コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 chemic pattern (ST-segment elevation or left bundle branch block).
2 -segment elevation or depression and/or left bundle-branch block).
3 LVP and BiVP in the failing heart with left bundle branch block.
4 duction system abnormalities, including left bundle branch block.
5 e branch block is far more common than right bundle branch block.
6 tivity and specificity in patients with left bundle branch block.
7 cal septal myectomy, 47 (40%) developed left bundle branch block.
8 ol septal ablation, 21 (36%) developed right bundle branch block.
9 spected AMI and ST segment elevation or left bundle branch block.
10 h of symptom onset with ST elevation or left bundle branch block.
11 egree atrioventricular block with associated bundle branch block.
12 (1.6%) had left (n = 131) or right (n = 289) bundle branch block.
13 95% CI, 1.08-2.42) for developing a complete bundle branch block.
14 60 patients with mild heart failure and left bundle branch block.
15 ose with QRS width 120 to 149 ms or non-left bundle branch block.
16 nts with heart failure (HF) and without left bundle branch block.
17 dent predictors of new onset persistent left bundle branch block.
18 morphology into those with and without left bundle-branch block.
19 lockade, and the presence of a complete left bundle-branch block.
20 ith or without systolic dysfunction and left bundle-branch block.
21 patients with systolic dysfunction and left bundle-branch block.
22 ventricular ejection fraction <35% and left bundle-branch block.
23 frequently accompanied by an apparent right bundle-branch block.
24 ng could differentiate between BrS and right bundle-branch block.
25 mal conduction, of whom 38.8% developed left bundle branch block, 1.1% developed right bundle branch
26 attempted permanent HBP in 27 patients (left bundle branch block 17, intraventricular conduction defe
27 QRSd >120 ms in 57 (39%) patients (27 right bundle branch block, 18 left bundle branch block, and 12
28 lts: (1) ST elevation or presumably new left bundle branch block, (2) other ECG signs indicating myoc
29 1.44) versus 1.04 (0.78, 1.51), de novo left bundle-branch block (4% versus 0%) and Q waves (5.3% ver
31 ing ECG abnormalities were as follows: right bundle-branch block, 65.2%; left bundle-branch block, 43
32 icular conduction delay, or incomplete right bundle branch block, a much higher proportion than in th
33 pacing/stimulation in DCM patients with left bundle-branch block acutely enhances systolic function w
34 possibly with harm in patients without left bundle-branch block (adjusted hazard ratio for death fro
36 y 17 patients (2.0%) had new Q waves or left bundle-branch block after surgery; however, cTnT elevati
41 with Lugol's solution induced complete right bundle branch block and converted the bidirectional VT i
43 hic VT (n = 23) or PVCs (n = 47) with a left bundle branch block and inferior QRS axis morphology.
44 rietal band VAs were characterized by a left bundle branch block and left inferior (n=12) or superior
45 to have nonischemic cardiomyopathy and left bundle branch block and less likely to have renal dysfun
46 failure with QRS duration >/=150 ms and left bundle branch block and less predictable in those with Q
50 cular ventricular tachycardia, and the right bundle branch block and ST segment elevation syndrome of
51 n association with typical or atypical right bundle branch block and ST-segment elevations (Brugada s
52 in; ventricular tachycardias (VTs) with left bundle branch block and V(3) transition remain a challen
54 nduction disturbances, mainly new-onset left bundle-branch block and advanced atrioventricular block
56 justment for other significant factors, left bundle-branch block and intraventricular conduction dela
57 icular fibrillation characterized by a right bundle-branch block and ST elevation in the right precor
59 er implantation after TAVR, with prior right bundle-branch block and transcatheter valve type and imp
60 ts reviewed presenting ECGs to identify left bundle-branch block and, in the absence of left bundle-b
61 ients (27 right bundle branch block, 18 left bundle branch block, and 12 intraventricular conduction
63 od pressure <100 mm Hg, anterior MI, or left bundle branch block, and as "low clinical risk" if none
65 smoking, left ventricular hypertrophy, left bundle branch block, and diabetes mellitus; it had good
67 rbidities, ischemic cardiomyopathy, non-left bundle branch block, and lower %CRT pacing vs. responder
68 ents with left bundle branch block, non-left bundle branch block, and right bundle branch block morph
69 isease), normal QT intervals, apparent right bundle branch block, and sudden cardiac death, particula
70 le-branch block (LBBB), 228 (13%) with right bundle-branch block, and 308 (17%) with nonspecific intr
71 ent smoking, body mass index >35 kg/m2, left bundle-branch block, and left ventricular hypertrophy.
