ライフサイエンスコーパス: breathe

1 here in the lung while the patient is freely breathing.

2 tivated immediately after birth and supports breathing.

3 sleep apnea (OSA), a widespread disorder of breathing.

4 unction parameters were clear during resting breathing.

5 right CSR, and 297 patients (52%) had normal breathing.

6 ve decline in patients with sleep-disordered breathing.

7 mimic the shear stress associated with tidal breathing.

8 oxygen, carbon dioxide, and pH, and regulate breathing.

9 is necessary for the chemosensory control of breathing.

10 RSA) while estimating the resting f(H) while breathing.

11 te disodium and gadoterate meglumine at free breathing.

12 3-targeted shRNA survived with no changes in breathing.

13 ngle radial sparse parallel sequence at free breathing.

14 iratory muscle adaptations to disruptions in breathing.

15 a novel pharmacological strategy to improve breathing.

16 d DS patients' frequently exhibit disordered breathing.

17 nt for sleep-wake state-dependent control of breathing.

18 the remarkable robustness and flexibility of breathing.

19 lasticity may be a novel strategy to improve breathing.

20 lation of either RTN or C1 neurons activates breathing.

21 rom appetite and digestion to heart rate and breathing.

22 f inspiratory flow around 1.0L/s due to rest breathing.

23 val {CI}: 0.007, -0.024]; rho = 0.97 vs free breathing, -0.004 [95% CI: 0.007, -0.016]; rho = 0.91).

24 In a mouse model of FRDA, breathing 11% O(2) attenuates the progression of ataxia,

25 diaphragmatic contraction during spontaneous breathing, 2) reduce expiratory flow and make lung compa

26 icantly lower in the HD patients during deep breathing (3.686 +/- 1.567 cm/s vs. 4.410 +/- 1.720 cm/s

27 s and Methods A heart rate-independent, free-breathing 3D T2 mapping technique at 3.0 T that can be c

28 d against tumor motions measured in the free-breathing 4DCT scans.

29 rmocapnia) and during hypercapnia induced by breathing 5% CO(2) in air.

30 ttenuates the progression of ataxia, whereas breathing 55% O(2) hastens it.

31 Sleep-disordered breathing accounted for 9 to 10% of ethnic differences i

32 -T MRI scanner and compared with a fast free-breathing acquisition technique for ADC mapping (approxi

33 antitative parameter maps from a single free-breathing acquisition with the potential to reduce exam

34 expiratory pressure both without spontaneous breathing activity (0.029 [0.027-0.030] vs 0.044 [0.041-

35 .041-0.065]; p = 0.004) and with spontaneous breathing activity (0.032 [0.028-0.043] vs 0.057 [0.042-

36 oups (n = 6/group; 12 hr): 1) no spontaneous breathing activity and positive end-expiratory pressure

37 atory pressure - 4 cm H2O, 2) no spontaneous breathing activity and positive end-expiratory pressure

38 piratory pressure + 4 cm H2O, 3) spontaneous breathing activity and positive end-expiratory pressure

39 piratory pressure + 4 cm H2O, 4) spontaneous breathing activity and positive end-expiratory pressure

40 al ventilation, independent from spontaneous breathing activity.

41 pendent zones in the presence of spontaneous breathing activity.

42 Individuals with obstructive sleep apnea can breathe adequately when awake but experience repeated ep

43 e, we identify two primary changes to murine breathing after administering opioids.

44 re even able to venture on land and possibly breathe air [11].

45 le cigarette smoke (WCS) under physiological breathing airflow, are lacking.

46 this technique on a healthy human volunteer breathing along different respiratory patterns during th

47 umonia (cough lasting <14 days or difficulty breathing, along with visible indrawing of the chest wal

48 ere measured continuously during ambient air breathing (Amb) and a 6 min inhalation of the vasodilato

49 nputs to mediate inspiratory activity during breathing and are constrained to fire in a pattern that

50 ctions, and monitored firing rate changes in breathing and blood glucose modulated conditions.

51 ls and adjusts homeostatic functions such as breathing and cardiovascular tone accordingly.

52 We hypothesized that during both spontaneous breathing and controlled mechanical ventilation, externa

53 ne of these substitutions increased envelope breathing and decreased scavenger receptor class B type

54 ce placed in the patient's trachea to assist breathing and delivering oxygen into the lungs.

