ライフサイエンスコーパス: bulimia

1 se of procedural justice than did those with bulimia.

2 with 12 female volunteers who had never had bulimia.

3 OCD symptoms persist after recovery from bulimia.

4 elated and contribute to the pathogenesis of bulimia.

5 mptoms persist after long-term recovery from bulimia.

6 r between alcohol dependence and anorexia or bulimia.

7 r disorder, alcohol addiction, anorexia, and bulimia.

8 examined the benefits of two treatments for bulimia: 1) fluoxetine, an antidepressant medication, an

9 ine dependence but not alcohol dependence or bulimia; (2) greater number of episodes, more impairment

10 l disorders, the highest ORs being seen with bulimia and alcohol and other drug dependence.

11 therapy may also be applicable to adolescent bulimia and binge eating.

12 Women with bulimia and those who had recovered from bulimia had sim

13 such as neurodegenerative diseases, obesity, bulimia, and anorexia nervosa.

14 ssions of OCD, the binge eating behaviors of bulimia, and the self-starvation of anorexia.

15 olics after controlling for other disorders, bulimia did occur at a greater than expected rate.

16 subscales of the Eating Disorders Inventory (bulimia, drive for thinness, maturity fears, perfectioni

17 lting positive feedback cycle escalates into bulimia for many, and anorexia in a few.

18 ervosa, 29 women who had been recovered from bulimia for more than 1 year, and 19 healthy female comp

19 ith bulimia and those who had recovered from bulimia had similar Yale-Brown scale scores and endorsed

20 he Yale-Brown scale scores of the women with bulimia (mean = 13.1, SD = 10.6) and those who had recov

21 SD = 10.6) and those who had recovered from bulimia (mean = 7.9, SD = 7.0) were significantly higher

22 1%]), anorexia nervosa (6 of 79 [7.6%]), and bulimia nervosa (7/79 [8.9%]).

23 into two groups: patients with anorexia and bulimia nervosa (ABN; n = 30) and control patients (CN;

24 ong, positive dose-response association with bulimia nervosa (adjusted hazard ratio = 1.15, 95% confi

25 We enrolled patients with severe bulimia nervosa (at least seven coupled binge/vomit epis

26 hotherapy in reducing behavioral symptoms of bulimia nervosa (binge eating and vomiting).

27 on in anorexia nervosa (AN) or overeating in bulimia nervosa (BN) a form of addiction?

28 EDs) such as binge eating disorder (BED) and bulimia nervosa (BN) among military personnel, less is k

29 Bulimia nervosa (BN) and binge eating disorder (BED) are

30 Anorexia nervosa (AN) and bulimia nervosa (BN) are disorders characterized by abno

31 ED data are presented here and compared with bulimia nervosa (BN) data in the World Health Organizati

32 effective treatments, many individuals with bulimia nervosa (BN) do not receive evidence-based thera

33 Bulimia nervosa (BN) has been associated with dysregulat

34 Women with bulimia nervosa (BN) have disturbances of mood and behav

35 the development of anorexia nervosa (AN) and bulimia nervosa (BN) in women.

36 Bulimia nervosa (BN) is characterized by dysregulated in

37 Bulimia nervosa (BN) is strongly familial, and additive

38 t as follows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n = 82; Recovered AN n = 90; Health

39 Neuroimaging studies of bulimia nervosa (BN) report reduced activity in frontost

40 hree 'classical' EDs [anorexia nervosa (AN), bulimia nervosa (BN), and binge eating disorder (BED)].

41 with future onset of anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), and p

42 trol circuits in adolescents and adults with bulimia nervosa (BN), but less is known about the micros

43 disorders, such as anorexia nervosa (AN) and bulimia nervosa (BN), have genetic and environmental und

44 in adolescent and adult female subjects with bulimia nervosa (BN).

45 =13.9), significantly longer than for either bulimia nervosa (mean=5.8 years, SD=9.1) or anorexia ner

46 either DSM-IV anorexia nervosa (n = 136) or bulimia nervosa (n = 110) between January 1, 1987, and D

47 tracts addressing anorexia nervosa (n = 13), bulimia nervosa (n = 6), and binge-eating disorder (n =

48 ficantly diminished and women recovered from bulimia nervosa (N=14) had significantly elevated hemody

49 as there were about anorexia nervosa and/or bulimia nervosa (N=169).

50 diagnoses of binge eating disorder (N=131), bulimia nervosa (N=17), and anorexia nervosa (N=18).

