ライフサイエンスコーパス: bronchoalveolar lavage fluid

1 llikrein (KLK) 5 and KLK14, were assessed in bronchoalveolar lavage fluid.

2 sed the production of TNF-alpha and MIP-2 in bronchoalveolar lavage fluid.

3 of increased macrophages and eosinophils in bronchoalveolar lavage fluid.

4 creased the numbers of inflammatory cells in bronchoalveolar lavage fluid.

5 ed in tissue extracts, nasal secretions, and bronchoalveolar lavage fluid.

6 ung epithelial cells and readily detected in bronchoalveolar lavage fluid.

7 1) with the percentage of eosinophils in the bronchoalveolar lavage fluid.

8 reased protein and cytokine concentration in bronchoalveolar lavage fluid.

9 ction and asthma-associated cytokines in the bronchoalveolar lavage fluid.

10 and was highly correlated with IFN levels in bronchoalveolar lavage fluid.

11 HR or total inflammatory cell numbers in the bronchoalveolar lavage fluid.

12 Cells were counted in bronchoalveolar lavage fluid.

13 L-13, IL-1beta, IL-33, and eotaxin levels in bronchoalveolar lavage fluid.

14 lls and 20-35% reduction in Th2 cytokines in bronchoalveolar lavage fluid.

15 s product of cellular necrosis, in cell-free bronchoalveolar lavage fluid.

16 ed the influx of inflammatory cells into the bronchoalveolar lavage fluid.

17 ers were assessed in lung tissue, serum, and bronchoalveolar lavage fluid.

18 d resulted in IL-1beta accumulation into the bronchoalveolar lavage fluid.

19 levels of iron-carrier proteins in cells or bronchoalveolar lavage fluid.

20 t least 10(4) colony forming units per mL of bronchoalveolar lavage fluid.

21 r protein leak and inflammatory cells in the bronchoalveolar lavage fluid.

22 Recruited eosinophils were enumerated in bronchoalveolar lavage fluid.

23 lung while also reducing LPA 18:2 content in bronchoalveolar lavage fluid.

24 rase chain reaction using RNA extracted from bronchoalveolar lavage fluid.

25 release from lung epithelium as detected in bronchoalveolar lavage fluid.

26 in (TSLP), IL-9, and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

27 n, and surfactant protein A concentration in bronchoalveolar lavage fluid.

28 the macrophage chemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

29 0.00003), and similar results were found in bronchoalveolar lavage fluids.

30 , IL-5, and IL-13 measured by ELISA in their bronchoalveolar lavage fluids.

31 rol and PNE rat pups: 1) the 5-HT content in bronchoalveolar lavage fluid, 2) the apneic response to

32 lmost 10-fold-higher bacterial burden in the bronchoalveolar lavage fluid 3 h following infection.

33 2 ligands was significantly increased in the bronchoalveolar lavage fluid 48 hours after segmental al

34 The specimens (5 cerebrospinal fluid, 7 bronchoalveolar lavage fluid, 5 plasma, 2 serum, and 1 n

35 o DC subsets (DC2/3 and DC5) are expanded in bronchoalveolar lavage fluid 8 h after lipopolysaccharid

36 urbiprofen augmented the release of IL-33 in bronchoalveolar lavage fluid after Alternaria challenge,

37 okines such as IL-5, IL-13, and eotaxin-1 in bronchoalveolar lavage fluid after OVA challenge.

38 chronic rhinosinusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen c

39 iration caused increased ALI (as assessed by bronchoalveolar lavage fluid albumin concentration) in b

40 ased airway hyperreactivity to methacholine, bronchoalveolar lavage fluid albumin, and serum IgE leve

41 HIV has been recovered from cell-free bronchoalveolar lavage fluid, alveolar macrophages, and

42 concentration was also induced in the serum, bronchoalveolar lavage fluid, alveolar type II epithelia

43 ere associated with sHLA-G concentrations in bronchoalveolar lavage fluid among asthmatic subjects wi

44 healthy volunteers and examine the impact on bronchoalveolar lavage fluid and blood MP repertoire.

