コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 factors, such as the phytohormones auxin and brassinosteroid.
2 auxin, strigolactone, gibberellic acid, and brassinosteroids.
3 of responses to the plant hormones auxin and brassinosteroids.
4 5, two defective transmembrane receptors for brassinosteroids.
5 and in alterations in response to auxin and brassinosteroids.
6 ptional pathway and can act independently of brassinosteroids.
7 NSTITUTIVE PHOTOMORPHOGENIC DWARF, DWF4, and BRASSINOSTEROID-6-OXIDASE 2 Phenotypic characterization
8 to three brassinosteroid-biosynthesis genes (BRASSINOSTEROID-6-OXIDASE, CONSTITUTIVE PHOTOMORPHOGENIC
13 uxin affected the expression of a few genes; brassinosteroid and gibberellin had only modest effects.
14 tly, TT8 affects stress response, along with brassinosteroid and jasmonic acid biosynthesis, by direc
15 mutant displayed reduced responses to GA and brassinosteroid and showed decreased expression of sever
17 alyses also suggested an induction of auxin, brassinosteroid, and gibberellin signatures and the invo
18 growth was found to be independent of auxin, brassinosteroid, and reactive oxygen species signaling p
20 ontrolled temporally and spatially by auxin, brassinosteroids, and light to result in AM initiation o
27 fied a R2R3-MYB transcription factor, BRAVO (BRASSINOSTEROIDS AT VASCULAR AND ORGANIZING CENTER), act
28 ants, steroid hormone action occurs when the brassinosteroids bind a membrane tyrosine kinase recepto
29 id, carotenoid, oxylipin, glucosinolate, and brassinosteroid biosyntheses and have shown that both P4
30 ng sturdy culm, specific for deficiencies in brassinosteroid biosynthesis and signaling in semidwarf
33 igned more than 20 historic mutants to three brassinosteroid-biosynthesis genes (BRASSINOSTEROID-6-OX
34 e ubiquitous in land plants, suggesting that brassinosteroid biosynthetic pathways differ between ear
35 studies have demonstrated that cytokinin and brassinosteroid (BR) act as regulatory inputs to elevate
36 to investigate the relationship between the brassinosteroid (BR) and gibberellin (GA) hormones acros
37 A paradigmatic case is the concurrence of brassinosteroid (BR) and gibberellin (GA) signaling in t
40 e show here that noncoding variations of the brassinosteroid (BR) biosynthesis gene DWARF1 (DWF1) lea
44 coding sterol C-22 hydroxylases that control brassinosteroid (BR) hormone levels using a promoter tha
48 BES1 functions as a master regulator in the brassinosteroid (BR) pathway that promotes plant growth.
49 onic acid (JA) pathway was induced while the brassinosteroid (BR) pathway was suppressed in infected
50 In Arabidopsis (Arabidopsis thaliana), the brassinosteroid (BR) receptor BR INSENSITIVE1 (BRI1) und
51 nase that functions as a coreceptor with the brassinosteroid (BR) receptor BRI1 and the flagellin rec
52 urally defective yet biochemically competent brassinosteroid (BR) receptor BRI1, resulted in identifi
53 urally imperfect yet biochemically competent brassinosteroid (BR) receptor bri1-9, causing its increa
57 nome-wide screen for mutants showing altered brassinosteroid (BR) sensitivity or bri1-like phenotypes
60 s GSK3-like kinase that negatively regulates brassinosteroid (BR) signaling by phosphorylating BES1 (
68 s in plant immunity, cell-death control, and brassinosteroid (BR) signaling, all four functional SERK
69 NSITIVE2 (BIN2), a key negative regulator of brassinosteroid (BR) signaling, can phosphorylate Arabid
