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1 n the oral mucosa measured in the tongue and buccal mucosa.
2 ocytes, urinary epithelial cells (UECs), and buccal mucosa.
3 tion occurred across inflamed but not normal buccal mucosa.
4 lesions, but also in grossly normal adjacent buccal mucosa.
5 rboring the mutation in her blood, skin, and buccal mucosa.
6 s of the surface of the dorsal tongue or the buccal mucosa.
7  the superficial squamous cell layers of the buccal mucosa.
8  connective tissue of the facial flap to the buccal mucosa.
9 e skin biopsy, as well as from the patient's buccal mucosa.
10  be taken from either perilesional or normal buccal mucosa.
11  (n = 678) gel, 0.5 mL/kg, massaged into the buccal mucosa 1 hour after birth.
12 ne were initiated at three sites through the buccal mucosa: 1) 6 mm apical to the cemento-enamel junc
13 n blood (15%), urinary epithelium (75%), and buccal mucosa (58%).
14  and, surprisingly, also in normal-appearing buccal mucosa adjacent to HNSCC (see the related article
15 membrane (blister formation) of normal human buccal mucosa, after 48 hours, in organ culture.
16         Parents were asked to collect both a buccal mucosa and an anal swab from their children once
17 were compared, to optimize cellular yield in buccal mucosa and cornea.
18  major salivary glands, tongue, gingiva, and buccal mucosa and detected beta-defensin peptides in sal
19  to have tongue malignancy (82%) followed by buccal mucosa and gingivobuccal sulcus malignancy (18%).
20  from peripheral samples (blood, saliva, and buccal mucosa) and correlate it to phenotypic outcomes.
21  tract and by pigmented macules of the lips, buccal mucosa, and digits.
22 the m.14487T>C mutation in his blood, urine, buccal mucosa, and hair follicle DNA samples, while the
23 dles eliciting greater immunogenicity in the buccal mucosa, and shorter microneedles eliciting greate
24  in plaque, others in keratinized gingiva or buccal mucosa, and some oligotypes were characteristic o
25 dents of three oral habitats: tongue dorsum, buccal mucosa, and supragingival plaque.
26  was detected in the gingiva, parotid gland, buccal mucosa, and tongue.
27 tion of the caudal oral mucosa, alveolar and buccal mucosa, and varying degrees of periodontal diseas
28 amples from the tongue, tonsils, throat, and buccal mucosa; and stimulated and unstimulated saliva.
29                                          The buccal mucosa appeared as an intermediate ecological nic
30 o detect chromatin packing domain changes in buccal mucosa as a lung cancer biomarker: chromatin-sens
31 s, we have investigated the potential of the buccal mucosa as an alternative delivery route for ShK b
32 ening sites included the forehead, nostrils, buccal mucosa, axilla, antecubital fossa, groin, and toe
33 ted AKT (pAKT) in 15 paired tumor, skin, and buccal mucosa biopsies (at baseline and after 1 month of
34 om LL explants compared to explants from the buccal mucosa (BM), HP, and transition zone of the lower
35 hesized that chromatin structural changes in buccal mucosa can be predictive of lung cancer.
36  with higher than 90% accuracy in diagnosing buccal mucosa cancer.
37   Prior studies have shown that cells in the buccal mucosa carry a molecular signature of lung cancer
38 seems to bind more to the tongue than to the buccal mucosa cell line, but this difference is overcome
39 objective was to monitor lycopene changes in buccal mucosa cells (BMCs) in response to 3 vehicles for
40 ion protein Cx43 were markedly diminished in buccal mucosa cells from arrhythmogenic cardiomyopathy p
41                                              Buccal mucosa cells from arrhythmogenic cardiomyopathy p
42 esmosomal protein desmoplakin was reduced in buccal mucosa cells from patients with mutations in DSP,
43 mosomal protein plakophilin-1 was reduced in buccal mucosa cells in patients with PKP2 mutations but
44    Inhibition of prelamin A farnesylation in buccal mucosa cells of patients treated with SCH66336 wa
45 , and 15 wk and 2 mo after the study, serum, buccal mucosa cells, and adipose tissue were analyzed fo
46  By comparing the DNA from leukemic cells to buccal mucosa cells, LOH was detected with markers D5S47
47 potential surrogate tissue, we characterized buccal mucosa cells.
48  patients, the mutation was also detected in buccal mucosa cells.
49  unfixed methods applied to exfoliated oral (buccal) mucosa cells.
50  including decreases in microcirculations of buccal mucosa, cerebral microvascular flow was preserved
51  A body tissue that is easy to access is the buccal mucosa (cheek cells).
52                   It was specific, as normal buccal mucosa did not share these genomic alterations.
53  The simultaneous presence of lesions on the buccal mucosa, grade of lesion extension, and presence o
54            The delivery of drugs through the buccal mucosa has attracted much research interest over
55 aps more germane to epithelial malignancies, buccal mucosa have provided surrogate tissues that allow
56             This study demonstrated that the buccal mucosa is a promising administration route for th
57 ups based on similar community compositions: buccal mucosa, keratinized gingiva, hard palate; saliva,
58 (+) cells equally distributed throughout the buccal mucosa of OPC(-) persons (HIV(-) or HIV(+)), irre
59 elialized surface of the facial flap and the buccal mucosa of the vestibule.
60 plant mucosa, but the effect of grafting the buccal mucosa on buccal bone thickness (BBT) has not bee
61 of selegiline, which is absorbed through the buccal mucosa producing higher plasma levels of selegili
62 c verrucous papules on the lips, tongue, and buccal mucosa refractory to multiple excisions.
63 th the best sensitivity were the leg and the buccal mucosa, respectively (82.6% and 52.2%; P = 0.003)
64                                              Buccal mucosa samples and peripheral blood mononuclear c
65 ryptic population of R. mucilaginosa in many buccal mucosa samples.
66 ue derived from lymphoblastoid cell lines or buccal mucosa scrapings.
67                              A total of 7366 buccal mucosa swabs and 5907 anal swabs were analyzed.
68 f fluorescent 5-Fam-ShK to untreated porcine buccal mucosa, there was no detectable peptide in the re
69 f the attached gingiva, alveolar mucosa, and buccal mucosa to gain insight into human tissue performa
70  cancer, and simultaneous involvement of the buccal mucosa, tongue, and palate was more common in thi
71 nols, with two oral models (tongue (HSC3) or buccal mucosa (TR146) was evaluated.
72 f PBD-1 in the dorsal tongue surface and the buccal mucosa was estimated at 20 to 100 micrograms/ml.
73 nactivation patterns in peripheral blood and buccal mucosa were compared between monochorionic MZ (MC