ライフサイエンスコーパス: bulimia nervosa

1 a nervosa and higher in women recovered from bulimia nervosa).

2 ng disorders, including anorexia nervosa and bulimia nervosa.

3 l of the brain mu-opioid receptor (mu-OR) in bulimia nervosa.

4 r the compulsive eating behavior observed in bulimia nervosa.

5 entified as a common feature of anorexia and bulimia nervosa.

6 ty factor for developing anorexia nervosa or bulimia nervosa.

7 life (OHRQoL) in patients with anorexia and bulimia nervosa.

8 h (1.3; 95% confidence interval, 0.0-7.2) in bulimia nervosa.

9 ome in women diagnosed as having anorexia or bulimia nervosa.

10 ory of a desensitized dopaminergic system in bulimia nervosa.

11 sfunction occurs frequently in patients with bulimia nervosa.

12 more inpatient treatment than did women with bulimia nervosa.

13 apy to decrease binge and purge behaviors in bulimia nervosa.

14 ies for relapse of susceptible patients with bulimia nervosa.

15 oral and neural reward-effort integration in bulimia nervosa.

16 present in patients who have recovered from bulimia nervosa.

17 erations in subjects who have recovered from bulimia nervosa.

18 n the pathophysiology of the eating disorder bulimia nervosa.

19 he preferred psychotherapeutic treatment for bulimia nervosa.

20 st effective psychotherapeutic treatment for bulimia nervosa.

21 mptoms are common in people who are ill with bulimia nervosa.

22 ilial aggregation was independent of that of bulimia nervosa.

23 49 females (0.30%), and 61 males (0.01%) had bulimia nervosa.

24 ilial vulnerability for anorexia nervosa and bulimia nervosa.

25 have been implicated in the neurobiology of bulimia nervosa.

26 on of psychiatric disorders for anorexia and bulimia nervosa.

27 independent of that of anorexia nervosa and bulimia nervosa.

28 arly 20% continued to meet full criteria for bulimia nervosa.

29 patterns were found for anorexia nervosa and bulimia nervosa.

30 is the psychological treatment of choice for bulimia nervosa.

31 questions based on the DSM-III criteria for bulimia nervosa.

32 for males, especially for those who develop bulimia nervosa.

33 observed deficit in satiety in patients with bulimia nervosa.

34 of abnormal eating patterns in patients with bulimia nervosa.

35 0.2% of the males were classified as having bulimia nervosa.

36 current diagnostic criteria for anorexia or bulimia nervosa.

37 sults for anorexia nervosa, and partially in bulimia nervosa.

38 aminergic dysfunction in the pathogenesis of bulimia nervosa.

39 icipants and women with anorexia nervosa and bulimia nervosa.

40 r anorexia nervosa and 1374 (11.8%) were for bulimia nervosa.

41 or maternal exposure to anorexia nervosa and bulimia nervosa.

42 version of psychoanalytic psychotherapy for bulimia nervosa.

43 by metabolic state (being fasted or fed) in bulimia nervosa.

44 behavioral therapy (CBT) in the treatment of bulimia nervosa.

45 ervosa and may be efficacious for youth with bulimia nervosa.

46 eating in anorexia nervosa and overeating in bulimia nervosa.

47 norexia nervosa and promising for adolescent bulimia nervosa.

48 oods and avoid weight gain that characterize bulimia nervosa.

49 and conflict resolution in adolescents with bulimia nervosa.

50 anorexia nervosa (AN) and other EDs such as bulimia nervosa.

51 including the subtypes anorexia nervosa and bulimia nervosa.

52 focused on the treatment of adolescents with bulimia nervosa.

53 determine how purging disorder differs from bulimia nervosa.

54 lower in adult men (anorexia nervosa, 0.12%; bulimia nervosa, 0.08%; binge eating disorder, 0.42%).

55 ia nervosa, 1.42 [95% CI, 1.23-1.63]; HR for bulimia nervosa, 1.91 [95% CI, 1.43-2.54]; and HR for un

56 male patients with eating disorders (10 with bulimia nervosa, 16 with anorexia nervosa) and 19 health

57 0 years after they appeared as patients with bulimia nervosa, 177 women (participation rate=79.7%) co

58 ervosa among women might be up to 4%, and of bulimia nervosa 2%.

