ライフサイエンスコーパス: bone marrow-derived macrophage

1 liensis infection of BALB/c (wild-type [WT]) bone marrow derived macrophages.

2 wever expression is increased over 2-fold in bone marrow derived macrophages.

3 Typhimurium, in a comparable manner to mouse bone marrow derived macrophages.

4 vo cytokine secretion by peritoneal cells or bone marrow derived macrophages.

5 inflammasome is attenuated in Nrf2-deficient bone marrow-derived macrophages.

6 teraction was enhanced by LPS stimulation in bone marrow-derived macrophages.

7 poorly and induced rapid cell death in mouse bone marrow-derived macrophages.

8 phage cell line RAW264.7, but not in primary bone marrow-derived macrophages.

9 abolished in Toll-like receptor (TLR2)(-/-) bone marrow-derived macrophages.

10 uced mRNA and protein expression of IL-18 in bone marrow-derived macrophages.

11 ta and interleukin 6 messenger RNAs in mouse bone marrow-derived macrophages.

12 potently inhibited Leishmania replication in bone marrow-derived macrophages.

13 uced IL-1beta secretion and NO production in bone marrow-derived macrophages.

14 phage phenotype was absent in peritoneal and bone marrow-derived macrophages.

15 RIM21 upon S Typhimurium infection of murine bone marrow-derived macrophages.

16 re supported by experiments with Ifitm3(-/-) bone marrow-derived macrophages.

17 optotic cells and are better phagocytes than bone marrow-derived macrophages.

18 man monocyte-derived macrophages, and murine bone marrow-derived macrophages.

19 duced chemokine secretion by neutrophils and bone marrow-derived macrophages.

20 s, but GM-CSF has also been used to generate bone marrow-derived macrophages.

21 39 significantly regulated 105 probe sets in bone marrow-derived macrophages.

22 cytokines, which is attenuated in Mrp14(-/-) bone marrow-derived macrophages.

23 ec kinases and LPS in primary peritoneal and bone marrow-derived macrophages.

24 10, and GM-CSF, was also markedly reduced in bone marrow-derived macrophages.

25 viral infection on inflammasome responses in bone marrow-derived macrophages.

26 in murine RAW 264.7 macrophages and primary bone marrow-derived macrophages.

27 rease of RANKL-induced osteoclastogenesis in bone marrow-derived macrophages.

28 n monocytes, murine RAW 264.7, and wild-type bone marrow-derived macrophages.

29 emphasis on proteolytic efficiency in murine bone marrow-derived macrophages.

30 ace of lipopolysaccharide-stimulated primary bone marrow-derived macrophages.

31 ulus for interleukin (IL)-1beta secretion by bone marrow-derived macrophages.

32 ed in MHV-infected Ifit2(-/-) relative to wt bone marrow-derived macrophages.

33 also activating ERK MAPK pathways in murine bone marrow-derived macrophages.

34 D88-NF-kappaB-dependent mode of induction in bone marrow-derived macrophages.

35 163 and functionally distinct from classical bone marrow-derived macrophages.

36 ory cytokines nor inflammasome activation in bone marrow-derived macrophages.

37 to embed Raw264.7 cell line and primary rat bone marrow-derived macrophages.

38 1 phosphorylation and cytokine production in bone marrow-derived macrophages.

39 odels, and in vitro on endothelial cells and bone marrow-derived macrophages.

40 response could be recapitulated in vitro in bone marrow-derived macrophages.

41 C class II expression in ZIP macrophages and bone marrow-derived macrophages.

42 in osteoclast precursors (OCPs) derived from bone marrow-derived macrophages.

43 versus hypoxia serum starvation-VEGFR1(+/-) bone marrow-derived macrophages.

44 uli in human monocytic cell lines and murine bone marrow-derived macrophages.

