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1 reaction of the enolate ion of 3-mercapto-2-butanone.
2 nogen, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.
3 ts of 4-(methylnitrosoamino)-1-(3-pyridyl)-1-butanone.
4 nogen, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.
5 ene or 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.
6 doacetate, 1,3-dibromoacetone, and 1-bromo-2-butanone.
7 ced by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.
8 )P and 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.
9 on of two of the compounds, 2-propanol and 2-butanone.
10 appetitive olfactory learning in response to butanone.
11 androsterone to approximately 940-fold for 2-butanone.
12 molecule(-1) s(-1) for 3-methyl-3-nitrooxy-2-butanone.
13 ly depended on the proportions of hexane and butanone.
14 inary solvent system, composed of hexane and butanone.
15 cleavage in 2-butanol partial oxidation to 2-butanone.
16 wo ion peaks, corresponding to benzene and 2-butanone.
17 thyl salicylate, naphthalene, benzene, and 2-butanone.
18 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.
19 obtained with dimethyl methylphosphonate and butanone.
20 carcinogen 4-methylnitrosamino-1-3-pyridyl-1-butanone.
21 [e.g., 4-[methylnitrosamino]-1-(3-pyridyl)-1-butanone].
22 , trans-2-hexenal (0.49-17.49%), 3-hydroxy-2-butanone (0.08-7.39%), 1-hexanol (0.56-16.39%), 1-octano
23 ohexanol (23 mV; 2575th, 19-38), 3-hydroxy-2-butanone (16 mV, 9-26), 3-methylbutanal (20 mV; 14-26),
24 ring-containing ketones, such as 4-phenyl-2-butanone (1a) and 1-phenyl-1,3-butadione (2a), were redu
25 acetaldehyde, acetic acid, ethyl acetate, 2-butanone, 2,3,-butanedione, and acetone, or the monoterp
26 bon disulfide, dimethyldisulfide, acetone, 2-butanone, 2,3-butanedione, 6-methyl-5-hepten-2-one, indo
31 methylpyrazine, 2-vinylpyrazine, 3-hydroxy-2-butanone, 3-(methylsulfanyl)-propanal, and 5-methyl-2-fu
32 methyl 2-propenal), four ketones (acetone, 2-butanone, 3-buten-2-one, and 6-methyl-5-hepten-2-one), o
33 sugar damage to DNA: glycolate, 3-hydroxy-2-butanone, 3-phenylbutyraldehyde, and alpha-hydroxy-gamma
36 he Escherichia coli gene for 3,4-dihydroxy-2-butanone 4-phosphate synthase, a key enzyme for riboflav
37 previously shown to code for 3,4-dihydroxy-2-butanone-4-phosphate synthase (DHBP synthase), an enzyme
40 rotein GTP cyclohydrolase II/3,4-dihydroxy-2-butanone-4-phosphate synthase and a negative regulator o
41 his bacterial gene encodes the 4-dihydroxy-2-butanone-4-phosphate synthase enzyme of the riboflavin b
42 -ray crystal structures of L-3,4-dihydroxy-2-butanone-4-phosphate synthase from Magnaporthe grisea ar
43 ioning GTP cyclohydrolase II-3,4-dihydroxy-2-butanone-4-phosphate synthase, an enzyme essential for r
45 significantly higher amounts of 3-hydroxy-2-butanone (acetoin), 6-methyl-5-hepten-2-one (sulcatone),
48 nogen 4-(methylnitrosamino)-1-(3-pyridyl)-1- butanone also had a low incidence (16.7%) of ER methylat
49 Methyl-2-buten-1-ol (prenol) and 3-Hydroxy-2-butanone (AMC) were selected for further behavioral assa
50 droxylation, 4-(methylamino)-1-(3-pyridyl)-1-butanone (aminoketone), can be converted to the potent t
51 2) enables an efficient direct conversion of butanone ammoximation to butanone oxime, showing a high
52 and octanal), sauce-like flavor (3-hydroxy-2-butanone and 2-furfural), and sweet, caramel (2,3-octane
56 orea and Ruminococcus and higher levels of 2-butanone and 4-methyl-2-pentanone compared to CTRLs.
60 gens 4-(methylnitrosamino)-1-(3-pyridinyl)-1-butanone and N-nitroso nornicotine were also detected.
61 (TSNA) 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone and N-nitrosonornicotine by repairing O(6)-[4-o
62 exhaust, oil refining, and gasoline leaks (2-butanone and xylenes), and plastics (acetone and styrene
63 3) cm(3) molecule(-1) s(-1) for 3-nitrooxy-2-butanone, and (2.6 +/- 0.9) x 10(-13) cm(3) molecule(-1)
64 anol, 3,7-dimethyl-1,3,6-octatriene, hydroxy butanone, and 1-octen-3-ol perceived aroma for longan.
66 m enolates derived from acetone, pinacolone, butanone, and 3-methyl-2-butanone to give fair to good y
67 ncies of alpha-nitrooxyacetone, 3-nitrooxy-2-butanone, and 3-methyl-3-nitrooxy-2-butanone have been d
70 anal, 2-pentylfuran, 2-heptanone, nonanal, 2-butanone, and heptanal, while stored samples were associ
71 g. hexanal, (E)-2-nonenal, (E)-2-heptenal, 2-butanone, and hexadecane), lipids, myoglobin, total vola
72 ake of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, and nicotine, respectively, in relation to lun
75 (5 mol %) and oxalic acid (10 mol %) with 2-butanone as a solvent for the racemization of a range of
76 migration as evident from the formation of 2-butanone as the major product via the enol 2-hydroxy-2-b
77 e obtained in high yields and selectivity in butanone by changing the ligand to 1,3,5,7-tetramethyl-2
78 four analytes (methanol, ethanol, acetone, 2-butanone) by systematically varying the oscillating freq
79 during prolonged starvation, nor butanol or butanone, compounds closely related to BOH, had an effec
80 ceae, 4-methyl-2-pentanone, 1-butanol, and 2-butanone could discriminate NAFLD patients from CTRLs.
