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1 pthatic pneumonia syndrome (IPS) following a bone marrow transplant.
2 41 years in patients who had not received a bone marrow transplant.
3 but were normalized in mice that received a bone marrow transplant.
4 s to improve outcomes in patients undergoing bone marrow transplant.
5 the individuals were successfully treated by bone marrow transplant.
6 in the peripheral blood of NHP recipients of bone marrow transplant.
7 mphoid organs of recipients of an allogeneic bone marrow transplant.
8 al treatments, such as radiation therapy and bone marrow transplant.
9 perimental parent C57BL/6-->CB6F1 allogeneic bone marrow transplant.
10 and Functional Assessment of Cancer Therapy-Bone Marrow Transplant.
11 obilization in preparation for an autologous bone marrow transplant.
12 ng repopulation of the immune system without bone marrow transplant.
13 (CML) after treatment with IFN or allogeneic bone marrow transplant.
14 injury after myeloablative chemotherapy and bone marrow transplant.
15 s hematopoietic recovery in mice receiving a bone marrow transplant.
16 mprove the homing of HSC after an autologous bone marrow transplant.
17 rotein, used as an immunostimulant following bone-marrow transplant.
18 TBI and were able to prevent engraftment of bone marrow transplants.
19 d is predicated on >40 years of success with bone marrow transplants.
20 mismatched) or female (sex-matched, control) bone marrow transplants.
21 aematopoietic cells following congenic mouse bone marrow transplants.
22 patients with hematological malignancies and bone marrow transplants.
23 rmed in Bambi(+/+) mice receiving Bambi(-/-) bone marrow transplants.
24 e cortex were found in HD mice that received bone marrow transplants.
25 t clinical data from 21 patients following a bone marrow transplant, 13 of which progress to idiopath
27 do not develop efficiently in the thymus of bone marrow transplanted adult tgepsilon26 mice, which h
28 lovirus (CMV) infection following allogeneic bone marrow transplant (allo-BMT) is controlled by donor
30 ibute to activation of APCs after allogeneic bone marrow transplant (alloBMT), we examined whether th
31 IV susceptibility in circulating T cells via bone marrow transplants allowed some individuals with HI
32 o syngeneic wild-type mice via Tgfbr2(Myeko) bone marrow transplant and can be rescued in Tgfbr2(Myek
33 rts of 121 pediatric patients who received a bone marrow transplant and subsequently required mechani
34 nfarct fibroblasts using lineage tracing and bone marrow transplants and a robust marker for cardiac
35 ss has led to effective therapies, including bone marrow transplants and gene therapy, that would hav
36 m the lungs and by rejection of incompatible bone marrow transplants and in vitro by cytolysis of YAC
38 y observed in recipients of solid organs and bone marrow transplants and is associated with increased
39 hat CHQ may impair rejection of incompatible bone marrow transplants and other functions mediated by
41 pe bone marrow-transplanted mice, ROCK2(+/-) bone marrow-transplanted and ROCK2(-/-) bone marrow-tran
42 marrow and its differentiated progeny after bone marrow transplant, and several mouse hematopoietic
43 patients (10.3%) proceeded to an allogeneic bone marrow transplant, and the overall response rate wa
44 fection, leukemia, lymphoma, solid organ and bone marrow transplants, and inherited immune deficienci
46 application of hematopoietic stem cells for bone-marrow transplant, and further elucidation of homin
47 y detected by histology in tumors taken from bone marrow transplanted animals, they were spatially is
49 acute phase response proteins at the time of bone marrow transplant are highly likely to develop IPS
52 b; 1 died 6 months after a matched unrelated bone marrow transplant as a result of undefined encephal
57 toGvHD occurs spontaneously after autologous bone marrow transplant (autoBMT) in the absence of CsA i
58 ociated multifactorial disease of allogeneic bone marrow transplant (BMT) -induced graft-vs.-host dis
59 logenous leukemia (AML) and those undergoing bone marrow transplant (BMT) are at greatest risk for co
60 locus were nonrandomly assigned to receive a bone marrow transplant (BMT) by using oral busulfan (16
62 ntinued over several months before and after bone marrow transplant (BMT) from his RSV-immune father.
63 ion-negative patients received an allogeneic bone marrow transplant (BMT) in first complete remission
65 fatal myeloproliferative disease in a murine bone marrow transplant (BMT) model, whereas T/T(F) cause
70 all the hematopoietic tissues of 16 primary bone marrow transplant (BMT) recipient mice and 14 secon
71 L challenge in both syngeneic and allogeneic bone marrow transplant (BMT) recipients at both early an
72 driven responses, was that rearrangements in bone marrow transplant (BMT) recipients exhibited much l
75 graft tolerance in mice receiving allogeneic bone marrow transplant (BMT) with minimal conditioning.
