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1 late to inflammatory changes in the lung and bronchi.
2 lergen-induced contraction of isolated human bronchi.
3 ase stimulated contraction of isolated human bronchi.
4 e fields were located in the trachea or main bronchi.
5 terized by inflamed and dilated thick-walled bronchi.
6 vessels, bile ducts, esophagus, trachea, and bronchi.
7 rmal numbers of mast cells in their skin and bronchi.
8  in the respiratory epithelia of trachea and bronchi.
9 o model for studies of EIB in isolated human bronchi.
10 tal and acquired stenosis of the trachea and bronchi.
11 pithelium including nasopharynx, trachea and bronchi.
12 terol delivery in a model of the trachea and bronchi.
13 ute to the epithelial layer in more than 99% bronchi.
14  a prejunctional mechanism in human isolated bronchi.
15 e connected to the airways and shaped by the bronchi.
16 ion of cytokines, in particular IL-4, in the bronchi.
17 y used in vivo model of human small-diameter bronchi.
18 (Golf and AC3) in the smooth muscle of human bronchi.
19 teroids on mast cell localisation within the bronchi.
20 n, and saline in three distinct subsegmental bronchi.
21 ads to tracheal agenesis and ectopic primary bronchi.
22 d mean wall area percentage in six segmental bronchi.
23 P63 into the prospective trachea and primary bronchi.
24 like structures in the trachea and main-stem bronchi.
25 number and shape of cartilage in trachea and bronchi.
26 is when lesions are located centrally in the bronchi.
27 roups in bacterial colonization of the lungs/bronchi.
28 had fluid in the subglottic trachea and main bronchi.
29 blish an ex vivo model of EIB in human small bronchi.
30 on and immunohistochemical analysis in human bronchi.
31 epithelium, glands, and endothelium of human bronchi.
32 ge pulmonary arteries and veins and mainstem bronchi.
33 er lobar bronchi 45%, left 55%; middle lobar bronchi 21%, lingula 26%; right lower lobar bronchi 28%,
34  bronchi 21%, lingula 26%; right lower lobar bronchi 28%, left 29%.
35 N THE TYPICAL ONES WAS IN: right upper lobar bronchi 45%, left 55%; middle lobar bronchi 21%, lingula
36                                     In human bronchi, a similar role of Rac1 was observed during bron
37       This experimental study in human small bronchi aimed to provide insight into which of the type
38                   A parasitology exam from a bronchi alveolar lavage yielded an immature rhabditiform
39 ere common abnormalities such as hyparterial bronchi and absence of middle lobe fissure on CTscans su
40 (AAR) is typically used to identify enlarged bronchi and bronchiectasis; however, current imaging met
41 lammatory changes and epithelial necrosis of bronchi and bronchiolar epithelium.
42 al passage of neutrophils into the pulmonary bronchi and bronchioles of lungs infected with P. haemol
43 ipheral lung samples (in epithelium of small bronchi and bronchioles or lymphoid aggregates).
44                        More than 30% of both bronchi and bronchioles were obstructed by cast formatio
45                      The conducting airways (bronchi and bronchioles) and peripheral gas exchange (al
46 bular septa, centrilobular region, and small bronchi and bronchioles), abnormal findings (reticulatio
47 ient neutrophils into the walls and lumen of bronchi and bronchioles.
48 gh the extensive extracellular matrix of the bronchi and bronchioles.
49 sia of neuroendocrine cells involving distal bronchi and bronchioles.
50  the effects of both IL-13 and IL-4 in human bronchi and human airway smooth muscle cells.
51 ied a dominant adventitial niche around lung bronchi and larger vessels in multiple tissues, where IL
52  isotropic, and submillimeter imaging of the bronchi and lung parenchyma with high CNR and SNR and ma
53 binding model in membranes prepared from all bronchi and lung parenchyma.
54 efects in the smooth muscle component of the bronchi and major pulmonary vessels with decreased Fgf10
55 efects in the smooth muscle component of the bronchi and major pulmonary vessels.
56  interstitial proliferation around the small bronchi and Masson's bodies.
57 s were found on postmortem human trachea and bronchi and on upper airways in 2 compartments, cartilag
58 ude the ciliated epithelial cells lining the bronchi and oviduct, as well as in the developing sperma
59 ng sites (Bmax) was reduced significantly in bronchi and parenchyma from rejecting lungs with or with
60 gned to characterize endothelin receptors in bronchi and parenchyma of transplanted lungs during acut
61 ssociation between structural alterations of bronchi and PH in COPD.
