ライフサイエンスコーパス: bulge

1 n of the anterior urethra ending in a smooth bulge.

2 in/Wnt pathway activity in the dental lamina bulge.

3 of stem cells out of their niche in the hair bulge.

4 t decatenation activity on DNA braids with a bulge.

5 ved in RluF and so RluF cannot stabilize the bulge.

6 trated with a backbone nick and extrahelical bulge.

7 zzled-related protein 1 disappearance in the bulge.

8 bluish discoloration of a palpebral surface bulge.

9 cle stem cells residing in the telogen basal bulge.

10 he midvein, and appearance of the primordium bulge.

11 rs, three double-chain-reversal loops, and a bulge.

12 ons in different compartments, including the bulge.

13 ighly conserved stem-loop I and a downstream bulge.

14 with their stoma complicated by a parastomal bulge.

15 ct on HRQoL was independent of time with the bulge.

16 ns compared to patients without a parastomal bulge.

17 stline lies along the ice sheet's peripheral bulge.

18 iligo bulge compared with untreated vitiligo bulge.

19 instead dispersing to form galactic disks or bulges.

20 nd afterwards migrate inwards to form galaxy bulges.

21 ified at different positions with 1- to 5-bp bulges.

22 nts are interspersed with internal loops and bulges.

23 uster around the S6 gate and distinctive pai-bulges.

24 braids of intact dsDNA and nicked dsDNA with bulges.

25 g the star-formation histories of the galaxy bulges.

26 nts are interspersed with internal loops and bulges.

27 to mitigate the risk of port-site hernia and bulging.

28 of interest were SSI, hernia recurrence, and bulging.

29 s 34.1%; P < .01) but more clinical cases of bulging (21.5% vs 1.3%; P < .01) and port-site hernia (2

30 By contrast, single-base bulges 6'U7' and 6'A7' on the target strand are looped-o

31 Our studies establish that single-base bulges 7T8, 5A6 and 4A5 on the guide strand are stacked-

32 sts of a 2'-5' phosphodiester bond between a bulged adenosine and the 5' end of the intron.

33 ds the Dicer processing site and an adjacent bulge, affording a 100-fold increase in potency.

34 itial, partially subtended spherical bulge ("bulging") after cell wall digestion occurs on a characte

35 terior muscle herniated and produced a focal bulge along the anterior aspect of the leg.

36 nique structural features such as an adenine bulge and a G.G.T base triple capping structure formed b

37 aining a precisely located single-nucleotide bulge and a GNRA tetraloop.

38 The associated sequence pattern, a 3-nt bulge and G-A, A-G base-pairs, generates an angle of app

39 ilinear contact length of 1.5 cm or capsular bulge and irregularity were grade 1, both features were

40 ar contact length, 88 of 208 (42%); capsular bulge and irregularity, 78 of 175 (45%); and EPE visible

41 s by a thymine, which forms a single-residue bulge and is projected out of the G-tetrad core.

42 We show the bulge and nexus are necessary for DNA cleavage and demon

43 f-target sites, each harboring a single-base bulge and one to three mismatches to the guide strand.

44 ramolecular G-quadruplex containing a single bulge and present evidence for extensive occurrence of b

45 n 15 negative (K15-) stem cells in the upper bulge and remained associated with the K15- upper bulge

46 hich enters the TAR major groove between the bulge and the central loop.

47 o conserved motifs, the Adenine-guanine (AG) bulge and the distal loop.

48 genic factor Egfl6 was expressed by the K15- bulge and was localized adjacent to the vascular annulus

49 Bulged and mispaired eG contexts, which can form during

50 rease of label-retaining cells (LRCs) in the bulges and enhanced CFE in vitro.

51 also analyzed the most frequently occurring bulges and internal loops for each RNA class and found t

52 ynamic ensembles of flexible RNAs containing bulges and internal loops.

53 cal disorder in noncanonical residues within bulges and loops and resulted in 0.3-4-fold reduction in

54 hal: cells become filamentous, form envelope bulges and lyse, resembling treatment with beta-lactam a

55 g support for genomic searching with DNA/RNA bulges and mismatches of arbitrary size as specified by

56 roduce inaccurate results for motifs such as bulges and mismatches.