59 ia nervosa, 2.04 [95% CI, 1.58-2.63]; HR for bulimia nervosa, 2.70 [95% CI, 1.68-4.32]; and HR for un

60 with eating disorders other than anorexia or bulimia nervosa (255%; from 0.6 per 10 000 population to

61 Thirty-one female patients with bulimia nervosa, 29 women who had been recovered from bu

62 high lifetime rates of depression (76.3% for bulimia nervosa, 65.5% for binge-eating disorder, and 49

63 1%]), anorexia nervosa (6 of 79 [7.6%]), and bulimia nervosa (7/79 [8.9%]).

64 als with anorexia nervosa, 1258 (19.0%) with bulimia nervosa, 757 (11.4%) with binge-eating disorder,

65 into two groups: patients with anorexia and bulimia nervosa (ABN; n = 30) and control patients (CN;

66 ong, positive dose-response association with bulimia nervosa (adjusted hazard ratio = 1.15, 95% confi

67 binge eating disorder, anorexia nervosa, and bulimia nervosa among sexual and gender minority childre

68 Eight women with bulimia nervosa and 10 age- and weight-matched control s

69 d, medication-free, normal-weight women with bulimia nervosa and 14 age-matched healthy female contro

70 ndent response in 18 female adolescents with bulimia nervosa and 18 healthy female age-matched subjec

71 al data acquired from 22 women remitted from bulimia nervosa and 20 group-matched controls who comple

72 Eight women with bulimia nervosa and 8 female controls underwent brain MR

73 ipants and 130 female participants with EDs (bulimia nervosa and anorexia nervosa subtypes).

74 found empirical support for conceptualizing bulimia nervosa and binge eating disorder as discrete sy

75 y remain the most established treatments for bulimia nervosa and binge eating disorder, with stepped-

76 utrient and food group intakes of women with bulimia nervosa and binge-eating disorder during pregnan

77 Eating disorders (anorexia nervosa, bulimia nervosa and binge-eating disorder) are a heterog

78 al therapy, may be effective, especially for bulimia nervosa and binge-eating disorder.

79 to measure the prevalence and correlates of bulimia nervosa and bulimic behaviors in a sample of und

80 nal distress compared with participants with bulimia nervosa and controls.

81 ialization affects the risk for anorexia and bulimia nervosa and disordered eating in members of oppo

82 ality ratios were significantly elevated for bulimia nervosa and eating disorder not otherwise specif

83 ndardized mortality ratios were elevated for bulimia nervosa and eating disorder not otherwise specif

84 compare serotonin function in patients with bulimia nervosa and healthy controls.

85 The neurocomputational processes underlying bulimia nervosa and its primary symptoms, out-of-control

86 ell-validated disorders anorexia nervosa and bulimia nervosa and likely represents a stable syndrome.

87 mptoms, with the exception of fluoxetine for bulimia nervosa and lisdexamfetamine for binge-eating di

88 n appear less likely to exhibit anorexia and bulimia nervosa and more likely to exhibit binge eating

89 red in humans with binge eating disorder and bulimia nervosa and that the degree of alteration correl

90 cles published about anorexia nervosa and/or bulimia nervosa and the number of articles published abo

91 imaging assessments of anorexia nervosa and bulimia nervosa and uncovers neurobiological differences

92 alized anxiety disorder, panic disorder, and bulimia nervosa), and four were "externalizing" (nicotin

93 s), 11% of this sample met full criteria for bulimia nervosa, and 0.6% met full criteria for anorexia

94 individuals with anorexia nervosa, 282 with bulimia nervosa, and 293 with anorexia nervosa and bulim

95 tes were 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for eating disorder not otherw

96 ering the threshold for anorexia nervosa and bulimia nervosa, and adding BED as a specified eating di

97 Eating disorders (eg, binge eating disorder, bulimia nervosa, and anorexia nervosa) are a group of ps

98 examined the prevalence of anorexia nervosa, bulimia nervosa, and binge eating disorder in a geograph

99 d lifetime prevalences for anorexia nervosa, bulimia nervosa, and binge eating disorder in adult wome

100 Individuals with anorexia nervosa, bulimia nervosa, and binge-eating disorder have high lif

101 me prevalence estimates of anorexia nervosa, bulimia nervosa, and binge-eating disorder were 0.3%, 0.