51 ing-type anorexia nervosa (N=24), women with bulimia nervosa (N=19), and healthy comparison women (N=

52 Women diagnosed with either anorexia or bulimia nervosa (N=246) completed prospective evaluation

53 with restricting anorexia nervosa (n=26) or bulimia nervosa (n=47), control women (n=44), and first-

54 5 individuals with anorexia nervosa (N=177), bulimia nervosa (N=906), or eating disorder not otherwis

55 to assess diagnoses of anorexia nervosa and bulimia nervosa (per DSM-IV and broad criteria).

56 .38-9.54] vs adjusted, 2.67 [1.78-4.01]) and bulimia nervosa (suicide attempts: crude, 6.26 [5.73-6.8

57 inin release compared with participants with bulimia nervosa (t(76.44) = 2.51; P = .01) and did not d

58 Nine women who had recovered from bulimia nervosa (they had no episodes of binge eating or

59 ervosa among women might be up to 4%, and of bulimia nervosa 2%.

60 Eight women with bulimia nervosa and 10 age- and weight-matched control s

61 d, medication-free, normal-weight women with bulimia nervosa and 14 age-matched healthy female contro

62 ndent response in 18 female adolescents with bulimia nervosa and 18 healthy female age-matched subjec

63 al data acquired from 22 women remitted from bulimia nervosa and 20 group-matched controls who comple

64 Eight women with bulimia nervosa and 8 female controls underwent brain MR

65 found empirical support for conceptualizing bulimia nervosa and binge eating disorder as discrete sy

66 y remain the most established treatments for bulimia nervosa and binge eating disorder, with stepped-

67 utrient and food group intakes of women with bulimia nervosa and binge-eating disorder during pregnan

68 Eating disorders (anorexia nervosa, bulimia nervosa and binge-eating disorder) are a heterog

69 al therapy, may be effective, especially for bulimia nervosa and binge-eating disorder.

70 to measure the prevalence and correlates of bulimia nervosa and bulimic behaviors in a sample of und

71 nal distress compared with participants with bulimia nervosa and controls.

72 ialization affects the risk for anorexia and bulimia nervosa and disordered eating in members of oppo

73 ality ratios were significantly elevated for bulimia nervosa and eating disorder not otherwise specif

74 ndardized mortality ratios were elevated for bulimia nervosa and eating disorder not otherwise specif

75 compare serotonin function in patients with bulimia nervosa and healthy controls.

76 ell-validated disorders anorexia nervosa and bulimia nervosa and likely represents a stable syndrome.

77 n appear less likely to exhibit anorexia and bulimia nervosa and more likely to exhibit binge eating

78 cles published about anorexia nervosa and/or bulimia nervosa and the number of articles published abo

79 t while menstrual irregularities are common, bulimia nervosa appears to have little impact on later a

80 ecovery rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, respectively; little is

81 Both anorexia nervosa and bulimia nervosa are associated with increased mortality.

82 Anorexia and bulimia nervosa are associated with poor periodontal con

83 Anorexia nervosa and bulimia nervosa are common and severe eating disorders (

84 Anorexia nervosa and bulimia nervosa are common problems facing adolescents a

85 Both anorexia nervosa and bulimia nervosa are familial.

86 -based treatment trials for adolescents with bulimia nervosa are largely absent.

87 These results support the validity of bulimia nervosa as a diagnostic category that is distinc

88 ht to investigate the predictive validity of bulimia nervosa as a diagnostic category.

89 ivate to the same degree in adolescents with bulimia nervosa as in healthy comparison subjects.

90 Women with bulimia nervosa before and during pregnancy and those wi

91 Women with the diagnosis of bulimia nervosa between 1981 and 1987 who participated i

92 itudinal distinction of anorexia nervosa and bulimia nervosa but do not support the anorexia nervosa

93 er mistakes was associated with anorexia and bulimia nervosa but not with other psychiatric disorders

94 ubtypes over time; one-third crossed over to bulimia nervosa but were likely to relapse into anorexia

95 Anorexia and bulimia nervosa can have significant effects on oral hea

96 ed in participants with purging disorder and bulimia nervosa compared with controls but did not diffe

97 ognize hunger signals, whereas overeating in bulimia nervosa could represent an exaggerated perceptio

98 women who continue to meet full criteria for bulimia nervosa declines as the duration of follow-up in