57 Spontaneous formation of ssDNA bulges and their diffusive motion along SSB surface was

58 IC, and leads to K(+)-mediated mitochondrial bulging and depolarization.

59 Here, we analyze the dynamics of membrane bulging and lysis in Escherichia coli, in which the form

60 d with fewer SSIs but more clinical cases of bulging and with the risk of developing a port-site hern

61 rs, a folded 2X2 (GU/UA) internal loop, a UU bulge, and a flexible AGUGA apical loop.

62 The ion exchange reactions affected bump, bulge, and crack formation on the biotite basal plane, a

63 he 2-helix junction structure contains a GGA bulge, and purine-rich bulges are common motifs in RNA s

64 s than RuvC, cleaving a variety of branched, bulged, and flap-containing substrates.

65 evealed the formation of helical turns, beta-bulges, and alpha-turns in cyclic peptides cross-linked

66 lial stem cells (EpSCs) in the hair follicle bulge are required for hair follicle growth and cycling.

67 t Tcf3-expressing cells in the hair follicle bulge are self-renewing stem cells with multilineage pot

68 ucture contains a GGA bulge, and purine-rich bulges are common motifs in RNA secondary structure.

69 l self-cleaving ribozymes containing adenine bulges are consistent with the hypothesis that these sma

70 lls-epithelial cells located in the follicle bulge-are activated by periodic beta-catenin activity, w

71 The bulge area of the hair follicle contains hair-follicle-a

72 ld also open the possibilities of exploiting bulges as recognition elements for interactions between

73 er than being halo stars passing through the bulge, as expected for stars formed at redshifts greater

74 condary structure as in the ribosome, with a bulge at A2602, but with 5-FU2605 flipped into the activ

75 form by attachment to a surface via a large bulge at the base of the flagellum, which is then remode

76 a stable alignment for the looped-out 6'N7' bulge base, which stacks on the unpaired first base of t

77 s of different lengths and the presence of a bulge between the G-quartets are structural elements tha

78 This progresses to gagging, gasping, eye bulging, bradycardia, cyanosis, and vomiting.

79 ernia recurrences and more clinical cases of bulging (bulging not associated with recurrence or serom

80 of an initial, partially subtended spherical bulge ("bulging") after cell wall digestion occurs on a

81 HR is associated with more clinical cases of bulging but fewer SSIs.

82 m ( approximately 10 mum thick) causes it to bulge by tens of microns.

83 Our results show that bulges can be formed in many different situations within

84 We show that bulging can be energetically favorable due to the relaxa

85 the CD200+/K15+ bulge compared to other non-bulge CD34+ and K15+ progenitor compartments and found t

86 r, Brg1 is indispensable for maintaining the bulge cell reservoir.

87 of this model reproduce straight, bent, and bulged cell shapes, and we discuss how this model is con

88 Without Brg1, bulge cells are depleted over time, partly through the e

89 Ablating Wnt signaling in the bulge cells causes them to lose their stem cell potency

90 Bulge cells express secreted Wnt inhibitors, including D

91 e into the cytokeratin 6-positive (K6) inner bulge cells in telogen, which regulate the quiescence of

92 h, whereas paracrine Wnt inhibition of inner bulge cells reinforces differentiation.

93 Bulge cells show increased levels of epigenetic repressi

94 dgehog (Shh) signals Gli to activate Brg1 in bulge cells to begin hair regeneration, whereas Brg1 rec

95 eling machinery, is dynamically expressed in bulge cells to control tissue regeneration and repair.

96 Hair follicle stem cells (bulge cells) are essential for hair regeneration and ear

97 gical abnormalities, including shortened and bulging cells, suggesting a cell wall defect.

98 al" i.e. their seed region comprises G:U and bulge combinations.

99 ive cell cycle inactivity of the CD200+/K15+ bulge compared to other non-bulge CD34+ and K15+ progeni

100 nocytes captured from NBUVB-treated vitiligo bulge compared with untreated vitiligo bulge.

101 insertions ('DNA bulge') or deletions ('RNA bulge') compared to the RNA guide strand, and Cas9 nicka

102 ite induces an intermediate state in which a bulge conformation at Ser-382 in a transmembrane helix i

103 in which the peptides adopted distinct super-bulged conformations.

