ライフサイエンスコーパス: brain tissue

1 on the rigidity and the permeability of the brain tissue.

2 orms a gel immediately upon contact with the brain tissue.

3 space between an impermeable artery and the brain tissue.

4 in the size of glycolytic larval muscle and brain tissue.

5 vailable RNA expression data from postmortem brain tissue.

6 studies are demonstrating aluminium in human brain tissue.

7 s from corticobasal degeneration (CBD) human brain tissue.

8 CS also modulates the substance transport in brain tissue.

9 d to associate with mechanical trauma to the brain tissue.

10 se factors are frequently dysregulated in MS brain tissue.

11 d higher C4A transcript levels in postmortem brain tissue.

12 ce of ABCA1 and elevated abundance of APP in brain tissue.

13 GL activity and elevation of 2-AG content in brain tissue.

14 ls, receptors, and transporters expressed in brain tissue.

15 for the delivery of bioactive molecules into brain tissue.

16 ese cells also interact with the surrounding brain tissue.

17 cell culture, chick embryos and adult mouse brain tissue.

18 nging or impossible, for example in heart or brain tissue.

19 rimary B cells and monocytes, thymus, and MS brain tissue.

20 association of CHRNA2 expression with CUD in brain tissue.

21 sample preparation was optimized using mouse brain tissue.

22 o measure Abeta turnover in blood and within brain tissue.

23 ng changes in a wide range of metabolites in brain tissue.

24 ication of intact ubiquitin in rat and mouse brain tissue.

25 mate 5 (mGlu(5)) receptor activity in intact brain tissue.

26 -regulation in both the mouse astrocytes and brain tissue.

27 tch between the artificial interface and the brain tissue.

28 technology in confining light through mouse brain tissue.

29 antifying synaptic protein or mRNA levels in brain tissue.

30 s (>5,600 unique N-glycopeptides) from mouse brain tissue.

31 rocytes seal focal injuries off from healthy brain tissue.

32 uring neurodifferentiation, and in liver and brain tissue.

33 rion disease that is causing rapid damage of brain tissue.

34 ls of extracellular ascorbic acid present in brain tissue.

35 l polarity, and normal dispersion within the brain tissue.

36 to study due to limited access to functional brain tissue.

37 e assessment of deconvolution algorithms for brain tissue.

38 phy of parvalbumin expression in post-mortem brain tissue.

39 of iPSC-derived neuronal cultures and human brain tissue.

40 represent the extracellular matrix (ECM) of brain tissue.

41 tasis and regulates solute permeability into brain tissue.

42 if there is imaging evidence of salvageable brain tissue.

43 are tissue-specific, especially enriched in brain tissues.

44 onal changes in queen abdominal fat body and brain tissues.

45 s of intact, interweaving neuron clusters in brain tissues.

46 dentical to that of the ribosomes from the 3 brain tissues.

47 th of approximately 2 mm in glycerol-cleared brain tissues.

48 inct cell populations within patient-derived brain tissues.

49 cell types with a focus on neurogenesis and brain tissues.

50 ensory retina as a unique window into deeper brain tissues.

51 nces was isolated from the infected lung and brain tissues.

52 nd are more complex than the patterns in non-brain tissues.

53 scores and enriched risk gene expression in brain tissues; (2) Atypical MPDs (40% of MPDs) had decre

54 so show that base editing corrects, in mouse brain tissue, a mutation that causes Niemann-Pick diseas

55 With brain tissue, a pixel size of 600 nm was achieved.

56 ng chromatin interaction profiles from human brain tissue across two developmental epochs and two bra

57 t detectable amount of naloxone was found in brain tissue after naloxone-methiodide administration, p

58 using phosphorylated Cytc purified from pig brain tissue and a recombinant phosphomimetic mutant (S4

59 s protein, an index of neuronal activity, in brain tissue and benchmarked it against two widely-used

60 (NMR) metabolomics measurements were made in brain tissue and blood.

61 Comparisons of brain tissue and cerebrospinal fluid proteomes reveal bi

62 xine was shown to detect aggregated Abeta in brain tissue and cerebrospinal fluid samples of AD mice.

