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1 ltage value for that same segment in the EMM bull's eye.
2 nding beyond the arcades; and 1 (2.5%) had a bull's-eye appearance.
3 autofluorescent lesions around fovea (double bull's eye), areas of hyper/hypoautofluorescence, and ex
4 migration and consolidation that produce the bull's-eye colonies typically associated with P. mirabil
5                      This cycle produces the bull's-eye colony often associated with cultures of P. m
6 o change except in severe cases in which the bull's-eye damage expanded progressively.
7 nal pigment epithelium), and severe (visible bull's-eye damage).
8                     The ultimate therapeutic bull's-eye for CLL is to eliminate the disease and achie
9 in structure, with Th2 cells failing to form bull's-eye IS.
10                    Th1 cells formed typical "bull's-eye" IS with a ring of adhesion molecules surroun
11                             The fundi showed bull's-eye macular atrophy and widespread RPE thinning.
12 esentation, fundus examination revealed left bull's eye maculopathy and right normal fundus.
13 short-term users or severe retinal toxicity (bull's eye maculopathy).
14  20/200 or worse, color vision disturbances, bull's eye maculopathy, and peripheral pigmentary retino
15 ngs included markedly reduced visual acuity, bull's eye maculopathy, foveal hyperpigmentation, peripa
16 es, such as a Stargardt-like flecked fundus, bull's eye maculopathy, or pattern dystrophy.
17                    A 43-year-old female with bull's eye maculopathy, whose sister was diagnosed with
18 fter exclusion of other causes of unilateral bull's eye maculopathy.
19 r disease or evidence for any other cause of bull's eye maculopathy.
20 ng the p.Arg420Ser mutation presented with a bull's eye maculopathy.
21 er diagnosis, 35% of CD and 51% of CRD had a bull's eye maculopathy; 70% of CRD showed absolute perip
22 phy (SD OCT) were performed in patients with bull's-eye maculopathy (BEM) to identify phenotypic mark
23 ge at onset, imaging and ERG: cone dystrophy-bull's-eye maculopathy (CD-BEM, 40 eyes), cone-rod dystr
24                 Seven patients had bilateral bull's-eye maculopathy at presentation.
25                               Cone dystrophy-bull's-eye maculopathy eyes typically had complete outer
26 assess the effect of the processing schemes: bull's-eye map (BEM) uniformity, contrast between the le
27 PCs) form an "immunological synapse" (IS), a bull's-eye pattern composed of a central supramolecular
28 ar, a P. mirabilis colony grows outward in a bull's-eye pattern formed by consecutive waves of rapid
29 actions help to shape and maintain the final bull's-eye pattern of the IS.
30 th the Wallowa Mountains in the centre of a 'bull's eye' pattern of valleys and low-elevation mountai
31 thy does not always develop in a parafoveal (bull's eye) pattern, and a pericentral pattern of damage
32 y anomalies over lunar impact basins display bull's-eye patterns consisting of a central positive (ma
33                                              Bull's-eye plots indicated that the (111)In signal from
34 he centerline chord method and visualized on bull's-eye plots.
35             Using EMM software, we created a bull's eye precisely matched to that generated by DE-MRI
36                               The unilateral bull's eye presentation which occurred in the eye with C
37 y of linear sine-wave gratings over proposed bull's-eye radial gratings is discussed.