104 Midcell bulges contained bands or foci of FtsA-GFP and FtsZ-GFP

105 e antimelanoma tissue habitat, namely in the bulge, could help to recreate a similar melanoma-suppres

106 morphologies, including filamentation, polar bulging, curving, and, surprisingly, loss of viability o

107 We identify two bulge-depleted regions on the miRNA stem, located approx

108 The height of the bulge deposited directly next to the channel was reduced

109 Cav1.2 regulates production of the bulge-derived BMP inhibitor follistatin-like1 (Fstl1), d

110 Reducing the bulge does not interfere with miR398-mediated regulation

111 The distance distributions for the AAA bulge duplex (3A-DNA) with six different Au-Au pairs pro

112 Dkk3 continues to be localized to the inner bulge during the hair cycle growth phase.

113 vo expansion of hEPI-NCSC isolated from hair bulge explants, manipulating the WNT, sonic hedgehog and

114 alization within the bulge to the entire sub-bulge follicle and cyst, and ectopic expression of kerat

115 standard k-turn comprises a three-nucleotide bulge followed by G.A and A.G pairs.

116 53 infants were reported to have a bulging fontanelle; 32 (0.3%) in the vitamin A group and

117 ant roothairless 6 (rth6) are arrested after bulge formation during the transition to tip growth and

118 description to include the possibilities of bulge formation should allow the identification of more

119 ce the feature size of microchannels and the bulges formed at the rim of the channel during CO2 laser

120 hod was further extended to assigning a 6 nt bulge from a 61 nt viral RNA element justifying its use

121 A large bulge >10 cm impaired HRQoL (P < 0.01) across all stoma

122 these, 693 (55%) patients with a parastomal bulge had significantly impaired (P < 0.01) HRQoL across

123 loss of aPKClambda altered the fate of lower bulge/hair germ stem cells.

124 metal, and a new DNAzyme (named Ce13) with a bulged hairpin structure was isolated and characterized.

125 The formation of bulges has also been observed in two different G-quadrup

126 o have thicker sclera (P = .067) and greater bulge height (P = .079).

127 The CCT was positively correlated to macular bulge height but not to BCVA.

128 eyes and significantly increased for macular bulge height greater than 350 mum (P = 0.0047).

129 The mean macular bulge height was 407.7 +/- 215.1 mum (120-1130) and was

130 inal, choroidal, and scleral thicknesses and bulge height were measured on SD OCT.

131 Choroidal thickness, scleral thickness, and bulge height were positively correlated (P < .01).

132 ing between the single-stranded regions of a bulged helix and a hairpin loop.

133 However, once activated, bulge HF-SCs begin to differentiate into epidermal cells

134 GT) embryonic cilia exhibited shortening and bulging, highly similar to the characterised retrograde

135 We further discuss how mimicking the bulge immune privilege may be an effective melanoma prev

136 nign diagnosis or an ileostomy, a parastomal bulge impacted significantly on Social Functioning and M

137 Importantly, IRES mutations that delete the bulge impair viral translation and completely inhibit re

138 t to the outer domain into the emerging hair bulge in Arabidopsis (Arabidopsis thaliana).

139 The same process also generates a bulge in the non-target DNA strand, enabling its handove

140 with disease progression that might cause a bulge in the prostate gland.

141 A hydrogen-bonding network stabilizes the bulge in the RluB-RNA but is not conserved in RluF and s

142 off-target analysis related to DNA and sgRNA bulges in addition to base mismatches, and suggest speci

143 present evidence for extensive occurrence of bulges in different G-quadruplex contexts.

144 port a model in which the internal loops and bulges in HDV RNA contribute flexibility to the quasi-do

145 es used for paired nicking can also tolerate bulges in one of the guide strands.

146 ile hosting fully grown and already quenched bulges in their cores.

147 We further reveal the prevalence of axon bulging in the brain cortex in vivo after mild compressi

148 As the new hair emerges, the entire old bulge, including its reserve HFSCs and SC-inhibitory inn

149 extremely metal-poor stars in the Milky Way bulge, including one star with an iron abundance about 1

150 es 'closed' loops of various types (hairpin, bulge, internal, and junction loops) and pseudoknots of

151 nus, a disease in which the cornea thins and bulges into a conical shape.

152 r, these findings demonstrate that the upper bulge is associated with a perivascular niche during the

153 ur data suggest that the non-template strand bulge is extruded into solvent in complexes containing a

154 In addition, a thymine bulge is found between G8 and G9.

155 ses HFSCs for the next hair cycle, the older bulge is left unanchored.