55 ation was mostly at birth with hypotonia and breathing and feeding difficulties often requiring venti

56 decreased from 24% to 16% during spontaneous breathing and from 32% to 18% during controlled mechanic

57 ptin acts on LepR(b) in the CBs to stimulate breathing and HVR, which may protect against sleep disor

58 WHO cutoffs for fast breathing and hypoxaemia overlap with RR and SpO(2) valu

59 with 394 (281, 554) kcal/d in spontaneously breathing and mechanically ventilated patients, respecti

60 with 639 (479, 723) kcal/d in spontaneously breathing and mechanically ventilated patients, respecti

61 spectively (P = 0.949).Both in spontaneously breathing and mechanically ventilated patients, the best

62 elated with higher REE both in spontaneously breathing and mechanically ventilated patients.

63 ctive equations, separately in spontaneously breathing and mechanically ventilated patients.

64 -related disparities center on sleep-related breathing and medication use, and racial disparities rel

65 sleep produces arousal, which helps restore breathing and normalizes blood gases.

66 assess associations between sleep-disordered breathing and outcomes, adjusted for sociodemographics,

67 Thus, zolpidem may stabilise breathing and reduce OSA severity without CPAP.

68 he efficacy of therapeutic interventions for breathing and sleep anomalies.

69 ther are involved in chemosensory control of breathing and sleep homeostasis.

70 "6-foot rule." Here we analyze flows during breathing and speaking, including phonetic features, usi

71 Both regulate breathing and the cardiovascular system but in ways that

72 the amplitude of nucleosome motions such as breathing and twisting are enhanced in nucleosomes with

73 jury and investigate the impact of injury on breathing and voice outcomes.

74 alveolar ventilation, both during unassisted breathing and with different modes of ventilatory assist

75 st is the unexpected loss of heart function, breathing, and consciousness and is commonly the result

76 maneuvers of normal breathing, talking, deep breathing, and coughing.

77 case during normal breathing, talking, deep breathing, and coughing.Conclusions: Oxygen delivery mod

78 rts promote ineffective triggering, periodic breathing, and diaphragmatic atrophy.

79 A) were recorded at supine rest, during deep breathing, and during a Valsalva manoeuvre.

80 cy and adversely affects speech, swallowing, breathing, and hearing.

81 ciency improvements limited by difficulty in breathing, and no safe reusability), which have yet to b

82 ical stress associated with sleep-disordered breathing, and this measure predicts incident cardiovasc

83 not associated with birth weight, difficulty breathing, apnea or upper or lower respiratory infection

84 and phase difference around the frequency of breathing (approximately 0.3 Hz) and around the frequenc

85 nical signs of insomnia and sleep-disordered breathing are common in mid-to-late pregnancy, but most

86 The neural systems that regulate breathing are fragile early in development, and it is no

87 source of the CO(2)/H(+)-dependent drive to breathe, are hyper-excitable in slices from Scn1a(DeltaE

88 Healthy participants were asked to breathe as long as possible until task failure against a

89 rature-dependent neutralization (e.g., virus breathing) assays indicated that both HVR1 and protectiv

90 f a peptidergic neural circuit that supports breathing at a particularly vulnerable period in life.

91 d it is not clear how they adjust to support breathing at birth.

92 onal changes necessary for adaptation to air breathing at birth.

93 o CO(2) homeostasis and to the regulation of breathing automaticity during sleep and wake.

94 rorespirator and a smoke machine to simulate breathing behavior and smoking topography parameters suc

95 Moreover, we predict the breathing behavior of other potentially interesting sequ

96 In the case of flexible MOFs, the breathing behavior, and in particular the pressure at wh

97 C-1) and find that it exhibits guest-induced breathing behavior.

98 s in MOFs, however, typically refers to the "breathing" behavior of cavities, where pores open and cl

99 Sniffing, the active control of breathing beyond passive respiration, is used by mammals

100 gmatic pressure decreased during spontaneous breathing by >10%, 2) expiratory flow was reduced and th

101 TASK2 is involved in the regulation of breathing by chemosensory neurons of the retrotrapezoid

102 monitored by EEG and neck EMG recordings and breathing by whole-body plethysmography.

