ライフサイエンスコーパス: bone marrow cell

1 d a growth advantage in the presence of host bone marrow cells.

2 chemotherapy-resistant MM, making up 90% of bone marrow cells.

3 arked mature peritoneal B-1a cells and adult bone marrow cells.

4 production in MDSCs when compared to control bone marrow cells.

5 locus yeast artificial chromosome (beta-YAC) bone marrow cells.

6 on, thus promoting replenishment of depleted bone marrow cells.

7 /2 in mouse lineage marker negative (Lin(-)) bone marrow cells.

8 h Ldlr(-/-) mice transplanted with wild-type bone marrow cells.

9 ivo, and the same was observed with CD11b(-) bone marrow cells.

10 ent with an in vitro perturbation of Klf7 in bone marrow cells.

11 , and solute transporters but not markers of bone marrow cells.

12 ral toxicity of shRNAs, we used normal mouse bone marrow cells.

13 2 cells on the gel as bone lining cells, and bone marrow cells.

14 wsh) mice and were labeled with transplanted bone marrow cells.

15 or successful in vitro osteoclastogenesis of bone marrow cells.

16 , were upregulated by GRO-gamma-primed mouse bone marrow cells.

17 markedly impaired as compared with wild-type bone marrow cells.

18 x-2 gene and activates beta-catenin in mouse bone marrow cells.

19 losuppression and by using moderate doses of bone marrow cells.

20 1-mutated patients but not of normal CD34(+) bone marrow cells.

21 number and function of fetal liver (FL) and bone marrow cells.

22 st, impaired hematopoietic reconstitution of bone marrow cells.

23 cant downregulation of miR-30c expression in bone marrow cells.

24 autoimmunity, and direct viral infection of bone marrow cells.

25 and osteoclasts were generated in vitro from bone marrow cells.

26 e receiving a transplant of Cited2 deficient bone marrow cells.

27 factors in leptin-receptor-positive stromal bone marrow cells.

28 the CD11c(+) and CD11b(+) populations in the bone marrow cells.

29 nd 3) mixtures of 4T1 tumor cells and murine bone marrow cells.

30 es diminished the pro-metastatic behavior of bone marrow cells.

31 wild-type but not HIF-1alpha(fl/fl) LysM-Cre bone marrow cells.

32 production by Rag2(-/-)gamma(c)(-/-) myeloid bone marrow cells.

33 eased colony forming units osteoblastic from bone marrow cells.

34 rtially recapitulated in murine Dnmt3a (-/-) bone marrow cells.

35 tion of myeloablated adult mice similarly to bone marrow cells.

36 ted pit formation caused by RANKL-stimulated bone marrow cells.

37 tion of exosomes produced by GM-CSF-expanded bone marrow cells.

38 performed whole-genome ChIP-Seq using mouse bone marrow cells.

39 OCs, in primary cultures of RANKL-stimulated bone marrow cells.

40 myeloid leukemia (AML) compared with normal bone marrow cells.

41 clusion IRE can induce nanopore formation in bone marrow cells.

42 e marrow proviral load 14-fold, and infected bone marrow cells 7-fold, but no inhibition was observed

43 es as well as numbers of splenic B cells and bone marrow cells after different immunization protocols

44 Purpose To assess for nanopore formation in bone marrow cells after irreversible electroporation (IR

45 After elimination of pre-osteoclasts from bone marrow cells, alendronate did not affect osteoblast

46 The immense plasticity of bone marrow cells allows them to populate diverse tissue

47 experiments using FLVCR-deleted and control bone marrow cells, along with wild-type competitor cells

48 n, followed by transplantation of 10 million bone marrow cells and 20 million splenocytes from either

49 We studied primary MDS-enriched bone marrow cells and bone marrow sections by western bl

50 AF6 is expressed in the cytoplasm of healthy bone marrow cells and controls rat sarcoma viral oncogen

51 itively correlated with MYC expression in MM bone marrow cells and correspondingly, genetic knockdown