156 Finally, we show that when the old bulge is lost with each hair cycle, overall levels of SC

157 m cells leading to their accumulation in the bulges is indicated by a strongly reduced response to mo

158 e further demonstrate that the occurrence of bulges is not a rare phenomenon and should be accounted

159 in violence tend to follow population "youth bulges." Large numbers of adolescents in equatorial regi

160 road conformational ensemble with increasing bulge length.

161 In particular, bulge loop initiation, multibranch loop initiation, AU/G

162 that uranyl binds between T23 and C25 in the bulge loop, G11 and T12 in the stem loop of the enzyme s

163 t RNA complexes of E. coli DsrA-rpoS derived bulge-loop interactions, which underlines the potential

164 e chemically engineered to create and attack bulge-loop regions upon hybridization to target RNA.

165 such as non-canonical, primarily U-G pairs, bulge loops and three-way junctions.

166 y fashion forming hairpins interspersed with bulge loops at distinct positions that enlarged the bind

167 ic manner, with up to 90% cleavage from 5-nt bulge-loops (BC5-alpha and BC5L-beta anomers) through mu

168 st that autocrine Wnt signaling in the outer bulge maintains stem cell potency throughout hair cycle

169 trategy, we found that repigmentation in the bulge MC precursors is driven by KCTD10, a signal with u

170 ranscriptome RNA sequencing of hair follicle bulge melanocyte precursors and compared their gene sign

171 e potential key players in the activation of bulge melanocyte precursors during vitiligo repigmentati

172 of TNC, GJB6, and THBS1 in the hair follicle bulge melanocytes and of TYR in the epidermal melanocyte

173 n and established the role of signature UCAA bulge motif in RT-aptamer interaction.

174 HIV-1 TAR RNA is a two-helix bulge motif that plays a critical role in HIV viral repl

175 is of miR-122-dependent binding revealed a G-bulged motif in addition to canonical motifs.

176 ations in the nucleosomal DNA resulting in a bulge near the SHL2 site.

177 tain histone tail conformations promoted DNA bulging near its entry/exit sites, resulting in the form

178 low-induced elastic deformation manifests as bulging near the cell outflow; bulges that come into con

179 urrences and more clinical cases of bulging (bulging not associated with recurrence or seroma).

180 s may have a modified 8/4 structure with one bulge nucleotide.

181 contains several unique features including a bulged nucleotide and the first crystal structure observ

182 adruplexes in the asymmetric unit, while the bulged nucleotide mediates crystal contacts.

183 ected features that include three individual bulge nucleotides and a C(+)*G-C triple adjacent to a st

184 stabilizes extrahelical conformations of the bulge nucleotides, thereby promoting coaxial stacking of

185 ons between residues on the C-helix and beta-bulge of cytP450 and residues at the end of helix alpha4

186 that vascular annuli formed around the upper bulge of de novo-reconstituted hair follicles before the

187 melanocyte stem cells (McSCs) located in the bulge of hair follicles can regenerate mature melanocyte

188 est stem cells [hEPI-NCSC(s)] present in the bulge of hair follicles.

189 e in its 5'-untranslated region (UTR) with a bulge of six nucleotides opposite to the 5' region of th

190 p putative stem cells to the distal enlarged bulge of the dental lamina that contains quiescent odont

191 thy, but specifically confined in the inward bulge of the staphyloma and secondary to excessive scler

192 OCT (P < .001) were noted inside the inward bulge of the staphyloma between eyes with and without se

193 ratinocyte precursors from the hair follicle bulge of untreated vitiligo skin and vitiligo skin treat

194 n increasing bend angle of DNA duplexes with bulges of one, three, and five adenosine residues, consi

195 n which clumps survive feedback and grow the bulges of present-day galaxies.

196 Stem cells located at the bulges of the hair follicles are responsible for hair cy

197 his entity is characterized by a small focal bulging of the aortic wall outline and/or a limited roun

198 ally be found today in the central regions ('bulges') of galaxies, because they formed in the largest

199 Once locked, Charon raises a permanent tidal bulge on Pluto, which greatly enhances the gravity signa

200 the substorms and the formation of the three bulges on the plasmasphere.

201 e, the size, the position, and the number of bulges on the structure and stability of G-quadruplexes.

202 ays that independently monitor the impact of bulges on TtAgo-mediated cleavage reaction.

203 nd frequent off-target sites with a one-base bulge or up to 13 mismatches between the single guide RN

204 ed interactions are noncanonical, containing bulged or mismatched nucleotides.