102 DSM-IV classifications of anorexia nervosa, bulimia nervosa, and binge-eating disorder.

103 current classifications of anorexia nervosa, bulimia nervosa, and binge-eating disorder.

104 sorder diagnosis including anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

105 1998 and followed them for anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

106 y was to determine whether anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

107 Research into treatment has focused on bulimia nervosa, and evidence-based management of this d

108 ssion and co-twin risk for anorexia nervosa, bulimia nervosa, and obesity.

109 it/hyperactivity disorder, bipolar disorder, bulimia nervosa, and other personality disorders.

110 miological studies, short-term treatment for bulimia nervosa, and outcome in anorexia nervosa.

111 ree diagnostic categories: anorexia nervosa, bulimia nervosa, and the atypical eating disorders.

112 her SUDs with mortality in anorexia nervosa, bulimia nervosa, and unspecified eating disorder compare

113 sted hazard ratios for the anorexia nervosa, bulimia nervosa, and unspecified eating disorder patient

114 ns and modifications to anorexia nervosa and bulimia nervosa; and the inclusion of binge eating disor

115 t while menstrual irregularities are common, bulimia nervosa appears to have little impact on later a

116 ecovery rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, respectively; little is

117 Both anorexia nervosa and bulimia nervosa are associated with increased mortality.

118 Anorexia and bulimia nervosa are associated with poor periodontal con

119 Anorexia nervosa and bulimia nervosa are common and severe eating disorders (

120 Anorexia nervosa and bulimia nervosa are common problems facing adolescents a

121 Both anorexia nervosa and bulimia nervosa are familial.

122 -based treatment trials for adolescents with bulimia nervosa are largely absent.

123 g disorders, especially anorexia nervosa and bulimia nervosa, are more common among white women than

124 These results support the validity of bulimia nervosa as a diagnostic category that is distinc

125 ht to investigate the predictive validity of bulimia nervosa as a diagnostic category.

126 ivate to the same degree in adolescents with bulimia nervosa as in healthy comparison subjects.

127 We enrolled patients with severe bulimia nervosa (at least seven coupled binge/vomit epis

128 Women with bulimia nervosa before and during pregnancy and those wi

129 Women with the diagnosis of bulimia nervosa between 1981 and 1987 who participated i

130 hotherapy in reducing behavioral symptoms of bulimia nervosa (binge eating and vomiting).

131 e four new categories are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eating disor

132 sed in diagnostic systems: anorexia nervosa, bulimia nervosa, binge eating disorder, avoidant-restric

133 ipants with a diagnosis of anorexia nervosa, bulimia nervosa, binge eating disorder, or eating disord

134 n eating disorders include anorexia nervosa, bulimia nervosa, binge-eating disorder, and avoidant/res

135 ervosa (AN) and other eating disorders (OED: bulimia nervosa, binge-eating disorder, and eating disor

136 han black women (N=4, 0.4%) met criteria for bulimia nervosa; binge eating disorder also was more com

137 on in anorexia nervosa (AN) or overeating in bulimia nervosa (BN) a form of addiction?

138 EDs) such as binge eating disorder (BED) and bulimia nervosa (BN) among military personnel, less is k

139 Bulimia nervosa (BN) and binge eating disorder (BED) are

140 While bulimia nervosa (BN) and binge eating disorder (BED) are

141 Anorexia nervosa (AN) and bulimia nervosa (BN) are disorders characterized by abno

142 ED data are presented here and compared with bulimia nervosa (BN) data in the World Health Organizati

143 effective treatments, many individuals with bulimia nervosa (BN) do not receive evidence-based thera

144 Bulimia nervosa (BN) has been associated with dysregulat

145 Women with bulimia nervosa (BN) have disturbances of mood and behav

146 the development of anorexia nervosa (AN) and bulimia nervosa (BN) in women.