45 ar lavage fluid was evaluated by microscopy; bronchoalveolar lavage fluid and blood were assessed by

46 PM2.5 increased neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronc

47 measured by changes in inflammatory cells in bronchoalveolar lavage fluid and cytokine expression in

48 rs of T cells and expression of TNF-alpha in bronchoalveolar lavage fluid and deteriorates lung funct

49 Higher levels of Th2 cytokines were found in bronchoalveolar lavage fluid and draining lymph node cel

50 ifested persistent inflammation in blood and bronchoalveolar lavage fluid and exacerbations despite h

51 recruitment of monocytes and neutrophils in bronchoalveolar lavage fluid and increased neutrophils i

52 augmented by 4MDM, the levels of PGP in the bronchoalveolar lavage fluid and infiltration of neutrop

53 fspring had lower white blood cell counts in bronchoalveolar lavage fluid and less pronounced peribro

54 ikingly reduced numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inf

55 ed a significantly reduced viral load in the bronchoalveolar lavage fluid and lower respiratory tract

56 ine expression in ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assess

57 Flow cytometry and cytokine measurements in bronchoalveolar lavage fluid and lung tissue were follow

58 ical studies and biochemical measurements in bronchoalveolar lavage fluid and lung tissue.

59 uce eosinophil extracellular traps (EETs) in bronchoalveolar lavage fluid and lung tissue.

60 phil infiltration and levels of cytokines in bronchoalveolar lavage fluid and lung tissue.

61 antioxidant responses were assessed in both bronchoalveolar lavage fluid and lung tissue.

62 nd primary lung monocytes/macrophages, mouse bronchoalveolar lavage fluid and lung tissues, and AHR i

63 Bronchoalveolar lavage fluid and lungs were harvested fo

64 neutrophils, lymphocytes, and macrophages in bronchoalveolar lavage fluid and lungs; inflammatory cel

65 Bronchoalveolar lavage fluid and matched blood were obta

66 NETs were also measured in bronchoalveolar lavage fluid and plasma from lung transp

67 vascular tissue to extracellular fluid (eg, bronchoalveolar lavage fluid and plasma).

68 SM reduces inflammatory cell accumulation in bronchoalveolar lavage fluid and proinflammatory cytokin

69 We detected the presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can b

70 ophilic and neutrophilic inflammation in the bronchoalveolar lavage fluid and Th1, Th2, and Th17 infl

71 d polymorphonuclear leukocytes (PMNs) in the bronchoalveolar lavage fluid and the wet-to-dry lung wei

72 MMP-9 is the major MMP found in the bronchoalveolar lavage fluids and bronchial biopsies fro

73 c antibodies were detected in nasal wash and bronchoalveolar lavage fluids and in sera from mice intr

74 ed hyperoxia-induced accumulation of IL-6 in bronchoalveolar lavage fluids and NADPH oxidase (NOX) 2

75 urement and polymorphonuclear recruitment in bronchoalveolar lavage fluid), and lethality were evalua

76 e lungs, (ii) higher amounts of IL-17 in the bronchoalveolar lavage fluid, and (iii) more neutrophils

77 d lower total cell counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of

78 nd disaturated PC in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased

79 were sensitized and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collect

80 alpha and the chemokines eotaxin and CCL2 in bronchoalveolar lavage fluid; and decreased pulmonary in

81 ive plus had greater than 20% lymphocytes in bronchoalveolar lavage fluid; and three had findings on

82 No changes in bronchoalveolar lavage fluid angiotensin-converting enzy

83 l counts with increased extracellular DNA in bronchoalveolar lavage fluid as well as in lung tissue,

84 and nonlymphoid tissues, including lung and bronchoalveolar lavage fluid, as measured by H2-Db NP366

85 ane B2 and immunoglobulin E were measured in bronchoalveolar lavage fluid at baseline and 24 h after

86 oxidative stress), and IgE were measured in bronchoalveolar lavage fluid at baseline and 24 hours af

87 -, and 2.1-fold greater drug levels in lung, bronchoalveolar lavage fluid (BAL), and plasma compared

88 tion of virus-specific memory CD8 T cells in bronchoalveolar lavage fluid (BAL), lungs, and spleen.