52 n, plasma cells in popliteal lymphoid nodes, bone marrow cells and granulocyte-macrophage progenitor

53 umor cells enhances differentiation of mouse bone marrow cells and human PBMC into immunosuppressive

54 her SCL expression upregulates Kit in normal bone marrow cells and increases chimerism after bone mar

55 Ectopic expression of C/EBPalpha in mouse bone marrow cells and monocyte/macrophage cells, in the

56 Bone marrow cells and mouse embryonic fibroblasts derive

57 Despite decreases in ROS generation in bone marrow cells and p47(phox)-Nox2 signaling in osteob

58 g misoprostol decreased M-CSFR expression in bone marrow cells and reduced the number of peritoneal m

59 Cell Atlas Census of Immune Cells dataset of bone marrow cells and show that it substantially improve

60 PI3 kinase/Akt or Raf/MEK/ERK activation in bone marrow cells and transplanted them into recipient m

61 receptor family, is expressed in myeloid and bone marrow cells and was implicated as a checkpoint reg

62 CCR5-/- mice reconstituted with wild-type bone marrow cells and wild-type mice reconstituted with

63 Bone marrow cells and, alternatively, fetal liver cells

64 itors, increased long-term reconstitution of bone marrow cells, and a chronic myeloproliferative neop

65 c stem cells, reduced telomerase activity in bone marrow cells, and altered the gut microbiota.

66 ice, which are reconstituted with Tlr2 (-/-) bone marrow cells, and in germ-free mice.

67 e most abundant PGE(2) receptor expressed by bone marrow cells, and its expression further increased

68 cells represent a relatively small subset of bone marrow cells, and need to be enriched prior to anal

69 We analyzed blood, lymph node, bone marrow cells, and serum before desensitization, aft

70 Here, we tested the hypothesis that bone marrow cell APOE genotype modulates pathological pr

71 romal-interacting molecule 1 (STIM1) gene in bone marrow cells are more susceptible to bacterial infe

72 When WT beta-YAC bone marrow cells are treated with the OGA inhibitor Thi

73 ke cell line, designated DC9, from Irf8(-/-) bone marrow cells as a model for DC development and func

74 5RA(-)CD123(-)CD71(+)CD41a(-)CD105(-)CD36(-) bone marrow cells as EEP giving rise to BFU-E, and Lin(-

75 clastogenesis assays using RAW264.7 cells or bone marrow cells as osteoclast precursors, addition of

76 Mechanistic studies define bone marrow cells as the source of the IL-10 signal and

77 e in telomere length in peripheral blood and bone marrow cells, as well as improved blood counts.

78 kemia-associated mutations in at least 5% of bone marrow cells at remission.

79 living donor or a deceased donor with frozen bone marrow cells available.

80 Clinical trials of bone marrow cell-based therapies after acute myocardial

81 d chemotherapy-resistant MM making up 80% of bone marrow cells before treatment.

82 These subsets also play a role in bone marrow cell (BMC) allograft rejection.

83 was added to either D3- or PTH-treated mouse bone marrow cell (BMC) cultures.

84 beta/Fc, or IL-2/Fc would enhance allogeneic bone marrow cell (BMC) engraftment and promote tolerance

85 ding the uncertainties about the outcomes of bone marrow cell (BMC) therapy for heart repair, further

86 In various organs, bone marrow cell (BMC) therapy has shown promising preli

87 nthusiasm, the therapeutic benefits of adult bone marrow cell (BMC) transplantation in patients with

88 Bone marrow cell (BMC)-based treatment for critical limb

89 asis, we found that a specific population of bone marrow cells (BMC) upregulated CSF-1R and secreted

90 transfected a)K562 cells; b)murine beta-YAC bone marrow cells (BMC); c)human haematopoietic progenit

91 CXCR4-positive bone marrow cells (BMCs) are critically involved in card

92 immunological tolerance using high doses of bone marrow cells (BMCs) has been demonstrated in mixed

93 n mice transplanted with Gfi-1(-/-);Id2(+/-) bone marrow cells (BMCs) in comparison with Gfi-1(-/-) B

94 c, effect of DG172 on the differentiation of bone marrow cells (BMCs).

