ライフサイエンスコーパス: brain MRI

1 from 135 high risk patients with CIS (>/= 2 brain MRI lesions and >/= 2 oligoclonal bands) enrolled

2 ease (SVD) and performed standardized 3-T 3D brain MRI studies.

3 A total of 2,362 3T brain MRI scans were acquired from 469 subjects.

4 T1-weighted 3T brain MRI data of 15 patients suffering from motor CD (n

5 ignificant nonlinear associations with all 4 brain MRI metrics, with mid-range CGG repeat carriers ev

6 In total, 222 brain CT, 47 brain MRI, and seven spinal MRI examinations were perfor

7 A brain MRI was obtained at baseline in 166 patients asses

8 sual attention task (n = 35; phase 2b) and a brain MRI after traumatic brain injury (n = 23; phase 2c

9 Each patient had a brain MRI scan at entry and 6 months later using a stand

10 ldren were clinically assessed and 351 had a brain MRI scan.

11 A subgroup of participants also underwent a brain MRI examination.

12 their healthy full-term newborns underwent a brain MRI including diffusion tensor imaging at approxim

13 nd in 5 of 29 patients (17%) who underwent a brain MRI.

14 eimer's disease pattern similarity scores (a brain-MRI measured neuroanatomical risk for Alzheimer's

15 Abnormal brain MRI findings were associated with the presence of

16 ies, spasticity or ataxic gait, and abnormal brain MRI.

17 neurologic manifestations, and (d) abnormal brain MRI findings.

18 volving the brain; 30% of cases had abnormal brain MRI at onset and 75% by follow up.

19 Conclusion A high incidence of abnormal brain MRI examinations and nonlongitudinal extensive SC

20 estations that were associated with abnormal brain MRI scans.

21 pite being four times faster, DL-accelerated brain MRI was interchangeable with conventional MRI for

22 Background Clinically acquired brain MRI scans represent a valuable but underused resou

23 ns in relapse rate and development of active brain MRI lesions, measures considered to reflect CNS in

24 Acute brain MRI and available follow-up MRIs were reviewed.

25 ma which was a radiological suggestion after brain MRI).

26 Although brain MRI findings are not characteristic for ephedrone

27 searchers worldwide to automatically analyse brain MRIs at a higher level of granularity, NextBrain h

28 We analysed brain MRIs and neuropsychological test scores for 228 ne

29 g multicenter neuroimaging data, we analyzed brain MRI scans from 2028 schizophrenia patients and 254

30 Anatomic brain MRI scans were obtained from 15 psychiatrically he

31 Thirteen healthy children for whom anatomic brain MRI scans were obtained every 2 years, for 8-10 ye

32 Anatomic brain MRIs for 57 boys with ADHD and 55 healthy matched

33 Anatomical brain MRI was performed at baseline and follow-up in 19

34 lgorithmically calculated for 108 anatomical brain MRI scans from 50 patients (20 of whom were female

35 In a cross-sectional design, 3 T anatomical brain MRI was acquired in 27 medication-free youth with

36 itive testing, stool microbiota analysis and brain MRI analysis.

37 underwent clinical cognitive assessments and brain MRI.

38 ospital's website for vaginal childbirth and brain MRI collected from representative US hospitals bet

39 ations between cord atrophy and clinical and brain MRI measures were investigated with multiple regre

40 l linear regression to identify clinical and brain MRI predictors of change in FMA-UE.

41 tory study, we assessed whether clinical and brain MRI variables predict response to treatment.

42 istory of dementia, changes in cognition and brain MRI outcomes from baseline to year 3 did not diffe

43 , and time interval between abdominal CT and brain MRI.

44 uring gross motor function deterioration and brain MRI.

45 underwent neuropsychological evaluation and brain MRI at exam 7.

46 ercise stress testing, cardiac CT, heart and brain MRI, serial vascular tonometry and accelerometry)

47 the evaluation of cognition and memory, and brain MRI or FDG-PET abnormalities less frequently restr

48 Neuropsychological and brain MRI variables were related to cardiac MRI-assessed

49 d transbronchial needle aspiration, PET, and brain MRI) by a clinical tumor board served as the refer

50 d children based on the presence of rash and brain MRI findings.