89 a-induced lung injury was evaluated by using bronchoalveolar lavage fluid (BALF) analysis and patholo

90 tracellular vesicles (EVs) are detectable in bronchoalveolar lavage fluid (BALF) and culture medium o

91 Lung tissue, bronchoalveolar lavage fluid (BALF) and draining lymph n

92 im of this study was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicti

93 infected with Streptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values

94 d temporal kinetics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumi

95 s the overlap in metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifi

96 tinfection and included cellular profiles in bronchoalveolar lavage fluid (BALF) and serum IgG and Ig

97 tized hosts to assess airway inflammation in bronchoalveolar lavage fluid (BALF) and Th2 responses in

98 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophi

99 -type lymphocytes were assessed in lungs and bronchoalveolar lavage fluid (BALF) by multiparametric f

100 Bacterial burdens, bronchoalveolar lavage fluid (BALF) cell counts, cell ty

101 We compared lung bacterial clearance, bronchoalveolar lavage fluid (BALF) characterization, lu

102 sion, PLTP activity decreased by 80% in COPD bronchoalveolar lavage fluid (BALF) due to serine protea

103 understanding of the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients

104 We studied bronchoalveolar lavage fluid (BALF) from 36 patients wit

105 etabolic profiling of serum, lung tissue and bronchoalveolar lavage fluid (BALF) from a non-lethal mo

106 cytokine/chemokine profiles were measured in bronchoalveolar lavage fluid (BALF) from children with C

107 identification of two biomarkers present in bronchoalveolar lavage fluid (BALF) from chlorine gas ex

108 alysis on gene expression data from cells in bronchoalveolar lavage fluid (BALF) from COVID-19 patien

109 us 6B (HHV-6B) DNA is frequently detected in bronchoalveolar lavage fluid (BALF) from immunocompromis

110 y flow cytometry on neutrophils in blood and bronchoalveolar lavage fluid (BALF) from mechanically ve

111 with human MSCs when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with A

112 by using mass spectrometry) were measured in bronchoalveolar lavage fluid (BALF) from patients with N

113 HR, airway inflammation and cytokines in the bronchoalveolar lavage fluid (BALF) in a murine model of

114 gation & density-gradient fractionation from bronchoalveolar lavage fluid (BALF) in vivo.

115 cytomegalovirus (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV repli

116 Bronchoalveolar lavage fluid (BALF) levels of SOCS3 were

117 ken together with the observation that local bronchoalveolar lavage fluid (BALF) levels of vitamin D

118 emic stroke caused a significant increase in bronchoalveolar lavage fluid (BALF) macrophages and neut

119 induce apolipoprotein E (APOE) expression by bronchoalveolar lavage fluid (BALF) macrophages from ast

120 We measured 13 cytokines in bronchoalveolar lavage fluid (BALF) of 88 children with

121 CD4(+) T cells in the bronchoalveolar lavage fluid (BALF) of Af5517-aspirated

122 We measured iron and ferritin levels in the bronchoalveolar lavage fluid (BALF) of participants enro

123 a diffuse lung injury marked by increases in bronchoalveolar lavage fluid (BALF) protein and histoche

124 ive to air-exposed controls, ozone increased bronchoalveolar lavage fluid (BALF) protein, a marker of

125 rformed a retrospective multicenter study on bronchoalveolar lavage fluid (BALF) samples obtained fro

126 All archived bronchoalveolar lavage fluid (BALF) samples that had pre

127 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of

128 Bronchoalveolar lavage fluid (BALF) was analysed for tot

129 levels and in CA04-neutralizing activity of bronchoalveolar lavage fluid (BALF) was observed followi