95 ated wild-type recipients of PDIA6-deficient bone marrow cells, both in the absence or presence of co

96 RANTES production by Rag2(-/-)gamma(c)(-/-) bone marrow cells, but the presence of gamma(c) did not

97 rofiles for macrophages, derived from murine bone marrow cells by each CSF.

98 response was defined as < 1 tumor cell/10(4) bone marrow cells by flow cytometry.

99 Transplantation of MDSCs, generated from bone marrow cells by granulocyte-macrophage colony-stimu

100 otent stem cells such as C3H10T1/2 and human bone marrow cells can be committed to the osteogenic lin

101 osteoclastogenesis of C/EBPalpha(-/-) mouse bone marrow cells can be rescued by c-fos overexpression

102 estrogen-regulated form of Hoxb8 into mouse bone marrow cells can be used along with Flt3 ligand to

103 Previous studies have shown that mouse bone marrow cells can produce mast cells when stimulated

104 he demonstration that intravenously injected bone marrow cells can rescue irradiated mice from lethal

105 stromal compartment provides a comprehensive bone marrow cell census and experimental support for can

106 nced differentiation into osteoclasts of the bone marrow cells co-cultured with galectin-8-treated os

107 sm after transplantation of 20 million total bone marrow cells, compared with <= 2.1% hematopoietic c

108 increased cytokine expression in mesenchymal bone marrow cells contributes to the inflammatory phenot

109 ed (i) a reduced spleen weight; (ii) reduced bone marrow cell counts and proliferation (BrdUrd flow c

110 In the bone marrow cell culture, the conditioned medium from ML

111 aP and TCDD enhanced osteoclast formation in bone marrow cell cultures and gavage with BaP stimulated

112 Studies using ex vivo bone marrow cell cultures showed that TSH inhibits and s

113 mice in the aorta, draining lymph nodes, and bone marrow cell cultures, indicating that IRF5 maintain

114 In ex vivo bone marrow cell cultures, SCI increased the number of o

115 ene imaging in mice with tMCAO revealed that bone marrow cell cycling peaked 4 days after stroke (P<0

116 ylaxis, and 1.5 x 10(7) haploidentical donor bone marrow cells (day 0).

117 RF-4 also functions as a tumor suppressor in bone marrow cells deficient in MyD88, an IRF-4-interacti

118 reported that ApoeKO mice transplanted with bone marrow cells deficient in the Transient Receptor Po

119 chimeric mice implanted with P2X7R-deficient bone marrow cells, defining hematopoietic cells as a suf

120 cells as well as the use of Hsf1 null mouse bone marrow cells demonstrated that 17-AAG-enhanced oste

121 F and receptor activator of NF-kappaB ligand bone marrow cells derived from BLT1(+/+) and/or BLT1(-/-

122 ChIP-Seq analysis of bone marrow cells derived from six acute myeloid leukemi

123 press PML-RARA, RUNX1-RUNX1T1, or MLL-AF9 in bone marrow cells derived from WT or DNMT3A-deficient mi

124 Treatment with specific bone marrow cell-derived secreted proteins may provide a

125 proresolution mediator resolvin E1 (RvE1) on bone-marrow-cell-derived osteoclasts in an in vitro muri

126 use IM insensitivity, because primary murine bone marrow cells engineered to express low amounts of B

127 terozygous inactivation of Nf1 (Nf1(+/-)) in bone marrow cells enhances neointima formation following

128 ex vivo generated MDSC from cytokine-treated bone marrow cells (evMDSC) suppressed neovascularization

129 dlr(-/-)) mice transplanted with Sptlc2(+/-) bone marrow cells exhibited significantly fewer atherosc

130 ne methylation patterns occurred in cultured bone marrow cells exposed to mammary tumor-conditioned c

131 tmentin vitro In this study, we transplanted bone marrow cells expressing green fluorescent protein,

132 d poorly in wild-type (WT) recipients and KO bone marrow cells failed to radioprotect lethally irradi

133 Cultivation of pig bone marrow cells for 5-7 d in presence of human rCSF-1

134 caerulein injections and isolated acinar and bone marrow cells for ex vivo studies.

