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1 d the impact of primary graft dysfunction on bronchiolitis obliterans syndrome.
2 gnificant risk factor for the development of bronchiolitis obliterans syndrome.
3 rences were observed in the overall onset of bronchiolitis obliterans syndrome.
4 recipients is limited by the development of bronchiolitis obliterans syndrome.
5 y may improve lung function in patients with bronchiolitis obliterans syndrome.
6 the main risk factor for the development of bronchiolitis obliterans syndrome.
7 ection has been linked to the development of bronchiolitis obliterans syndrome.
8 defined as idiopathic pneumonia syndrome or bronchiolitis obliterans syndrome.
9 fts due to acute rejection (AR) or developed bronchiolitis obliterans syndrome.
10 Four patients developed bronchiolitis obliterans syndrome.
11 ociated with an increased risk of developing bronchiolitis obliterans syndrome.
12 n of epithelial to mesenchymal transition in bronchiolitis obliterans syndrome.
13 ortality after lung transplantation (LTX) is bronchiolitis obliterans syndrome.
14 ng transplant patients prior to diagnosis of bronchiolitis obliterans syndrome.
15 reduce the incidence of post-lung transplant bronchiolitis obliterans syndrome.
16 th chronic lung allograft rejection known as bronchiolitis obliterans syndrome.
17 he surviving study subjects remain free from bronchiolitis obliterans syndrome.
19 antly elevated within 3 months of developing bronchiolitis obliterans syndrome (8.3 [1.4-25.1] vs. 3.
22 epatitis C viral RNA (HCV RNA), freedom from bronchiolitis obliterans syndrome, acute rejection, and
24 Thin-section CT studies in six patients with bronchiolitis obliterans syndrome (age range, 2 months t
25 with human cytomegalovirus increases risk of bronchiolitis obliterans syndrome (aHR, 2.88; 95% CI, 1.
26 reported risk factor for the development of bronchiolitis obliterans syndrome, an important cause of
27 smatch model of multiorgan system cGVHD with bronchiolitis obliterans syndrome and a minor MHC mismat
28 study was to investigate the development of bronchiolitis obliterans syndrome and graft loss after L
29 onic lung allograft rejection in the form of bronchiolitis obliterans syndrome and its histopathologi
30 distinct obstructive and restrictive forms: bronchiolitis obliterans syndrome and restrictive allogr
31 term comprises 2 major clinical phenotypes: bronchiolitis obliterans syndrome and restrictive allogr
33 valuates the current diagnostic criteria for bronchiolitis obliterans syndrome and reviews the epidem
34 rom lung transplant recipients who developed bronchiolitis obliterans syndrome and were compared to s
37 hromycin, and montelukast should be used for bronchiolitis obliterans syndrome; and the addition of n
38 tervention in five patients with progressive bronchiolitis obliterans syndrome, anti-TNFalpha treatme
40 odel demonstrated that the increased risk of bronchiolitis obliterans syndrome associated with primar
43 emic steroids are the standard treatment for bronchiolitis obliterans syndrome (BOS) after allogeneic
45 genetic polymorphisms on the development of bronchiolitis obliterans syndrome (BOS) after lung trans
48 the internationally recognized definition of bronchiolitis obliterans syndrome (BOS) and longer follo
49 for chronic graft dysfunction manifested as bronchiolitis obliterans syndrome (BOS) and worse posttr
51 mplications occur frequently post-HSCT, with bronchiolitis obliterans syndrome (BOS) being the most c
54 unosuppressive therapy for the management of bronchiolitis obliterans syndrome (BOS) has been sparsel
55 d for multiorgan system cGVHD and associated bronchiolitis obliterans syndrome (BOS) in a murine mode
56 plant operation on survival and the onset of bronchiolitis obliterans syndrome (BOS) in consecutive l
57 rentiation is associated with development of bronchiolitis obliterans syndrome (BOS) in human lung al
58 after lung transplantation fails to prevent bronchiolitis obliterans syndrome (BOS) in many patients
59 lowing lung transplantation fails to prevent bronchiolitis obliterans syndrome (BOS) in many patients
65 Chronic allograft rejection manifested as bronchiolitis obliterans syndrome (BOS) is the leading c
71 allograft dysfunction (CLAD), presenting as bronchiolitis obliterans syndrome (BOS) or restrictive a
72 ll lung transplant recipients suffering from bronchiolitis obliterans syndrome (BOS) or restrictive a
73 n (AR) and development of chronic rejection, bronchiolitis obliterans syndrome (BOS) remain major lim
76 -obliteration of the allograft airway during bronchiolitis obliterans syndrome (BOS) that occurs afte
78 ement of six lung transplant recipients with bronchiolitis obliterans syndrome (BOS), a condition pre
79 s limited by infectious complications and by bronchiolitis obliterans syndrome (BOS), a form of chron
81 w long-term survival and a high incidence of bronchiolitis obliterans syndrome (BOS), an inflammation