51 Head CT scan and brain MRI showed a huge intra-axial right temporo-pariet

52 (t-tau), phosphorylated tau 181 (p-tau) and brain MRI examination.

53 MR angiography in TOF technique and brain MRI in T1- and T2-weighted images, FLAIR and DWI s

54 nt subsequent neuropsychological testing and brain MRI at a mean follow-up of 17 months.

55 examination, neuropsychological testing and brain MRI.

56 Clinical information and brain MRIs were collected from 221 healthy individuals a

57 f FBDS alone, patients had normal sodium and brain MRIs, but electroencephalography demonstrated icta

58 Patients with MS also underwent annual brain MRI scans.

59 umine, and had the baseline scan and another brain MRI scan available for comparison.

60 rs completed neurocognitive testing, another brain MRI, and their parents completed neurobehavioural

61 ment, gait and balance assessment as well as brain MRI.

62 This was a retrospective study assessing brain MRI of 26 patients with natalizumab-associated PML

63 atients with 19 common and rare diagnoses at brain MRI acquired between January 2008 and January 2018

64 for generation of differential diagnoses at brain MRI compared with radiologists.

65 al diagnoses for rare and common diseases at brain MRI has not been demonstrated.

66 Background Abnormal findings at brain MRI in patients with neurologic Wilson disease (WD

67 matoma evacuation, or autopsy) and available brain MRI sequences of adequate quality, including T2-we

68 ive, blinded analysis of the first available brain MRIs from 192 patients at Oxford University Hospit

69 ions sharing the "molar tooth sign" on axial brain MRI, together with cerebellar vermis hypoplasia, a

70 106 patients had evaluable post-baseline brain MRI scans and were assessed for efficacy in the fe

71 etween 18 and 90 years old at their baseline brain MRI and had never received a GBCA, had undergone t

72 ischaemic stroke (n = 100) were assessed by brain MRI at 3 T including diffusion weighted imaging.

73 ite matter hyperintensities were assessed by brain MRI.

74 level, myopathic electrodiagnostic changes, brain MRI with cobblestone complex, and mutation in the

75 of childhood-onset chorea and characteristic brain MRI showing symmetrical bilateral striatal lesions

76 Clinical brain MRI is normal in the majority of patients with ant

77 A total of 13,215 clinical brain MRI studies were categorized to training (74%), va

78 spital cardiac arrest and who had a clinical brain MRI or MRS performed within 14 days postarrest wer

79 s (161 females; 48 males) underwent clinical brain MRI and were genotyped for the BDNF rs6265 Val66Me

80 We collected brain MRI scans from 615 healthy young adult twins and s

81 Conventional brain MRI with intravenous administration of contrast ma

82 Conventional brain MRI with intravenous administration of contrast ma

83 sts detect findings not seen on conventional brain MRI that sometimes result in substantial changes t

84 Using conventional brain MRI, T1-weighted intensity-based measures of chron

85 blinded standardised review of conventional brain MRIs of 30 patients with MS, 31 patients with LHON

86 A blinded review of conventional brain MRIs shows that patients with LMS have a scan appe

87 onald criteria, a relapsing clinical course, brain MRI lesions consistent with multiple sclerosis, an

88 ed text-vision framework accurately detected brain MRI abnormalities without expert-labeled datasets.

89 All three subjects presented a distinctive brain MRI pattern characterized by cavitating leukodystr

90 Being aware of TH during brain MRI evaluation will help prevent possible misdiagn

91 normal admission head CT had abnormal early brain MRI.

92 In addition to admission head CT, early brain MRI was performed 12 +/- 3.9 days after injury.

93 test in routine practice, and gradient echo brain MRI will identify all symptomatic cases.

94 The SI for up to eight brain MRI examinations per patient was measured, and rel

95 ctroencephalography (EEG), electromyography, brain MRI, CSF analysis, or a combination of these analy

96 s were also assessed by electroretinography, brain MRI and magnetic resonance spectroscopy (MRS), and

97 rmance in automatic protocoling of emergency brain MRI scans based on text from clinical referrals.

98 These patients also had gadolinium enhanced brain MRI scans and were divided into relapsing-remittin

99 comparison was made with gadolinium-enhanced brain MRI performed approximately 9 years earlier (Figs

100 comparison was made with gadolinium-enhanced brain MRI performed approximately 9 years earlier.