63 a novel, automated tool to quantify cells in brain tissue and complements current, open-sourced metho

64 the inaccessibility and complexity of human brain tissue and consequent lack of insight into disease

65 els are seen in ApoE4(+/+) mouse hippocampal brain tissue and cultured neurons when compared to ApoE3

66 xerted by a growing tumor on the surrounding brain tissue and delineates the physical versus biologic

67 to enable quick capture of SVs directly from brain tissue and determined the neurotransmitter profile

68 Immunohistochemical analysis of brain tissue and dura revealed reactive gliosis in the c

69 Human postmortem brain tissue and fibroblasts from subjects with DS and h

70 expression in response to virus infection in brain tissue and in cells from patients with STING-assoc

71 age of the cell adhesion molecule L1 (L1) in brain tissue and in cultured cerebellar neurons results

72 HFOs are a proposed biomarker of epileptic brain tissue and may also be useful for seizure forecast

73 th enrichment for enhancers found in primary brain tissue and neuronal progenitor cells.

74 markers, we time-dependently evaluated mouse brain tissue and neuronally derived plasma extracellular

75 pplicability, this assay was used to analyze brain tissue and study the transport of TAT-MeCP2 varian

76 irected mAbs showed far less avid binding to brain tissue and were consistently detected in the serum

77 as somatic mosaicism at ~15.1% in dysplastic brain tissue and ~11% in blood, and the MTOR p.S2215F va

78 enome architecture in human neurogenesis and brain tissues and also demonstrated that a rigorous Hi-C

79 rmatic analyses, based on gene expression in brain tissues and cells, showed that genes differentiall

80 nt LMTK2 immunohistochemistry on post-mortem brain tissues and compared them to age-matched controls

81 ormance was assessed using murine kidney and brain tissues and demonstrate that high-spatial-resoluti

82 lls to amyloid-beta plaques and dendrites in brain tissues and elastic fibers in developing cartilage

83 Na+/K+-ATPase, which is highly expressed in brain tissues and has previously been related to familia

84 ic profiles of SLC30A9 and BEND4 in multiple brain tissues and was previously associated with depress

85 mg/kg; at which naloxone was undetectable in brain tissue) and found that this drug still powerfully

86 ting preadipocytes in vitro, (2) fresh mouse brain tissue, and (3) human frozen adipose tissue (AT) f

87 re, we purified an INF2 inhibitor from mouse brain tissue, and identified it as a complex of lysine-a

88 We also analysed matched CSF, post-mortem brain tissue, and iPSCs from the same participant with t

89 uman glioma tissues, when compared to normal brain tissue, and that the extent of upregulation positi

90 between rs9315202 and advanced epigenetic to brain tissue, and to identify the effects of rs9315202 o

91 analyses of implicated genes and proteins in brain tissues, and tests of the effects of risk variants

92 e 3D organizations of tumor spheroids, mouse brain tissues, and tumor xenografts.

93 nvasive glioma cells infiltrated into normal brain tissue are responsible for tumor recurrence.

94 l consequences of these physical stresses on brain tissue are unknown.

95 aging (lymph nodes, vasculature, tumors, and brain tissue) are discussed in detail.

96 giomas (MENs), which contain less water than brain tissue, are characterized by fluid-like behavior.

97 rted results for the herniated volume of the brain tissue as a function of the intracranial pressure

98 n particular, histology can be used to study brain tissue at the microscopic level, using a wide arra

99 h the same direction of effect in the second brain tissue (BA25, N = 60) from the same individuals an

100 ia grown on ECM coatings from decellularized brain tissue (bECM) or MaxGel, a non-tissue-specific ECM

101 method to measure mitochondrial function in brain tissue biopsy punches excised from anatomically de

102 llion mass resolution for lipid imaging from brain tissue, but explicitly show such mass resolution i

103 215F variant, detected as ~8.8% mosaicism in brain tissue, but not in blood.

104 The compression of brain tissue by a tumour mass is believed to be a major

105 e apply existing deconvolution algorithms to brain tissue by using marker sets derived from human bra

106 romelanin and associated metal ions in human brain tissue can be achieved using synchrotron scanning

107 ery, release of the nanocluster payload into brain tissue can be triggered by a second focused ultras

108 ypothesized that the scRNA-seq data of mouse brain tissue can be used to complete human data to infer

109 ic scale of transcriptomic analyses of human brain tissue can provide an unbiased approach for identi

110 ttern, exert solid stress on the surrounding brain tissue, causing a decrease in local vascular perfu

111 nnotations across all (respectively blood or brain) tissues/cell-types in meta-analyses across all (r

112 he work protocol was repeated, and blood and brain tissue collected.