79 of systemic lupus erythematosus, were given bone marrow transplants (BMT) at 20 wk of age using MHC-
82 trated that SHIP(-/-) mice accept allogeneic bone marrow transplants (BMT) without significant acute
86 ukemia (CML) underwent unrelated donor (URD) bone marrow transplants (BMTs) facilitated by the Nation
87 consolidation chemotherapy and an autologous bone marrow transplant, but eventually died 22 months af
88 KGF-/- recipients of syngeneic or allogeneic bone marrow transplant, but using KGF-/- mice as a donor
89 ells (HSCs) are the therapeutic component of bone marrow transplants, but finding immune-compatible d
91 tment of tumor-bearing Tg/NCD mice or Tg/NCD bone marrow transplanted C3H mice (Tg/NCD-C3H) resulted
92 cts that can be corrected with an allogeneic bone marrow transplant can theoretically also be treated
93 ther specific subsets of cells in allogeneic bone marrow transplants can effectively treat the BCL(1)
94 xty-three percent of 24 NCs collected from a bone marrow transplant center contained biofilms compris
95 signaling in PcP-related lung injury, murine bone marrow transplant chimeras of wild-type, C57BL6/J,
100 the adult brain 1 to 6 months after an adult bone marrow transplant demonstrates a remarkable plastic
101 d that myeloablative irradiation followed by bone marrow transplant-derived microglia engraftment, ra
102 Ldlr(-/-) mice with a Nur77(-/-)-deficient bone marrow transplant developed 2.1-fold larger atheros
103 ients who responded to therapy who underwent bone marrow transplant did not show any benefit from thi
105 suggested, and indicate that improvement of bone marrow transplant efficiency may be possible in the
106 diated in vivo LTbetaR stimulation following bone marrow transplant enhances initial thymus recovery
116 not affect leukocyte functions in vitro, and bone marrow transplant experiments suggest that host Akt
120 s in Functional Assessment of Cancer Therapy-Bone Marrow Transplant (FACT-BMT) score relative to base
121 the Functional Assessment of Cancer Therapy-Bone Marrow Transplant (FACT-BMT) were received from 109
122 in individuals who had previously received a bone marrow transplant for hematological disorders.
124 nza B infection in a child who had undergone bone marrow transplant for X-linked severe combined immu
126 osis and (2) a female patient who received a bone marrow transplant from a male donor and subsequentl
130 d in nontransgenic SCID mice that received a bone marrow transplant from GFP-expressing SCID mice.
131 Rats status post 5/6 nephrectomy underwent bone marrow transplant from human placental alkaline pho
136 inflammation and liver damage in mice given bone marrow transplants from Cyp27a1(-/-) mice and place
138 ion was inhibited, irradiated mice receiving bone marrow transplants from heterozygous GFP+ mice reve
141 ported by experiments in which recipients of bone marrow transplants from SR-uPA+/0 donors but not no
142 onto Fgfbp1 GFP-knock-in reporter hosts and bone marrow transplants from the GFP-reporter model into
144 ite numerous reports of GBS in recipients of bone marrow transplants, GBS has rarely been reported in
145 ." Ablation of the immune system followed by bone marrow transplant has been shown to cure experiment
146 re, predictors for 5-year mortality included bone marrow transplant (hazard ratio, 3.66; 95% CI, 2.26
147 reover, generating chimeric mice via UPRT(+) bone marrow transplants identifies immune versus niche s
150 have been utilized extensively in allogeneic bone marrow transplants in order to purge the allograft
151 ould potentially overcome the limitations of bone marrow transplants, including graft rejection and t
152 years after the first successful allogeneic bone marrow transplants, infection remains the most comm
156 experiments involving H. felis infection of bone marrow transplanted irradiated mice have suggested
157 suggest that one mechanism for relapse after bone marrow transplant is acquired tolerance of allogene
158 opoietic stem cell transplantation (HSCT, or bone marrow transplant), is common even after transplant
163 show that in both the steady-state and after bone marrow transplant, lymphotoxin beta receptor (LTbet
165 flammation in patients undergoing autologous bone marrow transplant may reduce the subsequent develop
166 res: Functional Assessment of Cancer Therapy-Bone Marrow Transplant, Mental Health Inventory, occupat
168 cent studies have shown that in radiated and bone marrow transplanted mice, bone marrow-derived cells
169 of a polymeric IL-8 delivery system into GFP bone marrow-transplanted mice revealed that localized IL
170 +/-) bone marrow-transplanted and ROCK2(-/-) bone marrow-transplanted mice showed substantially less
173 -expressing Tregs over time in an allogeneic bone marrow transplant model and demonstrated colocaliza
174 major histocompatibility complex-mismatched bone marrow transplant model in which graft-versus-host
179 uce a myeloproliferative disease in a murine bone marrow transplant model, but are not sufficient to
187 gnificant prolongation of survival in murine bone marrow transplant models of FGFR3 TDII-induced pre-
188 hough cytokine levels are elevated in murine bone marrow transplant models of leukemia using tyrosine
192 ) or respiratory (odds ratio, 1.56) reasons, bone marrow transplant (odds ratio, 1.53), previous ICU
194 or alpha(V)beta(3), we performed reciprocal bone marrow transplants on wild-type and beta3(-/-) mice
195 replacement therapy, or more effectively by bone marrow transplant or HSC gene therapy (HSC-GT).