103 rpretation of PPV is unreliable (spontaneous breathing, cardiac arrhythmias) or doubtful (low Vt).

104 These changes implicate the brainstem's breathing circuitry which we confirm by locally eliminat

105 cleus (RTN) and adjacent C1 neurons regulate breathing, circulation and the state of vigilance, but p

106 by the purely mechanosensitive dynamics (to breathing clean air) nor by the response amplitudes acro

107 ury had significantly worse patient-reported breathing (Clinical Chronic Obstructive Pulmonary Diseas

108 eased markedly by 138+/-71% ( P<0.001) after breathing CO gas, 2.8 times more than the increase induc

109 The five breathing conditions led to highly significant differenc

110 y due to the contribution of RAP during both breathing conditions.

111 nd 20 s of normoxia (n = 9), or a 40 min air-breathing control (n = 7).

112 important physiological functions, including breathing control.

113 Growing evidence supports the Awakening and Breathing Coordination, Delirium monitoring/management,

114 Use of WHO definitions for fast breathing could result in misclassification of pneumonia

115 rmines the energy delivered to the lungs per breathing cycle.

116 posing healthy males to 40 consecutive 1 min breathing cycles, each comprising 40 s of hypercapnic hy

117 eurones resulted in attenuation of irregular breathing, decreased apnoea-hypopnoea incidence (11.1 +/

118 ating the respiratory rhythm-phenocopied the breathing deficits observed after RTN deletion of PACAP,

119 sion of PACAP in RTN neurons corrected these breathing deficits.

120 Breathing depends on pulmonary surfactant, a mixture of

121 sal obstruction, and PC2 was associated with breathing difficulties and lean body mass, although EDCs

122 y-onset myasthenic syndrome with feeding and breathing difficulties.

123 ep apnea, a common and serious sleep-related breathing disorder.

124 ostructural characteristics of sleep-related breathing disorders.

125 al results in some species, particularly air-breathing divers.

126 e significantly at the frequency of periodic breathing during acute and sustained normobaric and hypo

127 xia, which likely compensates and stabilizes breathing during injury or disease and has significant t

128 obese db/db mice and examine its effects on breathing during sleep and wakefulness and on HVR.

129 In adjusted analysis, spontaneous breathing during the first 2 days was not associated wit

130 and C1 neurons regulate distinct aspects of breathing (e.g., frequency, amplitude, active expiration

131 Breathing, ECG and microvascular blood flow were simulta

132 ck from lower limbs to modify the EPR, while breathing either ambient air, normocapnic hypoxia (S(a)

133 e conduction, ventilation variables, work of breathing, electrical stimulation variables, stimulation

134 romuscular function significantly influenced breathing endurance, timing and loading compensations.

135 r pancreatic imaging in the presence of free-breathing exam.

136 During spontaneous breathing, expiratory diaphragmatic contraction countera

137 igns; history in the past 24 h of difficulty breathing, fast breathing, runny nose, or nasal congesti

138 awing of the chest wall with or without fast breathing for age).

139 ced robust arousals and similar increases in breathing frequency and amplitude compared with RTN stim

140 RTN stimulation robustly increased breathing frequency and amplitude; it also triggered str

141 In particular, whether breathing frequency is inherently proportional to limb v

142 rotein genes, some expressed specifically in breathing gills of aquatic nymphs, suggesting a novel se

143 ow weight for height, unresponsiveness, deep breathing, hypoxemia, grunting, and the absence of cough

144 es in abdominal MRI have enabled rapid, free-breathing imaging without the need for intravenous or or

145 During respiration, humans breathe in more than 10,000 liters of non-sterile air da

146 report direct observation of spontaneous DNA breathing in atomistic molecular dynamics simulations, d

147 , which may protect against sleep disordered breathing in obesity.

148 these results support the use of spontaneous breathing in patients with acute respiratory distress sy

149 a region that contains neurons that regulate breathing in response to changes in CO(2) /H(+) , has be

150 Respiratory chemoreceptors regulate breathing in response to changes in tissue CO(2)/H(+).

151 ation for the evolution of costal aspiration breathing in stem amniotes.

