points were bacteremia (daily), body temperature and heart rate (continuously monitored

79 esent findings reveal fluctuations in distal body temperature and heart rate variability that consist

80 For this purpose, energy expenditure, body temperature and locomotion were measured in seven n

81 onal adjustment of their energy expenditure, body temperature and locomotion, under its natural high

82 WS), as well as the circadian rhythmicity of body temperature and locomotor activity.

83 he novel finding of increased metabolism and body temperature and lower BW in BAT-Kiss1r KOs reveal a

84 because of the tight coupling between their body temperature and metabolic rate.

85 ugh the continuous homeostatic regulation of body temperature and metabolism(1,2), is a defining feat

86 of care, but the inverse association between body temperature and mortality was robust and remained c

87 essment of symptom scores and measurement of body temperature and plasma histamine levels.

88 solid-to-liquid phase change of the metal at body temperature and probe shape deformation to provide

89 Bolus feeding increased body temperature and reduced weight gain compared with c

90 pro-social behavior without decreasing core body temperature and selectively enhanced nucleus accumb

91 or necrosis factor-alpha, sickness symptoms, body temperature and self-reported fatigue, and depressi

92 Here we show that body temperature and sleep responses to acute light expo

93 ls that initiate light's profound effects on body temperature and sleep.

94 p to 18 days, which are characterized by low body temperature and suppressed metabolism.

95 mia, and have a markedly accelerated loss of body temperature and survival after cold exposure compar

96 and glands), that is critical for regulating body temperature and the retention of bodily fluids, gua

97 fluorodeoxyglucose PET to maintain a normal body temperature and to avoid fluorodeoxyglucose uptake

98 SARS-CoV-2-infected ferrets exhibit elevated body temperatures and virus replication.

99 vores both directly (e.g. through changes in body temperature) and indirectly (e.g. through changes i

100 Clinical data (including body temperature) and plasma cytokine concentrations wer

101 ated by lower symptom scores, less change in body temperature, and a lower increase of plasma histami

102 is regulated by the daily subtle rhythms in body temperature, and a new study by Gotic and colleague

103 ite C. neoformans being able to grow at bird body temperature, and are able to escape from bird macro

104 RO5263397 on sleep/wake, locomotor activity, body temperature, and cataplexy were assessed in two mou

105 ol relevant to regulation of blood pressure, body temperature, and cerebral blood flow.

106 pn5-null mice show overactive BAT, increased body temperature, and exaggerated thermogenesis when col

107 likely being a physiologic response to lower body temperature, and has recently been associated with

108 EEG, EMG, body temperature, and locomotor activity data were colle

109 e and structure specific, sensitive to human body temperature, and manipulatable with small molecules

110 cribe a previously unrecognized role of host body temperature, and provide a potential explanation fo

111 camera CCD to monitor the activity, surface body temperature, and respiratory rate of the meadow jum

112 ely correlates with standard metabolic rate, body temperature, and serum thyroxine level.

113 kg subcutaneous) on locomotor activity, core body temperature, and social behavior (social interactio

114 ram, electromyogram, locomotor activity, and body temperature, and the efficacy of the TAAR1 agonist,

115 Changes in body mass, urine osmolality, body temperature, and thirst were monitored.

116 T-ablated mice were able to maintain optimal body temperature ( approximately 35-37 degrees C) during

117 environmental temperature and regulation of body temperature are integral determinants of behaviour

118 Most mammals maintain their body temperature around 37 degrees C, whereas in hiberna

119 erms, which often experience fluctuations in body temperature as a result of their environment.

120 es discover that the immune system regulates body temperature as a strategy to regulate metabolic rat

121 ith a greatly decreased metabolic rate and a body temperature as low as 20 degrees C(6), is regulated

122 rature >=32 degrees C to maintain their core body temperature at 33.5 degrees C had a high likelihood

123 nts a pathogen strictly adapted to the human body temperature, B. bronchiseptica causes infection in

124 lammatory response as assessed by changes in body temperature, basophil activation, and basophil depl

125 thms in host locomotor activity patterns and body temperature become differentially disrupted and in

126 marked improvement in survival, body weight, body temperature, behavior, neuropathology, and disease

127 ity of anaphylaxis as measured by decline of body temperature, behavioral effects, serum IL-4, IgE, a

128 More than half of patients had a body temperature below 38.3 degrees C.

129 TRalpha and TRbeta, most notably heart rate, body temperature, blood glucose, and triglyceride concen

130 mine the effects of IV acetaminophen on core body temperature, blood pressure, and heart rate in febr

131 depletion of SREBP prevented maintenance of body temperature both during circadian cycles as well as

132 hypothalamic signaling pathways that control body temperature (BT), blood pressure (BP), and energy b

133 The preoptic area (POA) regulates body temperature, but is not considered a site for body

134 diac rhythm at baseline, but increasing core body temperature by as little as 3 degrees C causes cove

135 The preoptic area (POA) controls body temperature by modulating BAT activity, but its rol

136 lso modulate localized changes in a wearer's body temperature by nearly 10-fold.