45 ome and Ingenuity Pathway Analysis of murine bone marrow-derived macrophages after exposure to this v

46 nscriptome analysis of mouse lymph nodes and bone marrow-derived macrophages after incubation with ac

47 have used a novel platform that integrates a bone marrow-derived macrophage and live H. pylori co-cul

48 Using bone marrow-derived macrophages and a murine model of pe

49 as LVS clpB grew similarly to LVS in primary bone marrow-derived macrophages and a variety of cell li

50 MV treatment of bone marrow-derived macrophages and bone marrow progenit

51 strongly impairs many effector functions of bone marrow-derived macrophages and bone marrow-derived

52 Whereas TLR9-induced TNF-alpha secretion of bone marrow-derived macrophages and conventional dendrit

53 also observed by in vitro experiments, using bone marrow-derived macrophages and dendritic cells as r

54 TARM1 expression was also upregulated by bone marrow-derived macrophages and dendritic cells foll

55 Bone marrow-derived macrophages and dendritic cells, lam

56 t cytokine that prompts the proliferation of bone marrow-derived macrophages and granulocytes.

57 rden was dramatically reduced in both murine bone marrow-derived macrophages and hamsters, in associa

58 A-induced macrophage cell death with primary bone marrow-derived macrophages and high-fat diet-induce

59 Murine bone marrow-derived macrophages and human monocyte-deriv

60 wild-type and T2S mutant bacteria in murine bone marrow-derived macrophages and human U937 cells.

61 cluded IL-12, TNF-alpha, and IL-6 in primary bone marrow-derived macrophages and LPS-induced IL-12/18

62 s isolated from Anxa2-deficient (Anxa2(-/-)) bone marrow-derived macrophages and lung parenchyma disp

63 In vitro, bone marrow-derived macrophages and lymphoid endothelial

64 In murine bone marrow-derived macrophages and murine embryonic fib

65 Using STAT3-deficient bone marrow-derived macrophages and pharmacologic inhibi

66 transendothelial migration (TEM) activity of bone marrow-derived macrophages and Raw264.7 cells accor

67 In vitro, both the WT bone marrow-derived macrophages and renal mesangial cell

68 TEM activity was decreased in Ninjurin1 KO bone marrow-derived macrophages and siNinj1 Raw264.7 cel

69 l changes were shown to be similar in murine bone marrow-derived macrophages and TAMs isolated from m

70 ted peritoneal macrophages) as compared with bone marrow-derived macrophages and the RAW264 cell line

71 Using Tmem203 knockout bone marrow-derived macrophages and transient knockdown

72 function of anti-inflammatory intestinal and bone-marrow-derived macrophages and their ability to sec

73 enterica serovar Typhimurium-infected murine bone-marrow-derived macrophages and thrombin activated h

74 e protocol that details several in vitro (in bone marrow-derived macrophages) and in vivo (in mice) s

75 tates its survival and replication in murine bone marrow-derived macrophages, and E. tarda infection

76 ity in vivo by tracking fluorescence-labeled bone marrow-derived macrophages, and found that tPA-defi

77 ly active, rescue replication of MHV(Mut) in bone marrow-derived macrophages, and inhibit RNase L-med

78 more, after phagocytosis of myelin in vitro, bone marrow-derived macrophages are much more susceptibl

79 ymosan-induced peritonitis, M1- and M2a-like bone marrow derived macrophages, as well as by mesotheli

80 ating factor)-induced activation of Rac1, in bone marrow-derived macrophages; (b) TRPV4 directly inte

81 pro-inflammatory cytokines were increased in bone marrow derived macrophage (BMDM) from PLTP-/-, whil

82 ignificantly higher IL-6 secretion by murine bone marrow derived macrophages (BMDM) compared to cultu

83 Further investigation revealed that bone marrow derived macrophages (BMDM) from the Muc-1(KO

84 d that treatment of RANKL-stimulated primary bone marrow-derived macrophage (BMDM) cultures with smal

85 rom 129 mice is sufficient to enhance the B6 bone marrow-derived macrophage (BMDM) inflammasome respo

86 ulture of atrophied myotubes with or without bone marrow-derived macrophages (BMDM) and/or M-CSF reve

87 lations of wild-type mice with Nrp1-depleted bone marrow-derived macrophages (BMDM) confers resistanc

88 LRP1-deficient bone marrow-derived macrophages (BMDM) expressed higher

89 Here we show that bone marrow-derived macrophages (BMDM) from high fat-fed

90 Using bone marrow-derived macrophages (BMDM) from wild-type an

91 this study, we confirm that tolerized mouse bone marrow-derived macrophages (BMDM) selectively incre

92 of the eicosanoid metabolic network in mouse bone marrow-derived macrophages (BMDM) upon stimulation

93 Primary bone marrow-derived macrophages (BMDM) was isolated from

94 e treated rat neonatal cardiomyocytes, mouse bone marrow-derived macrophages (BMDM), or mouse neutrop

95 n vivo and by isolated resident alveolar and bone marrow-derived macrophages (BMDM).