196 from five females who had received either a bone-marrow transplant or an allogeneic mobilised periph
197 status (P =.04), and early disease status at bone marrow transplant (P =.05) were associated with sup
198 CMV and human herpesvirus type 6 (HHV-6) in bone marrow transplant patients (causing marrow suppress
199 that it is possible to estimate dd-cfDNA in bone marrow transplant patients that are unrelated or th
201 report GBS in organ transplant patients and bone marrow transplant patients, both of whom have iatro
202 ective in certain high-risk patients such as bone marrow transplant patients, few studies have focuse
209 provide a functional cure, as observed in a bone marrow transplant performed with hematopoietic stem
216 emia after months of apparent cure in recent bone marrow transplant recipients and an immediately-tre
217 in immunocompromised individuals, including bone marrow transplant recipients and cancer and AIDS pa
219 TLs for adoptive immunotherapy of HLA-A*0201 bone marrow transplant recipients from 200 mL donor bloo
220 pecific DNA in the peripheral blood of islet/bone marrow transplant recipients of same sex cynomolgus
221 phomas is seen in solid organ transplant and bone marrow transplant recipients receiving immunosuppre
222 number of features that distinguish them as bone marrow transplant recipients that must be understoo
224 prognosis generally is poor among pediatric bone marrow transplant recipients who subsequently requi
225 kinase (SR-uPA(+/0) mice) and of SR-uPA(+/0) bone marrow transplant recipients, and we used bioinform
227 ncluding patients with AIDS, solid organ and bone marrow transplant recipients, patients with leukemi
228 mmunodeficiency virus-positive patients, and bone marrow transplant recipients, were tested for cytom
239 9 to 1998 and were reported to International Bone Marrow Transplant Registry (IBMTR) or Autologous Bl
240 white patients reported in the International Bone Marrow Transplant Registry (IBMTR), 30% of 341 MDS
242 chronic phase provided by the International Bone Marrow Transplant Registry and the National Marrow
243 1968 to 1996, reported to the International Bone Marrow Transplant Registry and/or National Marrow D
244 r results were reported to the International Bone Marrow Transplant Registry by 144 participating ins
246 ed with those from 509 similar International Bone Marrow Transplant Registry patients who underwent t
252 62) were recruited through the International Bone Marrow Transplant Registry/Autologous Blood and Mar
253 mune and neurodegenerative states, organ and bone marrow transplant rejection, and tumor response to
254 a data set from the Center for International Bone Marrow Transplant Research (CIBMTR) registry databa
256 develop myeloid and lymphoid cells received bone marrow transplants resulting in donor-derived micro
258 the Functional Assessment of Cancer Therapy-Bone Marrow Transplant Scale (FACT-BMT) questionnaire, w
259 observations, we found that both genetic and bone marrow-transplanted SCD mice had greater mortality
262 ransplantation conditioning regimen prior to bone marrow transplant significantly increased the risk
266 n unexpected finding was noted in allogeneic bone marrow transplant studies using IL-7 receptor null
268 vestigations of animals and short-term human bone marrow transplants suggest that bone marrow can rep
270 ased bone mass could partially be rescued by bone marrow transplants supporting our hypothesis that r
271 survivors and siblings participating in the Bone Marrow Transplant Survivor Study (BMTSS) completed
275 In addition, chimeric mice were generated by bone marrow transplant to limit expression of IL7Ralpha
277 Here, we compare CD13 wild-type and null bone marrow-transplanted tumor-bearing mice to show that
280 semiallogeneic T cells, long-term after the bone marrow transplant using G-Neutrophils, were confirm
281 and cancer cells in leukemia patients after bone marrow transplants, using a system of six delay dif
282 METHODS AND Using genetic lineage tracing or bone marrow transplant, we found no evidence for collage
283 virus cases occurring in children undergoing bone marrow transplant, we undertook norovirus genome se
285 tients aged 2-70 years receiving their first bone marrow transplant were eligible for inclusion in th
287 Patients with hematologic malignancy or bone marrow transplant were more likely to develop a com
288 A total of 960 recipients of solid organ or bone marrow transplants were identified from Veterans He
292 traperitoneally thioglycollate in Abcg1(-/-) bone marrow-transplanted, Western diet-fed, Ldlr-deficie
294 from stem cells residing in the bone marrow, bone marrow transplanted wild-type mice were treated wit
295 d human leukocyte antigen-matched kidney and bone marrow transplant with lead follow-up time of more
297 is during zymosan-induced peritonitis, using bone marrow transplants with transgenic mice deficient i
299 low-density lipoprotein receptor (LDLR) were bone-marrow transplanted with NLRP3-deficient, ASC (also
300 ontaminating cancerous cells from autologous bone marrow transplants without perturbing the engraftme