62 imulated submucosal gland secretion in human bronchi and smooth muscle contraction in mouse intestine
63 ontraction of isolated pulmonary vessels and bronchi and stimulates proliferation of smooth muscle ce
64 onchial smooth muscle and in human embryonic bronchi and surrounding mesenchyme, strongly suggesting
65 ial or endothelial cells that line the human bronchi and the aorta express nicotinic acetylcholine re
66 ls of sphingosine in the luminal membrane of bronchi and the trachea without morphological side effec
67                                         Main bronchi and tracheal ASM were significantly hyposensitiv
68 release a variety of signals that target the bronchi and vasculature and recruit other immune cells t
69 s (eg, small vessels, arterial walls, distal bronchi, and bone trabeculations) and their pathologies,
70 ory epithelium of the nasal cavity, trachea, bronchi, and bronchioles with accompanying inflammation.
71 ertrophy of epithelial cells of the trachea, bronchi, and bronchioles.
72 es, including neural tubules, gut, pulmonary bronchi, and hepatic cells.
73 , in airway smooth muscle of the trachea and bronchi, and in the smooth muscle layers of all abdomina
74                                     Trachea, bronchi, and lungs from 11 California sea lions, 2 North
75 epresenting 4 IAV subtypes infected trachea, bronchi, and lungs of macaques and marine mammals with v
76 inal size reduction in the trachea, mainstem bronchi, and proximal airways.
77 y using visual scoring, numbering of visible bronchi, and quantitative measurement of the apparent co
78 ependent induction of MMCs in trachea, large bronchi, and small intestine provides numbers but does n
79  were taken from the trachea, the five lobar bronchi, and the five lobar parenchyma, for qualitative
80 arterial smooth muscle cells, the developing bronchi, and the urogenital ridge and bladder.
81 sets of epithelial cells lining the trachea, bronchi, and tracheal glands.
82 dow's inner portion disappeared, and dilated bronchi appeared.
83  lineages of conducting airways (trachea and bronchi), as distinct from those of peripheral airways (
84                   In freshly harvested human bronchi, ASL thickness was 55 +/- 5 microm, [Na+] was 10
85 ronchial tissue oximetry of native and donor bronchi at 0, 3, and 30 days after transplantation (n =
86 VF contents in the lower trachea, carina and bronchi at baseline and at LAP +10 mmHg were 52.1 +/- 1.
87 ion therapy-induced consolidation in ectatic bronchi at least 9 months after completion of radiation
88                                The main-stem bronchi bifurcate from this common structure and connect
89 s present in basal epithelial cells of large bronchi, both histologically normal (26%) and hyperplast
90 nal arborized network of conducting airways (bronchi, bronchioles) and gas-exchanging units (alveoli)
91 ted, by immunostaining, in the epithelium of bronchi, bronchioles, and alveolar walls.
92 mmatory cell infiltrates in the lungs around bronchi, bronchioles, and pulmonary arteries and veins;
93  cell types and structures that resemble the bronchi/bronchioles of the developing human airway surro
94                    However, only in the main bronchi, but not in the trachea, did the loss of SM or c
95  guinea pig trachea and isolated human small bronchi challenged with antigen and anti-IgE, respective
96                   RNA sequencing of isolated bronchi confirmed the IL-13-mediated upregulation of H(1
97                             Trachea and main bronchi did not show significant differences in reactivi
98 blished place in diagnostics of tracheal and bronchi disorders and its potential has not been examine
99                       THE FREQUENCY OF LOBAR BRONCHI DIVISIONS OTHER THAN THE TYPICAL ONES WAS IN: ri
100 vessels that supply the trachea and mainstem bronchi do not penetrate into the intraparenchymal airwa
101 astrointestinal system (GI), followed by the bronchi, endocrine glands-like C cells of the thyroid (m
102 he lack of vascularization within obstructed bronchi establishes a paradigm for antimycobacterial dru
103 mutant mice was also decreased and the large bronchi extended to the periphery.
104 ination of glutaraldehyde-fixed medium-sized bronchi from acute Kawasaki disease fatalities and analy
105                                  Third-order bronchi from autografted or allografted lungs were eithe
106  receptors) caused comparable contraction of bronchi from autotransplanted and allotransplanted rejec
107 in A and B receptors) caused contractions of bronchi from autotransplanted lungs which were not diffe
108  facto the contralateral lung or nonbleeding bronchi from blood aspiration.
109                                              Bronchi from COX-1(-/-) mice were hyperresponsive to bro
110 made from parasympathetic ganglia located on bronchi from human lungs in order to determine the level
111 tions caused by endothelin 3 were reduced in bronchi from rejecting allotransplanted lungs.
112 es of bronchial reactivity were performed on bronchi from smooth muscle (SM)-specific Rac1 knockout m
113 racheas (605/827 glands, 15 subjects) and in bronchi from subjects who were transplanted because of o
114                                              Bronchi from wild-type mice contained predominantly COX-
115 pithelium, and adventitia of bronchioles and bronchi in lungs of calves with BLAD compared to normal
116                             Branching of the bronchi in lungs of mutant mice was also decreased and t
117 E(4) mediate antigen-induced constriction of bronchi in tissue obtained from subjects with asthma.
118                                           CF bronchi in vivo exhibited the same pattern of expression
119                                        Small bronchi (inner diameter, 0.5-2 mm) from macroscopically
120                                              Bronchi isolated from 10 of 16 lungs contracted in respo
121                                          The bronchi isolated from 8 of 16 lungs also exhibited relax
122                              Explanted small bronchi isolated from human lung tissue and human airway
123 ryonic murine craniofacial cartilage, heart, bronchi, kidney and vertebral bodies.