137 Upon contact with serum and at body temperature, C. albicans performs a regulated switc

138 Body temperature can be reliably measured in the oesopha

139 Changes in body temperature can profoundly affect survival.

140 These results suggest body temperature can significantly affect plasma cortico

141 one to precipitation in poikilotherms (their body temperature can vary considerably) needs a companio

142 red metabolic rate) and hypothermia (lowered body temperature) can be effective physiological strateg

143 It is well documented that elevated body temperature causes tumors to regress upon radiother

144 Humans' core body temperature (CBT) is strictly controlled within a n

145 Core body temperature (CBT), salivary melatonin, subjective a

146 physical activity, sleep disruption, or core body temperature (CBT).

147 Order effects, core body temperature changes due to menstruation, and circad

148 ce thermogenesis (primary outcome) and alter body temperature, cold and hunger sensations, plasma met

149 a continuous and more rapid decline in core body temperature compared to low rebound [LR] mattress t

150 of both UCP1 and UCP3 accelerate the loss of body temperature compared to UCP1KO alone during acute c

151 Body temperature control is a critical brain function.

152 Body temperature control is essential for survival.

153 e reproductive system: high frequency distal body temperature (DBT), sleeping heart rate (HR), sleepi

154 al medial preoptic area (vMPO) that mediates body temperature decrease, a negative feedback loop for

155 C2 KO mice were lower body weight, increased body temperature, decreased levels of total cholesterol,

156 ng torpor and hibernation-during which their body temperature decreases far below its homeostatic set

157 y on the planet consists of ectotherms whose body temperature depends on the environmental temperatur

158 Homeotherms maintain a stable internal body temperature despite changing environments.

159 able in TN goats, but was masked by elevated body temperature due to heat load in HS goats.

160 n BAT and is necessary to maintain circadian body temperature during chronic cold exposure.

161 mplant recipients demonstrated elevated core body temperature during cold challenges, enhanced respir

162 exercise capacity, failure to maintain core body temperature during cold stress, and reduced ability

163 However, HFpEF mice failed to regulate body temperature during cold temperature exposure.

164 us, where pregnant females maintained stable body temperatures during winter, while non-pregnant fema

165 impaired cardiac function and maintenance of body temperature, effects that could be rescued by exoge

166 -eye movement sleep (NREMS) and decreases in body temperature, energy expenditure and food intake.

167 llular growth combined with survival at bird body temperature explains the ability of birds to effici

168 Body temperatures for these two species indicate that va

169 Endotoxemia increased body temperature from 36.9 +/- 0.4 degrees C to 38.6 +/-

170 ) with criteria for suspected enteric fever (body temperature >/=38.0 degrees C for >/=4 days without

171 at least two of the following signs: fever (body temperature >38 degrees C), pulmonary infiltrates,

172 7,170 (68.6%) had fever (body temperature >= 37.5 degrees C), and none developed

173 We postulated that body temperature has decreased over time.

174 Continuous assessment of body temperature has traditionally required surgical imp

175 ion of nosocomial infections, and changes in body temperature, heart rate, and minute ventilation.

176 ed for monitoring human vital signs, such as body temperature, heart rate, respiration rate, blood pr

177 In mammals, rhythms in body temperature help to entrain and synchronize circadi

178 throughout the day is controlled not only by body temperature, hormone levels, motivation and mood st

179 n cerebral blood flow (CBF), as well as core body temperature; however, the isolated influence of tem

180 mer gel (NiPAAm-co-AAm) that contracts above body temperature (i.e. at 45 degrees C) to release compo

181 d beta in BAT allowed mice to maintain their body temperature in chronic cold.

182 To maintain core body temperature in cold conditions, mammals activate a

183 ramatically impaired ability to defend their body temperature in coldness.

184 Locomotor activity and body temperature in combination provide more sensitive m

185 n adipose tissue, contributes to maintaining body temperature in mammals through nonshivering thermog

186 150,280; 2007-2017)--we determined that mean body temperature in men and women, after adjusting for a

187 Meal ingestion increases body temperature in multiple species, an effect that is

188 ognized role for FGF21 in the maintenance of body temperature in response to cold.