113 ethods are used for obtaining and processing brain tissue coming from humans and experimental animals

114 gyrus [MFG]), we tested associations between brain tissue concentrations of 26 metabolites and the fo

115 Levels of APP and Tau in brain tissue correlated with the abundance of Nef.

116 kers which can accurately indicate damage to brain tissue could yield molecular diagnostics with the

117 he effective solute diffusion coefficient of brain tissue (D(eff)) was determined from the spatio-tem

118 signal intensity on T1-weighted MRI reflect brain tissue damage that may develop within newly formed

119 at greater risk of mechanical damage during brain tissue deformation from traumatic brain injury.

120 munoperoxidase analysis of macaque and human brain tissue demonstrate a conserved pattern for PI5P4Ka

121 Images of rat and mouse brain tissue demonstrate spatial variation in the concen

122 Quantitative assessment of brain tissue demonstrated that spaceflight caused an up

123 es extracted from cell cultures and human AD brain tissues, demonstrating the ability of the Hsp70 ma

124 Particularly, fetal brain tissue-derived ECM supported long-term maintenance

125 ce risk genes are predominantly expressed in brain tissues, especially prenatally, and are enriched f

126 04-scFv antibodies to inhibit the seeding by brain tissue extracts from different donors with tauopat

127 target protein in raw plasma samples and in brain tissue extracts from healthy individuals and post

128 P concentration in murine liver, kidney, and brain tissues, finding that it ranges from 0.4 mumol.kg(

129 protecting the downstream capillary bed and brain tissue from adverse pressure fluctuations.

130 erns of molecular convergence in post-mortem brain tissue from autistic subjects.

131 cies associated with amyloid polymorphism in brain tissue from individuals with sporadic AD (s-AD) an

132 ypermethylated in blood and downregulated in brain tissue from individuals with WS.

133 ental disease there are few similar data for brain tissue from non-neurologically impaired donors.

134 , blood-derived macrophages, whole blood and brain tissue from patients with C9-ALS/FTD all show an e

135 duals with an antemortem MRI and post-mortem brain tissue from the Mayo Clinic Alzheimer's Disease Re

136 Immunohistochemical analysis of brain tissue from two CYLD p.M719V mutation carriers sho

137 tal histopathological approach, in available brain tissue from up to 8 cortical regions bilaterally.

138 Analysis of brain tissues from GBM patients shows that lipid droplet

139 oradiographic studies using postmortem human brain tissues from healthy individuals and ALS patients,

140 We examined postmortem brain tissues from patients with sporadic deep ICH.

141 n of the TS and focal arteriolar SMC loss in brain tissues from patients with sporadic deep ICH.

142 Through mouse brain tissue G4-RNA immunoprecipitation, we further conf

143 ines, peripheral blood mononuclear cells and brain tissue, generating 36 libraries in six separate ex

144 itivity mass spectrometric quantification of brain tissue glutamine.

145 ble CA inhibitor that is unable to reach the brain tissue, had no effect.

146 e active, but finding the active synapses in brain tissue has been a technical challenge.

147 Prolonged persistence in brain tissue has implications for otherwise resolved hum

148 ery of magnetic nanoparticles (NPs) in human brain tissue has raised concerns regarding their source

149 ariants affecting SLC1A1 expression in human brain tissue have been associated with OCD.

150 Brain tissue heating by continuous three-photon imaging

151 position of ribosomes from 3 different mouse brain tissues, i.e., hippocampus, cortex, and cerebellum

152 33.90 +/- 9.06% of injected dose per gram of brain tissue (%ID/g) in the cortex and 17.09 +/- 7.22%ID

153 ch reflects the degree of neuronal damage in brain tissue in a CJD subtype manner.

154 and decellularized extracellular matrix from brain tissue in a compartmentalized cancer-stroma concen

155 us conclusions that the aluminium content of brain tissue in Alzheimer's disease, autism spectrum dis

156 oth quantified and imaged aluminium in human brain tissue in neurodegenerative and neurodevelopmental

157 enhances virus entry in cells as well as in brain tissue in vivo.