124       We found that, in the trachea and main bronchi, loss of SM or cartilage resulted in an increase
125 st susceptible to airway obstruction (nose < bronchi &lt;< bronchioles).
126      Filling in of previously patent ectatic bronchi occurred in the 20 patients with recurrent disea
127 f a single somatic p53 point mutation in the bronchi of a smoker suggests that a single progenitor br
128 al similarities in the primary and secondary bronchi of birds and crocodilians suggest that these str
129  a subset of SCGB3A2-expressing cells within bronchi of both mouse and neonatal human lungs.
130                                              Bronchi of chronic obstructive pulmonary disease (COPD)
131 ive immunity and chronic inflammation in the bronchi of COPD patients.
132 sion molecule (CEACAM) family members in the bronchi of patients with severe asthma.
133                               Intrapulmonary bronchi of sensitized monkeys had focal mucus cell hyper
134 s also expressed strongly in the trachea and bronchi of the adult lung.
135                           In the trachea and bronchi of the mouse, airway smooth muscle (SM) and cart
136 us 2 (SARS-CoV-2) infections initiate in the bronchi of the upper respiratory tract and are able to d
137 roid, and thymic epithelia, but not trachea, bronchi, or gastrointestinal tract occurred when mice we
138 tive stress (p = 0.002), airway obstruction (bronchi: p = 0.001, bronchioli: p = 0.013), parenchymal
139 n through most of the tubular gas-exchanging bronchi (parabronchi), whereas in the lungs of mammals a
140                                           In bronchi precontracted by carbachol, the IP receptor agon
141 rations of PGE2 (0.01-1 mumol/L) relaxed the bronchi precontracted by histamine.
142 r malacia that involved the trachea and main bronchi (reduction in cross-sectional area of more than
143 ecting emphysematous parenchyma with central bronchi, releasing trapped air.
144 stable radiation-induced fibrosis and patent bronchi remained disease free.
145                       DAC of vehicle-treated bronchi resulted in late-phase airway obstruction (appro
146 eversed these obstructions, highlighting the bronchi's contribution to fentanyl-induced airflow obstr
147 pled receptors on the smooth muscle of human bronchi suggests unappreciated therapeutic targets in th
148 ammation that is detected in the trachea and bronchi (termed inflammatory airway disease [IAD]) is mo
149 ns of the paranasal sinuses, middle ear, and bronchi that begin during infancy.
150 n IL-13-induced hyperresponsiveness in human bronchi, the increased Ca(2+) mobilization, or the enhan
151 oduce all tissue components of the lung from bronchi to alveoli by embryo complementation.Objectives:
152          Preliminary data showed that 90% of bronchi (up to third order) measureable on multiplanar C
153 d spacer on filter placed at the ends of the bronchi was measured by spectrophotometry (246 nm).
154 or 10 s every 100 s) of human isolated human bronchi was studied.
155 further characterize the antigen in acute KD bronchi, we examined paraffin-embedded ciliated bronchia
156      Because CXCL17 is strongly expressed in bronchi, we measured it in bronchoalveolar lavage fluids
157   Airway nodules in the trachea and mainstem bronchi were all benign, while lobar and segmental airwa
158                       Human trachea and main bronchi were dissected free of epithelium and connective
159                In each patient six segmental bronchi were evaluated for CT morphometric indices of br
160                          Sections containing bronchi were evaluated for epithelial cell proliferation
161                                     Specific bronchi were exposed to a single DAC on five consecutive
162                      Subsuperior bronchus or bronchi were found on the right side in 44% and on the l
163 he total study population (in whom both main bronchi were imaged) included 25 men and 26 women (mean
164                          Decellularized left bronchi were procured from decellularized porcine lungs
165                                              Bronchi were visible consistently up to the fourth gener
166  tract, from the epiglottis to the secondary bronchi, were imaged.
167 lso holds for the lung CTMCs in the proximal bronchi, whereas the induced MMCs express only four prot
168 ed IgE-receptor-induced contraction of human bronchi, which was blocked by SUCNR1 antagonism.
169 lung, toxic milk expression is restricted to bronchi, while mottled expression is diffuse.
170 nd relaxation were studied in isolated human bronchi with an inner diameter of 1 mm or less.
171 by treatment of both mouse trachea and human bronchi with specific SFK inhibitors.
172                 The trachea, larynx and main bronchi with the right vagus nerve and nodose ganglion w
173 ed efficiently in explants from tracheas and bronchi, with limited replication in alveolar cells.

 
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