戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 we characterized DCs that were exposed to L. braziliensis.
2 ishmania: Leishmania infantum and Leishmania braziliensis.
3  on a cohort of 62 patients infected with L. braziliensis.
4 eous leishmaniasis (CL) caused by Leishmania braziliensis.
5 versus patients with symptomatic CL due to L braziliensis.
6 ging and severe form of disease caused by L. braziliensis.
7 ith Leishmania (Viannia) panamensis and L.V. braziliensis.
8 ctive ulcers and positive PCR for Leishmania braziliensis.
9 sed in light of the present findings with L. braziliensis.
10 risk for CL development caused by Leishmania braziliensis.
11 ed with the prevalent related species L.(V.) braziliensis.
12 d an effective treatment for CL caused by L. braziliensis.
13 reatment of tegumentary disease caused by L. braziliensis.
14 eous leishmaniasis (CL) caused by Leishmania braziliensis.
15 bundant in skin lesions caused by Leishmania braziliensis.
16 cutaneous leishmaniasis caused by Leishmania braziliensis.
17  brucei and to a lesser extent in Leishmania braziliensis.
18 the clinical course of diseases caused by L. braziliensis.
19 to LCTAS, were isolated and mapped onto a L. braziliensis 250 kb multicopy minichromosome and the L.
20 onovani, Leishmania infantum, and Leishmania braziliensis, a capacity to generate intra- and interspe
21 interleukin-4 than did mice infected with L. braziliensis alone.
22  Here we show that ROS inhibits growth of L. braziliensis amastigotes in resting monocytes, and that
23    Monocytes from PBMC were infected with L. braziliensis and cocultured with CD8(+) T cells, and the
24  cases, macrophages were found to take up L. braziliensis and degrade them rapidly in contrast to liv
25          Patients coinfected with Leishmania braziliensis and helminths took longer to heal (relative
26                                  For both L. braziliensis and L. guyanensis, RNAi-derived LRV1-negati
27 S in the telomeric region differs between L. braziliensis and L. major: in L. major the LCTASs are ta
28 d with drug-treatment failures in Leishmania braziliensis and Leishmania guyanensis infections.
29 ll RNAs derived from LRV1 in both Leishmania braziliensis and Leishmania guyanensis, mapping across b
30 results against Trypnosoma cruzi, Leishmania braziliensis and Leishmania infantum were not clinically
31  300 telomere-derived clones from Leishmania braziliensis and Leishmania major, a conserved 100 bp se
32              Dogs from an area of Leishmania braziliensis and Leishmania peruviana endemicity were sc
33 nuclear cells from patients infected with L. braziliensis and reduced IL-1beta levels after cure.
34  a joint evolutionary analysis of Leishmania braziliensis and their dsRNA Leishmania virus 1.
35 cessfully generated axenic amastigotes of L. braziliensis and used them to test the hypothesis that L
36  second Brazilian sample (also exposed to L. braziliensis) and in Iranians infected with Leishmania t
37 Lutzomyia intermedia, a vector of Leishmania braziliensis, and evaluated the seroreactivity in expose
38 hmania (Leishmania) amazonensis, L (Viannia) braziliensis, and L (V) guyanensis] and samples from pat
39 were established, parasites were typed as L. braziliensis, and the presence of LRV1 was determined by
40 th cutaneous leishmaniasis due to Leishmania braziliensis are CD4(+)CD25(-)CD127(-/low)FOXP3(-) cells
41 rd against which future interventions for L. braziliensis are compared.
42                                   Leishmania braziliensis are intracellular parasites that cause uniq
43 C) or asymptomatic infection with Leishmania braziliensis are largely unknown.
44 vidence that photodynamically inactivated L. braziliensis are susceptible to the degradative pathway
45 interfering RNAs (siRNAs) associated with L. braziliensis Argonaute1 (LbrAGO1).
46 are of patients with CL caused by Leishmania braziliensis, because screening for and treatment of hel
47 ows that ROS are important for control of L. braziliensis both at the initial stages of infection, as
48              Leishmania major and Leishmania braziliensis both cause cutaneous leishmaniasis, but the
49 ric oxide, also contributed to killing of L. braziliensis by IFN-gamma activated monocytes.
50                    The control of Leishmania braziliensis by individuals with subclinical infection (
51                                         (V.) braziliensis by the parasitologic approach; for the othe
52                         Leishmania (Viannia) braziliensis can cause localized cutaneous leishmaniasis
53                         Leishmania (Viannia) braziliensis causes localized cutaneous leishmaniasis (L
54 esional pentamidine; 20 patients) against L. braziliensis CL in Bolivia.
