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1  population but do not usually contribute to brain pathology.
2 n levels being associated with more advanced brain pathology.
3 thologically verified AD and correlates with brain pathology.
4 ese systems and the degree of Alzheimer type brain pathology.
5 equire greater recognition as a mechanism of brain pathology.
6 at oral intake of DHA reduces Alzheimer-like brain pathology.
7 sis stages and occur mostly independent from brain pathology.
8 tween neuronal vimentin expression and local brain pathology.
9 d in the Tg2576 mouse model of AD-like Abeta brain pathology.
10 ven before the development of substantial AD brain pathology.
11 ausal TSE virions that induce late-stage PrP brain pathology.
12 s with HS and 12 patients without structural brain pathology.
13  addition to the activation that accompanies brain pathology.
14  in vivo neuroepigenome editing for treating brain pathology.
15 s in the brains of transgenic animals causes brain pathology.
16 ction in TNF-alpha levels, leukocytosis, and brain pathology.
17 , coinciding with cortical demyelination and brain pathology.
18 lowed the progression of motor phenotype and brain pathology.
19 ities are an important contributor to ageing brain pathology.
20  CD8(+) T cells to the brain, and negligible brain pathology.
21 o clinical outcome and precedes the onset of brain pathology.
22 tive, but unspecific tool to quantify active brain pathology.
23 hiatric symptom in relation to biomarkers of brain pathology.
24 ouse with high probability in the absence of brain pathology.
25 n reveal possible mechanisms contributing to brain pathology.
26 duced the infiltration of CD8(+) T cells and brain pathology.
27 onsidered a promising blood marker of active brain pathology.
28 e and AD patients did not have any effect in brain pathology.
29 isturbances in the absence of any structural brain pathology.
30 pacity to maintain physical function despite brain pathology.
31 may hold promise as a biomarker for early AD brain pathology.
32 volvement early in PD, which correlates with brain pathology.
33 observations, and more closely represent HIV brain pathology.
34 immune responses in the brain, contribute to brain pathology.
35 roglial physiology and their contribution to brain pathology.
36 flect the complex spatiotemporal features of brain pathology.
37 microbial infections, that could precipitate brain pathology.
38 l was extended to 462 days with reduction in brain pathology.
39 ted on cognitive functions and on markers of brain pathology.
40 maintenance, or relative lack of age-related brain pathology.
41 lable postnatal, pre-operative brain MRI for brain pathology.
42 ccumulating mosaic somatic mutations promote brain pathology.
43  it may provide a noninvasive way to examine brain pathology.
44  life for their memory, learning ability and brain pathology.
45  underlying adaptation, stress response, and brain pathology.
46 creased blood-brain barrier permeability and brain pathology.
47  or risk adjustment, and how this relates to brain pathology.
48  group A PfEMP1 may be effective at reducing brain pathology.
49 tant role for monocytes/microglia in causing brain pathology.
50 anoids to pioneer new understanding of human brain pathology.
51 meningeal-located immune cells in modulating brain pathology.
52 significantly associated with all markers of brain pathology.
53 sy development, even in the absence of major brain pathology.
54 ology after traumatic brain injury and other brain pathologies.
55  system and is important in stroke and other brain pathologies.
56 lood-brain barrier, thus benefiting numerous brain pathologies.
57 ype could be useful for treatment of various brain pathologies.
58  Hcy-thiolactone and lead to vascular and/or brain pathologies.
59 eficial role in neuroprotection from various brain pathologies.
60 ain yet have different disease latencies and brain pathologies.
61 y prediabetes but also cognitive decline and brain pathologies.
62 hout significant levels of neurodegenerative brain pathologies.
63 nd cervical lymph nodes (CLNs) is crucial in brain pathologies.
64 enetration of drugs used in the treatment of brain pathologies.
65 ations (SDs) have been identified in various brain pathologies.
66 ered our understanding of the eCSF's role in brain pathologies.
67 lopment and homeostasis-and in a plethora of brain pathologies.