96 d rectifier potassium channels (Kir) in mice bone marrow-derived macrophages (BMDM).

97 uction in response to LPS treatment in mouse bone marrow-derived macrophages (BMDM).

98 eficient RAW cells and primary PHD2 knockout bone marrow-derived macrophages (BMDM).

99 In this study, we found that murine bone marrow-derived macrophages (BMDMs) and a human leuk

100 In addition, infection of bone marrow-derived macrophages (BMDMs) and mice (C57BL/

101 Bone marrow-derived macrophages (BMDMs) are recruited to

102 3 or Pyrin inflammasome activation in murine bone marrow-derived macrophages (BMDMs) as an indicator

103 Using bone marrow-derived macrophages (BMDMs) derived from wil

104 In vitro, LPS-treated LysM PTP1B bone marrow-derived macrophages (BMDMs) displayed increa

105 onse to LPS challenge, MEK2-deficient murine bone marrow-derived macrophages (BMDMs) exhibited lower

106 d NLRP3 was revealed when Nlrc4(S533A/S533A) bone marrow-derived macrophages (BMDMs) expressing phosp

107 ate that LPS/IFNgamma polarization decreased bone marrow-derived macrophages (BMDMs) formation of pro

108 We found that bone marrow-derived macrophages (BMDMs) from BTK-deficie

109 Infected Chil1-deficient mice and bone marrow-derived macrophages (BMDMs) from Chil1-defic

110 Bone marrow-derived macrophages (BMDMs) from I/LnJ mice

111 aB pathway (IKKalpha/beta, NF-kappaB p65) in bone marrow-derived macrophages (BMDMs) from knockout mi

112 n of IFN regulatory factor 3 (IRF3), whereas bone marrow-derived macrophages (BMDMs) from mice carryi

113 Mirn23a (-/-) bone marrow-derived macrophages (BMDMs) have an attenuat

114 otes NLRP3 inflammasome activation in murine bone marrow-derived macrophages (BMDMs) infected with Gr

115 Elavl1Mo KO), we show that HuR expression in bone marrow-derived macrophages (BMDMs) is needed to mai

116 Both lung macrophages and bone marrow-derived macrophages (BMDMs) isolated from p4

117 Bone marrow-derived macrophages (BMDMs) isolated from RO

118 We report that Galpha(i2) in murine bone marrow-derived macrophages (BMDMs) regulates IL-1be

119 Mechanistic studies using bone marrow-derived macrophages (BMDMs) showed that LPS

120 Interestingly, IL-4 treatment of bone marrow-derived macrophages (BMDMs) significantly do

121 Following exposure of murine bone marrow-derived macrophages (BMDMs) to inactivated F

122 and adoptive transfer studies revealed that bone marrow-derived macrophages (BMDMs) traffic to the l

123 S induced secretion of IL-6 and TNF-alpha by bone marrow-derived macrophages (BMDMs) was significantl

124 esser extent at Il1a) reaches high levels in bone marrow-derived macrophages (BMDMs), and the enhance

125 In LPS/ATP-stimulated bone marrow-derived macrophages (BMDMs), CLIC1 or CLIC4

126 In bone marrow-derived macrophages (BMDMs), the IRAK2-TRAF6

127 In bone marrow-derived macrophages (BMDMs), we also observe

128 fy H. capsulatum genes required for lysis of bone marrow-derived macrophages (BMDMs), we identified a

129 nstituted mouse bone marrow neutrophils, and bone marrow-derived macrophages (BMDMs), we showed that

130 or NF-kappaB activation in HEK 293 cells and bone marrow-derived macrophages (BMDMs).