130 Lung tissue and bronchoalveolar lavage fluid (BALF) were analyzed for in

131 In parallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by ap

132 d higher levels of Th2 and Th17 cytokines in bronchoalveolar lavage fluid (BALF), accompanied by an i

133 Bronchoalveolar lavage fluid (BALF), airway inflammation

134 iveness, cytokine levels in spleen cells and bronchoalveolar lavage fluid (BALF), and cellular distri

135 4, 17, 21, 25, or 33 d after exposure, SpO2, bronchoalveolar lavage fluid (BALF), and histologic anal

136 mpared with WT mice, with fewer cells in Wsh bronchoalveolar lavage fluid (BALF), despite similar lev

137 Lung function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs we

138 ted biomarkers of aspiration (bile salts) in bronchoalveolar lavage fluid (BALF).

139 in mice resulted in increased OPN levels in bronchoalveolar lavage fluid (BALF).

140 examined the content in MMP active forms of bronchoalveolar lavage fluids (BALf) from male C57BL/6 m

141 d with the concentration of total protein in bronchoalveolar lavage fluids (BALF) from patients with

142 RP3 inflammasome activation, was detected in bronchoalveolar lavage fluids (BALF) in a macrophage- an

143 Quantitative bacterial culture of bronchoalveolar lavage fluids (BALF) is labor-intensive,

144 s) of the allergic response were assessed in bronchoalveolar lavage fluids (BALFs) after allergen cha

145 damage and increased lung injury scores and bronchoalveolar lavage fluid cell numbers in control but

146 Total bronchoalveolar lavage fluid cell numbers increased from

147 bronchitis, perivasculitis, and increases in bronchoalveolar lavage fluid cell numbers were detected

148 resulting in increase in lung injury scores, bronchoalveolar lavage fluid cell numbers, and cellular

149 n reduced the immunosuppressive functions of bronchoalveolar lavage fluid cells, inhibited bone marro

150 generation by severe asthma whole blood and bronchoalveolar lavage fluid cells.

151 evels of interleukin-1beta, tended to reduce bronchoalveolar lavage fluid CINC-3 levels, but no effec

152 ated by quantifying cellular accumulation in bronchoalveolar lavage fluid, collagen levels, and histo

153 s extracted from endobronchial brushings and bronchoalveolar lavage fluid collected from 39 asthmatic

154 Analysis of bronchoalveolar lavage fluid collected from human patien

155 eduction of eosinophil and T cell numbers in bronchoalveolar lavage fluid compared with those in dilu

156 2 223K/K mice had persistent eosinophilia in bronchoalveolar lavage fluid compared with wild-type and

157 tion and IL-17A/F production in the lung and bronchoalveolar lavage fluid compared with wild-type mic

158 a, accompanied with significant reduction in bronchoalveolar lavage fluid concentration of IL-5, a cy

159 Airway inflammation and function, bronchoalveolar lavage fluid cytokine levels, and flow c

160 Outcome measures included: bronchoalveolar lavage fluid cytology to assess airway e

161 d tumor-promoting cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expressio

162 Bronchoalveolar lavage fluid demonstrates greater than 2

163 Quantitative microbiology applied to bronchoalveolar lavage fluid derived from infected segme

164 dies hint toward the proinflammatory role of bronchoalveolar lavage fluid-derived exosomes in asthmat

165 ed levels of cytokines and chemokines in the bronchoalveolar lavage fluid, enhanced bronchoalveolar c

166 ILC2 numbers, proliferation (Ki-67(+)), and bronchoalveolar lavage fluid eosinophil numbers were mea

167 , IL-33(KO)/Tg+ mice had complete absence of bronchoalveolar lavage fluid eosinophilia, accompanied w

168 iduals with cystic fibrosis underwent annual bronchoalveolar lavage fluid examination, and chest comp

169 Our findings validate bronchoalveolar lavage fluid exosomal shuttle RNA as a s

170 Bronchoalveolar lavage fluid exosomes were collected fro

171 fferential cell counts were performed on the bronchoalveolar lavage fluid, followed by histological a