189 GF21 and its site of action to regulate core body temperature in response to cold.

190 Increased body temperature in survivors was also associated with s

191 and F-IR/IGFRKO mice were unable to maintain body temperature in the cold and developed severe diabet

192 fants being allowed to passively lower their body temperature in the days after birth, is an emerging

193 of critically ill septic patients, increased body temperature in the emergency department was strongl

194 especially mammals, which maintain constant body temperature in the face of environmental temperatur

195 Both compounds reduced body temperature in the two narcolepsy models at the hig

196 nd Trpv1(-/-) mice, but only restored normal body temperature in Trpa1(-/-) and Trpa1(-/-)/Trpv1(-/-)

197 ian dinosaurs and that not all dinosaurs had body temperatures in the range of that seen in modern bi

198 rms of the similarity in 24 h rhythm of core body temperature, in weeks when food was only available

199 Body temperature increased 2 degrees C, and gut permeabi

200 E was 65.7 (13.5) minutes; and the mean core body temperature increased by 0.46 degrees C (0.20 degre

201 Glu(2/3) receptor agonist (LY379268)-induced body temperature increases in mice lacking the Grm3, but

202 tinic acid, did not affect sleep amounts and body temperature, indicating that it is not nicotinamide

203 on norm slopes but have diverged in baseline body temperature (intercepts), being higher for the more

204 Maintaining a stable body temperature is a fundamental requirement for homeot

205 Body temperature is an important physiological parameter

206 erosis, exacerbation of symptoms with rising body temperature is associated with impulse conduction f

207 Homeostatic control of core body temperature is essential for survival.

208 Core body temperature is normally tightly regulated to within

209 urons being GABAergic.SIGNIFICANCE STATEMENT Body temperature is regulated by the CNS.

210 e survival at 90 days and time to reach core body temperature less than 34 degrees C.

211 mptoms, resulting in resistance to decreased body temperature, less severe reactions, and maintained

212 tress, and some changes (PS bouts, SWS time, body temperature, locomotor activity) persisted after th

213 ch is a candidate region associated with the body temperature maintenance phenotype in Siberian cattl

214 h for detecting genomic intervals related to body temperature maintenance under acute cold stress.

215 Body temperature may affect glucocorticoid concentration

216 In ectotherms, behaviors that elevate body temperature may decrease parasite performance or in

217 Antipyretic therapy decreased body temperature (mean difference, -0.38 degrees C; 95%

218 anaphylaxis, reflected by increased drop in body temperature, most likely due to accelerated histami

219 es at mid-latitudes where the hottest hourly body temperatures occurred; by contrast, the marine spec

220 Four HCWs reached or exceeded a mean core body temperature of >/= 38.5 degrees C.

221 lled at ~38-40 degrees C (slightly above the body temperature of 37 degrees C) for future in vivo USF

222 orted severe fever in the previous 24 h or a body temperature of 37.8 degrees C or more at presentati

223 ich plays essential roles in maintaining the body temperature of infants.

224 ss to the core neural circuit regulating the body temperature of mammals.

225 f the temperature of the environment and the body temperatures of the animals.

226 LR) OR, 1.14/increase (p < 0.001)], elevated body temperature on admission [OR, 1.53/ degrees C incre

227 e the impact of feeding-induced increases in body temperature on energy balance, we compared rats fed

228 nd field measurements to test the effects of body temperature on plasma corticosterone (predominant g

229 as the regulation of blood pressure and core body temperature, oncogenesis, and immune function(3).

230 Plasma hyperosmolality delayed the mean body temperature onset of sweating (+1.24 +/- 0.18 vs. +

231 Importantly, no changes in body temperature or food intake were observed.

232 hen determinations were based on either core body temperature or plasma melatonin measurements, as co

233 ithout altering food intake, lean body mass, body temperature, or biochemical and haematological mark

234 The rise of the body temperature, or fever, is an important brain-orches

235 We measured core body temperature over most of the gestation period in bo

236 Core body temperature, PET imaging of glucose uptake and VO(2

237 To check the influence of subject-specific body temperature, pH, H2(18)O, and cellular produced CO2

238 emperature similar to torpor, as measured by body temperature, physical activity, indirect calorimetr

239 Anaphylactic symptom scores, core body temperatures, plasma histamine levels, basophil num

240 es were necessary and sufficient to increase body temperature postprandially, a process that required

241 d computational analyses, we show that human body temperature promotes unfolding of the central dimer

242 ible pharmacological response as measured by body temperature reduction.