131 rous chemokines and cytokines that activated bone marrow-derived macrophages (BMDMs).

132 ived LPS enhanced the level of miR-15a/16 in bone marrow-derived macrophages (BMDMs).

133 of CD40 on the surface of T. gondii-infected bone marrow-derived macrophages (BMdMs).

134 of inflammatory factors in BV2 microglia and bone marrow-derived macrophages (BMDMs).

135 veral inflammasome-activating signals in the bone marrow-derived macrophages (BMDMs).

136 Aim2(-/-), Casp1/11(-/-) and Asc(-/-) murine bone-marrow derived macrophages (BMDMs) were infected wi

137 7BL/6, and CD1 mice models and C57BL/6 mouse bone-marrow-derived macrophages (BMDMs) were used as inf

138 uction in lipopolysaccharide (LPS)-activated bone marrow derived-macrophages (BMM) was detected in 6-

139 Using bone marrow-derived macrophages (BMM s) and CD4(+) T cel

140 In vitro priming of bone marrow-derived macrophages (BMM s) with IL-4 or TSG

141 nsistent with the in vivo data, infection of bone marrow-derived macrophages (BMM) with lethal Ehrlic

142 RNase L activation and replicates poorly in bone marrow-derived macrophages (BMM), while ns2(H126R)

143 ates HuR expression in cardiac- and cultured bone marrow-derived macrophages (BMMO) and stimulates Hu

144 CD200R-deficient bone marrow derived macrophages (BMMPhi) were used to de

145 ining 3 (NLRP3) inflammasome in primed mouse bone marrow-derived macrophages (BMMPhi), inducing a rob

146 the effect of Fh12 on the function of mouse bone marrow-derived macrophages (bmMPhis).

147 irus replication was attenuated in wild-type bone marrow-derived macrophages (BMMs) and partially res

148 in LPS (1 ng/ml)-stimulated wild-type murine bone marrow-derived macrophages (BMMs) but failed to do

149 response to L. monocytogenes, P2X5-deficient bone marrow-derived macrophages (BMMs) exhibit defective

150 ogenesis was greatly enhanced in cultures of bone marrow-derived macrophages (BMMs) from Notch2(tm1.1

151 lysis, the bacterial modulation of miRNAs in bone marrow-derived macrophages (BMMs) in which activity

152 racellular concentrations of R. australis in bone marrow-derived macrophages (BMMs) of TLR4(-/-) and

153 r ligation were severely reduced in Hck(-/-) bone marrow-derived macrophages (BMMs) or in RAW/LR5 mac

154 entration of R. australis in Atg5(flox/flox) bone marrow-derived macrophages (BMMs) than in Atg5(flox

155 Subsequently, rat bone marrow-derived macrophages (BMMs) were cultured in

156 Stimulation of bone marrow-derived macrophages (BMMs) with endotoxin re

157 ligand (RANKL)-induced osteoclastogenesis in bone marrow-derived macrophages (BMMs).

158 in the RAW 264.7 macrophage line and primary bone marrow-derived macrophages but did not affect LXR-d

159 n rates and mROS expression in mock-infected bone marrow-derived macrophages but reduced caspase-depe

160 deficiency adversely affected the ability of bone marrow-derived macrophages, but not dendritic cells

161 L-1beta secretion was impaired in Pgam5(-/-) bone marrow-derived macrophages, but not in Ripk3(-/-) b

162 RP3-dependent interleukin-1beta secretion by bone marrow-derived macrophages by activating nuclear fa

163 id compartments, causes long-term changes in bone marrow-derived macrophages by suppressing interleuk

164 significantly higher by wild-type (C57BL/6) bone marrow-derived macrophages compared with TLR2-defic

165 Our in vitro data suggest that bone marrow-derived macrophage-conditioned media induces

166 Bone marrow-derived, macrophage-CSF-induced cells with a

167 hemozoin was purified and added to in vitro bone marrow-derived macrophage cultures concurrently exp

168 Bone marrow-derived macrophage cultures from the Notch2(

169 ent livers enhanced macrophage activation in bone marrow-derived macrophage cultures.