152 ia, effects of disease and age on control of breathing, inaccuracy of pulse oximetry at low oxygen sa

153 nts aerosolised coronavirus released through breathing increases the chance of spreading the disease.

154 nd significantly attenuated sleep-disordered breathing independently of body weight.

155 RC %), breaths per minute (BPM), and labored breathing index (LBI) on an iPad.

156 ry pressure generation and the rapid shallow breathing index) were also assessed.

157 tic resonance images of 17 nonsedated, quiet-breathing infants with severe bronchopulmonary dysplasia

158 Case patients who did not have difficulty breathing infected 0.05 times as many contacts (95% CI,

159 ibility that arousal-dependent modulation of breathing involves recruitment of cholinergic projection

160 that RVLM-C1 neurones play a pivotal role in breathing irregularities in volume overload HF, and medi

161 Sleep-disordered breathing is associated with worse functional and cognit

162 Spontaneous breathing is common in patients with acute respiratory d

163 on is the standard treatment when volitional breathing is insufficient, but drawbacks include muscle

164 Spontaneous breathing is not associated with worse outcomes and may

165 electrode over cycles, termed as mechanical breathing, is a crucial issue limiting the quality and l

166 ucing ICAN is predicted to slow down or stop breathing; its contributions to motor pattern would be r

167 nduced hypoxia in humans evokes a pattern of breathing known as periodic breathing (PB), in which the

168 ly to the structural parameters of adjacent "breathing lattice" SrCuO(2) (SCO).

169 iquid interfaces, with compression/expansion breathing-like dynamics enhancing rapid interface-assist

170 ses indicated that greater activation during breathing load anticipation was associated with past bul

171 During breathing load anticipation, the RBN group, relative to

172 motion face-processing task, (2) inspiratory breathing load task, and (3) fear conditioning and extin

173 gm displacement from quiet breathing to deep breathing ( m) were lower in HD patients than in control

174 ) was higher during breath hold than at free breathing (mean +/- standard deviation in milliliters of

175 cuff BP were recorded before and after slow breathing, mental arithmetic, cold pressor, and sublingu

176 In order to determine the optimal breathing method for childhood lung sound analyses, it i

177 es within the olfactory epithelium in freely breathing mice, we find widespread antagonistic interact

178 osure, focusing specifically on their radial breathing mode (RBM).

179 bond length change associated with molecular breathing mode can be tracked with a sub-Angstrom resolu

180 Dramatic overdamping of the breathing modes indicates that dynamic stripe phase may

181 oherent phonons are identified: localized 0D breathing modes of isolated superatom, 2D synchronized t

182 The impact of this concept on "breathing" MOFs is discussed. I(2) sorption, both from g

183 ously determined RSV F structures, reveals a breathing motion of the prefusion conformation.

184 pairing is important because the underlying "breathing" motion between the two conformations can sign

185 , together with synchronous and asynchronous breathing motions of the enzyme, underlie allosteric coo

186 Inspiratory breathing movements depend on pre-Botzinger complex (pre

187 ning whether and how the rhythms of limb and breathing movements interact is highly informative about

188 the autonomic nervous system, fetal body and breathing movements, and from baroreflex and circadian p

189 te synthetic MRI ventilation scans from free-breathing MRI (deep learning [DL] ventilation MRI)-deriv

190 work we investigate the use of a novel free-breathing multi-echo Dixon technique for quantitative my

191 ed to observe the DNA of an ensemble of such breathing nucleosomes through x-ray diffraction with con

192 s into the sequence dependence of nucleosome breathing observed in the experiment and allows us to de

193 .96) and was more likely to require assisted breathing (odds ratio, 6.55; 95% CI, 1.17-36.67); depend

194 al [CI]: 1.45 to 10.76; p = 0.007 vs. normal breathing; odds ratio: 4.01; 95% CI: 1.54 to 10.46; p =

195 ifically to a single-stranded region and, on breathing of the inhibitory structure, relocate to the R

196 tween HA head domains; reversible molecular "breathing" of the HA trimer can expose the interface to

197 my tube for 24 hours to see whether they can breathe on their own.

198 s underlying the effects of sleep-disordered breathing on the brain.