101 Participants had clinical evaluations, brain MRI, and blood draws annually.

102 construction of three-dimensional (3D) fetal brain MRI have led to significant improvements in the qu

103 onal volumes that were registered to a fetal brain MRI atlas with 28 anatomic regions of interest.

104 Materials and Methods Consecutive fetal brain MRI examinations performed between January 2014 an

105 ning these results with an independent fetal brain MRI cohort (n = 228; 21-36 gestational weeks), we

106 f this atlas and additional individual fetal brain MRI atlases for completely automatic multi-atlas s

107 automatic multi-atlas segmentation of fetal brain MRI.

108 This is the first brain MRI study of the impact of the 16p11.2 distal CNV,

109 dbirth in 14% (3 of 22) of hospitals and for brain MRI in 19% (9 of 47) of hospitals.

110 s providing both online and phone prices for brain MRI, prices were within 25% of each other for 66%

111 e neurologic manifestations and referral for brain MRI.

112 atients matched for age and sex referred for brain MRI under general anesthesia.

113 testing of a portable prototype scanner for brain MRI that uses a compact and lightweight permanent

114 lusion An artificial intelligence system for brain MRI approached overall top one, top two, and top t

115 ying headaches and detecting biomarkers from brain MRI data.

116 For example, shape measurements derived from brain MRI scans are multidimensional geometric descripti

117 We present detailed findings from brain MRI in three mutation-positive individuals.

118 h and without FXTAS) with five outcomes from brain MRI imaging and 22 peripheral bioenergetic outcome

119 performed repeated structural and functional brain MRI in 108 Barcelona participants.

120 t the relationship between input data (e.g., brain MRIs) and output variables (e.g., diagnosis).

121 ants who had previously received gadobutrol, brain MRI showed no differences relative to healthy cont

122 ar bundle RNFL correlated with higher global brain MRI lesion burden index (R(2) = 0.35, P = .001) an

123 , 50 HE subjects, and 43 AD/MCI patients had brain MRI and PiB PET.

124 65.1 [13.8] years; 126 men [57.5%]) who had brain MRI data were included.

125 brain atrophy was found in 12 of 14 who had brain MRI.

126 in volumes using a dataset of 10,099 healthy brain MRIs.

127 ork obtained from tractography of 14 healthy-brain MRIs.

128 We analyzed retinal leakage, histopathology, brain MRI, and associations with death and neurological

129 underwent (i) clinical evaluations; and (ii) brain MRI scans analysed using whole-brain voxel-based m

130 contributor to the performance of GPT-4V in brain MRI differential diagnosis, followed by the medica

131 Distortions in brain MRI caused by gradient nonlinearities may reach se

132 ssion of signal intensity changes visible in brain MRI is not associated with clinical condition impr

133 s to identify the presence of 4 variables in brain MRIs including abnormal, acute infarction, acute h

134 , patients referred for clinically indicated brain MRI from January 2006 through May 2016 were evalua

135 Initial brain MRI revealed multiple, disseminated lesions that w

136 Initial brain MRI was abnormal in 54 (67%).

137 article presents clinical symptoms, initial brain MRI findings and characteristics of changes observ

138 n blinded visual assessment of their initial brain MRI scans.

139 Wechsler Abbreviated Scale of Intelligence), brain MRI, medical records, and structured interviews wi

140 een 2002 and 2006, more than 26 years later, brain MRIs were performed.

141 year period, based on analyzing longitudinal brain MRIs from 12 COS patients and 12 healthy controls

142 ogressive multiple sclerosis who had monthly brain MRI studies for 4 months (one baseline and three f

143 nium-enhanced T1-weighted lesions on monthly brain MRI scans from weeks 8 to 24.

144 aging studies (consisting of whole-body MRI, brain MRI, breast MRI, mammography, abdominal and pelvic

145 In this case-controlled, multimodal brain MRI study of 44 carefully phenotyped subjects, we

146 The evolution of multimodal brain MRI demonstrates that neuroimaging findings of tis

147 In this retrospective study, multimodal brain MRI scans from 212 patients (mean age, 55 years +/

148 Subjects also underwent multimodal brain MRI.

149 eurodevelopmental outcome we used multimodal brain MRI to study a large cohort of preterm infants.