205 when DNA sequences contain insertions ('DNA bulge') or deletions ('RNA bulge') compared to the RNA g

206 ilure requiring retreatment or self-reported bulge; or anatomic POP failure requiring retreatment or

207 that longer peptides can also bind by either bulging out of the groove in the middle of the peptide o

208 The amnioserosa tissue bulges outward during the early-to-mid stages of closure

209 ct binding mechanism, namely "bypassing" the bulged peptide and making extensive contacts with the ex

210 ises a range of TCRs that can interact with "bulged" pHLA-I epitopes using unpredictable strategies,

211 go) ternary complexes containing single-base bulges positioned either within the seed segment of the

212 king interactions in a conserved 5'-AUAGC-3' bulge preorganize the adjacent helices at nearly orthogo

213 anocyte (MC) precursors in the hair follicle bulge proliferate, migrate, and differentiate to repopul

214 Structural features, like bulges, provide opportunities for selective chemical tar

215 cked in place because of the permanent tidal bulge raised by Charon.

216 e normal cylindrical shape to a branched and bulging, ramified shape, which we call 'coli-flower'.

217 rst time, the existence of a lipid-dependent bulge region in the pore-forming module of lysenin, whic

218 e emission of annihilation gamma-rays in the bulge region of our Galaxy.

219 e cellular origin of melanoma, reside in the bulge region of the hair follicle (HF), an immune-privil

220 ion in the tissue and, interestingly, in the bulge region of the HF, a major reservoir of epidermal s

221 lanocyte progenitors reside in the bulge/sub-bulge region of the lower permanent portion of the hair

222 escent stem cells (SC) are maintained in the bulge region, and hair bulbs at the base contain rapidly

223 However, Amt-Nea,Azq binds at the bulged region in a similar way than Amt-NeaG4.

224 etantrone intercalates in the characteristic bulged region.

225 etate-induced proliferation and migration of bulge-region stem cells in vivo.

226 in ssDNA regions rather than dsDNA, loop or bulge regions, with flanking bases influencing the degre

227 ing a 5-mer RNA, whereas the template strand bulge remains within the template strand tunnel, exertin

228 ed Ki-67 expression and EdU incorporation in bulge resident K15+ cells, but not in supra/proximal bul

229 a global change in strand register, in which bulge residues pair up with residues in the upper stem,

230 s, one containing a gapped RNA and another a bulged RNA, reveal that conformational changes of an RNA

231 ived progenitor cells, rather than quiescent bulge SCs, for anagen HF repair can be a potential appro

232 (+) basal hair bulb progenitors, rather than bulge SCs, were quickly activated to replenish matrix ce

233 Mutating the bulge sequence to 5'-ACCCC-3' ablates base stacking in t

234 ble from changes in ionic conditions and the bulge sequence, effects that can be understood in terms

235 intraplacental T2-hypointense bands, uterine bulge, serosal hypervascularity, and signs of extrauteri

236 with conformational changes localized to the bulge site, thereby having minimal impact on the cleavag

237 We confirm that most of the metal-poor bulge stars are on tight orbits around the Galactic Cent

238 ce lacking TRs do not have a decrease of the bulge stem cell population.

239 Keratinocytes of the bulge stem cells (SCs) niche of Ctip2(ep-/-) mice prolif

240 by a gradual loss of quiescent hair follicle bulge stem cells and a temporary increase in proliferati

241 a reduction in the number or function of the bulge stem cells could be responsible for this phenotype

242 and intercellular signaling (Shh) to control bulge stem cells during tissue regeneration.

243 Fbeta Smad signalling, which is localized to bulge stem cells in both normal human and murine skin.

244 factor expressed primarily in hair follicle bulge stem cells in mice.

245 Altered function of the bulge stem cells is associated with aberrant activation

246 ifferentiation, and maintenance of epidermal bulge stem cells likely through its role in balancing sy

247 hout the hair cycle quiescent phase in outer bulge stem cells that produce their own Wnt signals.

248 Hair follicle bulge stem cells were completely absent, despite the con

249 expansion and later decline of hair follicle bulge stem cells, accompanied by an enrichment of commit

250 h denervation altered the phenotype of upper bulge stem cells, the vascular annulus persisted in surg

251 ne network governing the homeostasis of hair bulge stem cells, we developed a Keratin 15-driven genet

252 ls but results in depletion of hair follicle bulge stem cells.