99 zygosity; the prevalence of DSM-IV or broad bulimia nervosa did not differ in women from opposite- v

100 essary to resolve conflict, adolescents with bulimia nervosa displayed abnormal patterns of activatio

101 tine versus placebo in preventing relapse of bulimia nervosa during a 52-week period after successful

102 howed symptoms of either anorexia nervosa or bulimia nervosa during pregnancy had a higher frequency

103 ple of individuals with anorexia nervosa and bulimia nervosa from the Price Foundation collaborative

104 a nervosa groups and on the left side in the bulimia nervosa group relative to the healthy comparison

105 educed in the recovered anorexia nervosa and bulimia nervosa groups and predicted sensitivity to rewa

106 Women who had recovered from bulimia nervosa had a reduction of medial orbital fronta

107 In the fed state, women remitted from bulimia nervosa had attenuated prediction-error-dependen

108 mately 50% of women initially diagnosed with bulimia nervosa had fully recovered from their disorder,

109 In addition, women with bulimia nervosa had less activation in the lateral and a

110 As bulimia nervosa has been associated with a dysregulated

111 tions decrease binge eating in patients with bulimia nervosa has fueled interest in the hypothesis th

112 Recent research on Anorexia Nervosa and Bulimia Nervosa has yielded an increasingly detailed und

113 Two treatments for bulimia nervosa have emerged as having established effic

114 The treatment of patients with bulimia nervosa in a primary care setting is hampered by

115 haviours in patients with severe and chronic bulimia nervosa in a randomised, double-blind, placebo-c

116 g the usefulness of family interventions for bulimia nervosa in adolescents.

117 ervosa and possibly lower incidence rates of bulimia nervosa in Caucasian North American and Northern

118 on; and increasing rates of binge eating and bulimia nervosa in Hispanic and Black American minority

119 Bulimia nervosa in men was too rare to be assessed by zy

120 ta on outcome for individuals diagnosed with bulimia nervosa in order to better understand long-term

121 whether treatments known to be effective for bulimia nervosa in specialized treatment centers can be

122 due to all causes of death for subjects with bulimia nervosa in these studies was 0.3% (seven deaths

123 ate genes (n = 13), focusing on anorexia and bulimia nervosa in very small samples with considerable

124 Bulimia nervosa is a common eating disorder in adolescen

125 The etiology of bulimia nervosa is complex, with biologic, psychological

126 acy of fluoxetine in the acute management of bulimia nervosa is well established; however, few contro

127 ver" among the anorexia nervosa subtypes and bulimia nervosa may reflect problems with the validity o

128 nervosa n = 91; other specified EDs n = 34; bulimia nervosa n = 56; binge ED n = 16), and 120 health

129 r of eating that appears to be distinct from bulimia nervosa on subjective and physiological response

130 th binge eating and purging behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purgin

131 ften useful as adjuncts to psychotherapy for bulimia nervosa or binge-eating disorder; in the case of

132 acial/ethnic difference in the prevalence of bulimia nervosa or bulimic behaviors; 1.5% of the whites

133 y (CBT) guided self-care in adolescents with bulimia nervosa or eating disorder not otherwise specifi

134 Eighty-five adolescents with bulimia nervosa or eating disorder not otherwise specifi

135 were randomly assigned to family therapy for bulimia nervosa or individual CBT guided self-care suppo

136 isorders, 0.8% had partial- or full-criteria bulimia nervosa or purging disorder and 2.9% had partial

137 The participants were 80 mothers with bulimia nervosa or similar eating disorder who were atte

138 A total of 120 women with bulimia nervosa participated in a randomized, placebo-co

139 mined menstrual and reproductive function in bulimia nervosa patients retrospectively, with 10-15-yea

140 Adolescents with and without bulimia nervosa performed similarly on the task.

141 rial was conducted in which 70 patients with bulimia nervosa received either 2 years of weekly psycho

142 Long-term outcome for women diagnosed with bulimia nervosa remains unclear.

143 outcome studies of both anorexia nervosa and bulimia nervosa show that no significant differences exi

144 ompared with control subjects, patients with bulimia nervosa showed a blunting of postprandial cholec

145 More research on bulimia nervosa specific to adolescence is needed.

146 h higher in people with anorexia nervosa and bulimia nervosa than in a nonclinical group of women in

147 in engendering improvement in patients with bulimia nervosa than IPT.