150 onclusion Deep learning analysis of neonatal brain MRI yielded high performance for predicting 2-year

151 ticularly unsupervised) analysis of neonatal brain MRI's.

152 omen) with 35 neurologic diseases and normal brain MRI scans obtained between January 2008 and Januar

153 (infantile encephalopathy and largely normal brain MRI) to that of NALCN-related infantile encephalop

154 We prospectively obtained brain MRIs and cognitive testing in healthy controls and

155 e scanner could improve the accessibility of brain MRI at the point of care, particularly for critica

156 We performed secondary analyses of brain MRI GWAS and exome sequencing data from adults in

157 A systematic analysis of brain MRI features revealed a common diagnostic signatur

158 bellum as revealed by volumetric analysis of brain MRI scans.

159 Analysis of brain MRI studies identified non-specific neuroimaging f

160 tients were highly correlated with degree of brain MRI involvement.

161 of this study is to evaluate the efficacy of brain MRI without anesthesia in infants younger than 3-m

162 Re-evaluation of brain MRI images of four individuals showed a shared dis

163 Results of brain MRI were abnormal in 18 (31.6%) of 57 patients: 8

164 avity, assessed by blinded central review of brain MRI scans by the study neuroradiologist in the mod

165 rom JXG with CNS involvement and the role of brain MRI including DWI and PWI in the evaluation of bra

166 Over the past two decades, thousands of brain MRI scans from healthy youth and those with neurop

167 On brain MRI, within 1 year there was stabilization of T2/F

168 (0->=2) and/or new T2 lesions (<3 or >=3) on brain MRI.

169 Prevalence of abnormalities on brain MRI by category of finding (no referral necessary,

170 rmine the long-term risk of abnormalities on brain MRI for the development of multiple sclerosis and

171 anges and subtle structural abnormalities on brain MRI.

172 lar ataxia and white matter abnormalities on brain MRI.

173 are inversely associated with MS activity on brain MRI.

174 ncluding ataxia and/or cerebellar atrophy on brain MRI.

175 Here, based on brain MRI, we apply a 3D surface-based morphometry metho

176 of microbleeds were assessed at baseline on brain MRI of 4759 participants aged >/=45 years.

177 ive impairment and a thin corpus callosum on brain MRI.

178 teral involvement of the internal capsule on brain MRI was associated with poorer survival [20 +/- 18

179 y, chronic insomnia, white matter changes on brain MRI, dysmorphic features, decreased stature, and d

180 between groups and no significant effects on brain MRI measures were noted in either group (mean T2 l

181 demyelination and gadolinium enhancement on brain MRI.

182 We found that ependymitis on brain MRI was the best predictor of higher log(sCD27) le

183 imaging pipeline detected 11 key features on brain MRI scans with 89% accuracy (sensitivity, 81%; spe

184 rintensity (WMH) burden is commonly found on brain MRI among patients with atrial fibrillation (AF).

185 es and types of incidental findings found on brain MRI in patients with HIV infection, who may be at

186 (FLAIR) hyperintense arteries (FLAIR-HAs) on brain MRI and prognosis after acute ischaemic stroke (AI

187 silent infarcts were defined as infarcts on brain MRI in patients without a clinical history of stro

188 f imaging characteristics of inflammation on brain MRI scans of inflammatory NTZ-PML patients.

189 tural, hippocampal, or neocortical lesion on brain MRI is not always indicative of the site of seizur

190 he number of gadolinium-enhancing lesions on brain MRI scans for both RRMS studies.

191 gadolinium-enhancing T1-weighted lesions on brain MRI, clinical relapses, and disability (Expanded D

192 and 25.8 +/- 2.9 in those without LNCCIs on brain MRI (p < 0.001).

193 ies (WMH), lacunes, and microbleeds (MBs) on brain MRI.

194 inium contrast-enhancing lesions measured on brain MRI scans every 4 weeks between weeks 8 and 24.

195 he highly characteristic molar tooth sign on brain MRI.

196 loped a deep neural network model trained on brain MRI scans of healthy people to predict "healthy" b

197 nd scored available postnatal, pre-operative brain MRI for brain pathology.

198 gnificant, and no changes on the ECG, EEG or brain MRI were observed.