172 We also detect Siglec-F ligand activity in bronchoalveolar lavage fluid fractions containing polyme

173 Bronchoalveolar lavage fluid from 23 lean, 12 overweight

174 17A, neutrophil counts, and total protein in bronchoalveolar lavage fluid from acute respiratory dist

175 Bronchoalveolar lavage fluid from Ad-MD-2s mice transfer

176 Microbiology was done on bronchoalveolar lavage fluid from all patients and venti

177 CG and NE in bronchoalveolar lavage fluid from CF patients both contr

178 ein levels were significantly upregulated in bronchoalveolar lavage fluid from HIV-infected smokers,

179 In bronchoalveolar lavage fluid from human lung transplant

180 In bronchoalveolar lavage fluid from humans with ARDS, gut-

181 patients with severe asthma compared with in bronchoalveolar lavage fluid from individuals without as

182 was consistently inhibited by treatment with bronchoalveolar lavage fluid from inhibitory kappaB kina

183 cell counts were significantly higher in the bronchoalveolar lavage fluid from OVA/OVA than OVA/PBS m

184 s of IL-1beta and IL-8 rapidly determined in bronchoalveolar lavage fluid from patients randomised to

185 vo, histone-C1INH complexes were detected in bronchoalveolar lavage fluid from patients with acute re

186 we found significant elevation of IL-17A in bronchoalveolar lavage fluid from patients with ARDS, an

187 he protein expression patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

188 We use our method to characterize bronchoalveolar lavage fluid from patients with asthma,

189 nase activities were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asth

190 anges in the miRNA composition of EVs in the bronchoalveolar lavage fluid from patients with IAV-indu

191 pulmonary eosinophilia were measured in the bronchoalveolar lavage fluid from patients with mild ast

192 r, specific ceramide species were altered in bronchoalveolar lavage fluid from patients with severe a

193 F-specific IgG is elevated in both serum and bronchoalveolar lavage fluid from Rasgrp1-deficient mice

194 Additionally, bronchoalveolar lavage fluid from this group of hepSTAT3

195 ory cells and cytokines were compared in the bronchoalveolar lavage fluid from WT and SP-D(-/-) mice

196 Treatment of alveolar epithelial cells with bronchoalveolar lavage fluids from ARDS patients drove b

197 Bronchoalveolar lavage fluids from ozone-treated rats re

198 Low concentrations of IL-1beta and IL-8 in bronchoalveolar lavage fluid have been validated as effe

199 und significantly elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tis

200 beta, and chemokines in lung homogenates and bronchoalveolar lavage fluid; however, PMN recruitment i

201 ase-9 and proinflammatory mediator levels in bronchoalveolar lavage fluid, ii) lung parenchymal leuko

202 al aimed to determine whether measurement of bronchoalveolar lavage fluid IL-1beta and IL-8 could eff

203 ry inflammation, eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adop

204 Bronchoalveolar lavage fluid IL-9 and IL-10, serum IL-9,

205 eficient mice had fewer DEP exposure-induced bronchoalveolar lavage fluid immune cells and proinflamm

206 (IL-28A/B) production was attenuated in the bronchoalveolar lavage fluid in all factor-deficient mic

207 ntrations and absolute neutrophil numbers in bronchoalveolar lavage fluid in GSTM1+ but not GSTM1null

208 s, angiotensin-converting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elder

209 Following repetitive O3 exposure, higher bronchoalveolar lavage fluid inflammatory cells were obs

210 Furthermore, a delayed (6-8 hr) increase in bronchoalveolar lavage fluid interleukin-6 concentration

211 ezolid administration sufficiently decreased bronchoalveolar lavage fluid levels of IFN-gamma at day

212 ance responses was associated with increased bronchoalveolar lavage fluid levels of IL-10 and TGF-bet

213 Hypothermia reduced bronchoalveolar lavage fluid levels of interleukin-1beta

214 es in lung epithelial cell proliferation and bronchoalveolar lavage fluid levels of keratinocyte grow