148 lescents and adults with anorexia nervosa or bulimia nervosa that, together, implicate dysregulation

149 sover longitudinally in anorexia nervosa and bulimia nervosa to inform the validity of the DSM-IV-TR

150 of the studies linking anorexia nervosa and bulimia nervosa to substance use disorders suffer from p

151 Cognitive behavioural therapy for bulimia nervosa was assigned grade A because of the evid

152 al behavior therapy adapted for treatment of bulimia nervosa was associated with a promising decrease

153 Bulimia nervosa was more common than anorexia nervosa in

154 the risk of ASD in children to mothers with bulimia nervosa was no longer significant.

155 obands; the corresponding relative risks for bulimia nervosa were 4.2 and 4.4 for female relatives of

156 enty patients meeting DSM-III-R criteria for bulimia nervosa were allocated at random to 19 sessions

157 en partial syndromes of anorexia nervosa and bulimia nervosa were considered, relative risks fell by

158 tes of full and partial anorexia nervosa and bulimia nervosa were determined in first-degree relative

159 omen with a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7 years; weekly eating

160 Antidepressants for bulimia nervosa were given grade B.

161 A total of 173 women with bulimia nervosa were interviewed an average of 11.5 year

162 ing female patients with anorexia nervosa or bulimia nervosa were interviewed every 6 months for a me

163 who had recovered from anorexia nervosa and bulimia nervosa were studied to avoid confounding effect

164 Women with bulimia nervosa were unlikely to cross over to anorexia

165 th fluoxetine is useful for individuals with bulimia nervosa who do not respond to psychotherapy or r

166 Twenty-two patients with bulimia nervosa who had not responded to, or had relapse

167 e study examined relapse in 48 patients with bulimia nervosa who had responded to cognitive behaviora

168 y be a useful intervention for patients with bulimia nervosa who have not responded adequately to psy

169 d treatment with fluoxetine in patients with bulimia nervosa who responded to acute treatment with fl

170 formation exists on relapse in patients with bulimia nervosa who responded with complete abstinence f

171 194 women meeting the DSM-III-R criteria for bulimia nervosa who were treated with 18 sessions of man

172 of attrition and outcome in the treatment of bulimia nervosa with cognitive behavior therapy.

173 e recorded (lifetime anorexia nervosa, N=14; bulimia nervosa with no history of anorexia nervosa, N=2

174 orexia nervosa and 2.33 (95% CI=0.3-8.4) for bulimia nervosa with no history of anorexia nervosa.

175 hout obsessive-compulsive features; and LC4, bulimia nervosa with self-induced vomiting as the sole f

176 (LC1) resembled restricting AN; LC2, AN and bulimia nervosa with the use of multiple methods of purg

177 Since bulimia nervosa's introduction to the psychiatric nomenc

178 (any eating disorder, anorexia nervosa, and bulimia nervosa) identified by any lifetime diagnoses re

179 alized anxiety disorder, panic disorder, and bulimia nervosa), and four were "externalizing" (nicotin

180 a nervosa and higher in women recovered from bulimia nervosa).

181 lower in adult men (anorexia nervosa, 0.12%; bulimia nervosa, 0.08%; binge eating disorder, 0.42%).

182 ia nervosa, 1.42 [95% CI, 1.23-1.63]; HR for bulimia nervosa, 1.91 [95% CI, 1.43-2.54]; and HR for un

183 male patients with eating disorders (10 with bulimia nervosa, 16 with anorexia nervosa) and 19 health

184 0 years after they appeared as patients with bulimia nervosa, 177 women (participation rate=79.7%) co

185 ia nervosa, 2.04 [95% CI, 1.58-2.63]; HR for bulimia nervosa, 2.70 [95% CI, 1.68-4.32]; and HR for un

186 Thirty-one female patients with bulimia nervosa, 29 women who had been recovered from bu

187 high lifetime rates of depression (76.3% for bulimia nervosa, 65.5% for binge-eating disorder, and 49

188 als with anorexia nervosa, 1258 (19.0%) with bulimia nervosa, 757 (11.4%) with binge-eating disorder,

189 s), 11% of this sample met full criteria for bulimia nervosa, and 0.6% met full criteria for anorexia

190 individuals with anorexia nervosa, 282 with bulimia nervosa, and 293 with anorexia nervosa and bulim

191 tes were 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for eating disorder not otherw

192 ering the threshold for anorexia nervosa and bulimia nervosa, and adding BED as a specified eating di

193 Eating disorders (eg, binge eating disorder, bulimia nervosa, and anorexia nervosa) are a group of ps

194 examined the prevalence of anorexia nervosa, bulimia nervosa, and binge eating disorder in a geograph

195 d lifetime prevalences for anorexia nervosa, bulimia nervosa, and binge eating disorder in adult wome

196 Individuals with anorexia nervosa, bulimia nervosa, and binge-eating disorder have high lif

197 me prevalence estimates of anorexia nervosa, bulimia nervosa, and binge-eating disorder were 0.3%, 0.