73 ude various RSR' patterns, without a typical bundle-branch block are markers of altered ventricular d
74 athophysiologic and clinical aspects of left bundle branch block, as well as current and future strat
75 us persistent ST-segment elevation and right bundle branch block, as well as the effectiveness of sod
77 ricular (AV) block, bifascicular block, left bundle branch block, atrial fibrillation, and left ventr
80 versus 85 to 99 ms and >/=100 ms (excluding bundle branch block [BBB]) and QRS morphology in those w
84 io-ventricular conduction block (AVB), right bundle branch block, bradycardia, and the Brugada syndro
85 as applied in the presence of complete right bundle branch block by atrial-synchronized RV free wall
87 Atrial Fibrillation, Myocardial Infarction, Bundle Branch Block, Cardiomyopathy, Dysrhythmia, and Hy
88 es were significant among patients with left bundle branch block conduction disturbance (n = 1,204, p
89 y in those with QRS >/= 150 ms and with left bundle branch block conduction disturbance, respectively
90 ctural heart disease associated with a right bundle branch block conduction pattern and ST-segment el
91 In patients with mild heart failure and left bundle branch block, decreased RWT was associated with a
92 dle-branch block and, in the absence of left bundle-branch block, definite STEMI (according to both c
94 vival benefit of CRT-D in patients with left bundle-branch block did not differ significantly accordi
95 structural heart disease, paced rhythms, and bundle branch block during sinus rhythm were excluded.
96 toms, left ventricular dysfunction, and left bundle-branch block, early intervention with CRT-D was a
98 nsustained ventricular tachycardia with left bundle branch block excluding right ventricular outflow
99 and 24 patients with heart failure and left bundle branch block followed by computer simulations of
100 55; p = 0.033) and new-onset persistent left bundle-branch block following TAVR (HR: 2.26, 95% CI: 1.
102 left ventricular ejection fraction and left bundle-branch block; group 3 (n=20), left ventricular ej
103 We identified 12 affected individuals (right bundle-branch block, > or =1-mm STE) with an autosomal d
108 HFEs was pronounced among patients with left bundle branch block (HR: 0.38, 95% CI: 0.29 to 0.49, p <
109 was neutral, regardless of morphology, right bundle branch block (HR=1.01, P=0.975), and intraventric
111 (ii) heart with a conduction disorder (right bundle branch block) (iii) focal activation initiated by
113 n resulted in ST-segment elevation and right bundle branch block in all patients in group A and in al
116 Idiopathic ventricular arrhythmias of left bundle branch block inferior axis morphology are usually
117 hic pattern associated with this VT was left bundle branch block, inferior axis and early precordial
121 premature ventricular contractions with left bundle branch block/inferior axis pattern in 16 ARVD/C p
122 ch block/inferior axis, case 3 showed a left bundle branch block/inferior axis, and case 4 showed a n
123 h block/superior axis, case 2 showed a right bundle branch block/inferior axis, case 3 showed a left
125 t bundle branch block (LBBB; including right bundle branch block, intraventricular conduction delay)
129 pa 0.52), and Q waves (kappa 0.44), good for bundle branch block (kappa 0.78), and very good for atri
131 baseline, while MB-LATER score (Male gender, Bundle branch block, LA, AF Type, Early Recurrences) 3 m
132 a pronounced effect among patients with left bundle branch block (LBBB) (hazard ratio [HR]: 0.58; p <
133 82], p = 0.001) and CRT-D patients with left bundle branch block (LBBB) (HR: 0.51 [95% CI: 0.35 to 0.
134 evolution in ST segment, T or Q wave or left bundle branch block (LBBB) abnormalities between the pre
135 ronization Therapy) study patients with left bundle branch block (LBBB) and 0, 1, 2, or >/=3 comorbid
136 ific syndrome characterized by isolated left bundle branch block (LBBB) and a history of progressive
137 t to separate the effects of associated left bundle branch block (LBBB) and coronary artery disease (
138 de (VAQRS), halfway between that during left bundle branch block (LBBB) and LV pacing, reflects optim
139 ata on the clinical impact of new-onset left bundle branch block (LBBB) and permanent pacemaker impla
140 t guidelines suggest that patients with left bundle branch block (LBBB) be treated with cardiac resyn
142 tion therapy (CRT) in patients with non-left bundle branch block (LBBB) conduction abnormality have n
143 ild heart failure (HF) patients without left bundle branch block (LBBB) did not derive a significant
144 and death events in CRT-D patients with left bundle branch block (LBBB) enrolled in MADIT-CRT (Multic
145 rognostic value of new-onset persistent left bundle branch block (LBBB) in patients undergoing transc
148 a suspected acute coronary syndrome and left bundle branch block (LBBB) present a unique diagnostic a
149 no benefit of CRT-D in patients without left bundle branch block (LBBB) regardless of patient sex.