68  new perspectives for detecting and treating brain pathologies.
69 nt and clinical manifestation of age-related brain pathologies.
70 icant association with any of the underlying brain pathologies.
71 rously test them against complex and diverse brain pathologies.
72 ure would be more sensitive to the effect of brain pathologies.
73 xhibited prediabetes, cognitive decline, and brain pathologies.
74 ain for the early detection and treatment of brain pathologies.
75 ling, identifying, and treating the affected brain pathologies.
76 d is increasingly associated with many other brain pathologies.
77 of targeting RyR2 hyperactivity in heart and brain pathologies.
78 k and vasculature are ubiquitous in numerous brain pathologies.
79 NA editing process itself is dysregulated by brain pathologies.
80  of biological data across a growing list of brain pathologies.
81 lia, potentially implicating Cyp1b1 in other brain pathologies.
82 ell cycle checkpoint control with a focus on brain pathologies.
83 ledged as prime regulators involved in human brain pathologies.
84 ons have been extensively studied in various brain pathologies.
85 ctive processes and is highly susceptible to brain pathology across common disorders.
86 ican-style football (ASF) has been linked to brain pathology, along with physical and mental distress
87                                              Brain pathologies amplify this variability through disco
88 ring stroke, brain injury, and certain other brain pathologies, anaerobic glycolysis produces severe
89 perinatal period to block the progression of brain pathologies and allow normal brain development to
90 y of LATE-NC and its associations with other brain pathologies and cognition in a population aged >=
91 ntrations, and dynamics cell volume in other brain pathologies and normal brain function.
92 edents with annual motor testing, postmortem brain pathologies and quantified 226 proteotypic peptide
93 tween some measures of ALF and biomarkers of brain pathologies and subjective cognitive decline in co
94 ting immune cells are needed for coping with brain pathologies and that their optimal effect is depen
95 ies have demonstrated a relationship between brain pathology and AD progression, the Alzheimer's path
96 T are informative metrics for characterizing brain pathology and alternative energy use, and may prov
97 rozygous TSC animal models do not show gross brain pathology and are seizure-free, suggesting that su
98 ia cannot encase Abeta plaques, accelerating brain pathology and behavioral deficits.
99 ssion were assessed at 15 and 24 months, and brain pathology and biochemistry were assessed at 24 mon
100 sive, affordable bedside tool for predicting brain pathology and death in TBM.
101 e and affordable bedside tool for predicting brain pathology and death in TBM.
102 ationship between neocortical Alzheimer-type brain pathology and dementia in consecutive autopsies fr
103 o be identified (E46K) results in widespread brain pathology and early onset Parkinson symptoms.
104 omise in detecting markers of HIV-associated brain pathology and explore strategies to study the impa
105 have been proposed based on genetic studies, brain pathology and imaging, but a major impediment to t
106 rstanding the association between cell-level brain pathology and noncoding variants are lacking.
107 dvances understanding of Alzheimer's disease brain pathology and provides new insights into the subce
108 use of the many-to-many relationship between brain pathology and psychiatric symptoms, true natural d
109 oligodendrocyte Abeta production improves AD brain pathology and restores neuronal function in the mo
110 r a quarter of a century despite severe host brain pathology and with no evidence of immune response.
111 hagocytosis of viable neurons contributes to brain pathology and, surprisingly, blocking this process
112 ved in synapse homeostasis, or implicated in brain pathologies, and identified the phagosome as the s
113 ute to the development of lactic acidosis in brain pathologies, and suggest targeting it as a novel t
114            Complex interactions among sleep, brain pathology, and cardiovascular disease may occur in
115 showed viral persistence in the CNS, chronic brain pathology, and demyelination in the spinal cord wh
116 elates, in fatal cases, with the severity of brain pathology, and has diagnostic and prognostic signi
117 g can lead to deleterious neuroinflammation, brain pathology, and neurological dysfunction.