199 noncontributory data regarding brain MRI or brain MRI showing ischemic infarcts, cerebral venous thr

200 MRI, particularly brain MRI, has a pivotal role in the early diagnosis of

201 and two of the following criteria: positive brain MRI; positive spinal cord MRI; or positive cerebro

202 enrolled for preoperative and postoperative brain MRIs between 2001 and 2021 with a total of 466 MRI

203 Pre- and postprocedural brain MRI was performed on each patient within a week of

204 patients showed improvement on posttreatment brain MRI scans.

205 iles), measured on average 7 years preceding brain MRI, was associated with higher odds of having sev

206 ectively analyzed multisequence preoperative brain MRI from 199 adult patients with glioblastoma who

207 tal heart disease who underwent preoperative brain MRI as part of 2 separate prospective studies.

208 mean number of lesions noted on pretreatment brain MRI scans.

209 We performed a comparative prospective brain MRI study in patients with COS and pediatric patie

210 Conclusion DLMUSE enabled rapid brain MRI segmentation, with performance comparable to t

211 nsity on fluid-attenuated inversion recovery brain MRI within a vascular distribution.

212 th missing or noncontributory data regarding brain MRI or brain MRI showing ischemic infarcts, cerebr

213 alysed) and 254 (48%) underwent the required brain MRI protocol (122 in the start antiplatelet therap

214 able partial epilepsy, where high-resolution brain MRI disclosed a unilateral, focal, hippocampal, or

215 With high-resolution brain MRI scans, we created composite maps of cortical g

216 very (FLAIR) sequence is part of the routine brain MRI protocol.

217 d onto a surface rendering of each subject's brain MRI.

218 Non-sedated brain MRI was acquired for full-term (n = 13) and premat

219 Serial brain MRI scanning is widely used for assessing multiple

220 nts with childhood-onset schizophrenia share brain MRI abnormalities with the patients that may follo

221 All patients underwent a standard brain MRI during the first 30 days after ICU admission.

222 Baseline clinical data and standardized brain MRI scans from the Swiss Atrial Fibrillation cohor

223 A standardized brain-MRI protocol using a 3-Tesla machine and 16-channe

224 Structural brain MRI was obtained at term-equivalent age and DWMA v

225 Structural brain MRI was performed at term.

226 High field structural brain MRI images were acquired in a 3T scanner and analy

227 ased on measurements derived from structural brain MRI scans.

228 brain imaging studies, including structural brain MRI, magnetoencephalography and transcranial magne

229 Analysis of structural brain MRI scans showed a dose-dependent change in the ra

230 spaces, brain atrophy) as seen on structural brain MRI and of a global SVD score on the patients' per

231 r for dystonia diagnosis from raw structural brain MRIs of 612 subjects, including 392 patients with

232 er and gray matter on 10 different synthetic brain MRI images corrupted with 18 different combination

233 LNCCIs and other brain lesions on systematic brain MRI screening, and most of these lesions are clini

234 to only gadoterate meglumine underwent 3.0-T brain MRI with a dedicated head coil, including T1 mappi

235 Participants had 3 T brain MRI at baseline.

236 nal analysis, neurocognitive testing and 3 T brain MRI's were obtained in 101 survivors treated with

237 ative and quantitative neuroimaging with 3-T brain MRI and optical coherence tomography.

238 rial Fibrillation (MACPAF) study, serial 3-T brain MRIs and neuropsychological assessment were perfor

239 We emphasize the importance of 3D-T1 brain MRI with a standardized review protocol including

240 matched control subjects underwent a 7-Tesla brain MRI scan and a detailed cognitive assessment.

241 The brain MRI images of these patients were evaluated.

242 The brain MRI results were normal.

243 s in whom SWI acquisition formed part of the brain MRI protocol, and Group comprised patients who und

244 This study suggests that the brain MRI criteria for differentiating RRMS from NMOSD a

245 acute or subacute neurological symptoms; the brain MRIs indicating diffuse brain damage.

246 stroke and dementia and is diagnosed through brain MRI.

247 Thus, brain MRI findings were normal in the majority of patien

248 brain swelling or focal changes according to brain MRI.

249 sing step for machine learning approaches to brain MRI analysis.

250 In participants who underwent at least two brain MRI examinations (subgroup), the first and last av

251 ss-sectional study of outpatients undergoing brain MRI at 1 outpatient imaging facility was conducted

252 ional study included all patients undergoing brain MRI including SWI during a 6-month period.