135 HT7(-/-) mice and in mice reconstituted with bone marrow cells from 5-HT7(-/-) mice compared with con

136 Using a leukemic cell line and diagnostic bone marrow cells from 6 patients with B-progenitor cell

137 Towards this end, bone marrow cells from Axl(-/-) or wild-type mice were t

138 ice were irradiated and given transplants of bone marrow cells from C57BL6 mice, with or without the

139 Chimera mice reconstituted with bone marrow cells from CD11c-diphtheria toxin receptor (

140 Bone marrow cells from CLP-treated mice had normal OC pr

141 veloping in chimeric mice reconstituted with bone marrow cells from COL-EGFP mice very rarely showed

142 In addition, nonadherent bone marrow cells from Cx37(-/-) mice exhibit higher lev

143 on in mouse and human progenitors, including bone marrow cells from DBA patients.

144 restore expression of DNMT3A in transplanted bone marrow cells from Dnmt3a (-/-) mice.

145 Following transplantation with bone marrow cells from Foxp3-deficient but not wild-type

146 We transplanted bone marrow cells from Fx-/- or control mice (Ly5.2) int

147 Analysis of blood and bone marrow cells from healthy and eosinophilic donors a

148 imited changes in TNT formation similarly to bone marrow cells from healthy individuals.

149 ansplant experiments in which we transferred bone marrow cells from Id cDKO mice into lethally irradi

150 es were not altered by the reconstitution of bone marrow cells from IDO(+/+) mice.

151 sis after administration of DSS or TNBS, but bone marrow cells from Il1rl2(-/-) mice did not prevent

152 Bone marrow cells from immunocompetent mice were isolate

153 Furthermore, transplantation of bone marrow cells from integrin-beta3 KO mice into WT mi

154 17F mutation in hematopoiesis, we transduced bone marrow cells from Jak2(V617F) knockin mice with len

155 nd and a higher level of PARP3 expression in bone marrow cells from mice as compared with rats and hu

156 y in multiple myeloma cell lines or CD138(+) bone marrow cells from multiple myeloma patients comprom

157 -exposed mice promoted eosinophilopoiesis in bone marrow cells from naive mice, which was blocked by

158 myeloid leukemia (AML) compared with CD34(+) bone marrow cells from normal donors.

159 An analysis of bone marrow cells from patients and mice with X-CGD reve

160 formed a bioinformatic secretome analysis in bone marrow cells from patients with acute MI and discov

161 SULF2 in an in silico secretome analysis in bone marrow cells from patients with acute myocardial in

162 We found that bone marrow cells from patients with MM secreted higher

163 d in multiple myeloma cell lines and primary bone marrow cells from patients.

164 Bone marrow cells from preleukemic CD19-CreDeltaPB mice

165 Competitive transplantation of bone marrow cells from PTPsigma-deficient mice revealed

166 Using bone marrow cells from Rc3h1(gt/gt) mice transferred int

167 Bone marrow cells from the cured mice displayed normal c

168 Moreover, isolated bone marrow cells from the sclerostin null mice show imp

169 using transplantation with lineage-negative bone marrow cells from wild-type mice.

170 and microarray analysis in lineage-negative bone marrow cells from young and aged wild-type and apol

171 ould prolong VCA survival and required donor bone-marrow cells, given that bone-marrow might promote

172 Dectin-1-dependent manner in GM-CSF-derived bone marrow cells (GM-BM).

173 ce reconstituted with MTMR9 shRNA-transduced bone marrow cells had an elevated proportion of T-box tr

174 that AKT3(-/-) mice receiving AKT3-deficient bone marrow cells had elevated clinical scores relative

175 Fx-/- mice receiving control bone marrow cells had intestinal inflammation and dyspla

176 irradiated wild-type mice reconstituted with bone marrow cells harvested from 5-HT7(-/-) mice.