215 asthma resulted in a remarkable increase in bronchoalveolar lavage fluid levels of LPA enriched in p

216 Bronchoalveolar lavage fluid LT levels were increased in

217 kine and chemokine levels were determined in bronchoalveolar lavage fluid, lung homogenates and lung

218 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasa

219 fected myeloid cells were detected in blood, bronchoalveolar lavage fluid, lungs, spleen, and brain,

220 L-9(+) CD4(+) T cells was increased in lung, bronchoalveolar lavage fluid, lymph nodes, and blood of

221 ded throughout the study day, and plasma and bronchoalveolar lavage fluid markers of inflammation wer

222 I are seen in HAECs, in association with low bronchoalveolar lavage fluid mitochondrial DNA and more

223 ergic inflammation, including cell counts in bronchoalveolar lavage fluid, mucin production, ASM mass

224 utum (n = 128), tracheal aspirates (n = 71), bronchoalveolar lavage fluid (n = 152), pleural fluid (n

225 red soluble HLA-G (sHLA-G) concentrations in bronchoalveolar lavage fluid (n = 36) and plasma (n = 57

226 Bronchoalveolar lavage fluid neutrophils, total protein,

227 Bronchoalveolar lavage fluid obtained during this bronch

228 Similarly, bronchoalveolar lavage fluid obtained from human volunte

229 m asthmatic and control lung tissue, (ii) in bronchoalveolar lavage fluid obtained from non-severe an

230 cytokines, chemokines, and growth factors in bronchoalveolar lavage fluid of 20 stable patients, 20 p

231 P-ribosyl-HNP-(ornithine) were isolated from bronchoalveolar lavage fluid of a patient with idiopathi

232 cells in lymph nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques

233 1) levels were significantly elevated in the bronchoalveolar lavage fluid of all mice infected with C

234 OSM levels were also increased in bronchoalveolar lavage fluid of allergic asthmatic patie

235 Consistently, the IL-22 expression in bronchoalveolar lavage fluid of asthmatic patients inver

236 we report that SOCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria

237 icantly reduced the number of neutrophils in bronchoalveolar lavage fluid of C3aR-deficient mice.

238 Exosomes were isolated and detected from bronchoalveolar lavage fluid of control and asthmatic mi

239 g immunohistochemistry and Luminex assays in bronchoalveolar lavage fluid of healthy and asthmatic pa

240 med that leukotriene levels are increased in bronchoalveolar lavage fluid of HIV-infected patients.

241 As we found ILC3-like cells in the bronchoalveolar lavage fluid of individuals with asthma,

242 -promoting cytokines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

243 oxidase were more frequently detected in the bronchoalveolar lavage fluid of lung transplant patients

244 of desialylated MUC1-ED were elevated in the bronchoalveolar lavage fluid of mechanically ventilated

245 f RAGE was determined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum

246 5-HT levels are increased in bronchoalveolar lavage fluid of mice and people with ast

247 shown to reduce the total number of cells in bronchoalveolar lavage fluid of mice challenged with hou

248 F) in the medium of B20-treated cells and in bronchoalveolar lavage fluid of mice exposed to B20 were

249 crophages, neutrophils, and cytokines in the bronchoalveolar lavage fluid of mice than its mutant cou

250 identify host proteins contained within the bronchoalveolar lavage fluid of mice that are cleaved an

251 rply increased the levels of UDP detected in bronchoalveolar lavage fluid of mice.

252 NF-alpha, IL-9, CXCL1, CCL2, and CCL5 in the bronchoalveolar lavage fluid of RSV-infected mice, witho

253 lenge, and eosinophils were increased in the bronchoalveolar lavage fluid of wild-type mice.