118  production that promotes neuroinflammation, brain pathology, and seizure propagation.
119 lation of dural ELS varies according to age, brain pathology, and sex.
120                                         Many brain pathologies are associated with liver damage, but
121 hose aged 90 and older, other ageing-related brain pathologies are prevalent in addition to Alzheimer
122 ging, whereby the spatial extent and load of brain pathology are used to estimate the severity of Alz
123 ated with low level Alzheimer's Disease-like brain pathology as observed by (11)C-PiB PET and (18)F-F
124 ring strategy using PAH or PAL corrects both brain pathology as well as previously unknown lipid meta
125 studies have linked particulate exposures to brain pathologies associated with Parkinson's and Alzhei
126 tudies suggest that antibodies contribute to brain pathology associated with autoimmune diseases such
127                           In later adulthood brain pathology becomes common and trajectories of cogni
128 nsing platform for the detection of multiple brain-pathology biomarkers.
129 ributing to neurodegeneration in retinal and brain pathologies, but its role in retinitis pigmentosa
130 chiatric disorders classically lack defining brain pathologies, but recent work has demonstrated dysr
131  potential to tailor solutions for different brain pathologies by choosing the composition of the NDs
132 /activation or induction of dementia-related brain pathologies by the infection.
133 icroglial cells are considered as sensors of brain pathology by detecting any sign of brain lesions,
134 ful memory aging can be accomplished despite brain pathology by means of efficient compensatory and s
135 hed exosomes appear to exacerbate AD-related brain pathology by promoting the aggregation of Abeta.
136 t R47H and evaluated the variant's effect on brain pathology, cellular morphology, and expression of
137 lts suggest a likely contribution of diet to brain pathologies characterized by neuroinflammation and
138 severe weight loss and hypoventilation, with brain pathology characterized by TDP-43 immunostaining.
139  markedly reduced meningeal inflammation and brain pathology compared with wild type, despite similar
140 g to a shortened lifespan (mean 58 days) and brain pathology consistent with TSC.
141 repeats, supporting the idea that underlying brain pathology contributes to SCA1-like anxiety.
142                             A broad range of brain pathologies critically relies on the vasculature,
143                                  Analysis of brain pathology demonstrated cortical and hippocampal la
144 l experiments have not demonstrated enduring brain pathology despite chronic, high ethanol (EtOH) exp
145 tenuated Bartha strain does not cause severe brain pathology despite viral replication and spread thr
146                                              Brain pathology developed primarily on the ipsilateral s
147     These results suggest that expression of brain pathology differs between left-handed and right-ha
148 , similar to what has been observed in human brain pathology, diffuse deposits display higher levels
149         This report broadens the spectrum of brain pathologies elicited by protein misfolding and pro
150  that intestinal inflammation is linked with brain pathology even in the earliest disease stages.
151 ngivalis alone significantly increased all 7 brain pathologies examined: neuronal damage, activation
152  motor decline in models controlling for ten brain pathologies (FDR < 0.05).
153                          The independence of brain pathology from visceral complications in Niemann-P
154 pus represents a putative epicenter of early brain pathology from which dysfunction may spread to aff
155 riant in a Wnt pathway gene and ASD-relevant brain pathology has not been established.
156 ty, and the relationships of OE pathology to brain pathology have not been established.
157 lack of correlation between serum titers and brain pathology have, no doubt, contributed to a delayed
158 cterized by atrophy without any of the major brain pathologies, here termed Non-Vascular-Alzheimer-Sy
159  what causes it, how it leads to surrounding brain pathology, how researchers can investigate it with
160 ects of nec-1 and metformin on cognition and brain pathologies in prediabetes have not been investiga
161 min equally attenuated cognitive decline and brain pathologies in prediabetic rats.
162  CamKII-Cre driver and examined behavior and brain pathology in 12-18 month old animals.
163  lysates and purified protein can accelerate brain pathology in a manner similar to prions.