198 DSM-IV classifications of anorexia nervosa, bulimia nervosa, and binge-eating disorder.

199 current classifications of anorexia nervosa, bulimia nervosa, and binge-eating disorder.

200 sorder diagnosis including anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

201 1998 and followed them for anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

202 y was to determine whether anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

203 Research into treatment has focused on bulimia nervosa, and evidence-based management of this d

204 ssion and co-twin risk for anorexia nervosa, bulimia nervosa, and obesity.

205 it/hyperactivity disorder, bipolar disorder, bulimia nervosa, and other personality disorders.

206 miological studies, short-term treatment for bulimia nervosa, and outcome in anorexia nervosa.

207 ree diagnostic categories: anorexia nervosa, bulimia nervosa, and the atypical eating disorders.

208 sted hazard ratios for the anorexia nervosa, bulimia nervosa, and unspecified eating disorder patient

209 g disorders, especially anorexia nervosa and bulimia nervosa, are more common among white women than

210 e four new categories are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eating disor

211 sed in diagnostic systems: anorexia nervosa, bulimia nervosa, binge eating disorder, avoidant-restric

212 ipants with a diagnosis of anorexia nervosa, bulimia nervosa, binge eating disorder, or eating disord

213 n eating disorders include anorexia nervosa, bulimia nervosa, binge-eating disorder, and avoidant/res

214 ervosa (AN) and other eating disorders (OED: bulimia nervosa, binge-eating disorder, and eating disor

215 ), attention-deficit/hyperactivity disorder, bulimia nervosa, cataplexy, dysthymic disorder, fibromya

216 episodes of binge eating in individuals with bulimia nervosa, even in those without depression (fluox

217 e slowing of motor responses was impaired in bulimia nervosa, even when the likelihood of having to s

218 criteria for DSM-5 binge eating disorder or bulimia nervosa, had 12 months of continuous health care

219 rs, but not the women who had recovered from bulimia nervosa, had an age-related decline in 5-HT(2A)

220 Anorexia nervosa, but not bulimia nervosa, has one of the highest mortality rates

221 interventions are of established utility in bulimia nervosa, medications have no clear role in the t

222 a longitudinal study of anorexia nervosa and bulimia nervosa, now in its 12th year.

223 evidence that the risk for anorexia nervosa, bulimia nervosa, or disordered eating was associated wit

224 ot respond to cognitive behavior therapy for bulimia nervosa, potentially allowing early use of a sec

225 Patients who met DSM-IV criteria for bulimia nervosa, purging type, were assigned to single-b

226 For patients with bulimia nervosa, the fenfluramine-stimulated increase in

227 en substantial advances in the management of Bulimia Nervosa, the goal of offering effective treatmen

228 ed to reward-processing deficits in remitted bulimia nervosa, the purpose of this study was to identi

229 h AN and related eating disorders, including bulimia nervosa, was performed, resulting in only modest

230 diagnostic criteria of anorexia nervosa and bulimia nervosa, which emphasize intrapsychic conflicts

231 onwhites (n = 693) were classified as having bulimia nervosa, while 5.5% of the whites and 5.3% of th

232 focused on the treatment of adolescents with bulimia nervosa.

233 determine how purging disorder differs from bulimia nervosa.

234 ng disorders, including anorexia nervosa and bulimia nervosa.

235 l of the brain mu-opioid receptor (mu-OR) in bulimia nervosa.

236 r the compulsive eating behavior observed in bulimia nervosa.

237 life (OHRQoL) in patients with anorexia and bulimia nervosa.

238 entified as a common feature of anorexia and bulimia nervosa.

239 ty factor for developing anorexia nervosa or bulimia nervosa.

240 h (1.3; 95% confidence interval, 0.0-7.2) in bulimia nervosa.

241 ome in women diagnosed as having anorexia or bulimia nervosa.

242 ory of a desensitized dopaminergic system in bulimia nervosa.