199 d cough, runny nose, sore throat, difficulty breathing or myalgia, and collected data on other sympto

200 ignalling in this region influences baseline breathing or the ventilatory response to CO(2) in consci

201 atory conditions probably affecting stamina, breathing, or fatigue (0.75, 0.47-1.19; p=0.220; assesse

202 rve as the surfactant-producing cells of air-breathing organisms.

203 nce of the external gills that represent key breathing organs of bichir free-living embryos and early

204 oxia, and function can promptly improve upon breathing oxygen.

205 fty hemodynamically stable and spontaneously breathing patients equipped with a femoral (n = 21) or r

206 tory network that control the changes in the breathing pattern associated with elevated metabolic dem

207 Instead, a severe ataxic breathing pattern emerged with many apnoeas.

208 correlate with increasing irregularities in breathing pattern.

209 ehind the neural mechanisms that control the breathing pattern.

210 These results identify common breathing patterns in healthy young adults with distinct

211 hare features of chemoreflex-driven clinical breathing patterns that also occur primarily in males, w

212 ystems, baseline wander, normal and abnormal breathing patterns, changes in breathing rate, noise, an

213 increased sympathetic activity and irregular breathing patterns.

214 Periodic breathing (PB) occurs in most humans at high altitudes a

215 kes a pattern of breathing known as periodic breathing (PB), in which the regular oscillations corres

216 nickelates apart from that in cuprates where breathing phonons are not overdamped and point out remar

217 is an effective and safe treatment for fast-breathing pneumonia in children 2-59 months of age.

218 strength may help face an increased work of breathing postoperatively.

219 delie penguins and observations of other air-breathing predators (penguins, seals, and whales), all o

220 dge) or inaccessible (solid fast ice) to air-breathing predators.

221 5%), which was characterized by a history of breathing problems/eczema during infancy and non-respira

222 detailing a microscopic mechanism of the DNA breathing process.

223 ramme and a peer-led camp based on the Power Breathing Programme.

224 tion Ultrafine Particles in School Children (BREATHE) Project, we estimated residential [Formula: see

225 Free-breathing proton MRI may help quantify lung function usi

226 Net-based DCNN model was trained to map free-breathing proton MRI to hyperpolarized helium 3 ((3)He)

227 etworks generated ventilation maps from free-breathing proton MRI trained with a hyperpolarized noble

228 By enabling fast and free-breathing quantitation, MRF has the potential to add val

229 tivation by VNS: stimulus-elicited change in breathing rate (DeltaBR) and heart rate (DeltaHR), respe

230 Thus, breathing rate is a key modulator of cerebral oxygenatio

231 and abnormal breathing patterns, changes in breathing rate, noise, and artifacts.

232 = 8); educational (k = 4); peer-led (k = 5); breathing re-training (k = 1).

233 creased apnoeas and blunted CO(2)-stimulated breathing; re-expression of PACAP in RTN neurons correct

234 evel of RTN region could also be involved in breathing regulation.

235 Breathing results from sequential recruitment of muscles

236 Breathing results from the interaction of two distinct o

237 explanation for the acute O(2) regulation of breathing, reveal an unanticipated role of HIF2alpha, an

238 oxygen tension in the brain through carbogen breathing reversed the neuroprotective effects of FLASH,

239 site is the preBotzinger Complex, where the breathing rhythm originates, and use genetic tools to re

240 For breathing, rhythm generation is localized to a brainstem

241 We assessed the mechanism of mammalian breathing rhythmogenesis in the preBotzinger complex (pr

242 In sum, stimulating C1 or RTN activates breathing robustly, but only RTN neuron stimulation prod

243 the past 24 h of difficulty breathing, fast breathing, runny nose, or nasal congestion; and current

244 Sleep-disordered breathing (SDB) during pregnancy has been linked to adve

245 Rationale: Sleep-disordered breathing (SDB) is associated with increased vascular re

246 ior studies have found that sleep-disordered breathing (SDB) is common among those with left ventricu

247 Sleep-disordered breathing (SDB) is frequently associated with atrial arr

248 uration, sleep consistency, sleep-disordered breathing (SDB), and sleep architecture.

249 Obstructive sleep-disordered breathing (SDB), which includes primary snoring through

250 We quantify the breathing strain and the evolution of the mechanical pro

251 The discovery of air-breathing structures in eurypterids indicates that chara

252 s known as Kiemenplatten represent novel air-breathing structures.