243 bsequent Blo t 5 challenge induced decreased body temperature, reduction in movement, and a fall in s

244 c anaphylaxis, antigen-dependent decrease in body temperature, reflecting the anaphylactic reaction,

245 quired for growth, metabolism, breeding, and body temperature regulation (diurnal variation, response

246 tor 1 (TRPV1), besides being involved in the body temperature regulation and in the response to pain,

247 the immune response, insulin secretion, and body temperature regulation.

248 r growth, wound healing, water repulsion and body temperature regulation.

249 static control of breathing, heart rate, and body temperature relies on circuits within the brainstem

250 hese behavioral consequences, although basal body temperature remained elevated, comparable to tumor-

251 ogical processes and behaviors as diverse as body temperature, respiration, aggression, and mood.

252 Sleep, wakefulness and body temperature rhythm were monitored throughout.

253 maintenance of animal locomotor activity and body temperature rhythmicity.

254 and renders circadian locomotor activity and body temperature rhythms unstable.

255 Nontargeted screening measures, including body temperature screening among employees and visitors

256 coordinated depression of metabolic rate and body temperature similar to torpor, as measured by body

257 s had browning of SAT, based on increases in body temperature, starting 18 months before PDAC diagnos

258 Measurements of circadian rhythms in body temperature suggest a biological mechanism through

259 Mammals maintain a nearly constant core body temperature (T(b)) by balancing heat production and

260 Endotherms can maintain their body temperature (T(b)) over a wide range of ambient tem

261 se recordings as a proxy for continuous core body temperature (T(core)) measurements has not been ass

262 es (TPCs), which quantify how an ectotherm's body temperature (Tb ) affects its performance or fitnes

263 Rectal, mean skin and mean body temperature (Tb ), electromyographic activity (EMG)

264 to investigate the potential causes of high body temperature (Tb) during lactation in mice as a puta

265 This stereotypic rise in body temperature (Tb) in response to inflammatory stimul

266 sLH neurons attenuates the arousal, LMA, and body temperature (Tb) responses to a psychological stres

267 Mammals typically keep their body temperature (Tb) within a narrow limit with changin

268 EEG, EMG, body temperature (Tb), and locomotor activity (LMA) were

269 e scarce, endothermic animals can lower core body temperature (Tb).

270 are partly mediated by the lowering of core body temperature that occurs during CR.

271 f plasma uridine is required for the drop in body temperature that occurs during fasting.

272 how that marine ectotherms experience hourly body temperatures that are closer to their upper thermal

273 ysiological effect of norepinephrine on core body temperature, the fast increase of iBAT temperature

274 t calorie restriction, IGF-1R signaling, and body temperature, three of the main regulators of metabo

275 ptic area of the hypothalamus that regulates body temperature to counter fever.

276 at room temperature and rapidly rigidify at body temperature to form hydrogels mechanically and stru

277 s and ectotherms (e.g., fish) increase their body temperature to limit pathogen infection.

278 We recently found that distinct body temperature trajectories of infected patients corre

279 nisms, understanding the mechanisms by which body temperatures translate into major biogeographic pat

280 In mice, the hit compound increased body temperature, UCP1 protein levels, and thermogenic g

281 y shown to cause fever and viremia, and core body temperature, viremia, and blood cell and chemistry

282 Body temperature was associated independently with clini

283 Among vital signs, body temperature was best at predicting mortality.

284 Core body temperature was controlled using an intravascular d

285 tivity (SWA) was significantly decreased and body temperature was increased with a delay of 5-6 h.

286 Body temperature was measured and classified according t

287 Body temperature was measured by means of telemetry, Il3

288 rate and forearm blood flow relative to mean body temperature was unaffected by the OCP cycle (both P

289 fically in BAT influences BW, metabolism, or body temperature, we used Cre/lox technology to generate

290 nvolved in the regulation of blood pressure, body temperature, weight and the response to pain.

291 Decreased body temperature, weight loss, splenomegaly, and deforma

292 Cardiovascular measures and body temperature were also assessed.

293 /kg, i.v.) on food-maintained responding and body temperature were determined in these subjects prior

294 ylation in response to a 2 degrees C rise in body temperature were determined with the use of Caco-2

295 Odds ratios for mortality according to body temperature were estimated using multivariable logi

296 Changes in the body temperature were not correlated with changes in mea

297 Minute ventilation and body temperature were unchanged during dialysis (1.2 +/-

298 or mechanical ventilation, or changes in the body temperature) were independently associated with ace

299 vital signs (heart rate, pulse oximetry, and body temperature) were monitored at regular intervals.

300 at leucine enkephalin directly controls core body temperature when exogenously injected into the hypo

301 also manifested a defective control of their body temperature, which was overly reduced.