254 HDM, elevated the levels of chemerin in the bronchoalveolar lavage fluid of WT mice.

255 ncreased ATP concentrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

256 ar species and the level of ATX exoenzyme in bronchoalveolar lavage fluids of human patients with ast

257 5-HT levels were measured in bronchoalveolar lavage fluid or serum of animals with AA

258 ns and polymorphonuclear cell recruitment in bronchoalveolar lavage fluid (p<0.05 for both).

259 y/severity; (2) RSV-specific CD8+ T cells in bronchoalveolar lavage fluid preinfection (subjects with

260 e exposure of vehicle-treated rats increased bronchoalveolar lavage fluid protein, albumin, neutrophi

261 Th2 cytokines including IL-4 and IL-5 in the bronchoalveolar lavage fluid relative to control mice.

262 Higher numbers of Asm(-/-) T cells in bronchoalveolar lavage fluid released lower levels of IL

263 The assay is validated for testing bronchoalveolar lavage fluids, replacing the requirement

264 ay and analyses of the injury markers in the bronchoalveolar lavage fluid, respectively.

265 resulted in hypovirulence, while analysis of bronchoalveolar lavage fluid revealed that tumor necrosi

266 s with that of conventional culture by using bronchoalveolar lavage fluid samples from patients with

267 istress syndrome 1, we used paired serum and bronchoalveolar lavage fluid samples obtained within 48

268 Endobronchial biopsies and bronchoalveolar lavage fluid samples were collected from

269 tained pulmonary or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflam

270 ynthase similarly attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mic

271 ption factor GATA3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of

272 ith severe asthma had increased HA levels in bronchoalveolar lavage fluid that correlated with pulmon

273 ity in lymphoid tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate func

274 ther report that intra-alveolar coagulation (bronchoalveolar lavage fluid thrombin-antithrombin compl

275 ficantly reduced histopathologic changes and bronchoalveolar lavage fluid total protein (endothelial

276 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA

277 ressive alveolar neutrocytosis and increased bronchoalveolar lavage fluid tumor necrosis factor-alpha

278 yte count (p = 0.03) and a minor increase in bronchoalveolar lavage fluid tumor necrosis factor-alpha

279 CINC-3 levels, but no effect was observed on bronchoalveolar lavage fluid tumor necrosis factor-alpha

280 ation and analysis of Aspergillus conidia in bronchoalveolar lavage fluid using the combination of tr

281 determined by whole-body plethysmography and bronchoalveolar lavage fluid was analyzed for cellular c

282 Bronchoalveolar lavage fluid was analyzed for inflammato

283 gillus species, Streptococcus pneumoniae) in bronchoalveolar lavage fluid was associated with clinica

284 Bronchoalveolar lavage fluid was collected from 23 stero

285 Bronchoalveolar lavage fluid was collected from patients

286 One day after treatment, bronchoalveolar lavage fluid was collected to determine

287 Bronchoalveolar lavage fluid was evaluated by microscopy

288 Mouse bronchoalveolar lavage fluid was harvested for cell coun

289 had a separate donor; however, pretransplant bronchoalveolar lavage fluid was only available from the

290 The concentration of CXCL12 in plasma and bronchoalveolar lavage fluids was quantified by ELISA.

291 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower

292 third MR imaging examination, eosinophils in bronchoalveolar lavage fluid were counted.

293 Bacterial counts in homogenized lungs and bronchoalveolar lavage fluid were decreased after cranbe

294 Levels of cytokines and chemokines in bronchoalveolar lavage fluid were measured by enzyme-lin

295 Aliquots of blood, sputum, and bronchoalveolar lavage fluid were obtained from asthma s

296 While total and differential cell counts in bronchoalveolar lavage fluid were similar between the Sy

297 reased numbers of T cells and neutrophils in bronchoalveolar lavage fluid, whereas expression of Foxp

298 trophil apoptosis and reduce neutrophilia in bronchoalveolar lavage fluid, while IL-6 increases rapid

299 increased the numbers of eosinophils in the bronchoalveolar lavage fluid, while simultaneously decre

300 ocessed 25-kDa IL-33 protein was detected in bronchoalveolar lavage fluids without any exogenous stim