55                                   Against L. braziliensis CL, a prevalent, aggressive form of New Wor
56  in the peripheral blood of patients with L. braziliensis, compared with uninfected controls.
57                                           L. braziliensis, contrary to Leishmania species examined so
58                          LRV-1-positive L.V. braziliensis demonstrated significantly lower expression
59 ed Leishmania proteins, Leishmania (Viannia) braziliensis-derived Lb8E and Lb6H, and evaluated both i
60 that human patients infected with Leishmania braziliensis develop dysbiotic skin microbiota, characte
61  the first covalent inhibitors of Leishmania braziliensis dihydroorotate dehydrogenase (LbDHODH), a k
62 CL was diagnosed through documentation of L. braziliensis DNA by polymerase chain reaction or identif
63                          Many isolates of L. braziliensis (>25%) contain a double-stranded RNA virus
64 sions from patients infected with Leishmania braziliensis has been associated with inflammation induc
65 ngle lesions due to Bolivian Leishmania (v.) braziliensis in a placebo-controlled study.
66 eatment of 133 patients with CL caused by L. braziliensis in Bahia, Brazil.
67  vs Sbv in 133 patients with CL caused by L. braziliensis in Bahia, Brazil.
68 rd Sb v for the treatment of CL caused by L. braziliensis in Brazil and accelerate the healing time o
69 han Sbv for the treatment of CL caused by L. braziliensis in Brazil and accelerates the healing time.
70 eous leishmaniasis (CL) caused by Leishmania braziliensis in Brazil with pentavalent antimony (Sb(v))
71                        The assay detected L. braziliensis in dermal scrapings from cutaneous leishman
72 eous leishmaniasis patients infected with L. braziliensis in Rio de Janeiro, Brazil, an area where th
73  successful extension of this approach to L. braziliensis in the Viannia subgenus, viz.
74 dy, the role of AnxA1 was investigated in L. braziliensis-infected BALB/c mice.
75   The chemoattractant factors secreted by L. braziliensis-infected cells were highly efficient in rec
76 wide transcriptional profiling of Leishmania braziliensis-infected cutaneous lesions and normal skin
77                              In contrast, L. braziliensis-infected DCs failed to up-regulate activati
78 ood mononuclear cells (PBMC) from Leishmania braziliensis-infected human patients have demonstrated t
79 itical for modulating disease outcomes in L. braziliensis-infected mice colonized with S. aureus, giv
80  Tregs in S. aureus-colonized and Leishmania braziliensis-infected mice increased IFN-gamma, S. aureu
81 cytokines and prime naive CD4(+) T cells, L. braziliensis-infected MyD88(-/-) DCs exhibited less acti
82  drug treatment success and disease in 97 L. braziliensis-infected patients from Peru and Bolivia.
83  of an IL-4-mediated pathology in Leishmania braziliensis-infected pregnant women.
84                                  As such, L. braziliensis-infected TLR2(-/-) DCs were more competent
85 to rLinB-13 influences disease outcome in L. braziliensis infection and results indicate that positiv
86  We have previously reported that Leishmania braziliensis infection can activate murine dendritic cel
87  of inflammatory mediators in response to L. braziliensis infection contributes to cell recruitment a
88 and used them to test the hypothesis that L. braziliensis infection efficiently triggers innate respo
89                    We observed that while L. braziliensis infection induced the production of chemoki
90    Cutaneous leishmaniasis due to Leishmania braziliensis infection is an inflammatory disease in whi
91                AnxA1 increased also after L. braziliensis infection of BALB/c (wild-type [WT]) bone m
92                                 Secondly, L. braziliensis infection triggered transcription and phosp
93       Our results suggest that, following L. braziliensis infection, the production of multiple infla
94 revealed unique immunological features of L. braziliensis infection.
95 e that AnxA1 is actively expressed during L. braziliensis infection.
96 -up, 118 subjects (38.3%) had evidence of L. braziliensis infection.
97 tion against Leishmania major and Leishmania braziliensis infection.
98  cells (PBMCs) from healthy volunteers to L. braziliensis infection.