164 knockout of Sirt1 results in exacerbation of brain pathology in a mouse model of Huntington's disease
165 regates as a frequent and unexpected type of brain pathology in AD and CASP8-GGGAGA-AD-R1 alleles as
166 gyrus mean diffusivity as an early marker of brain pathology in children with OSAS.SIGNIFICANCE STATE
167 stand how CD4(+) T lymphocytes contribute to brain pathology in chronic intestinal inflammation, we i
168  Rhes alleviates motor deficits and improves brain pathology in HD mice.
169  between MCI+AD and LBD or detecting amyloid brain pathology in LBD.
170 aphy (PET) have enabled anatomic tracking of brain pathology in longitudinal studies of normal aging
171 artments, developmental stages, and types of brain pathology in mice.
172  to the generation of recurrent seizures and brain pathology in patients with medically intractable m
173 function associated with Alzheimer's disease brain pathology in terms of functional network organizat
174 1 gene rescued motor function and alleviated brain pathology in the MLIV mouse model.
175 te specific cognitive abnormalities to focal brain pathology in the presence of widespread disease su
176 to contribute directly to multiple sclerosis brain pathology in the vast majority of cases.
177                This study reports on a novel brain pathology in young patients with frontal lobe epil
178                                              Brain pathologies included AD pathology measured by silv
179 reases with age and is a robust predictor of brain pathology including Alzheimer's and other dementia
180 t signaling has been associated with several brain pathologies, including Alzheimer's disease.
181 of nitric oxide, is expressed in a number of brain pathologies, including cerebral ischemia.
182 brain edema are hallmark features of several brain pathologies, including traumatic brain injuries (T
183 in midlife might increase risk for late-life brain pathology, including dementia.
184                                              Brain pathology indices obtained by postmortem pathologi
185                               However, since brain pathology induces astrocytes to become reactive, i
186 cans were also performed in 51 patients with brain pathologies involving the blood-brain barrier, nam
187                                              Brain pathology is a poorly understood systemic manifest
188                                      Similar brain pathology is also seen in a familial FTD pedigree
189 acid (GABA)-A receptor during progression of brain pathology is challenging because of the lack of im
190                                       The AD brain pathology is characterized by deposits of amyloid-
191         These findings provide evidence that brain pathology is functionally associated with persiste
192                               Although gross brain pathology is not characteristic of autism, subtle
193 he role of locally produced antibodies in MS brain pathology is poorly understood.
194 l head impact and the pattern of progressive brain pathology is poorly understood.
195 global Alzheimer disease pathology, vascular brain pathologies, Lewy body, hippocampal sclerosis, and
196  infection, with recent work suggesting that brain pathology may be driven by immune responses.
197                                              Brain pathology measures included beta-amyloid load, pho
198 re birth is a major risk factor for multiple brain pathologies, notably periventricular leukomalacia
199 neural development, but when, where, and how brain pathology occurs remain elusive.
200 ctors can affect these measurements, such as brain pathologies, ocular axial length (AL) as well as a
201                           The 2 hallmarks of brain pathologies of prematurity are a restricted gestat
202 olfactory epithelium correlating well to the brain pathology of AD patients.
203  neurodegenerative disorder characterized by brain pathology of intracellular neurofibrillary tangles
204 evidence for the involvement of microglia in brain pathology of mouse models of the two diseases.
205                    In addition, the hallmark brain pathology of TSC, cortical tubers and giant cells
206     Many subjects with LATE-NC have comorbid brain pathologies, often including amyloid-beta plaques
207  cognition mediated the effects of different brain pathologies on neuropsychiatric symptoms.
208 actor that modifies or buffers the impact of brain pathology on cognitive function.
209 nxiety or depression stem from an underlying brain pathology or as a consequence of living with an un
210             However, we found no evidence of brain pathology or impacts on motor function in aged Csf
211 ever, L-DOPS treatment did not arrest global brain pathology or improve somatic growth, as gene thera
212 n decline in aging and enhances tolerance to brain pathology or that it does not affect cognitive dec
213 isplatin, GENUS alleviated cisplatin-induced brain pathology, promoted oligodendrocyte survival, and
214 cal roles in neurons and its contribution to brain pathology remain largely elusive.