253 hort (mean age 39.2 years, SD 8.4) underwent brain MRI between June, 2009 and June, 2010.

254 y-six patients with non-COVID ARDS underwent brain MRI during the index hospitalization, resulting in

255 troke or transient ischemic attack underwent brain MRI in 1992 and 1993, 1 to 2 years before echocard

256 ons centered on 4 Canadian cities, underwent brain MRI and simple tests of cognition and gait as part

257 imia nervosa and 8 female controls underwent brain MRI followed by (11)C-carfentanil PET.

258 group of 16 healthy term newborns underwent brain MRI at term-equivalent age.

259 e symptoms within 6 hours of onset underwent brain MRI followed by noncontrast CT.

260 The patient underwent brain MRI for further evaluation.

261 ation carriers and 10 NC relatives underwent brain MRI and clinical assessment.

262 All of them underwent brain MRI with the use of 1.5T scanners.

263 hildren, including 66 children who underwent brain MRI (median [IQR] age, 1.0 [0.0-3.0] years; 28 gir

264 Material/Patients with IIH who underwent brain MRI and contrast-enhanced MR venography before mea

265 %, 95% CI: 58, 87) in patients who underwent brain MRI, often with an ovoid shape suggestive of micro

266 ~70% of young carrier parents who underwent brain MRI.

267 ed at age 11 and 79 years, and who underwent brain MRI.

268 (47% males; range, 1 to 11 yr) who underwent brain MRI.

269 participants, aged 65 to 84 years, underwent brain MRI including DWI at baseline.

270 Unenhanced brain MRI was performed in 220 participants (76, 84, 25,

271 Purpose To perform follow-up brain MRI in volunteer participants who had previously r

272 We used brain MRI or post-mortem examination in 351 fullterm inf

273 ry and 30 age-matched healthy controls using brain MRI.

274 High-resolution in vivo brain MRI can be used to estimate genetic correlations (

275 AT, and VAT, they had undergone a volumetric brain MRI scan with measurements of total brain volume (

276 SM-IV) based history of enuresis, volumetric brain MRI scans and neuropsychological testing were obta

277 dementia-free survivors underwent volumetric brain MRI between 1999 and 2005, approximately 7.7 years

278 ce models were extracted from 82 T1-weighted brain MRI scans (256 x 192 x 124 volumes) of 42 subjects

279 ongitudinal conventional T2- and T1-weighted brain MRI scans, we measured the relative amount of chro

280 w-up on 81 such patients who had T2-weighted brain MRI at presentation.

281 nium-enhanced and proton density/T2-weighted brain MRI from months 0 to 6 and 18 to 24 to determine t

282 nt lesions of at least 3 mm on a T2-weighted brain MRI scan, and an Expanded Disability Status Scale

283 Patients also underwent T1- and T2-weighted brain MRI.

284 ents with PTEN pathogenic variants for which brain MRIs were available (age range 0.4-17 years).

285 e determined the contribution of these whole brain MRI markers to cognitive impairment in SVD.

286 nts into the brain of mice for in vivo whole brain MRI.

287 We report a whole-brain MRI morphometric survey of asymmetry in children w

288 Therefore, whole-brain MRI scans were acquired from 31 neurologically-hea

289 Structural T(1)-weighted whole-brain MRI data from healthy control subjects (N=5,827) a

290 79%) completed the trial with eligible whole-brain MRIs.

291 did not require ventilatory support for whom brain MRI was indicated.

292 Covert cSVD, which is detectable with brain MRI but does not manifest as clinical stroke, is h

293 pting to correlate locomotor disability with brain MRI findings.

294 der individuals with WMH were evaluated with brain MRI and detailed clinical and neuropsychological a

295 This article addresses findings with brain MRI that may underlie cognitive dysfunction in dia

296 ted in 4 sites; 60 patients were imaged with brain MRI after TAVR.

297 tes with CHD were studied prospectively with brain MRI: before surgery, within 2 weeks of surgery, an

298 a term applied to asymptomatic subjects with brain MRI abnormalities highly suggestive of multiple sc

299 f two sets of CTX patients, with and without brain MRI evidence of DN involvement, with a set of heal

300 f prospective follow-up that included yearly brain MRI and biannual clinical visits (n = 219).