150 Studies in canine hearts with acute left bundle branch block (LBBB) showed that endocardial left
151 nts with CRT-D in a very wide QRSD with left bundle branch block (LBBB) versus those without LBBB.
153 ead electrocardiographic morphology was left bundle branch block (LBBB), and in 15, it was nonspecifi
154 hy (DCM); in particular, the effects of left bundle branch block (LBBB), coronary artery disease (CAD
157 synchronization therapy candidates with left bundle branch block (LBBB)-like electrocardiogram morpho
163 Therapy (MADIT-CRT), patients with non-left bundle branch block (LBBB; including right bundle branch
164 strated an adverse impact of persistent left bundle-branch block (LBBB) after surgical aortic valve r
166 ree wall differed between patients with left bundle-branch block (LBBB) and normal QRSd and if synchr
167 go exercise testing develop a transient left bundle-branch block (LBBB) during exercise, but its prog
170 Septal activation in patients with left bundle-branch block (LBBB) patterns has not been describ
171 d to investigate the cardiac effects of left bundle-branch block (LBBB) using myocardial contrast ech
172 at baseline, there were 1281 (70%) with left bundle-branch block (LBBB), 228 (13%) with right bundle-
173 ardial infarction (MI) in patients with left bundle-branch block (LBBB), but the clinical impact of t
174 ough 30% to 50% of patients develop new left bundle-branch block (LBBB), its effect on clinical outco
175 similarly augment systolic function in left bundle-branch block (LBBB)-failing hearts despite differ
176 ts with (1) RV dilatation, (2) multiple left bundle-branch block (LBBB)-type VTs, and (3) an abnormal
180 rformed in anesthetized dogs with acute left bundle-branch block (LBBB, n=10) and chronic LBBB with t
181 5+/-12; ejection fraction 26+/-7%) with left bundle-branch block (LBBB; QRS duration 174+/-18 ms) wer
182 rdiographic abnormalities (long PR, complete bundle branch block, left ventricular hypertrophy voltag
183 tment group, age, gender, QRS duration, left bundle-branch block, left ventricular ejection fraction,
184 the 9 patients; 9 of the 11 VTs had a right bundle branch block/left-axis morphology that mimicked l
186 lvement, and electrical abnormalities (right bundle branch block/low voltages) was developed to predi
187 diothoracic ratio, higher prevalence of left bundle branch block, lower prevalence of atrial fibrilla
190 lure with reduced ejection fraction and left bundle branch block may respond positively to cardiac re
191 nts had nonischemic cardiomyopathy with left bundle branch block, median LVEF of 29%, and a mean QRS
192 tricular arrhythmias with a predominant left bundle branch block morphology and no ECG or echocardiog
193 hallenging because they present with a right bundle branch block morphology by electrocardiography.
194 rall, patients with mild HF but without left bundle branch block morphology did not derive clinical b
196 ricular arrhythmias with a predominant right bundle branch block morphology, 13 of 27 (48%) showed EC
197 ntricular arrhythmias with polymorphic/right bundle branch block morphology, without significant regu
203 CRT was performed in dogs with chronic left bundle-branch block (n=8) or atrioventricular (AV) block
205 clinical response between patients with left bundle branch block, non-left bundle branch block, and r
208 (paced rhythms, ventricular rhythms, or left bundle-branch block) on the ECG performed before thrombo
209 ere most pronounced among patients with left bundle branch block or a QRS duration at least 150 ms an
210 and major conduction abnormalities (new left bundle branch block or high-degree atrioventricular bloc
211 end point was a combination of complete left bundle branch block or high-degree atrioventricular bloc
212 d the clinical outcome of patients with left bundle branch block or intraventricular conduction delay
213 onged P-R interval, but not incomplete right bundle-branch block or early repolarization patterns, in
214 cardia, QRS prolongation resulting from left bundle-branch block or intraventricular conduction delay
215 patients with a non-LBBB QRS pattern (right bundle-branch block or intraventricular conduction distu
216 ry (OR 4.6; P = 0.016), the presence of left bundle branch block (OR 39; P = 0.002), and first-degree
217 2.1; 95% CI, 1.3 to 3.3), preoperative left bundle branch block (OR, 2.0; 95% CI, 1.3 to 2.9), preop
218 ration >/=150 ms (OR: 1.79; p = 0.007), left bundle branch block (OR: 2.05; p = 0.006), body mass ind
219 abnormalities, left ventricular hypertrophy, bundle branch block, or left-axis deviation) or exercise
220 irst-degree atrioventricular block, complete bundle branch block, or pacemaker insertion, and 1471 ag
223 h a higher rate of new onset persistent left bundle branch block, particularly in patients receiving
224 respectively) and nonsignificant in non-left bundle branch block patients (HR: 1.12, 95% CI: 0.77 to
225 long-term clinical outcomes of 537 non-left bundle branch block patients with mild HF enrolled in th
