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1 mune blistering skin diseases, pemphigus and bullous pemphigoid.
2 dase-IV inhibitor-associated noninflammatory bullous pemphigoid.
3 ermal blistering disease that closely mimics bullous pemphigoid.
4 associated with the blistering skin disease, bullous pemphigoid.
5 e relevant in the diagnosis and treatment of bullous pemphigoid.
6 pemphigoid and help distinguishing them from bullous pemphigoid.
7 as valuable medications in the treatment of bullous pemphigoid.
8 sh nomacopan as a new therapeutic option for bullous pemphigoid.
9 nd for the clinical benefit of patients with bullous pemphigoid.
10 ut blisters had immunopathologic findings of bullous pemphigoid.
11 hout skin lesions can be the only symptom of bullous pemphigoid.
12 en of the autoimmune skin blistering disease bullous pemphigoid.
13 s in a mouse model of the autoimmune disease bullous pemphigoid.
14 in NC16A are involved in the pathogenesis of bullous pemphigoid.
15 he development of the autoimmune response in bullous pemphigoid.
16 .2% female; specifically, 6951 patients with bullous pemphigoid, 1669 patients with pemphigus, and 79
17 ibodies directed against the NC16A domain of bullous pemphigoid 180 (collagen XVII), a transmembrane
18 s that bind to this immunodominant region of bullous pemphigoid 180 are capable of inducing a skin di
19 d patients, all of whom had circulating anti-bullous pemphigoid 180 autoantibodies, showed a specific
22 mice would also be resistant to experimental bullous pemphigoid, a disease with a pathogenesis though
23 ned autoantibody-reactive site recognized by bullous pemphigoid and herpes gestationis sera) and the
25 can be well tolerated in older patients with bullous pemphigoid and may have therapeutic benefits for
26 ednisolone for short-term blister control in bullous pemphigoid and significantly safer in the long-t
28 e dermal-epidermal-junction is a hallmark of bullous pemphigoid and was shown to be important for pat
29 , pemphigus foliaceous, pemphigus, vulgaris, bullous pemphigoid, and benign chronic pemphigus, howeve
31 vulgaris were diagnosed; there was 1 case of bullous pemphigoid; and 1 suspected case of paraneoplast
33 esmosomal transmembrane protein, the 180 kDa bullous pemphigoid antigen (BP180), also known as type X
34 n utilizing primers specific for the 230 kDa bullous pemphigoid antigen (BPAG1), the 180 kDa bullous
36 lous pemphigoid antigen (BPAG1), the 180 kDa bullous pemphigoid antigen (BPAG2), the alpha6 and beta4
46 led-coil domain of the epithelial isoform of bullous pemphigoid antigen 1, BPAG1-e (also known as BP2
49 he beta4 integrin-associated plakin protein, bullous pemphigoid antigen 1e (BPAG1e) functions as a sc
53 5 (p < 0.05), and in skin from patients with bullous pemphigoid antigen 2 mutations (n = 3) the count
54 roteins such as the integrin alpha6beta4 and bullous pemphigoid antigen 2 within the hemidesmosomes a
58 ression of zinc finger protein 185 (ZNF185), bullous pemphigoid antigen gene (BPAG1), and prostate se
59 and attest to the importance of the 180-kDa bullous pemphigoid antigen in the attachment of the epid
62 protein interacting with Nck (SPIN90/WISH), bullous pemphigoid antigen-1, and calcium channel beta2.