82 gnized, idiopathic pneumonia syndrome (IPS), bronchiolitis obliterans syndrome (BOS), and bronchiolit
83 ssion to chronic rejection that manifests as bronchiolitis obliterans syndrome (BOS), but no biomarke
84 tive (ELR(+)) CXC chemokines associated with bronchiolitis obliterans syndrome (BOS), but the effect
85 n after lung transplantation, manifesting as bronchiolitis obliterans syndrome (BOS), has become the
86 g to progressive airflow obstruction, termed bronchiolitis obliterans syndrome (BOS), is the major ca
87 man lung allograft rejection, represented by bronchiolitis obliterans syndrome (BOS), is the single m
88 ween these disorders and risk for subsequent bronchiolitis obliterans syndrome (BOS), mortality and g
90 allograft dysfunction (CLAD), and especially bronchiolitis obliterans syndrome (BOS), remain dominant
93 n the fibro-obliterative lesion found during bronchiolitis obliterans syndrome (BOS), we hypothesized
94 mputed tomography morphology, mortality, and bronchiolitis obliterans syndrome (BOS)-free survival we
112 The per-protocol analysis shows incidence of bronchiolitis obliterans syndrome (BOS): 1/43 in the Eve
113 enting chronic rejection of lung allografts (bronchiolitis obliterans syndrome [BOS]) and proinflamma
114 ce of all other causes (currently defined as bronchiolitis obliterans syndrome [BOS]) is considered t
115 ar that patients may develop an obstructive (bronchiolitis obliterans syndrome [BOS]) or a restrictiv
119 , 41 patients remained STA, and 37 had CLAD (bronchiolitis obliterans syndrome, BOS, [n = 32] or rest
120 sinophilic BAL predisposed to development of bronchiolitis obliterans syndrome but particularly to re
121 s that CMVIG prophylaxis reduces the risk of bronchiolitis obliterans syndrome, but a controlled tria
122 Acute rejection is a major risk factor for bronchiolitis obliterans syndrome, but noninvasive bioma
123 d a shorter survival and an earlier onset of bronchiolitis obliterans syndrome compared with patients
124 pha as a potential new therapeutic target in bronchiolitis obliterans syndrome deserving of a randomi
126 Adjustment for clinical variables including bronchiolitis obliterans syndrome did not change this re
128 Secondary outcomes included freedom from bronchiolitis obliterans syndrome (fBOS) and rates of ac
130 A trend, however, toward reduced onset of bronchiolitis obliterans syndrome grade 2 or 3 was obser
131 survival in a multivariable model including bronchiolitis obliterans syndrome grade and baseline FEV
134 o activate fibroblasts in the development of bronchiolitis obliterans syndrome has not been evaluated
135 ted rejection, acute cellular rejection, and bronchiolitis obliterans syndrome; however, the signific
136 enance macrolide therapy in the treatment of bronchiolitis obliterans syndrome in lung transplant rec
137 after heart transplantation, and potentially bronchiolitis obliterans syndrome in lung transplant rec
138 ges to the progress of medical management of bronchiolitis obliterans syndrome include difficulties a
139 TF4 axis in the altered airway epithelium of bronchiolitis obliterans syndrome, including substantial
140 tion is associated with an increased risk of bronchiolitis obliterans syndrome independent of acute r
147 ) chronic allograft dysfunction, manifest by bronchiolitis obliterans syndrome, is frequent and limit
149 and were divided into three groups: no CLAD (bronchiolitis obliterans syndrome level 0 [BOS 0]), earl
150 lantation imitate the in vivo development of bronchiolitis obliterans syndrome-like lesions and revea
152 o be important in obliterative bronchiolitis/bronchiolitis obliterans syndrome (OB/BOS), which severe
153 onic lung allograft dysfunction manifests as bronchiolitis obliterans syndrome or the recently descri
154 was not a risk factor for the development of bronchiolitis obliterans syndrome or worse overall survi
155 rvival (P = 0.09) and increased freedom from bronchiolitis obliterans syndrome (P = 0.03) was observe
157 sions in explanted lungs from four end-stage bronchiolitis obliterans syndrome patients was analyzed
160 genesis of chronic lung allograft rejection (bronchiolitis obliterans syndrome) remains to be elucida
161 trated that respiratory viral infection is a bronchiolitis obliterans syndrome risk factor and virus-
164 iated with a significantly increased risk of bronchiolitis obliterans syndrome stage 1 (grade 1: rela
165 acute rejection, lymphocytic bronchitis, and bronchiolitis obliterans syndrome stage 1, using univari
166 sponse to viruses and in the pathogenesis of bronchiolitis obliterans syndrome, the predominant manif
168 the association of bronchial dilatation with bronchiolitis obliterans syndrome was significant (P = .
169 s in the six patients with clinically proved bronchiolitis obliterans syndrome were mosaic perfusion
170 hown to be implicated in the pathogenesis of bronchiolitis obliterans syndrome, which is considered t