ライフサイエンスコーパス: burst-forming unit-erythroid

1 cient mice revealed a reduction in the early burst-forming unit-erythroid and an expansion of late-st

2 Similarly, in vitro burst-forming unit-erythroid and colony-forming unit-ery

3 Deletion of Stat1 increased burst-forming unit-erythroid and reduced colony-forming

4 It has been previously shown that burst-forming units-erythroid and colony-forming units-e

5 ma-globin expression in K562 cells and human burst-forming units-erythroid and that increase prolifer

6 colony-forming unit-granulocyte-macrophage, burst-forming unit-erythroid, and high proliferative pot

7 Normal burst forming unit-erythroid (BFU-E) growth (>30 bursts/

8 points representing the transition from late burst forming unit-erythroid (BFU-E) to basophilic eryth

9 nit-granulocyte-macrophage (CFU-GM)-derived, burst forming unit-erythroid (BFU-E)-derived, and CFU-me

10 ly supports the differentiation of wild-type burst-forming unit erythroid (BFU-e) and colony-forming

11 lf-renewal of an early erythroid progenitor, burst-forming unit erythroid (BFU-E), and increase the p

12 median slope per day, -0.57; P = .0008), and burst-forming unit-erythroid (BFU-E) (median slope per d

13 1 expression in human CD34(+) cells impaired burst-forming unit-erythroid (BFU-E) and colony-forming

14 le colony-forming unit-erythroid (CFU-E) and burst-forming unit-erythroid (BFU-E) colonies were not p

15 Burst-forming unit-erythroid (BFU-E) colony-formation wa

16 formation by promoting self-renewal of early burst-forming unit-erythroid (BFU-E) progenitors.

17 Studies with progeny of burst-forming unit-erythroid (BFU-E) suggest that the FR

18 colony-forming unit-macrophage (CFU-M), and burst-forming unit-erythroid (BFU-E) was markedly decrea

19 orming unit-granulocyte/macrophage (CFU-GM), burst-forming unit-erythroid (BFU-E), and CFU-erythroid

20 ming unit-granulocyte, -macrophage (CFU-GM), burst-forming unit-erythroid (BFU-E), and colony-forming

21 main (HBD), to promote adhesion of primitive burst-forming unit-erythroid (BFU-E), mature BFU-E, and

22 , levels of these factors were determined in burst-forming unit-erythroid (BFU-E)-derived cells at di

23 Burst-forming unit-erythroid (BFU-E)-derived colony grow

24 3 protein was also highly expressed in early burst-forming unit-erythroid (BFU-E)-derived erythroid p

25 ntirely in the SBA(+) fraction; in contrast, burst-forming unit-erythroid (BFU-E)-forming cells were

26 eous cell population, we analyzed individual burst-forming units-erythroid (BFU-E) and nonerythroid c

27 Progeny of normal human burst-forming units-erythroid (BFU-E) contained Mpl rece

28 Single burst-forming units-erythroid (BFU-E) from 1 patient wer

29 , and greater than 10-fold higher numbers of burst-forming units-erythroid (BFU-E) in the Wnt-express

30 Burst-forming units-erythroid (BFU-E) increased in the s

31 acrophage colony-forming cells (GM-CFC), and burst-forming units-erythroid (BFU-E) per kilogram in th

32 0(4)/kg, respectively; the median numbers of burst-forming units-erythroid (BFU-e) were 0.20, 6.94, a

33 ethasone selectively increased the number of burst-forming units-erythroid (BFU-E), whereas lenalidom

34 p67(+) cells were significantly enriched for burst-forming units-erythroid (BFU-Es) and depleted of c

35 nulocyte-macrophage [CFU-GM]; 17% inhibition burst forming unit-erythroid [BFU-E]) and 3.44 micromol/

36 Erythroid progenitors (burst-forming unit-erythroid [BFU-E]) were significantly

37 ny-forming unit-megakaryocyte [CFU-Meg], and burst-forming unit-erythroid [BFU-E]), and CD34(+) cells

38 ocyte, megakaryocyte [CFU-GEMM]), erythroid (burst-forming unit-erythroid [BFU-E]), and granulocyte-m

39 ing unit-granulocyte-macrophage [CFU-GM] and burst-forming unit-erythroid [BFU-E]).

40 and chemotherapy patient-derived erythroid (burst-forming units-erythroid [BFU-E]), myeloid (colony-

41 ently detected on red blood cell precursors, burst-forming unit-erythroid- (BFU-E) derived cells.

42 lony-forming unit granulocyte-macrophage and burst-forming unit erythroid cells revealed absence of t

43 lony-forming unit granulocyte-macrophage and burst-forming unit erythroid cells with the 11q deletion

44 oid progenitors (CFU-granulocyte-macrophage, burst-forming unit-erythroid, CFU-granulocyte-erythrocyt

45 yocyte [CFU-MK], CFU-granulocyte/macrophage, burst-forming unit-erythroid/CFU-erythroid, and CFU-gran

46 -EPOR antibodies stimulated the formation of burst forming unit erythroid colonies from human CD34(+)

47 Frs. 2 and 3, greater than 70% of the total burst-forming unit erythroid colonies in CB and PB were

48 Further, although burst-forming unit erythroid colonies of BM were distrib

49 Erythroblasts plucked from the burst-forming unit-erythroid colonies of one of these ch

50 onies, whereas Flt3low cells produced mostly burst-forming unit-erythroid colonies.

51 AT1 protein was detected in 7-d-old burst-forming units-erythroid colonies by Western blotti

52 progenitors, defined as increased numbers of burst-forming units-erythroid colonies.

53 y erythroid progenitors showed inhibition of burst-forming unit-erythroid colony formation when inter

54 y transiently delaying erythropoiesis at the burst-forming unit-erythroid/colony-forming unit-erythro

55 ing unit-granulocyte/macrophage (CFU-GM) and burst-forming unit-erythroid derived from CML over a 2-l

56 in other hematopoietic cells including human burst forming unit-erythroid-derived erythroblasts and T

57 Fura-2 loaded day-10 burst-forming units-erythroid-derived erythroblasts were

58 We focus on the regulated expansion of burst-forming unit-erythroid erythroid progenitors by gl

59 a and promoted the formation of CFU-GEMM and burst-forming unit-erythroid in methylcellulose cultures

60 iest erythroid-restricted precursors (BFU-E [burst-forming unit-erythroid]) is also detected in the I

61 lony-forming unit-granulocyte macrophage and burst-forming unit-erythroid numbers and preferentially

62 colony-forming unit-granulocyte-macrophage, burst-forming unit-erythroid, or colony-forming unit-ery

63 479 (Y8) were capable of supporting immature burst-forming unit-erythroid progenitor development.

64 ming granulocytic-macrophage) and erythroid (burst-forming unit-erythroid) progenitor colony formatio

65 Burst-forming unit erythroid progenitors (BFU-Es) are so

66 r, only gp55-P induces erythroid bursts from burst-forming unit-erythroid progenitors and only gp55-P

67 g unit-granulocyte-monocytic) and erythroid (burst-forming unit-erythroid) progenitors.

68 eLV-C impairs the in vivo differentiation of burst-forming unit-erythroid to colony-forming unit-eryt

69 lony-forming unit-granulocyte macrophage and burst forming unit-erythroid were detected in the BM of

70 Maximal numbers of CFU-E and burst-forming unit-erythroid were increased, and CFU-E d

71 lony forming unit granulocyte-macrophage and burst-forming unit erythroid, while treatment with block