253 h simultaneously remodels the structure of a bulge, stem, and apical loop.

254 In bulge-stem-loop constructs of HIV-1 transactivation resp

255 t tRNA-derived sequences with predicted stem-bulge-stem-loop structures are sufficient to mediate mRN

256 y uncharacterized noncanonical long loop and bulged structures.

257 Cs) and melanocyte progenitors reside in the bulge/sub-bulge region of the lower permanent portion of

258 treated cells formed midcell circumferential bulges, suggesting that interrupted PG synthesis destabi

259 y in vitro experiments showing low curvature bulges surrounded by I-BAR domains on giant unilamellar

260 istic timescale of 1 s and the growth of the bulge ("swelling") occurs on a slower characteristic tim

261 anatomic success), (2) no bothersome vaginal bulge symptoms, and (3) no re-treatment for prolapse at

262 tral nucleotides of the DNA form a prominent bulge that contacts the SH3-like domain, while the nucle

263 becomes localized to the Wnt-inactive inner bulge that contains differentiated cells.

264 racterization of pathways and signals in the bulge that control the repigmentation process.

265 is consistent with a frozen tidal-rotational bulge that formed later, at a semi-major axis of about 3

266 ugh FOXC1-deficient HFs can still form a new bulge that houses HFSCs for the next hair cycle, the old

267 by melanocyte precursors from hair follicle bulge that proliferate, migrate, and differentiate into

268 manifests as bulging near the cell outflow; bulges that come into contact with the rigid cell roof i

269 The DNA bulges that form within the transcription bubble in RPIT

270 ere, we show that EMT also occurs within the bulge, the epithelial stem cell (eSC) niche of human sca

271 In contrast to stem cells in the bulge, the identities of the progenitors and the mechani

272 and remained associated with the K15- upper bulge throughout the hair cycle.

273 idine-rich tract that base pairs to a G-rich bulge to form the pseudoknot.

274 -positive cells from localization within the bulge to the entire sub-bulge follicle and cyst, and ect

275 Deletion of the beta-bulge trigger-loop results in both a switch in the prefe

276 pe I' beta-turn and a misfolded state with a bulged turn, providing evidence for distinct conformatio

277 sical titrations reveal that the 5'-AUAGC-3' bulge undergoes a conformational change to assemble a fu

278 The Milky Way bulge underwent a rapid chemical enrichment during the f

279 ns, we substituted the universally conserved bulged uridine (U51) in the P4 helix of circularly permu

280 RNA with metal ions, demonstrating that the bulged uridine coordinates at least one catalytic metal

281 inferred midnight transit times of the three bulges, using the rotation rate of the Earth, coincide w

282 Applications to TAR bulge variants and more complex tertiary RNAs support th

283 CA5, SB27, and SB47, complexed with a "super-bulged" viral peptide (LPEPLPQGQLTAY) restricted by HLA-

284 Investigation of the architectural (i.e. bulge vs. contiguous pairs) and sequence (Watson-Crick v

285 Base stacking in the bulge was investigated using the fluorescent purine anal

286 The height of the macular bulge was measured, and the choroidal thickness was mapp

287 A foveal bulge was not present in 67% of patients.

288 lity regional registries was that parastomal bulging was associated with substantial and sustained im

289 Relative to unpaired adenines in a bulge, Watson-Crick A-T base pairs in dsDNA only conferr

290 Stem cells in the hair follicle bulge were largely protected from apoptosis upon p14(ARF

291 activity of RNAs in which internal loops and bulges were mutated and of synthetically designed RNAs d

292 t in the untreated and treated hair follicle bulge, whereas a possible secondary melanocyte germ comp

293 in2 expression remains confined to the outer bulge, whereas Dkk3 continues to be localized to the inn

294 observing period, the plasmasphere had three bulges which were located at different geomagnetic longi

295 s the tRNA anticodon, and the antiterminator bulge, which base pairs with the tRNA acceptor end.

296 The hair follicle bulge, which contains melanocyte stem cells, was also re

297 SMAD3 binds RNA with large internal loops or bulges with high apparent affinity.

298 cnemii across the whole stride cycle as they bulged within the shared anatomical space.

299 general understanding about the formation of bulges within G-quadruplexes, we systematically investig

300 cal G-quadruplex-forming sequence containing bulges within the PARP1 promoter.