177 in vivo labeling studies, where normal mouse bone marrow cells (i.e. high turnover) were evaluated po

178 In contrast, JAM-A deficiency in bone marrow cells impeded monocyte de-adhesion, thereby

179 he bcl-2/IgH translocation was determined on bone marrow cells in a centralized laboratory belonging

180 Measurement of protein synthesis within bone marrow cells in a cohort of six mice can be achieve

181 row transplantation of Apoe(-/-) Malat1(-/-) bone marrow cells in Apoe(-/-) Malat1(+/+) mice enhanced

182 kemia-associated mutations in at least 5% of bone marrow cells in day 30 remission samples was associ

183 elevant for irradiation-induced depletion of bone marrow cells in mice.

184 Nearly all the bone marrow cells in patients with myelodysplastic syndr

185 ent death of intestinal epithelial cells and bone marrow cells in response to double-strand DNA break

186 stimulation increased IL-1beta expression in bone marrow cells in wild-type, Tie2Cre-IL-1R1(r/r), and

187 tors that cause DNA damage in cord blood and bone marrow cells, including stem cells.

188 t the conditional abrogation of IRE1alpha in bone marrow cells increases bone mass as the result of d

189 es eosinophil differentiation of low-density bone marrow cells independent of further IL-5 stimulatio

190 ow bone mass phenotype, we demonstrated that bone marrow cell-induced osteoclastogenesis was reduced

191 uates the effect of repeated intramyocardial bone marrow cell injection in patients with residual or

192 Repeated bone marrow cell injection in previously responding pati

193 Intramyocardial bone marrow cell injection is associated with improvemen

194 Reconstitution of bone marrow cells into a TGF-alpha transgenic mouse mode

195 3(+/-); Apc(del/+); or Egr1(+/-), Apc(del/+) bone marrow cells into lethally irradiated Apc(del/+) re

196 nsdifferentiated human myeloid lineage CD34+ bone marrow cells into neural progenitors.

197 Transplantation of young bone marrow cells into old recipients prevented sarcopen

198 nsfer of Cx3cr1-proficient monocyte-enriched bone marrow cells into septic Cx3cr1-depleted mice preve

199 ction and perfusion with direct injection of bone marrow cells into the hearts of patients with ische

200 ued by adoptive transfer of affected HSCs or bone marrow cells into Treg-competent recipients.

201 Transplantation of Lrp5 null bone marrow cells into wild-type mice did not limit fibr

202 ther wild-type (WT) or Pax5+/- hematopoietic bone marrow cells into WT recipient mice revealed that t

203 Transplantation of old bone marrow cells into young animals reduced satellite c

204 studies demonstrated that LRG1 derived from bone marrow cells is required for normal wound healing,

205 cated that blocking inflammasome activity in bone marrow cells is sufficient to improve healing.

206 Bone marrow cells isolated based solely on Fgd5 reporter

207 It is noteworthy that transplantation of bone marrow cells isolated from Akt2(-/-) mice to Ldlr(-

208 Indeed, bone marrow cells isolated from Malat1(-/-) mice showed

209 mice, obtain cells of interest (here, we use bone marrow cells) just 1 h later, and quantify the amou

210 However, even when 95% of the donor bone marrow cells lacked FLVCR, all red cells in recipie

211 rs and that of inflammatory macrophages from bone marrow cells leads to macrophage heterogeneity.

212 Targeting senescence in the BMAd or other bone marrow cells may represent a novel therapeutic appr

213 of the bone marrow indicated that progenitor bone marrow cells MPP2 and CMP were upregulated in GT3-N

214 Here, we show that non-(plastic)-adherent bone marrow cells (NABMCs) are more potent osteoprogenit

215 of soluble intracellular contents from whole bone marrow cells, named "Bone Marrow (BM) Soup", was re

216 howed negligible cytotoxic effects in normal bone marrow cells not expressing activated STAT5 protein

217 ecific PAP-iPSCs were generated from CD34(+) bone marrow cells of a CSF2RA-deficient patient with PAP

218 The bone marrow cells of Dnmt3a+/- mice had a subtle but sta

219 CLP did not promote OC formation from bone marrow cells of Itch-/- mice in vitro nor induce bo

220 ing and cause leukemia when expressed in the bone marrow cells of mice.