253 during behavioral or metabolic challenges to breathing, such as changes in sensory feedback, sighing,

254 dicate eurypterids were capable of subaerial breathing, suggesting that book gills are the direct pre

255 ory distress syndrome model in spontaneously breathing surfactant-deficient newborn piglets to invest

256 e participants performed maneuvers of normal breathing, talking, deep breathing, and coughing.

257 This was the case during normal breathing, talking, deep breathing, and coughing.Conclus

258 vigated "Koosh ball" 3D UTE sequence at free breathing (TE, 0.03 msec).

259 ed screening tests such as liquid biopsy and breathing tests may transform the screening landscape.

260 te a robust, contrast agent-unenhanced, free-breathing three-dimensional (3D) cardiac MRI approach fo

261 Changes in diaphragm displacement from quiet breathing to deep breathing ( m) were lower in HD patien

262 [i.e., acid-activated neurons that regulate breathing to maintain a constant arterial PCO(2) (PaCO(2

263 points for future trials of sleep-disordered breathing treatment in stroke.

264 eiving mechanical ventilation, a spontaneous breathing trial consisting of 30 minutes of pressure sup

265 he opportune moment to conduct a spontaneous breathing trial.

266 tions of care on compliance with spontaneous breathing trials (odds ratio, 1.00; 95% CI, 0.95-1.07),

267 Daily spontaneous breathing trials (SBTs) are the best approach to determi

268 were measured immediately before spontaneous breathing trials and at 60 minutes after spontaneous bre

269 x increased significantly during spontaneous breathing trials in the failure group.

270 g trials and at 60 minutes after spontaneous breathing trials initiation.

271 h respiratory variability during spontaneous breathing trials is independently associated with extuba

272 n saturation (DeltaScvO2) during spontaneous breathing trials were independently associated with extu

273 for lung-protective ventilation, spontaneous breathing trials, and neuromuscular blockade, respective

274 of respiratory variables during spontaneous breathing trials, and the change in airway pressure duri

275 vidence-based processes of care (spontaneous breathing trials, lung-protective ventilation, and neuro

276 venous oxygen saturation, during spontaneous breathing trials, were independent predictors of weaning

277 manding ventilation strategy for spontaneous breathing trials.

278 Extubation after successful spontaneous breathing trials.

279 Spontaneous Awakening Trials and Spontaneous Breathing Trials; "C" for Choice of Analgesia and Sedati

280 s (RVLM-C1) modulate sympathetic outflow and breathing under normal conditions.

281 mporally and spatially controlled 'chromatin breathing' upon DNA damage, which we demonstrate fosters

282 ute normoxic and hyperoxic steady-state free-breathing UTE acquisitions.

283 Apnoea/hypopnoea incidence (AHI), breathing variability, respiratory-cardiovascular coupli

284 ustment, diaphragmatic excursion during deep breathing was associated with haemoglobin level (regress

285 Sleep-disordered breathing was associated with worse functional outcome (

286 Work of breathing was maintained between 0.2 and 2.0 joules/L 96

287 Spontaneous breathing was present in 67% of patients with mild acute

288 of apneic events and the percentage of time breathing was stable (r = -0.66, P = 0.03) during sleep.

289 show that this supramolecule can reversibly breathe water through lattice expansion and contraction,

290 i-compartment platform that bidirectionally 'breathes' WCS through microchannels of a human lung smal

291 ry flow around 2.0L/s due to slightly strong breathing were significantly higher than the lung sound

292 OHS in obese patients with sleep-disordered breathing when suspicion for OHS is not very high (<20%)

293 dults, both caused by deviations from normal breathing which we term deep breaths and bursts.

294 ibit a higher respiratory frequency (fR) and breathe with a more predictable pattern.

295 ht phrenic nerves and maintenance of work of breathing within defined limits for 80% of stimulated br

296 n with inspiration while maintaining work of breathing within defined limits.

297 RIP measures work of breathing (WOB) indices including phase angle ( ), perce

298 One subject had PSLs detected by the breathing zone air sampler.

299 on exposure on air exchange rate between his breathing zone and bedroom air (lambda(BZ)).

300 ants nor airborne contamination in subjects' breathing zones.