170 kout of ASC in lipopolysaccharide-stimulated bone marrow-derived macrophages depressed HMGB1 activity

171 d J20/A7(-/-) mice, whereas the capacity for bone marrow-derived macrophages derived from A7(-/-) mic

172 ed the regulation of AC7 with that of AC2 in bone marrow-derived macrophages devoid of AC7.

173 Bone marrow-derived macrophages did not release NE in re

174 PTEN in lipopolysaccharide-stimulated mouse bone marrow-derived macrophages enhanced beta-catenin ac

175 As in RAW264.7 cells, primary bone marrow-derived macrophages exposed to a sublethal d

176 In this study, bone marrow-derived macrophages exposed to excretory/sec

177 man monocyte-derived macrophages, and murine bone marrow-derived macrophages following infection with

178 Bone marrow derived-macrophages from Fgf2(LMW-/-) mice c

179 Similar to human cells, bone marrow-derived macrophages from A20-deficient mice

180 In tissue culture models, MaR1 treatment of bone marrow-derived macrophages from aged mice protected

181 a lower parasite intake, parasite burden in bone marrow-derived macrophages from AnxA1(-/-) mice was

182 Bone marrow-derived macrophages from Bbaa1 congenic mice

183 per 1 cells or T-helper 2 cells, we obtained bone marrow-derived macrophages from both strains and in

184 as chemotaxis in response to LPS and C5a in bone marrow-derived macrophages from BVR (fl/fl) and Lys

185 rom Cyp46a1(-/-) mice as well as retinal and bone marrow-derived macrophages from Cyp27a1(-/-) and Cy

186 parison, gene expression was investigated in bone marrow-derived macrophages from Cyp46a1(-/-) mice a

187 In bone marrow-derived macrophages from different knockout

188 duction was recapitulated ex vivo in primary bone marrow-derived macrophages from Dusp1-deficient mic

189 LPS-challenged peritoneal and bone marrow-derived macrophages from IkappaBzeta-deficie

190 ry cytokines and pro-inflammatory enzymes in bone marrow-derived macrophages from LKB1 KO than those

191 This response was lost in bone marrow-derived macrophages from mice deficient in A

192 d M2 macrophage activation were confirmed in bone marrow-derived macrophages from mice with the myelo

193 ng IL-31RA expression on both peritoneal and bone marrow-derived macrophages from mice.

194 Delivery of bone marrow-derived macrophages from miR106b-93-25(-/-)

195 ur findings in cultured macrophages, primary bone marrow-derived macrophages from MPV17(-/-) mice, a

196 The effects of estrogen are long-lasting; bone marrow-derived macrophages from ovariectomized mice

197 Our previous studies showed that bone marrow-derived macrophages from S100A4(-/-) mice ex

198 rd M2 macrophage activation was confirmed in bone marrow-derived macrophages from Slc7a2(-/-) mice.

199 hagic markers were constitutively induced in bone marrow-derived macrophages from Sphk dKO mice.

200 city and inflammatory cytokine production of bone marrow-derived macrophages from TLR2(-/-) mice.

201 Bone marrow-derived macrophages from Tlr4-deficient mice

202 iments were performed in naive and polarized bone marrow-derived macrophages from wild-type (WT) and

203 Experiments with LPS-activated bone marrow-derived macrophages from wild-type and GILZ

204 the expression and significance of GLUT6 in bone marrow-derived macrophages from wild-type and GLUT6

205 but complementary approaches: 1) we compared bone marrow-derived macrophages from wild-type and TIM4(

206 Using bone marrow-derived macrophages from WT and RIP2-KO mice

207 elin phagocytosis in vitro by LPS stimulated bone-marrow-derived macrophages from IL-10-null mice fai

208 Bone-marrow-derived macrophages from mice deficient for

209 Neutrophils, but not bone marrow-derived macrophages, from cmo mice secreted

210 n of caspase-1 inhibitors or the infusion of bone marrow-derived macrophages genetically engineered t

211 we report that IFN-beta signaling in murine bone marrow-derived macrophages has a cell-intrinsic pro

212 In mouse bone marrow-derived macrophages, heme induced HO-1, lipi

213 In a range of cell lines (murine bone marrow-derived macrophages, human monocytic THP-1 c

214 appaB reporter system in immortalized murine bone marrow-derived macrophages (iBMDM).