70 e pattern; instead, the detailed analysis of brassinosteroid (BR) signaling, innate immunity, and sym
71 PRs of either the negative regulators of the brassinosteroid (BR) signaling, the glycogen synthase ki
77 we show that the activity of two homologous brassinosteroid (BR) transcriptional effectors, BRASSINA
78 enous signals, including light, temperature, brassinosteroid (BR), and gibberellin (GA), regulate cel
79 monal signals, involving light, temperature, brassinosteroid (BR), and gibberellin, that inhibit the
81 nstrating the role of BRAVO in counteracting Brassinosteroid (BR)-mediated cell division in the QC ce
82 pattern (PAMP)-triggered immunity (PTI) and brassinosteroid (BR)-mediated growth was recently report
84 through TOR controls the accumulation of the brassinosteroid (BR)-signaling transcription factor BZR1
87 uxin, gibberellic acid (GA), cytokinin (CK), brassinosteroids (BR) and peptide hormones are also impl
90 well as intrinsic signals such as hormones (brassinosteroid [BR], auxin, cytokinin, ethylene) and nu
91 es have indicated that CYP734A1 binds active brassinosteroids, brassinolide and castasterone, as well
105 strong evidence that steroid hormones called brassinosteroids (BRs) are required to maintain position
110 ) is the main ligand-perceiving receptor for brassinosteroids (BRs) in Arabidopsis (Arabidopsis thali
111 to investigate a role for the phytohormones brassinosteroids (BRs) in specifying bristle identity an
126 at are coordinated by hormones including the brassinosteroids (BRs), a group of steroids with structu
129 terplay of light and plant hormones, such as brassinosteroids (BRs), in the regulation of plant growt
136 3 double mutant roots are insensitive toward brassinosteroids but have a phenotype different from bri
137 the largely differing response to zeatin and brassinosteroid by the metabolic pathways in chloroplast
140 nt defense and growth such as jasmonic acid, brassinosteroids, cytokinins, auxin and synthesis of fla
141 ctivities of downstream MAPKs are reduced in brassinosteroid-deficient mutants but increased by treat
144 iption and integrate the auxin response to a brassinosteroid-dependent molecular circuit that promote
145 nthesis of structural sterols from signaling brassinosteroid derivatives and are highly regulated.
146 A15 (SAUR15), a well-characterized auxin and brassinosteroid early response gene in Arabidopsis (Arab
147 the mechanism by which another phytohormone, brassinosteroid, elevates ethylene biosynthesis in etiol
148 Overexpression of the HBI1-related bHLHs brassinosteroid enhanced expression2 (BEE2) and cryptoch
149 helix (bHLH) transcription factor homolog of brassinosteroid enhanced expression2 interacting with IB
150 binding of either transcription factor, and brassinosteroid enhancement of MP/ARF5 binding required
151 tive LRR-RK leads to strong gain-of-function brassinosteroid, floral abscission, and stomatal pattern
152 S lines revealed downregulation of auxin and brassinosteroid genes, and upregulation of cytokinin tra
153 growth-regulating signals, including auxin, brassinosteroid, gibberellin, light, and temperature.
155 c activities shows that the enzyme regulates brassinosteroid homeostasis via mono- and diacetylation
156 tential to fine-tune the levels of different brassinosteroid hormones throughout plant growth and dev
158 , abscisic acid (ABA), cytokinins (CKs), and brassinosteroids in molding plant-pathogen interactions.