63 s of BMZ proteins: one enriched with the two bullous pemphigoid antigens (BP230, BP180) and one enric
68 has recently demonstrated that reactivity of bullous pemphigoid autoantibodies to the BP180 ectodomai
69 tandard techniques for detecting circulating bullous pemphigoid autoantibodies, including other enzym
70 h the 180 and 230 kDa proteins recognized by bullous pemphigoid autoantibodies, LABD97 has been thoug
71 t of the hemidesmosome plaque is the 230-kDa bullous pemphigoid autoantigen (BP230/BPAG1), which conn
73 tween serum levels of autoantibodies against bullous pemphigoid (BP) antigens 180 (BP180) and 230 (BP
75 munofluorescence, and specific reactivity to bullous pemphigoid (BP) autoantigens BP180 and BP230, co
76 Affiliated Hospitals with a new diagnosis of bullous pemphigoid (BP) between May 1, 1997 and Septembe
99 tibodies from the sera of some patients with bullous pemphigoid (BP) react with a 180 kDa protein ter
102 ation has long been a histologic hallmark of bullous pemphigoid (BP), a subepidermal autoimmune blist
104 targeted by autoantibodies in patients with bullous pemphigoid (BP), and absent in patients with one
105 sera from patients with pemphigus vulgaris, bullous pemphigoid (BP), and chronic atopic and chronic
106 tients with a blistering skin disease called bullous pemphigoid (BP), is a transmembrane component of
107 rom patients with MMP, 1 of 50 patients with bullous pemphigoid (BP), none of 7 with pemphigus, and 3
108 e used IgG passive transfer murine models of bullous pemphigoid (BP), pemphigus foliaceus (PF), and p
113 ns regarding an experimental murine model of bullous pemphigoid by showing that the plasminogen/plasm
116 th BP underwent clinical evaluation with the Bullous Pemphigoid Disease Activity Index (BPDAI); and 3
117 mean absolute and percentage changes in the Bullous Pemphigoid Disease Area Index (BPDAI) activity s
118 d diagnosis of pemphigus vulgaris/foliaceus, bullous pemphigoid, epidermolysis bullosa acquisita, muc
121 usion, there are now three bullous diseases, bullous pemphigoid, herpes gestationis, and cicatricial
122 , vitiligo (HR, 0.534; 95% CI, 0.254-1.123), bullous pemphigoid (HR, 0.524; 95% CI, 0.140-1.956), and
123 eports, including vitiligo (IC(025) = 4.87), bullous pemphigoid (IC(025) = 4.08), lichenoid dermatiti
124 ation on immune checkpoint inhibitor-induced bullous pemphigoid (ICI-BP) in patients with cancer, wit
125 ded, that is, chronic spontaneous urticaria, bullous pemphigoid, IgA (Henoch-Schonlein purpura) and I
132 e toxin-1-positive S. aureus is prevalent in bullous pemphigoid lesions and suggests that early imple
133 . aureus colonization was observed in 85% of bullous pemphigoid lesions, 3-6-fold higher than the nar
134 . aureus colonization was observed in 85% of bullous pemphigoid lesions, 3-6-fold higher than the nar
136 systemic lupus erythematosus (BSLE) (n = 3), bullous pemphigoid (n = 16), pemphigus (n = 11), and nor
137 Pemphigus vulgaris (n=40, USA and Japan), bullous pemphigoid (n=40, USA), and healthy donors (n=55
139 skin disorders, including atopic dermatitis, bullous pemphigoid, Netherton's syndrome, and prurigo no
141 ostic assays and expertise and classified as bullous pemphigoid or epidermolysis bullosa acquisita.
142 ovide a source of infection in patients with bullous pemphigoid, particularly in the setting of high-
143 stemic lupus erythematosus patients (n = 3), bullous pemphigoid patients (n = 15), and normal humans
148 reas the predominant epitope identified with bullous pemphigoid sera is located in the noncollagenous
151 inducing a skin disease that closely mimics bullous pemphigoid, supporting the hypothesis that epito
152 icture may be indistinguishable from that of bullous pemphigoid, the latter being the most common aut
153 p randomised controlled trial of adults with bullous pemphigoid (three or more blisters at two or mor
154 sults with a complement-targeting therapy in bullous pemphigoid, to our knowledge, and supports furth
155 mmon in elderly patients, including scabies, bullous pemphigoid, transient acantholytic dermatosis, a
157 A potential role of Staphylococcus aureus in bullous pemphigoid was explored by examining the coloniz
158 (median [range] age, 75 [55-85] years) with bullous pemphigoid were included in the trial, of whom 5
160 Earlier preliminary studies in humans with bullous pemphigoid, which is also associated with excess