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1 cells or primary cultures of F508/F508 human bronchial epithelia.
2 tr(F508/F508) immortalized and primary human bronchial epithelia.
3 focus of the resulting viral infection is in bronchial epithelia.
4 ffects in the mucociliary differentiation of bronchial epithelia.
5 a level 16% of that observed in normal human bronchial epithelia.
6 eration of differentiated human tracheal and bronchial epithelia.
7 hed release of type I and type III IFNs from bronchial epithelia.
8 n direct deposition onto reconstituted human bronchial epithelia.
9 dulate TGF beta-induced IL-8 secretion in CF bronchial epithelia.
10 onal expression at the apical PM in human CF bronchial epithelia.
11 nel in primary CFTRDeltaF508/DeltaF508 human bronchial epithelia.
12 K293T cells and in well-differentiated human bronchial epithelia.
13 Ca(2+)(i) signals in CF compared with normal bronchial epithelia, a response that reflects endoplasmi
14 differentiated primary cultures of tracheal/bronchial epithelia and found that transepithelial condu
15 ia co-expressed p120-3 and p120-1, including bronchial epithelia and mammary luminal epithelial cells
16 not increased in these cultured CF tracheal/bronchial epithelia and point to loss of anion transport
17 sites were significantly increased in normal bronchial epithelia and tumor cells derived from p18(-/-
18 e epithelium is disrupted (e.g. wounded skin/bronchial epithelia) and where T cells frequently are pr
20 clearly demonstrated OPN3 expression in lung bronchial epithelia as well as immune cells, while CHML
21 DeltaF508-CFTR in DeltaF508/DeltaF508 human bronchial epithelia but did not correct a different temp
22 ance tRNA that is particularly rare in human bronchial epithelia, but not in other human tissues, sug
23 colocalization with CFTR in CF human primary bronchial epithelia by proximity ligation assay, immunop
27 ell-differentiated primary cultures of human bronchial epithelia grown at an air-liquid interface.
28 s of primary human nasal epithelia and human bronchial epithelia have established many of the biophys
29 creased Cl(-) secretion in cultured human CF bronchial epithelia (HBE) carrying the G551D gating muta
30 s-treated) normal, and cystic fibrosis human bronchial epithelia (HBE), and a proliferating, single-c
33 ion transport in newborn nasal and tracheal/bronchial epithelia in tissues, cultures, and in vivo.
34 apeutically targeting either TLR2 or IL17 in bronchial epithelia, in the context of morphine, can res
35 30-40-day-old) primary cultures of DeltaF508 bronchial epithelia, indicating this response was indepe
36 xcision repair (NER) pathway in normal human bronchial epithelia (NHBE) and lung fibroblasts (NHLF).
37 first time the stressed mitochondria in the bronchial epithelia of high-fat- or high-fructose-fed mi
38 ithelial cells in the renal tubules), lungs (bronchial epithelia), thymus (epithelial cells inside th
39 restore ion transport in polarized CF human bronchial epithelia to levels comparable to those achiev
41 (6-11-day-old) primary cultures of DeltaF508 bronchial epithelia was lost in long-term (30-40-day-old
42 while deleterious effects were observed when bronchial epithelia were exposed to cysteamine plus the
43 tudy, air-liquid interface cultures of human bronchial epithelia were exposed to different series of