ライフサイエンスコーパス: bulbectomy

1 f the ipsilateral piriform cortex 24 h after bulbectomy.

2 wing lesioning of the olfactory bulb, termed bulbectomy.

3 hat were subjected to a unilateral olfactory bulbectomy.

4 neurons in rat olfactory cortex after total bulbectomy.

5 l or unilateral with contralateral olfactory bulbectomy.

6 ion after methyl bromide lesion or olfactory bulbectomy.

7 yl bromide lesion, but not at any time after bulbectomy, a large fraction of residual, marker-confirm

8 D2 mRNA was eliminated after bulbectomy, a manipulation known to cause retrograde deg

9 Olfactory bulbectomy, an established model of depression that redu

10 eneration of olfactory neurons by unilateral bulbectomy and methimazole treatment.

11 sal thalamic (MD) lesions plus contralateral bulbectomy and transection of the anterior commissure (A

12 a second group of rats received a unilateral bulbectomy and were sacrificed 24 h later.

13 of the effect was less than that produced by bulbectomy, and enhanced TUNEL labeling was apparent bot

14 In female degus, olfactory bulbectomies (BX) inhibit socially-facilitated reentrain

15 Bilateral olfactory bulbectomy (BX) or bilateral transection of the rostral

16 In addition, olfactory bulbectomy can be used for investigating molecular mecha

17 ed to assess antidepressant onset: olfactory bulbectomy, chronic mild stress, chronic forced swim tes

18 layer Ia that was markedly reduced following bulbectomy, consistent with expression of mGluR7 in mitr

19 sed rate of neurogenesis caused by olfactory bulbectomy had little effect on the transcriptional prof

20 Bulbectomy had no effect on MePD morphology with one exc

21 Bulbectomy impaired operant social interaction without a

22 osis following de-afferentation by olfactory bulbectomy in adult rats.

23 e expression of plunc was up-regulated after bulbectomy in respiratory epithelium.

24 Thus, the olfactory bulbectomy model of depression may share some similariti

25 Neither bulbectomy nor naris closure affected TUNEL labeling in

26 The present study examined if olfactory bulbectomy (OBX) altered defensive behaviors on the elev

27 perated rats, rats made anosmic by olfactory bulbectomy (OBX) failed to compensate for the dilution o

28 Olfactory bulbectomy (OBX) in SWR outbred male mice lengthened the

29 sensory neurons (OSNs) induced by olfactory bulbectomy (OBX) leads to the activation of resident mac

30 gorus) were subjected to bilateral olfactory bulbectomy (OBx) or a sham surgical procedure, and were

31 Olfactory bulbectomy (OBX) resulted in several transient, early-on

32 f young and aged rats subjected to olfactory bulbectomy (OBX), a procedure that reproduces many of th

33 nic mild stress (CMS) paradigm and olfactory bulbectomy (OBX).

34 iform cortex (PFx)-after bilateral olfactory bulbectomy (OBX).

35 gnificantly affected at 3 d or 3 weeks after bulbectomy (OBX).

36 First, we conducted bulbectomy on both male and female rats to evaluate the

37 Next, we assessed the effect of bulbectomy on rats given a choice between social interac

38 The effects of olfactory bulbectomy on the acoustic startle reflex and shock-indu

39 re-uptake inhibitor, reversed the effects of bulbectomy on the mean level of activity and significant

40 nic mild stress (CMS) paradigm and olfactory bulbectomy paradigm.

41 The results confirmed that unilateral bulbectomy produced a dramatic increase in TUNEL labelin

42 ute and sustained injury following olfactory bulbectomy remains intact even in very old animals.

43 These data suggest that olfactory bulbectomy results in an increased vulnerability to stre

44 Three days after a unilateral olfactory bulbectomy, Sema3A transcript levels increased in regene

45 ndergo classical apoptosis within 24 h after bulbectomy via transsynaptic, but not retrograde, signal