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1 sponse to up to 31 drugs within 5 days after bone marrow biopsy.
2 red with tryptase in estimating the need for bone marrow biopsy.
3 cell aggregates, and atypical mast cells on bone marrow biopsy.
4 ish M2, t[8;21][q22;q22.1]) was diagnosed on bone marrow biopsy.
5 fficient accuracy to replace routine staging bone marrow biopsy.
6 , urine and serum immunoelectrophoresis, and bone marrow biopsy.
7 the diagnosis usually depends on results of bone marrow biopsy.
8 emic mastocytosis) and thus candidates for a bone marrow biopsy.
9 ng pure red cell aplasia (PRCA) confirmed by bone marrow biopsy.
10 pared with hematologic response criteria and bone marrow biopsies.
11 ot spot density compared with normal control bone marrow biopsies.
12 nical relevance of expression in 55 archival bone marrow biopsies.
13 e myeloid leukemia (AML) routinely undergo a bone marrow biopsy 7-10 days after induction chemotherap
18 nohistochemically by factor VIII staining of bone marrow biopsies and quantified by assessment of mic
19 umbers of blood vessels were measured in 145 bone marrow biopsies and the levels of vascular endothel
20 Patients with low-risk MGUS can safely defer bone marrow biopsy and advanced imaging, and should unde
22 enia and negligible gene marking, diagnostic bone marrow biopsy and aspirate were performed at day 88
25 ermediate- and high-risk MGUS should trigger bone marrow biopsy and bone imaging to detect overt MM a
27 cible ischemia in Tc-99m SPECT who underwent bone marrow biopsy and were allocated to cells (n=16) or
28 cible ischemia in Tc-99m SPECT who underwent bone marrow biopsy and were allocated to cells (n=16) or
29 work-up consisted of a complete blood count, bone marrow biopsy, and immunohistochemical and histoche
30 agnosis, in the selection of those needing a bone marrow biopsy, and in the documentation of disease
31 ria to identify those patients who require a bone marrow biopsy, and whether the pathogenesis of IA i
32 s or suspicious lymphocytic infiltrates in a bone marrow biopsy as the sole suggestion of residual di
33 )At-radioimmunotherapy, after lymph node and bone marrow biopsies at 2-4 and/or 19 h after injection.
34 notyping, as well as follow-up allograft and bone marrow biopsies at 3 and 9 months, including analys
35 The diagnosis of 661 PMF patients with a bone marrow biopsy at presentation was revised according
36 one-metastatic CRPC who underwent transiliac bone marrow biopsy between October 2007 and March 2010.
38 B-cell lymphoma (DLBCL), the sensitivity of bone marrow biopsy (BMB) for the detection of bone marro
40 aled an IgG kappa monoclonal gammopathy, and bone marrow biopsy confirmed smoldering multiple myeloma
44 l and transformed plasma cells from the same bone marrow biopsy enables discovery of patient-specific
45 agrelide therapy should undergo surveillance bone marrow biopsy every 2 to 3 years and that those who
47 r urticaria pigmentosa or the characteristic bone marrow biopsy finding of multifocal mast-cell aggre
50 rt a comprehensive proteogenomic analysis of bone marrow biopsies from 252 uniformly treated AML pati
52 iption factor (MITF), is highly expressed in bone marrow biopsies from 9 of 10 patients with systemic
59 simultaneous analysis of WM patient sera and bone marrow biopsies identified a set of dysregulated cy
60 nohistochemical paraffin section staining of bone marrow biopsies in the staging of B-cell malignant
63 commonly used test to estimate the need for bone marrow biopsy in patients suspected to have indolen
65 tumor DNA (ctDNA) is directly comparable to bone marrow biopsy in representing the genomic heterogen
66 o detected by immunohistochemistry in normal bone marrow biopsies, indicating an in vivo function.
72 In-DOTA-anti-CD45 antibody (BC8) imaging and bone marrow biopsy measurements to ascertain the biodist
73 aranase activity in the plasma isolated from bone marrow biopsies of 100 patients reveals 86 positive
74 e to quantify IDO-1 expression on diagnostic bone marrow biopsies of AML patients in order to facilit
75 this cutoff correctly classifies diagnostic bone marrow biopsies of MPN,U patients specified upon fo
78 ic health record; review of laboratory data; bone marrow biopsy pathology analysis; and in vitro enzy
79 drome features with the exception that their bone marrow biopsy pathology revealed abundant neutrophi
80 decreased risk of identifying pathology on a bone marrow biopsy performed for a low WBC count (odds-r
82 e and post-treatment assessments were serial bone-marrow biopsy, peripheral blood collections, stagin
83 g/gm Cr corresponded with the high degree of bone marrow biopsies positive for atypical mast cells, t
88 a murine model of Ph+ B-ALL as well as human bone marrow biopsy samples to assess the fundamental nat
89 90 ng/mL, <15% infiltration by mast cells in bone marrow biopsy-sections, and diagnosis of ISM were i
97 The TMA was constructed using pretreatment bone marrow biopsy specimens from 64 adult patients with
98 orulae were detected on peripheral smear and bone marrow biopsy specimens, and PCR amplified Ehrlichi
108 ent monoclone of IgG1, the patient underwent bone marrow biopsy which revealed Monoclonal Gammopathy