243 sfunction occurs frequently in patients with bulimia nervosa.

244 more inpatient treatment than did women with bulimia nervosa.

245 apy to decrease binge and purge behaviors in bulimia nervosa.

246 ies for relapse of susceptible patients with bulimia nervosa.

247 oral and neural reward-effort integration in bulimia nervosa.

248 present in patients who have recovered from bulimia nervosa.

249 erations in subjects who have recovered from bulimia nervosa.

250 n the pathophysiology of the eating disorder bulimia nervosa.

251 he preferred psychotherapeutic treatment for bulimia nervosa.

252 st effective psychotherapeutic treatment for bulimia nervosa.

253 mptoms are common in people who are ill with bulimia nervosa.

254 49 females (0.30%), and 61 males (0.01%) had bulimia nervosa.

255 ilial aggregation was independent of that of bulimia nervosa.

256 ilial vulnerability for anorexia nervosa and bulimia nervosa.

257 have been implicated in the neurobiology of bulimia nervosa.

258 patterns were found for anorexia nervosa and bulimia nervosa.

259 on of psychiatric disorders for anorexia and bulimia nervosa.

260 independent of that of anorexia nervosa and bulimia nervosa.

261 arly 20% continued to meet full criteria for bulimia nervosa.

262 is the psychological treatment of choice for bulimia nervosa.

263 questions based on the DSM-III criteria for bulimia nervosa.

264 for males, especially for those who develop bulimia nervosa.

265 observed deficit in satiety in patients with bulimia nervosa.

266 of abnormal eating patterns in patients with bulimia nervosa.

267 0.2% of the males were classified as having bulimia nervosa.

268 current diagnostic criteria for anorexia or bulimia nervosa.

269 sults for anorexia nervosa, and partially in bulimia nervosa.

270 aminergic dysfunction in the pathogenesis of bulimia nervosa.

271 icipants and women with anorexia nervosa and bulimia nervosa.

272 version of psychoanalytic psychotherapy for bulimia nervosa.

273 r anorexia nervosa and 1374 (11.8%) were for bulimia nervosa.

274 behavioral therapy (CBT) in the treatment of bulimia nervosa.

275 ervosa and may be efficacious for youth with bulimia nervosa.

276 eating in anorexia nervosa and overeating in bulimia nervosa.

277 norexia nervosa and promising for adolescent bulimia nervosa.

278 oods and avoid weight gain that characterize bulimia nervosa.

279 and conflict resolution in adolescents with bulimia nervosa.

280 anorexia nervosa (AN) and other EDs such as bulimia nervosa.

281 including the subtypes anorexia nervosa and bulimia nervosa.

282 ial anxiety (8-13 years) to anorexia nervosa/bulimia nervosa/obsessive-compulsive/binge eating/cannab

283 ; other EDs: OR = 4.66, 95% CI = 4.47, 4.87; bulimia nervosa: OR = 5.01, 95% CI = 4.63, 5.41) and the

284 ns and modifications to anorexia nervosa and bulimia nervosa; and the inclusion of binge eating disor

285 han black women (N=4, 0.4%) met criteria for bulimia nervosa; binge eating disorder also was more com

286 e a patient they were currently treating for bulimia or anorexia.

287 d greater acceptability for adolescents with bulimia or eating disorder not otherwise specified.

288 er, the types of OCD symptoms experienced by bulimia patients do not vary dramatically with improveme

289 Persistent OCD symptoms after recovery from bulimia raise the possibility that these behaviors are t

290 Bulimia status was assessed using criteria from the Diag

291 ng Disorder Inventory drive for thinness and bulimia subscales and depressed mood.

292 on and was operationalized using the Revised Bulimia Test.

293 ptor binding in women who had recovered from bulimia-type AN but not restricting-type AN.

294 Women recovered from bulimia-type AN had significantly (P<.05) increased [11C

295 tively, for the alcohol-dependent women, and bulimia was observed in 1.35% of the alcoholic men.

296 Lifetime rates for anorexia and bulimia were 1.41% and 6.17%, respectively, for the alco

297 of the alcohol-dependent men; the rates for bulimia were 3.46% and 0.72%, respectively.

298 tric disorders, and symptoms of anorexia and bulimia were evaluated among alcohol-dependent probands,

299 a nervosa, and 293 with anorexia nervosa and bulimia were given the Structured Clinical Interview for

300 Treatments for bulimia yield extremely high relapse rates, but cognitiv