221 terminal erythropoiesis of lineage-negative bone marrow cells of Thra1 (PV/+) mice was rescued by th

222 lectron microscopy demonstrated nanopores in bone marrow cells only after IRE (P , .01).

223 mals in which iNOS or PARP1 was deleted from bone marrow cells only.

224 er obtained from in vitro differentiation of bone marrow cells or isolated from infarcted hearts alte

225 Using bone marrow cells or macrophages differentiated in vitro

226 entiation without general toxicity to normal bone marrow cells or non-MLL cells.

227 regeneration by the transdifferentiation of bone marrow cells or putative adult resident cardiac pro

228 physiological patterns of hematopoiesis and bone marrow cell outputs depend on the expression of ACK

229 The majority of WT recipients of Id cDKO bone marrow cells phenocopied Id cDKO cardiac fibrosis 4

230 ential for the differentiation of alphaLP, a bone marrow cell population that gives rise to all known

231 Mechanistically, stimulation of specific bone marrow cell populations in vivo using growth factor

232 the course of mast cell differentiation from bone marrow cells, procaspase-3 was present in cells of

233 t conditioned media from young, but not old, bone marrow cells promoted myoblast proliferation in vit

234 ether, our results demonstrate that aging of bone marrow cells promotes the age-related reduction of

235 Transplantation of Hb Null-derived bone marrow cells provides short-term radioprotection of

236 mice transplanted with Nras(G12D/+)p53(-/-) bone marrow cells rapidly develop a highly penetrant AML

237 xperiments indicated that CRAMP derived from bone marrow cells rather than structural cells was respo

238 NKL/M-CSF treatment of nonadherent Cx37(-/-) bone marrow cells rendered a 5-fold lower level of osteo

239 Bone marrow cells represent an additional source of GAD

240 Transplanting KO mice with wild-type bone marrow cells rescued the angiogenic defect and amel

241 9-Tert treatment after telomere attrition in bone marrow cells rescues aplastic anemia and mouse surv

242 ic day 18.5 fetal liver and adult spleen and bone marrow cells, respectively.

243 Bone marrow cell response after injury and during early

244 ent feedback mechanism through which myeloid bone marrow cells restore quiescence of myeloid-biased H

245 IL-5 stimulation of low-density bone marrow cells resulted in expression of a panel of c

246 Induced deletion of P38alpha in bone marrow cells retards the maturation of mast cells i

247 ut the presence of gamma(c) did not increase bone marrow cell sensitivity to IL-15.

248 In mice, FcgammaRIIA expression by bone marrow cells severely aggravated lupus nephritis an

249 lantation experiments indicated that A(2b)AR bone marrow cell signals alone were not sufficient to el

250 he murine context, silencing of AID in human bone marrow cells skews differentiation toward myelomono

251 osis in CKD mice with the deletion of MPO in bone marrow cells, strongly implicating bone-marrow-deri

252 nstituted with Adora2a(+/+) and Adora2a(-/-) bone marrow cells suggest that decreased IL-7Ralpha in n

253 significantly effecting the growth of normal bone marrow cells, suggesting a favorable therapeutic in

254 Pig BMDM could be generated from bone marrow cells that had been stored frozen and thawed

255 cells give rise to megakaryocytes, the giant bone marrow cells that in turn break down to form blood

256 ticated interaction network between multiple bone marrow cells that regulate different hematopoietic

257 rine vomeronasal sensory neurons but also in bone marrow cells, the primary source for immune cell re

258 signals enhance the homing and attachment of bone marrow cells to bone surfaces and the commitment to