159 and CYP72C1 in Arabidopsis diverge more than brassinosteroid inactivating P450s in other plants, this
160 YP734A1 and CYP72C1, have been identified as brassinosteroid-inactivating enzymes important for stero
161 rassinosteroid phytohormones and enzymes for brassinosteroid inactivation are present only in spermat
163 ls to the hypocotyl, where it triggers local brassinosteroid-induced cell elongation in seedling stem
165 Recently, two Arabidopsis receptor kinases--BRASSINOSTEROID INSENSITIVE 1 (BRI1) and BRI1-ASSOCIATED
168 ity of the dual-specificity receptor kinase, brassinosteroid insensitive 1 (BRI1), reflects the balan
169 ed in BR perception and signal transduction: brassinosteroid insensitive 1 (BRI1), which is the BR re
173 brane-resident RLKs, flagellin-sensing 2 and brassinosteroid insensitive 1-associated kinase 1 (BAK1)
174 phosphorylation on the plant protein kinase brassinosteroid insensitive 1-associated kinase 1 (BAK1)
175 atic embryogenesis receptor kinase 3 (SERK3)/brassinosteroid insensitive 1-associated kinase 1 (BAK1)
176 lagellin receptor FLAGELLIN SENSING 2 (FLS2)-BRASSINOSTEROID INSENSITIVE 1-ASSOCIATED KINASE 1 (BAK1)
177 LLIN-SENSING 2 (FLS2) with their co-receptor BRASSINOSTEROID INSENSITIVE 1-ASSOCIATED KINASE 1 (BAK1)
178 ll death was shown to require N. benthamiana Brassinosteroid insensitive 1-Associated Kinase 1 (NbBAK
181 ense responses required the known coreceptor BRASSINOSTEROID INSENSITIVE 1-ASSOCIATED RECEPTOR KINASE
182 elopment, and immunity, Arabidopsis thaliana BRASSINOSTEROID INSENSITIVE 1-associated receptor kinase
183 ctivation of the Arabidopsis thaliana kinase BRASSINOSTEROID INSENSITIVE 1-ASSOCIATED RECEPTOR-LIKE K
188 Arabidopsis thaliana receptor-like kinases, brassinosteroid-insensitive 1 (BRI1) and BRI1-associated
189 gh plasma membrane-localized receptor kinase brassinosteroid-insensitive 1 (BRI1), BRI1-associated re
191 Y5 enhances the activity of GSK3-like kinase BRASSINOSTEROID-INSENSITIVE 2 (BIN2), a key repressor of
192 lycogen synthase kinase 3 (GSK3)-like kinase brassinosteroid-insensitive 2 (BIN2), which has been wel
193 LYCOGEN SYNTHASE KINASE 3)-like kinase BIN2 (BRASSINOSTEROID-INSENSITIVE 2), a well established negat
195 ifferent ligand-perceiving receptors such as BRASSINOSTEROID INSENSITIVE1 (BRI1) and FLAGELLIN-SENSIT
200 plasma membrane through the receptor kinase BRASSINOSTEROID INSENSITIVE1 (BRI1) together with co-rec
201 of the leucine-rich repeat receptor kinase, brassinosteroid insensitive1 (BRI1), was shown to depend
205 kinase domains of the SERK-dependent LRR-RKs BRASSINOSTEROID INSENSITIVE1, HAESA and ERECTA form tigh
206 avily glycosylated brassinosteroid receptor, BRASSINOSTEROID INSENSITIVE1, while MNS1 to MNS3 appear
208 LecRK-VI.2 constitutively associates with BRASSINOSTEROID INSENSITIVE1-ASSOCIATED KINASE1 (BAK1) i
209 exes between the membrane-localized IOS1 and BRASSINOSTEROID INSENSITIVE1-ASSOCIATED KINASE1 (BAK1)-d
210 the leucine-rich repeat receptor-like kinase BRASSINOSTEROID INSENSITIVE1-ASSOCIATED KINASE1 (BAK1)/S
211 flagellin sensing2 and its signaling partner brassinosteroid insensitive1-associated kinase1 is obser
213 seedling lethal and cell death phenotypes of BRASSINOSTEROID INSENSITIVE1-associated receptor kinase1
215 d PM accumulation of FLS2 and its coreceptor BRASSINOSTEROID INSENSITIVE1-ASSOCIATED RECEPTOR KINASE1
216 dent on the regulatory receptor-like kinases brassinosteroid insensitive1-associated receptor kinase1
217 Fls3, independently of flg22/flgII-28 or of BRASSINOSTEROID INSENSITIVE1-ASSOCIATED RECEPTOR KINASE1
218 th these findings, the transcription factors BRASSINOSTEROID INSENSITIVE1-EMS SUPPESSOR1 and MONOPTER
220 ffectors, BRASSINAZOLE RESISTANT1 (BZR1) and BRASSINOSTEROID INSENSITIVE1-ETHYL METHANESULFONATE-SUPP
221 The leucine-rich repeat receptor-like kinase BRASSINOSTEROID-INSENSITIVE1 (BRI1) is the main ligand-p
223 al transduction cascade, as is the case with BRASSINOSTEROID-INSENSITIVE1 Associated Kinase1 and CPK5
227 Here, we demonstrate that the protein kinase BRASSINOSTEROID INSENSITIVE2 (BIN2), a key negative regu
229 of which carry gain-of-function mutations in BRASSINOSTEROID-INSENSITIVE2 (BIN2), one of the three me
230 In addition, auxin, cytokinin, ethylene, brassinosteroids, jasmonates, and salicylic acid also co
231 pression data, the SUB1A genotype had higher brassinosteroid levels after submergence compared to the
232 harmacologically and genetically manipulated brassinosteroid levels in poplar trees and assayed the e
238 haracterized roles in stamen development and brassinosteroid perception, SERK1 plays a unique role in
240 coding the complete biosynthetic pathway for brassinosteroid phytohormones and enzymes for brassinost
241 iterpenoids, which include membrane sterols, brassinosteroid phytohormones, and non-steroidal triterp
245 ns, gibberellins, cytokinins, abscisic acid, brassinosteroids, polyamines, strigolactones, and ascorb
248 ns of a structurally impaired variant of the brassinosteroid receptor BRASSINOSTEROID INSENSITIVE 1.