147 Bulimia nervosa (BN) is characterized by dysregulated in

148 Bulimia nervosa (BN) is strongly familial, and additive

149 t as follows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n = 82; Recovered AN n = 90; Health

150 Neuroimaging studies of bulimia nervosa (BN) report reduced activity in frontost

151 hree 'classical' EDs [anorexia nervosa (AN), bulimia nervosa (BN), and binge eating disorder (BED)].

152 hree eating disorders-anorexia nervosa (AN), bulimia nervosa (BN), and other eating disorders (OED)-a

153 with future onset of anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), and p

154 ucted in persons with anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), other

155 trol circuits in adolescents and adults with bulimia nervosa (BN), but less is known about the micros

156 disorders, such as anorexia nervosa (AN) and bulimia nervosa (BN), have genetic and environmental und

157 s with a diagnosis of anorexia nervosa (AN), bulimia nervosa (BN), or eating disorder not otherwise s

158 in adolescent and adult female subjects with bulimia nervosa (BN).

159 itudinal distinction of anorexia nervosa and bulimia nervosa but do not support the anorexia nervosa

160 er mistakes was associated with anorexia and bulimia nervosa but not with other psychiatric disorders

161 ubtypes over time; one-third crossed over to bulimia nervosa but were likely to relapse into anorexia

162 Anorexia and bulimia nervosa can have significant effects on oral hea

163 ), attention-deficit/hyperactivity disorder, bulimia nervosa, cataplexy, dysthymic disorder, fibromya

164 ed in participants with purging disorder and bulimia nervosa compared with controls but did not diffe

165 ognize hunger signals, whereas overeating in bulimia nervosa could represent an exaggerated perceptio

166 women who continue to meet full criteria for bulimia nervosa declines as the duration of follow-up in

167 zygosity; the prevalence of DSM-IV or broad bulimia nervosa did not differ in women from opposite- v

168 essary to resolve conflict, adolescents with bulimia nervosa displayed abnormal patterns of activatio

169 tine versus placebo in preventing relapse of bulimia nervosa during a 52-week period after successful

170 howed symptoms of either anorexia nervosa or bulimia nervosa during pregnancy had a higher frequency

171 episodes of binge eating in individuals with bulimia nervosa, even in those without depression (fluox

172 e slowing of motor responses was impaired in bulimia nervosa, even when the likelihood of having to s

173 ple of individuals with anorexia nervosa and bulimia nervosa from the Price Foundation collaborative

174 a nervosa groups and on the left side in the bulimia nervosa group relative to the healthy comparison

175 educed in the recovered anorexia nervosa and bulimia nervosa groups and predicted sensitivity to rewa

176 Women who had recovered from bulimia nervosa had a reduction of medial orbital fronta

177 In the fed state, women remitted from bulimia nervosa had attenuated prediction-error-dependen

178 mately 50% of women initially diagnosed with bulimia nervosa had fully recovered from their disorder,

179 In addition, women with bulimia nervosa had less activation in the lateral and a

180 criteria for DSM-5 binge eating disorder or bulimia nervosa, had 12 months of continuous health care

181 rs, but not the women who had recovered from bulimia nervosa, had an age-related decline in 5-HT(2A)

182 As bulimia nervosa has been associated with a dysregulated

183 tions decrease binge eating in patients with bulimia nervosa has fueled interest in the hypothesis th

184 Recent research on Anorexia Nervosa and Bulimia Nervosa has yielded an increasingly detailed und

185 Anorexia nervosa, but not bulimia nervosa, has one of the highest mortality rates

186 Two treatments for bulimia nervosa have emerged as having established effic

187 (any eating disorder, anorexia nervosa, and bulimia nervosa) identified by any lifetime diagnoses re

188 The treatment of patients with bulimia nervosa in a primary care setting is hampered by

189 haviours in patients with severe and chronic bulimia nervosa in a randomised, double-blind, placebo-c

190 g the usefulness of family interventions for bulimia nervosa in adolescents.