99 e findings uncover a dual role for DCs in L. braziliensis infection: T cell activation by bystander D
100                                   Leishmania braziliensis infections are often associated with exagge
101                                   Leishmania braziliensis is a parasitic infection that can result in
102 eous leishmaniasis (CL) caused by Leishmania braziliensis is associated with high levels of tumor nec
103 eous leishmaniasis (CL) caused by Leishmania braziliensis is characterized by a single ulcer or multi
104 eous leishmaniasis (CL) caused by Leishmania braziliensis is characterized by a strong Th1 response t
105                     An unusual feature in L. braziliensis is that the telomeric repeats are often com
106                         Leishmania (Viannia) braziliensis is the causative agent of cutaneous and muc
107                Leishmania (subgenus Viannia) braziliensis is the causative agent of mucocutaneous lei
108 iasis, caused in South America by Leishmania braziliensis, is difficult to cure by chemotherapy (prim
109 d SC expression associated with 2 primary L. braziliensis isolates from patients with LCL or MCL.
110 east 70-77% of their sterol from leucine; L. braziliensis, L. donovani and L. tropica apparently prod
111 riTrypDB integrates datasets from Leishmania braziliensis, L. infantum, L. major, L. tarentolae, Tryp
112 tro against Leishmania infantum , Leishmania braziliensis , Leishmania guyanensis , Leishmania amazon
113 on approved to treat CL caused by Leishmania braziliensis, Leishmania guyanensis, and Leishmania pana
114 omplexes of New World Leishmania (Leishmania braziliensis, Leishmania mexicana, and Leishmania donova
115  suggest that if local therapy for single L. braziliensis lesions is chosen, this treatment is attrac
116              Compared to LRV-1-negative L.V. braziliensis, LRV-1-positive strains of L.V. braziliensi
117                                   Leishmania braziliensis M2903 contains a highly amplified small chr
118              Our observations suggest that L braziliensis may evade microbicidal mechanisms of PMNs f
119 oll-like receptors (TLRs) are involved in L. braziliensis-mediated DC activation.
120                          Species included L. braziliensis (n = 7), L. panamensis (n = 5), Leishmania
121                           This killing of L. braziliensis occurred by a gamma interferon-dependent me
122 ed infection with doses of >10(4) Leishmania braziliensis parasites in BALB/c mice.
123                  Our understanding of how L. braziliensis parasites interact with dendritic cells (DC
124           We found that DCs cultured with L. braziliensis parasites up-regulated DC activation marker
125                      With a dose of 10(7) L. braziliensis parasites, 60 to 70% of the mice developed
126     Importantly, analysis of lesions from L. braziliensis patients revealed that low FOXP3 gene expre
127  upregulated following their loading with L. braziliensis photodynamically inactivated by both strate
128                        Mice infected with L. braziliensis plus saliva produced two- to threefold more
129                              Herein, L. (V.) braziliensis preparations were standardized to contain 1
130 lected tropical disease caused by Leishmania braziliensis, presents treatment challenges due to varyi
131 braziliensis, LRV-1-positive strains of L.V. braziliensis produced a predominant Th2-biased immune re
132 holog, and an EC(5)(0) of 11 +/- 5 muM in L. braziliensis promastigotes, with no cytotoxicity in THP-
133                                           L. braziliensis replication was increased (P < .05) in macr
134                        A signature of the L. braziliensis skin lesion was defined, which includes ove
135 onary history of parasites of the Leishmania braziliensis species complex based on whole-genome seque
136 history of diversification of the Leishmania braziliensis species complex in Peru.
137 s the human immune response, facilitating L. braziliensis survival in vitro, and increases the risk o
138 tion and had a greater ability to control L. braziliensis than cells from patients with CL.
139 oss-protected mice against infection with L. braziliensis that causes mucocutaneous leishmaniasis.
140 he LCTASs are tandemly repeated, while in L. braziliensis the LCTAS is present as a single copy per e
141 .6%) did not have evidence of exposure to L. braziliensis The ratio of infection to disease was 3.2:1
142  in vitro a role for LRV1 in virulence of L. braziliensis, the Leishmania species responsible for the
143  the generation of protective immunity to L. braziliensis, TLR2 seems to have a regulatory role durin
144                         Doubly sensitized L. braziliensis transfectants were photo-inactivated before
145 nsgenic strategy used previously produced L. braziliensis transfectants, which gave the same phenotyp
146                            In areas where L. braziliensis transmission is endemic, up to 15% of healt
147 en 1992 and 1998 in an area where Leishmania braziliensis transmission is endemic; 8 of the patients
148                      In a separate study, L. braziliensis was also found, like other Leishmania spp.,
149 w that IL-1beta production in response to L. braziliensis was dependent on NLRP3, caspase-1, and casp
150 lied in a second step, on single cells of L. braziliensis, which were sorted by fluorescence activate
151  has proven high efficacy in CL caused by L. braziliensis with a cure rate of 75%.
152 sine has proven efficacy for CL caused by L. braziliensis, with a cure rate (CR) of 75%.

 
Page Top