249 he auxin translocators PIN2 and AUX1 and the brassinosteroid receptor BRI1 into distinct endomembrane
250 d cell-death control by interacting with the brassinosteroid receptor BRI1, immune receptors, such as
251 ession of phenotypes caused by the defective brassinosteroid receptor bri1-9 strongly suggest that th
256 sfolded variants of the heavily glycosylated brassinosteroid receptor, BRASSINOSTEROID INSENSITIVE1,
257 ich are caused by ER retention and ERAD of a brassinosteroid receptor, BRASSINOSTEROID-INSENSITIVE 1
259 rmation with a second receptor kinase termed brassinosteroid receptor1 associated kinase1 (BAK1).
263 Here we demonstrate in Arabidopsis that brassinosteroid regulates stomatal development by activa
264 tudy provides insights into the mechanism of brassinosteroid regulation of root hair patterning.
267 tematically examined root hair phenotypes in brassinosteroid-related mutants, and found that brassino
269 teroid signaling components to attenuate the brassinosteroid response, thus illuminating one aspect o
271 Evidence points to the interplay of auxin, brassinosteroids, SHORTROOT/SCARECROW and INDETERMINATE
272 ant, whereas the contents of gibberellin and brassinosteroid showed no difference between the mutant
273 ein in rice directly interacts with critical brassinosteroid signaling components to attenuate the br
274 ssinosteroid-related mutants, and found that brassinosteroid signaling inhibits root hair formation t
277 ances brassinosteroid (BR) signaling through BRASSINOSTEROID SIGNALING KINASE 5 (BSK5) and leads to t
283 s associated with cell wall modification and brassinosteroid signaling were induced under enriched FR
284 and biomass accumulation and is regulated by brassinosteroid signaling, auxin transport, and gibberel
286 olved in auxin, abscisic acid, ethylene, and brassinosteroid signaling, peroxisome function, disease
287 OID-INSENSITIVE 2 (BIN2), a key repressor of brassinosteroid signaling, to repress hypocotyl elongati
290 HOTOMORPHOGENIC DWARF, and DIMINUTO) and one brassinosteroid-signaling gene (BRASSINOSTEROID-INSENSIT
291 e role of Arabidopsis (Arabidopsis thaliana) BRASSINOSTEROID-SIGNALING KINASE5 (BSK5), a member of th
292 regulated transcription factor PIF4, and the brassinosteroid-signaling transcription factor BZR1, int
293 lyses indicate that receptor kinase-mediated brassinosteroid signalling inhibits stomatal development
295 tension wood formation, while inhibition of brassinosteroid synthesis resulted in decreased growth a
296 members of the CYP734A subfamily inactivate brassinosteroids through C-26 hydroxylation, the biochem
297 ant MAPKKK to its upstream regulators and of brassinosteroid to a specific developmental output.
300 ASE1 (BAK1), the receptor and coreceptor for brassinosteroids--were shown to autophosphorylate on tyr