ライフサイエンスコーパス: bowel perforation

1 ed toxicity (five thromboembolic events, one bowel perforation).

2 Two patients had a delayed diagnosed bowel perforation.

3 lar access thrombosis (2%), stroke (2%), and bowel perforation (1%).

4 ed deaths (disseminated disease, 4 patients; bowel perforation, 1 patient).

5 ntestinal stoma formation in children, while bowel perforation (14, 31.8%) was the main indications i

6 with inadequate distribution (7%) and small-bowel perforation (3%) make the otherwise less toxic 32P

7 despite the PKD group's higher incidence of bowel perforation and increased age at time of transplan

8 colonoscopy-related complications, including bowel perforation and major bleeding.

9 On multivariable analysis, bile duct injury, bowel perforation, and high clinical severity were assoc

10 ocedures (hepatic arterial hemorrhage, small bowel perforation, and liver decompensation salvaged by

11 onal age, ductus closure, occurrence of NEC, bowel perforation, and mortality.

12 atment were gastrointestinal bleeding, small-bowel perforation, and the development of enterocolic fi

13 A on colonoscopy complications, specifically bowel perforation, aspiration pneumonia, and splenic inj

14 %) in emergency neonatal surgeries involving bowel perforation, bowel resection, congenital diaphragm

15 Bowel perforation can lead to significant bacterial spil

16 The primary outcome was bowel perforation, defined using a validated algorithm.

17 al adhesions are at potential higher risk of bowel perforation during implantation of an indwelling p

18 e that typhoid fever accounts for 43% of all bowel perforation during the period of enhanced surveill

19 Delayed small bowel perforation following BAT is thought to occur seco

20 is is the first case report of delayed small bowel perforation following BAT with extensive portomese

21 re of any other case report of delayed small bowel perforation following BAT without signs of intraab

22 Isolated small bowel perforation following blunt abdominal trauma (BAT)

23 sity contributed to extensive hemorrhage and bowel perforation for each tissue above a certain energy

24 cate that there is increased risk of NEC and bowel perforation in premature infants with PDA receivin

25 ctivated FXII (FXIIa) modifies the course of bowel perforation-induced peritoneal sepsis in mice.

26 idity and mortality rates, and delayed small bowel perforation is even rarer.

27 cations included bile duct injury (n = 397), bowel perforation (n = 96), and hemorrhage (n = 78).

28 As major complication one small bowel perforation occurred (1/59; 1.7%).

29 reased risk of aspiration pneumonia, but not bowel perforation or splenic injury.

30 No bowel perforations or fistulas occurred.

31 (OR 1.71, 95% CI 1.31-2.24), small and large bowel perforation (OR 4.33, 95% CI 4.12-4.56), and pepti

32 (OR 4.06, 95% CI 3.03-5.44), small and large bowel perforation (OR 6.97, 95% CI 6.60-7.37), and pepti

33 Progression-free survival (PFS) and bowel perforation rates were taken from recently reporte

34 cept for acute medical conditions, including bowel perforation (relative risk [RR] = 3.0, 95% confide

35 Vascular thromboses, bowel perforation, septicemia, and retransplantation, ea

36 Treatment with 14E11 within 12 hours after bowel perforation significantly improved survival compar

37 ications (bleeding, transfusion requirement, bowel perforation, surgical intervention, and graft loss

38 GOG 218 at the baseline estimates of PFS and bowel perforation, the cost of PC was $2.5 million, comp

39 Bowel perforation was noted in 27 cases (30%).

40 events such as hypertension, thrombosis, and bowel perforation were also observed at rates consistent

41 was seen with the 7-day infusions (including bowel perforation), with 600,000 IU/m2 as the maximum-to