215 uce mTOR signalling in the microglia but not bone marrow-derived macrophages in both in vitro and in

216 as used to examine gene regulation in murine bone marrow-derived macrophages in response to 90-mum-di

217 by nephritic glomeruli ex vivo and cultured bone marrow-derived macrophages in vitro, suggesting add

218 arly, FENDRR overexpression in primary mouse bone marrow-derived macrophages increased mRNA expressio

219 osome damage was observed in infected murine bone marrow-derived macrophages, increased with time, an

220 Bone marrow-derived macrophages incubated with or withou

221 MDP treatment of bone marrow-derived macrophages incubated with P. gingiv

222 LLO was able to infect and replicate within bone marrow-derived macrophages indistinguishably from t

223 Coculture of PyMT cells with bone marrow-derived macrophages induced a TAM-like macro

224 d AIM2 inflammasome activation and following bone marrow-derived macrophage infection with herpes sim

225 In primary cultures of mouse bone marrow-derived macrophages, inhibition of miR-29a,

226 During brain injury or infection, bone-marrow derived macrophages invade neural tissue, ma

227 ased cathepsin activity determined in StB KO bone marrow-derived macrophages is not essential for inf

228 The function of IL-37 in transfected bone marrow-derived macrophages is nucleotide-binding ol

229 Bone marrow derived macrophages isolated from WT or Notc

230 e very low levels of endogenous Cav-1 and in bone marrow-derived macrophages isolated from Cav-1(-/-)

231 s are confirmed by comparing Kir currents in bone marrow-derived macrophages isolated from Cav-1(-/-)

232 d pro-inflammatory enzymes were monitored in bone marrow-derived macrophages isolated from myeloid ce

233 lso confirmed enhanced osteoclastogenesis by bone marrow-derived macrophages isolated from the Kdm3C

234 le, if any, iRhom2 was detectable in mEFs or bone marrow-derived macrophages lacking ADAM17, suggesti

235 Bone marrow-derived macrophages lacking GPSM3 expression

236 uced by dsDNA and other microbial ligands in bone marrow-derived macrophages lacking p205 revealed th

237 rtin messenger RNA and protein expression in bone marrow-derived macrophages, liver, and spleen of mi

238 ne marrow-derived macrophages or VEGFR1(+/-) bone marrow-derived macrophages (M1-like phenotype), we

239 itional measurement of TNF-alpha shedding on bone marrow-derived macrophages, meprin beta/ADAM protea

240 g pathways are delayed in P2-deficient mouse bone marrow-derived macrophages, mouse embryonic fibrobl

241 In SIRT3 knock-out bone marrow-derived macrophages, NLRP3 activation promot

242 expression and cytokine production in mouse bone marrow-derived macrophages of different genotypes:

243 h MEK1/2 inhibitor U0126 or genetically with bone marrow-derived macrophages or DCs from Tpl2(-/-) mi

244 Mfrn1 (-/-)/Mfrn2 (-/-) bone marrow-derived macrophages or skin fibroblasts in v

245 d adoptive transfer of VEGF(165)b-expressing bone marrow-derived macrophages or VEGFR1(+/-) bone marr

246 M2 macrophage markers (Mrc1, Arg1, Il10) in bone-marrow-derived macrophages or in SAT from male or f

247 to measure the mRNA and miRNA expression in bone marrow-derived macrophages over a time-series of 8

248 embryonic precursors and become replaced by bone-marrow-derived macrophages over time.

249 TLR2-induced TNF-alpha production by murine bone marrow-derived macrophages (p < 0.001).

250 We have found that in murine bone marrow-derived macrophages, PGE2 via the cAMP/prote

251 n of MST1 in mouse embryonic fibroblasts and bone marrow-derived macrophages potentiated the TNFalpha

252 An expansion of bone marrow-derived macrophages preceded a second phase,

253 CD44(+/+) murine bone marrow-derived macrophages produced higher TNF-alph

254 Ex vivo, TLR9(-/-) bone marrow-derived macrophages produced more A20 than W

255 In bone marrow-derived macrophages, protective TLR ligands

256 otif) ligands 1, 10, and 12, which stimulate bone marrow-derived macrophage recruitment.