215 etween an acute stroke event and preexisting brain pathology remains a priority and will be critical
216 y syndrome (AIDS) dementia complex (ADC) and brain pathology remains uncertain.
217 ces can be revealed by the fact that certain brain pathologies render inhibitory processing ineffecti
218                    Regenerative processes in brain pathologies require the production of distinct neu
219  Tourette's syndrome, the absence of evident brain pathology resulted in vacillation between organic
220                 Examination of patients with brain pathologies revealed that slow tracer accumulation
221                                     Distinct brain pathologies seem associated with discrete microgli
222 of cervical spinal cord lesions and atrophy, brain pathology seems more strongly related to physical
223 istic linkage between A-to-I RNA editing and brain pathologies should be revisited.
224 etween IDO expression, IFN-gamma levels, and brain pathology signs observed in neuro-AIDS.
225 xpand proteins away from each other in human brain pathology specimens, including formalin-fixed para
226 in Alzheimer's disease is a manifestation of brain pathologies such as accumulation of amyloid-beta p
227 tion is an important pathogenetic factor for brain pathologies such as Parkinson's disease, ADHD, sch
228               Drug targeting to inflammatory brain pathologies such as stroke and traumatic brain inj
229 ated in migraine with aura and various other brain pathologies, such as ischemic stroke and traumatic
230 s accelerate astroglial activation and other brain pathologies, suggesting that daily functions in as
231 Tfpi-/- embryos completely ameliorated their brain pathology, suggesting that TFPI dampens FV-depende
232 tected animals against viral replication and brain pathology, suggesting that vaccination against HSV
233  and the influence of aging and co-occurring brain pathologies that commonly arise in the elderly.
234                          Given the number of brain pathologies that involve ferroptosis, defects in t
235  a previously unappreciated component of the brain pathologies that occur as a result of apolipoprote
236 associated with motor decline unexplained by brain pathologies that provide motor resilience.
237 users show early Alzheimer's disease-related brain pathology that may be the basis for cognitive impa
238 uman patients and Col4a1 mutant mice display brain pathology that typifies cerebral small vessel dise
239  wakefulness, and specific patterns of local brain pathology-thereby possibly providing a mechanistic
240 "DC8" and "DC13" have been proposed to cause brain pathology through interactions with endothelial pr
241 y are frequently used in patients with acute brain pathologies to facilitate sedation or surgical and
242 n eye on how tripartite synapses figure into brain pathologies underlying behavioral impairments in p
243  results suggest that TDP-43 is an important brain pathology underlying cognitive decline and dementi
244 ship between blood lipids and 12 age-related brain pathologies using a linear mixed model adjusted fo
245 cy of these results with previously reported brain pathology validates the use of either technique, a
246                                              Brain pathology was ascertained with standardized method
247 ical scrapie, but it was unclear whether the brain pathology was caused by damage to astrocytes, neur
248                            No improvement in brain pathology was observed.
249 ctive effects of fingolimod on twitcher mice brain pathology was reflected by an increased life span
250 YT1 knock-in mouse model to test efficacy on brain pathologies, we found that ritonavir restored mult
251 d substantially when common dementia-related brain pathologies were included in the multivariable-adj
252 or-beta/Smad2/3 activation and hydrocephalic brain pathology were evaluated at Day 14 and the inflamm
253              Memory deficits and majority of brain pathology were rescued in mice receiving HGSNAT ch
254                   Neurologic examination and brain pathology were scored by blinded observers.
255           Notably, neurological function and brain pathology were significantly improved in MPS I mic
256 uture perspectives for the study of aOPCs in brain pathologies which may instigate the development of
257 yte state that increased in association with brain pathology, which we termed disease-associated olig
258                                              Brain pathology with extensive microglial infiltration,

 
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