158 ed to study proteomes from heart, liver, and brain tissues in a late-stage amyloid precursor protein/

159 ause more severe disease and to replicate in brain tissues in animal models as well as their ability

160 and mortality, with an extended tropism for brain tissues in both ferret and mouse models.

161 In all 3 brain tissues, in both monosomal and polysomal ribosome

162 was colocalized with AADC transcript in rat brain tissues, in contrast to rat peripheral tissues whe

163 cribes the biomechanical response of healthy brain tissue-in terms of changes in ICP and edema-to a g

164 ingly, despite severe vascular anomalies and brain tissue infarction, the blood-brain barrier was mai

165 However, brain tissue integrity in cognition, anxiety, and depres

166 accurate estimates of non-linear changes in brain tissue intensity and shape with clear potential fo

167 Using MRI, we simultaneously estimated brain tissue intensity on T1- and T2-weighted scans as w

168 that involves: (i) a method for slicing rat brain tissue into sections with the same thickness and s

169 Brain tissue iron accumulates with age and co-localises

170 found QSM increases (consistent with higher brain tissue iron content) in PD compared with controls

171 sures and QSM, an MRI technique sensitive to brain tissue iron content.

172 ring late adolescence.SIGNIFICANCE STATEMENT Brain tissue iron is essential to healthy brain function

173 Brain tissue iron, measured using QSM, can track cogniti

174 n of volume electron microscopy data sets of brain tissue is challenging but can reveal invaluable in

175 d peristaltic deformation and the deformable brain tissue is modeled by means of an elastic Hooke's l

176 the structure of Abeta amyloid fibrils from brain tissue is poorly understood.

177 In neuroscience, the rarity of brain tissue is the bottleneck for such study.

178 Complete surgical resection of abnormal brain tissue is the most important predictor of seizure

179 mples, but their performance when applied to brain tissue is unclear.

180 lays (up to 5 hr) to mimic the way the human brain tissue is usually processed.

181 erwhelming brightness and slow expression in brain tissue limits the fidelity of dendritic spine anal

182 ndividuals with AUD demonstrate accelerating brain tissue loss with advanced age.

183 in multilayer gel arrangements and surrogate brain tissue models.

184 region is demonstrated on HeLa cells and rat brain tissue, monitoring molecules that were previously

185 istal middle cerebral artery occlusion mouse brain tissue more accurately than (18)F-GE-180 and appea

186 ls from both blood (N = 1132) and postmortem brain tissues (N = 61 samples from Brodmann Area 10, BA1

187 In brain tissue, neurogranin was detected in the cytoplasm,

188 (STED), to characterize in postmortem human brain tissue non-fibrillar Abeta structures in amyloid p

189 d DNA extracted from cerebrospinal fluid and brain tissue now offers another strategy for diagnosing

190 ophysiological recordings performed on fetal brain tissue obtained immediately following MRI demonstr

191 (IHC) dataset for five major cell-types from brain tissue of 70 individuals, who also have bulk corti

192 sibility and safety of FUS-LBx in the normal brain tissue of a porcine model.

193 of miR-195 are significantly lower in human brain tissue of ApoE4(+/-) patients with clinical diagno

194 oton microscopy imaged the spread across rat brain tissue of fluorescently-labeled solutes injected t

195 The abundance of ABCA1 in brain tissue of HIV-infected human subjects diagnosed wi

196 ntracellular inclusions in human post-mortem brain tissue of Lewy body disease cases, but not aged co

197 prion seeding activity was also detected in brain tissue of ovine PrP mice inoculated with limiting

198 ine model of demyelination and in postmortem brain tissue of patients with AD.

199 We isolated microglia from post-mortem brain tissue of patients with MDD (n = 13-19) and contro

200 m neurons and oligodendrocytes obtained from brain tissue of patients with schizophrenia and matched

201 LRP4 was reduced in postmortem brain tissues of AD patients.

202 sed gene sets extracted from either liver or brain tissues of BXD recombinant inbred mice in the Gene

203 sing well-characterized BDTOs, isolated from brain tissues of different tauopathies, including Alzhei

204 DNAm levels of target genes in brain tissues of the offspring were determined by pyrose

205 s as well as in mitochondria from postmortem brain tissues of unaffected individuals and HD patients,

206 o used mitochondria isolated from postmortem brain tissues of unaffected individuals and HD patients.