191 ervosa and possibly lower incidence rates of bulimia nervosa in Caucasian North American and Northern

192 on; and increasing rates of binge eating and bulimia nervosa in Hispanic and Black American minority

193 Bulimia nervosa in men was too rare to be assessed by zy

194 ta on outcome for individuals diagnosed with bulimia nervosa in order to better understand long-term

195 whether treatments known to be effective for bulimia nervosa in specialized treatment centers can be

196 due to all causes of death for subjects with bulimia nervosa in these studies was 0.3% (seven deaths

197 ate genes (n = 13), focusing on anorexia and bulimia nervosa in very small samples with considerable

198 Bulimia nervosa is a common eating disorder in adolescen

199 The etiology of bulimia nervosa is complex, with biologic, psychological

200 acy of fluoxetine in the acute management of bulimia nervosa is well established; however, few contro

201 hildren of mothers with anorexia nervosa and bulimia nervosa, it could not be explained by unmeasured

202 en they are in a fed state, individuals with bulimia nervosa may not effectively process unexpected i

203 ver" among the anorexia nervosa subtypes and bulimia nervosa may reflect problems with the validity o

204 =13.9), significantly longer than for either bulimia nervosa (mean=5.8 years, SD=9.1) or anorexia ner

205 interventions are of established utility in bulimia nervosa, medications have no clear role in the t

206 nervosa n = 91; other specified EDs n = 34; bulimia nervosa n = 56; binge ED n = 16), and 120 health

207 either DSM-IV anorexia nervosa (n = 136) or bulimia nervosa (n = 110) between January 1, 1987, and D

208 tracts addressing anorexia nervosa (n = 13), bulimia nervosa (n = 6), and binge-eating disorder (n =

209 ficantly diminished and women recovered from bulimia nervosa (N=14) had significantly elevated hemody

210 as there were about anorexia nervosa and/or bulimia nervosa (N=169).

211 diagnoses of binge eating disorder (N=131), bulimia nervosa (N=17), and anorexia nervosa (N=18).

212 ing-type anorexia nervosa (N=24), women with bulimia nervosa (N=19), and healthy comparison women (N=

213 Women diagnosed with either anorexia or bulimia nervosa (N=246) completed prospective evaluation

214 with restricting anorexia nervosa (n=26) or bulimia nervosa (n=47), control women (n=44), and first-

215 5 individuals with anorexia nervosa (N=177), bulimia nervosa (N=906), or eating disorder not otherwis

216 a longitudinal study of anorexia nervosa and bulimia nervosa, now in its 12th year.

217 ial anxiety (8-13 years) to anorexia nervosa/bulimia nervosa/obsessive-compulsive/binge eating/cannab

218 r of eating that appears to be distinct from bulimia nervosa on subjective and physiological response

219 th binge eating and purging behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purgin

220 ften useful as adjuncts to psychotherapy for bulimia nervosa or binge-eating disorder; in the case of

221 acial/ethnic difference in the prevalence of bulimia nervosa or bulimic behaviors; 1.5% of the whites

222 y (CBT) guided self-care in adolescents with bulimia nervosa or eating disorder not otherwise specifi

223 Eighty-five adolescents with bulimia nervosa or eating disorder not otherwise specifi

224 were randomly assigned to family therapy for bulimia nervosa or individual CBT guided self-care suppo

225 isorders, 0.8% had partial- or full-criteria bulimia nervosa or purging disorder and 2.9% had partial

226 The participants were 80 mothers with bulimia nervosa or similar eating disorder who were atte

227 evidence that the risk for anorexia nervosa, bulimia nervosa, or disordered eating was associated wit

228 ; other EDs: OR = 4.66, 95% CI = 4.47, 4.87; bulimia nervosa: OR = 5.01, 95% CI = 4.63, 5.41) and the

229 A total of 120 women with bulimia nervosa participated in a randomized, placebo-co

230 mined menstrual and reproductive function in bulimia nervosa patients retrospectively, with 10-15-yea

231 to assess diagnoses of anorexia nervosa and bulimia nervosa (per DSM-IV and broad criteria).