231 ferentiated from the A-LV summit VAs by left bundle branch block pattern, QRS duration </=175 ms, pre
232 icular fibrillation characterized by a right bundle-branch block pattern and ST elevation (STE) in th
237 entricular enlargement only, one with a left bundle branch block) presented with advanced HF and SCD,
238 and male sex, left ventricular hypertrophy, bundle branch block, previous myocardial infarction, and
239 tensive ST-segment elevation, new-onset left bundle branch block, previous myocardial infarction, Kil
240 raventricular conduction delay but not right bundle-branch block provided prognostic information abou
241 P<0.001) along with elimination of the right bundle branch block QRS morphology, increase in RV filli
242 ion (OR=0.32; 95% CI, 0.19 to 0.53) and left bundle-branch block QRS morphology (OR=0.21; 95% CI, 0.1
243 cular block (PR interval, >=0.2 s), complete bundle branch block (QRS, >=0.12 s), or pacemaker insert
244 score (female sex, nonischemic origin, left bundle-branch block, QRS >/=150 milliseconds, prior hosp
245 , SA was found to increase the risk of right bundle branch block (RBBB) (pooled odds ratio [OR]: 56.3
246 aventricular tachycardia with aberrant right bundle branch block (RBBB) and left anterior hemiblock (
247 ock (LBBB) was present in 65 patients, right bundle branch block (RBBB) in 48 patients, and nonspecif
248 ricular conduction delay (NICD) versus right bundle branch block (RBBB) in patients eligible for impl
249 udy sought to test the hypothesis that right bundle branch block (RBBB) patients have larger scar siz
250 ft bundle branch block, 1.1% developed right bundle branch block (RBBB), and 0.6% had complete heart
253 lock may be made when catheter-induced right bundle-branch block (RBBB) develops in patients with bas
257 of ventricular tachycardia (VT) with a right bundle-branch block (RBBB)-type pattern and an R-S ratio
261 nization therapy for heart failure with left bundle branch block reduces left ventricular (LV) conduc
263 excitation, as well as during right or left bundle branch block, resembled experimental measurements
264 ior hemiblock (RR: 1.62; p < 0.01), or right bundle branch block (RR: 2.89; p < 0.01) at baseline; an
265 ents with congenital heart disease and right bundle branch block, RV cardiac resynchronization therap
266 atients with systolic heart failure and left bundle-branch block scheduled for cardiac resynchronizat
267 , nonsustained ventricular tachycardia, left bundle branch block, signal-averaged electrocardiogram,
268 patients with QRS 120 to 149 ms or non-left bundle branch block, significant associations of baselin
270 eria (QRS duration of 120-149 ms or non-left bundle branch block), SSI>/=9.7% was independently assoc
271 ribed in patients with the syndrome of right bundle branch block, ST-segment elevation in leads V1 to
273 ectrocardiography showed sinus rhythm, right bundle branch block, T-wave inversion in V6, and evidenc
274 was relatively well preserved, and the right bundle-branch block type of BBR was frequently induced.
276 ree patients with systolic heart failure and bundle-branch block underwent implantation of biventricu
277 reverse remodeling is predicted by the left bundle branch block ventricular activation sequence, the
280 depression greater than 1 mm, complete left bundle-branch block, ventricular paced rhythm, preexcita
282 n 46% of septal myectomy patients, and right bundle branch block was evident in 58% of alcohol septal
283 in the VA interval with the development of a bundle branch block was the only tachycardia characteris
284 eath from any cause among patients with left bundle-branch block was 18% among patients randomly assi
285 an electrocardiographic pattern showing left bundle-branch block was associated with a significant re
289 tion, left ventricular hypertrophy, and left bundle branch block were strongly associated with HF in
290 urface electrocardiographic patterns of left bundle-branch block were all noted during intentional an
291 on-4, we studied patients with STEMI or left bundle branch block who received acute reperfusion with
292 associated with ST-segment elevation or left bundle branch block who underwent primary angioplasty.
293 dy group consisted of 245 patients with left bundle-branch block who underwent tomographic (single ph
294 associated with ST-segment elevation or left bundle-branch block who were treated with primary angiop
295 acute STEMI (including 3 patients with left bundle-branch block) who were undergoing infarct-artery
296 pidemia, and had more often a non-LBBB (left bundle branch block) wide QRS complex, and lower left ve
297 h NSVT or VT, the dominant morphology (right-bundle branch block with superior axis) was 100% predict
298 h a higher rate of new onset persistent left bundle branch block with the CoreValve (47.7% versus 35.
299 %, 55% ischemic cardiomyopathy, and 71% left bundle-branch block) with a follow-up of 29+/-11 months.
300 able adjustment, older age, female sex, left bundle branch block, worsened heart failure class, highe