207 ellular localization of MSI1 and MSI2 in the brains tissues of Alzheimer's disease (AD), amyotrophic

208 e lack of cell-specific targeting methods in brain tissue or living animals.

209 ables longitudinal imaging of the compressed brain tissue over several weeks or months as the screw i

210 We hypothesized that in postmortem brain tissue, OXTR binding in the hippocampal formation

211 tension less than 20 mm Hg versus normoxia (brain tissue oxygen tension > 20 mm Hg).

212 xygen tension, jugular venous bulb oxygen to brain tissue oxygen tension gradient, and cerebral perfu

213 ith brain hypoxia, as defined by parenchymal brain tissue oxygen tension less than 20 mm Hg versus no

214 In the hypoxia group, the mean brain tissue oxygen tension, jugular venous bulb oxygen

215 In the normoxia group, the mean brain tissue oxygen tension, jugular venous bulb oxygen

216 Hg), and the mean percentage of time with a brain tissue oxygenation below 20 mm Hg was 38% (6-100%)

217 e approached optimal mean arterial pressure, brain tissue oxygenation increased (p < 0.001).

218 The mean brain tissue oxygenation was 23 mm Hg (SD 8 mm Hg), and

219 p between optimal mean arterial pressure and brain tissue oxygenation.

220 (11)C-PiB signal from blood (P = 0.005) and brain tissue (P = 0.004) to the lateral ventricles and s

221 nd inflammation in cerebral microvessels and brain tissue paralleling hyperglycemia in mice of both D

222 To date, the brain tissue penetrance of intranasal oxytocin has not b

223 use cerebral vasoconstriction and compromise brain tissue perfusion.Objectives: To determine if the m

224 We present a tunable 3D bioengineered brain tissue platform by integrating microenvironmental

225 racterized by rapid cellular infiltration of brain tissue, raising the possibility that disease progr

226 and [(18)F]28 provided high tumor to normal brain tissue ratios, with maximal ratios of 3.5 and 4.1,

227 usly administered HN penetrated perihematoma brain tissue, reduced neurological deficits, and improve

228 sequencing of gliomas, different cell types, brain tissue regions and advances in bioinformatics algo

229 bled the detection of 30 proteins from mouse brain tissue, rising to 44 when quadrupole isolation was

230 ensitivity for Gln, thus enabling down-sized brain tissue sample volume procurement for quantificatio

231 cible quantifications of 80 small GTPases in brain tissue samples from 15 patients.

232 Additionally, the relatively small number of brain tissue samples may have limited our power to detec

233 fections, viral sequences were analyzed from brain tissue samples of a single SSPE case and compared

234 nt protocol is demonstrated to work on human brain tissue samples, a source that has proven to be dif

235 ection by shotgun analyses, crosschecking in brain tissue samples, and verification by targeted prote

236 ents that previously went undetected in bulk brain tissue samples.

237 bpopulations can also be estimated from bulk brain tissue samples.

238 ing Escherichia coli lipid extracts on mouse brain tissue sections and imaged by MOLI MSI.

239 Bacterial peptidoglycan was demonstrated in brain tissue sections from several MS subjects.

240 nal cultures, transfected Neuro2a cells, and brain tissue sections Nrg3 protein was localized to the

241 wer expression of the GluN2B subunits in ALS brain tissue sections than in healthy controls, in line

242 and axon tract orientation in prenatal human brain tissue sections that are not visible using brightf

243 ification of diverse classes of compounds in brain tissue sections, whereas simultaneously mapping th

244 ll as binding to both human and mouse BBB in brain tissue sections.

245 t types of samples, bacteria cocultures, and brain tissue sections.

246 ctive serotonin reuptake inhibitor, in mouse brain tissue sections.

247 iting LTP and is upregulated several-fold in brain tissue, serum, and CSF of Alzheimer's disease (AD)

248 Immunostaining of the patient's IgG on mouse brain tissue showed sparse but distinctive points of sta

249 yses suggest potentially causal genes in non-brain tissues, showing the utility of these tissues for

250 cerol lipase (MAGL) expression in postmortem brain tissue, such that regions with higher MAGL express

251 al or verbal prompts to identify and protect brain tissue supporting essential functions such as lang

252 brain as well as the herniated volume of the brain tissue that exits the skull following craniectomy.