232 Adolescents with and without bulimia nervosa performed similarly on the task.

233 ot respond to cognitive behavior therapy for bulimia nervosa, potentially allowing early use of a sec

234 Patients who met DSM-IV criteria for bulimia nervosa, purging type, were assigned to single-b

235 rial was conducted in which 70 patients with bulimia nervosa received either 2 years of weekly psycho

236 Long-term outcome for women diagnosed with bulimia nervosa remains unclear.

237 Since bulimia nervosa's introduction to the psychiatric nomenc

238 outcome studies of both anorexia nervosa and bulimia nervosa show that no significant differences exi

239 ompared with control subjects, patients with bulimia nervosa showed a blunting of postprandial cholec

240 More research on bulimia nervosa specific to adolescence is needed.

241 .38-9.54] vs adjusted, 2.67 [1.78-4.01]) and bulimia nervosa (suicide attempts: crude, 6.26 [5.73-6.8

242 inin release compared with participants with bulimia nervosa (t(76.44) = 2.51; P = .01) and did not d

243 h higher in people with anorexia nervosa and bulimia nervosa than in a nonclinical group of women in

244 in engendering improvement in patients with bulimia nervosa than IPT.

245 lescents and adults with anorexia nervosa or bulimia nervosa that, together, implicate dysregulation

246 For patients with bulimia nervosa, the fenfluramine-stimulated increase in

247 en substantial advances in the management of Bulimia Nervosa, the goal of offering effective treatmen

248 ed to reward-processing deficits in remitted bulimia nervosa, the purpose of this study was to identi

249 Nine women who had recovered from bulimia nervosa (they had no episodes of binge eating or

250 sover longitudinally in anorexia nervosa and bulimia nervosa to inform the validity of the DSM-IV-TR

251 of the studies linking anorexia nervosa and bulimia nervosa to substance use disorders suffer from p

252 Cognitive behavioural therapy for bulimia nervosa was assigned grade A because of the evid

253 al behavior therapy adapted for treatment of bulimia nervosa was associated with a promising decrease

254 Bulimia nervosa was more common than anorexia nervosa in

255 the risk of ASD in children to mothers with bulimia nervosa was no longer significant.

256 h AN and related eating disorders, including bulimia nervosa, was performed, resulting in only modest

257 obands; the corresponding relative risks for bulimia nervosa were 4.2 and 4.4 for female relatives of

258 enty patients meeting DSM-III-R criteria for bulimia nervosa were allocated at random to 19 sessions

259 en partial syndromes of anorexia nervosa and bulimia nervosa were considered, relative risks fell by

260 tes of full and partial anorexia nervosa and bulimia nervosa were determined in first-degree relative

261 omen with a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7 years; weekly eating

262 Antidepressants for bulimia nervosa were given grade B.

263 A total of 173 women with bulimia nervosa were interviewed an average of 11.5 year

264 ing female patients with anorexia nervosa or bulimia nervosa were interviewed every 6 months for a me

265 who had recovered from anorexia nervosa and bulimia nervosa were studied to avoid confounding effect

266 Women with bulimia nervosa were unlikely to cross over to anorexia

267 diagnostic criteria of anorexia nervosa and bulimia nervosa, which emphasize intrapsychic conflicts

268 onwhites (n = 693) were classified as having bulimia nervosa, while 5.5% of the whites and 5.3% of th

269 th fluoxetine is useful for individuals with bulimia nervosa who do not respond to psychotherapy or r

270 Twenty-two patients with bulimia nervosa who had not responded to, or had relapse

271 e study examined relapse in 48 patients with bulimia nervosa who had responded to cognitive behaviora

272 y be a useful intervention for patients with bulimia nervosa who have not responded adequately to psy

273 d treatment with fluoxetine in patients with bulimia nervosa who responded to acute treatment with fl

274 formation exists on relapse in patients with bulimia nervosa who responded with complete abstinence f

275 194 women meeting the DSM-III-R criteria for bulimia nervosa who were treated with 18 sessions of man

276 of attrition and outcome in the treatment of bulimia nervosa with cognitive behavior therapy.

277 e recorded (lifetime anorexia nervosa, N=14; bulimia nervosa with no history of anorexia nervosa, N=2

278 orexia nervosa and 2.33 (95% CI=0.3-8.4) for bulimia nervosa with no history of anorexia nervosa.

279 hout obsessive-compulsive features; and LC4, bulimia nervosa with self-induced vomiting as the sole f

280 (LC1) resembled restricting AN; LC2, AN and bulimia nervosa with the use of multiple methods of purg