259 suppressed Flt3-L-induced differentiation of bone marrow cells to pDCs in wild-type but not HIF-1alph

260 r, the capacity of genetically B2R-deficient bone marrow cells to promote endothelial repair in vivo

261 TBI activates p53 to decrease the ability of bone marrow cells to suppress lymphoma development throu

262 IMS: There is growing interest in the use of bone marrow cells to treat liver fibrosis, however, litt

263 +)c-kit(+) cells from mice transplanted with bone marrow cells transduced with a MSCV-HOXB4-ires-YFP

264 In mouse bone marrow cells transduced with MLL-AF9, we show that

265 ation of one such peptidomimetic, MM-102, in bone marrow cells transduced with MLL1-AF9 fusion constr

266 fted lethally irradiated recipient mice with bone marrow cells transduced with retroviruses expressin

267 ronchoalveolar lavage fluid cells, inhibited bone marrow cell transendothelial migration, and inhibit

268 tor knockout mouse model in combination with bone marrow cell transplantation, MRI, and neurocognitiv

269 Phenotypic correction of blood and bone marrow cells was shown by the acquired resistance o

270 asophil differentiation from SHP-1 deficient bone marrow cells was significantly reduced.

271 After 24 hours, whole bone marrow cells were analyzed in vitro: 1) colony-form

272 Approximately 85% of bone marrow cells were clonal in the myelodysplastic-syn

273 Splenocytes and bone marrow cells were exposed to nicotine ex vivo, and

274 Bone marrow cells were isolated from wild-type (WT) or i

275 Approximately 0.3% of bone marrow cells were LepR(+), 10% of which were CFU-Fs

276 and we found that factors released by young bone marrow cells were more supportive of myotube differ

277 nd wild-type mice reconstituted with CCR5-/- bone marrow cells were protected against PH, suggesting

278 Mice lacking S1P3 on bone marrow cells were protected from IRI, and S1P3-defi

279 CD133-sorted HPCs and CD133-depleted bone marrow cells were purified from bone marrow specime

280 Subsequently, modified bone marrow cells were transferred into irradiated mice,

281 A total of 15 x 10(6) BALB/c bone marrow cells were transplanted after varying doses

282 Bone marrow cells were transplanted from Il1rl2(-/-) to

283 In the present study, bone marrow cells were transplanted into P20 mice that e

284 In this study, wild-type (WT) bone marrow cells were transplanted into two lethally ir

285 MC3T3-E1 osteoblastic cells and bone marrow cells were used to verify if mechanical forc

286 We observe a more complex picture in primary bone marrow cells, where EVI1 suppresses Cebpa in stem c

287 n is required for Bv8 up-regulation in mouse bone marrow cells, whereas other Stat family members and

288 vivo assay, designed for peripheral blood or bone marrow cells, which can produce a clinical result w

289 marrow-derived mast cells (BMMCs) and mouse bone marrow cells, which contain hematopoietic progenito

290 id cell lineage displayed a dysregulation of bone marrow cells with a rapid decline in population at

291 at the transduction of CREB transgenic mouse bone marrow cells with a Sox4 retrovirus increases survi

292 ular similarities between megakaryocytes and bone marrow cells with an HSC cell-surface phenotype rem

293 We found that coculture of murine bone marrow cells with bladder tumor cells promoted stro

294 the frequency of MDSCs generated from mouse bone marrow cells with GM-CSF and IL-6 in vitro.

295 In vitro culture of mouse bone marrow cells with GM-CSF is a well-established meth

296 Finally, in vitro treatment of bone marrow cells with IL-33, but not IL-5, led to speci

297 recent observation of fusion of circulating bone marrow cells with, in particular, cerebellar Purkin

298 ematopoietic chimerism from 50 million total bone marrow cells without conditioning.

299 signature is robust and may be applicable to bone marrow cells without the need to isolate CD34+ cell

300 We characterized fluorescent bone marrow cells (YFP(+) BMCs) in the thy1-YFP mouse an