253 gation of a relatively slow wave in cortical brain tissue that is linked to a number of pathological

254 atomists ascertain the fine-scale pattern of brain tissue, the 'texture' formed by cellular organizat

255 Importantly, Me(2)SPM was detectable in brain tissue, the organ most affected in SRS, and was as

256 t is often difficult to access primary human brain tissue; therefore, approximations are made using a

257 globin is orders of magnitude higher than in brain tissue, though how this might impact NO signaling

258 , and the rerouting of blood to hypoperfused brain tissue through collateral anastomoses.

259 that provides three-dimensional coverage of brain tissue through multisite multifunctional fiber-bas

260 integrated human proteome data derived from brain tissue to evaluate whether targeted proteins putat

261 This study utilizes postmortem human brain tissue to investigate whether Purkinje cell degene

262 screw is progressively extended against the brain tissue to recapitulate tumor growth-induced solid

263 he newly developed co-localization score for brain tissues to score de novo mutations in whole genome

264 Optical clearing of brain tissue using hydrogel-based methods (e.g. CLARITY)

265 gle nanotube imaging can be achieved in live brain tissue using ultralow excitation doses (~0.1 kW/cm

266 holine (PC) lipid cations generated from rat brain tissue via matrix-assisted laser desorption/ioniza

267 sicles and had improved survival and reduced brain tissue virus levels.

268 ignificant correlations between the resected brain tissue volume and the ganglion cell layer region o

269 ogether, the present study demonstrates that brain tissue volume loss in schizophrenia is conditioned

270 l discrimination with structural measures of brain tissue volume was investigated using magnetic reso

271 RI markers of cerebral small vessel disease, brain tissue volumes, and white matter microstructural i

272 rs of cerebral small vessel disease, smaller brain tissue volumes, and worse white matter microstruct

273 The uptake of 1-L-[(18)F]FETrp in the normal brain tissue was low, suggesting that 1-L-[(18)F]FETrp m

274 C-methionine uptake above that of uninvolved brain tissue was observed in 18 of 22 baseline scans (82

275 Diagnostic assessment of surrounding brain tissue was significantly improved in VMI(>=100keV)

276 nstrated by a high fungal burden in lung and brain tissues, was associated with mouse mortality.

277 In white matter lesions from MS brain tissue, we noted the presence of bile acid recepto

278 ation analysis of DNA methylation from human brain tissues, we identified a differentially methylated

279 e the three-dimensional (3D) architecture of brain tissue.We employed human induced pluripotent stem

280 arcodes of the different biofluids and human brain tissue were acquired and biofluid- and tissue-asso

281 ohort wherein whole body autopsy reports and brain tissue were available for interrogation was establ

282 and accumbens nucleus (NAC)) from postmortem brain tissue were compared between 6 OCD and 8 control c

283 hromatin regions identified in human primary brain tissue were observed in organoids at different dev

284 cyte and microglia, and bacterial studies on brain tissue were used to characterize neurologic lesion

285 rning from the ISS, mice were euthanized and brain tissues were collected for analysis.

286 Gene expression profiles of brain tissues were extracted from 14 publicly available

287 nd functional dynamics of neural circuits in brain tissue, which is highly scattering to light.

288 MDD employed bulk homogenates of postmortem brain tissue, which obscures gene expression changes wit

289 the D2 receptor and tyrosine hydroxylase in brain tissue, while markers of neurodegeneration and neu

290 in pathways and deliver drug directly to the brain tissue whilst limiting systemic exposure.

291 nohistochemistry, and ex vivo stimulation of brain tissue with insulin, we assessed insulin signaling

292 intraoperative feedback to identify abnormal brain tissue with millimeter spatial resolution.

293 optical voltage recording with our probes in brain tissue with one-photon and two-photon fluorescence

294 We observed that aging brain tissues with APP had protective effects on ZIKV in

295 complimentary, information about underlying brain tissues - with each metric offering unique sensiti

296 Plasminogen protein was higher in MCI and AD brain tissue, with plasminogen mRNA not likewise elevate

297 te ([(13)C(5)]-5-formylTHF) concentration in brain tissues, with levels comparable to wild-type anima

298 urized neural interfaces that integrate into brain tissue without altering its properties.

299 n tumor tissues while undetectable in normal brain tissues, yet the identity and functions of lipid s

300 Furthermore, in human microcephalic fetal brain tissue, ZikV-NS5 persists at the base of the motil