ライフサイエンスコーパス: bulimic

1 rders were identified, of whom 62 (46%) were bulimic, 30 (22%) were anorexic, and 43 (32%) met criter

2 alence and correlates of bulimia nervosa and bulimic behaviors in a sample of undergraduate students

3 We concluded that the sex difference in bulimic behaviors reported in other studies may be due t

4 The prevalence of bulimic behaviors was 5.4% overall, 6.6% for females, an

5 Detailed information about anorectic and bulimic behaviors was assessed through personal intervie

6 meostatic perturbations may promote hallmark bulimic behaviors-binge eating, dietary restriction, and

7 epression were statistically associated with bulimic behaviors.

8 he whites and 5.3% of the nonwhites reported bulimic behaviors.

9 ence in the prevalence of bulimia nervosa or bulimic behaviors; 1.5% of the whites (n = 459) and 0.4%

10 ported high concerns with muscularity but no bulimic behaviors; 2.4%, high concerns with muscularity

11 ue; 2.5%, high concerns with thinness but no bulimic behaviors; and 6.3%, high concerns with thinness

12 e investigated the effects of ondansetron on bulimic behaviours in patients with severe and chronic b

13 The amount of time spent engaging in bulimic behaviours was decreased on average by 7.6 h per

14 e dropouts were characterized by more severe bulimic cognitions and greater impulsivity, but it was n

15 The strongest associations were found for bulimic disorders (OR: 4.38; 95% CI: 3.66, 5.23) and hyp

16 ed to improve treatment for individuals with bulimic disorders.

17 mary outcome was the change in the number of bulimic episodes between baseline and posttreatment.

18 nstrated a significantly greater decrease in bulimic episodes compared with the control group (Cohen

19 (SCOFF), and categories of ED (restrictive, bulimic, hyperphagic, and other types of EDs) were deter

20 uture increases in body mass index (BMI) and bulimic pathology; however, the mechanisms underlying th

21 ntation by diagnostic group; 42% of the male bulimic patients were identified as either homosexual or

22 e dependence was elevated among relatives of bulimic probands compared with relatives of anorexic pro

23 Relatives of anorexic and bulimic probands had increased risk of clinically subthr

24 and 12.3 in female relatives of anorexic and bulimic probands, respectively.

25 and 4.4 for female relatives of anorexic and bulimic probands, respectively.

26 ted in female relatives of both anorexic and bulimic probands.

27 , and neural responses directly, 17 remitted bulimic (rBN) and 21 healthy individuals (HC) received a

28 Bulimic status in 1982 conferred an approximately 15-fol

29 rences in mu-OR binding between controls and bulimic subjects and to correlate mu-OR binding with the

30 es that conducted follow-up assessments with bulimic subjects at least 6 months after presentation.

31 of the self-perpetuating behavioral cycle of bulimic subjects because the insula is the primary gusta

32 nding in the left insular cortex was less in bulimic subjects than in controls and correlated negativ

33 t self-reported abnormal eating behaviors in bulimic subjects.

34 g load anticipation was associated with past bulimic symptom severity and the duration of symptom rem

35 Secondary outcome measures included other bulimic symptoms and cost of care.

36 temporal stability and predictive utility of bulimic symptoms and related variables over the course o

37 The temporal stability of bulimic symptoms and related variables was relatively hi

38 ression predicts future increases in BMI and bulimic symptoms and whether suppressed resting metaboli

39 Furthermore, these results suggest that bulimic symptoms are associated with disorders involving

40 The main outcome measures were bulimic symptoms assessed by the Eating Disorder Examina

41 Bulimic symptoms display high temporal stability and thu

42 environment, rBN subjects experienced fewer bulimic symptoms than in the natural environment (uncont

43 Reduction in core bulimic symptoms was also more immediate for patients re

44 avoidant, and groups together patients with bulimic symptoms who are high functioning and self-criti

45 to reduce the confounding effects of active bulimic symptoms) and control women (CW; n = 25).

46 mately 30% of women experienced relapse into bulimic symptoms, and risk of relapse appeared to declin

47 predicts future increases in BMI but not in bulimic symptoms.

48 however, did not predict future increases in bulimic symptoms.

49 do not vary dramatically with improvement in bulimic symptoms.

50 received support as long-term predictors of bulimic symptoms.

51 t increase the probability of maintenance of bulimic symptoms.

52 gated as risk factors for the development of bulimic symptoms.