257 ll surface, but diminished TNF production by bone marrow-derived macrophages relative to wild type.

258 ine and paracrine proinflammatory effects on bone marrow-derived macrophages, renal endothelial cells

259 oxide addition to CB3-infected NOD.Ncf1(m1J) bone marrow-derived macrophages rescued the inflammatory

260 Silencing of NM23-M1 in mouse bone marrow-derived macrophages resulted in decreased ph

261 se-1 and -11 activation determined in StB KO bone marrow-derived macrophages resulted in enhanced IL-

262 Mechanistic analysis of superoxide-deficient bone marrow-derived macrophages revealed a marked diminu

263 Ex vivo studies using splenocytes and bone marrow-derived macrophages revealed significantly d

264 Like bone marrow-derived macrophages, RNA editing in MG leads

265 Bone marrow-derived macrophages secreted IL1B and IL18,

266 lity of these cells to condition co-cultured bone marrow-derived macrophages so that the macrophages

267 mokine gene expression was examined in mouse bone marrow-derived macrophages stimulated through vario

268 f IFN-beta expression was also identified in bone marrow-derived macrophages stimulated with B. burgd

269 ions of Rapamycin in LC3B and ATG5 deficient bone marrow derived macrophages, suggesting that non-aut

270 Co-culture with VDR-activated bone marrow-derived macrophages suppresses UPR target ge

271 a production and pyroptosis in primed murine bone marrow-derived macrophages that is mediated by the

272 Correspondingly, mouse bone marrow-derived macrophages that lack either thrombo

273 dulated the functional phenotype ascribed to bone marrow-derived macrophages: the B6 allele promoted

274 Exposure of bone marrow-derived macrophages to eRNA resulted in a co

275 at in vitro, CTLA4Ig synergizes with NO from bone marrow-derived macrophages to inhibit T cell prolif

276 ility group B1, stimulated Kupffer cells and bone marrow-derived macrophages to produce cytokines by

277 Adoptive transfer of FcgammaRIa(-/-) bone marrow-derived macrophages transfected with Fcgamma

278 In contrast, mice that received bone marrow-derived macrophages transfected with Fcgamma

279 Mechanistically, in vitro, bone marrow-derived macrophages treated with necrotic bo

280 licited in an analogous fashion using LPS in bone marrow-derived macrophages upon inhibition of caspa

281 ressed in immune cells and is upregulated in bone marrow-derived macrophages upon stimulation with in

282 r found that TMAO induces M1 polarization of bone marrow-derived macrophages via the nucleotide-bindi

283 Furthermore, the secretome of MaR1-treated bone marrow-derived macrophages was identified as osteoi

284 In additional studies using bone marrow-derived macrophages, we found that Tec and B

285 Using bone marrow-derived macrophages, we observed that alpha-

286 Using bone marrow-derived macrophages, we then observed that c

287 onal impact of GH on macrophage programming, bone marrow derived macrophages were treated with GH or

288 Bone marrow-derived macrophage were polarized to an M2 p

289 Consequently, murine bone marrow-derived macrophages were cocultured with tro

290 Murine bone marrow-derived macrophages were derived into mature

291 mpared with TLR2-deficient or CD14-deficient bone marrow-derived macrophages when stimulated with cur

292 In vitro, treatment of wild-type bone marrow-derived macrophages with advanced glycation

293 can be reproduced in vitro by stimulation of bone marrow-derived macrophages with conditioned media f

294 In the current study, in vitro treatment of bone marrow-derived macrophages with EDPs induced M1 mac

295 Coculture of bone marrow-derived macrophages with ERCs from DKO mouse

296 RNA sequencing (RNASeq) analysis showed that bone marrow-derived macrophages with IRE1alpha deletion

297 The study suggests that treating mouse bone marrow-derived macrophages with KLA and ATP produce

298 Co-culture of bone marrow-derived macrophages with organotypic tumour

299 The interaction of WT bone marrow-derived macrophages with renal microvascular

300 Treatment of mouse bone-marrow-derived macrophages with lipotoxic hepatocyt