82 . vaginalis T016 with >/=20 mM 1,4-diamino-2-butanone (DAB) to inhibit ODC resulted in growth arrest,
83 transformant was inhibited by 1, 4-diamino-2-butanone (DAB), a known inhibitor of eukaryotic ODC.
86 curves were produced for ethanol, acetone, 2-butanone, ethyl acetate and eucalyptol, yielding R(2) >
87 g 4-(N-methyl-N-nitrosamino)-1-(3-pyridyl)-1-butanone-exposed hamsters revealed that MUC1 mRNA increa
88 unds (acetaldehyde, diacetyl, acetoin, and 2-butanone) followed a sigmoidal trend described by the mo
90 trooxy-2-butanone, and 3-methyl-3-nitrooxy-2-butanone have been determined at room temperature at 100
91 well as hydroxy acetone (HA) and 1-hydroxy-2-butanone (HB) as donor substrates, in each case using gl
93 ion of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone-induced pulmonary adenoma formation by myo-inos
94 ocking 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone-induced short-term O(6)-methylguanine and long-
95 mine, 4-(methylnitrosoamino)-1-(3-pyridyl)-1-butanone, is activated to lung DNA methylating and pyrid
97 amine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, is one of the most potent and abundant procarc
99 THF was analyzed for a series of ketones (2-butanone, methyl acetoacetate, and N,N-dimethylacetoacet
100 The rate constants for the reduction of 2-butanone, methylacetoacetate, N, N-dimethylacetoacetamid
101 ion of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) (0.05 ng/L) and 4-(methylnitrosamino)-1-(
102 ncluding (methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and 4-(methylnitrosamino)-1-(3-pyridyl)-1
103 ure of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and benzo[a]pyrene (BaP) in A/J mice.
104 amines 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and N'-nitrosonornicotine (NNN) can under
105 amines 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and N-nitrosonicotine (NNN) are potent ca
106 amines 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and NNN and the addictive alkaloid nicoti
107 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and vinyl carbamate (VC) during mouse lun
108 nogens 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and vinyl carbamate, and the occupational
109 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) by measuring the frequency of NNK-induced
110 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) by s.c. injection at a dose of 1.5 mg/kg
111 cromol 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) given by gavage once weekly for 8 weeks.
113 ced by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) in male F344 rats that had been fed eithe
114 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) in people who had stopped smoking: 4-(met
115 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) induces Ca(2+) signalling, a mechanism th
116 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) into DNA-altering compounds that cause lu
117 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is an important component in cigarette sm
118 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is formed by nitrosation of nicotine and
119 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is formed by nitrosation of nicotine and
120 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is of particular interest due to its pote
121 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is the most potent carcinogen contained i
122 ion by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) or vinyl carbamate (VC) resulted in a sig
123 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) reproducibly induces pulmonary adenocarci
124 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) were previously shown to be highly persis
125 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) within DNA duplexes representing p53 muta
126 inogen 4-(methylnitrosamino)-I-(3-pyridyl)-1-butanone (NNK), 4ebp1(-/-)/4ebp2(-/-) mice showed increa
128 (NNN), 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), and 4-(methylnitrosamino)-1-(3-pyridyl)-
129 nogen, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), and its inhibition by black tea have bee
130 N) and 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), are human carcinogens with No Significan
131 samine-4-(methylnitrosamino)-1-(3 pyridyl)-1-butanone (NNK), BEAS-2B cells exhibited evidence of tran
134 amine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), is a chemical carcinogen thought to be i
135 he NOC 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), O(6)-methyl-dG (O(6)-Me-dG) and O(6)-pyr
136 amine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), selectively induces lung tumors in F344
137 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), the inflammatory agent lipopolysaccharid
138 ole in 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced lung cancer by producing carcinog
139 ine on 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced lung tumor progression from adeno
149 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK); and r-1-,t-2,3,c-4-tetrahydroxy-1,2,3,4-
150 sed to 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, NNK, a potent carcinogen in tobacco smoke.
151 4-(acetoxymethylnitrosamino)-1-(3-pyridyl)-1-butanone (NNKOAc), enhanced the tumorigenic activity of
153 irect conversion of butanone ammoximation to butanone oxime, showing a high conversion rate of 80.2%
154 ose of 4-(methylnitrosamino)-I-(3-pyridyl)-1-butanone (P = 0.03) per cigarette than noncarriers.
155 entanone (PF-2M3P), and perfluoro-3-methyl-2-butanone (PF-3M2B) using outdoor atmospheric simulation
156 .1 g/mol), 4-aminopiperidine, 3,3-dimethyl-2-butanone (pinacolone) and methylisobutyl ketone for whic
158 found to modify the kinetic parameters for 2-butanone reduction by R-HPCDH in a saturable fashion, wi
159 , and increasing the enantioselectivity of 2-butanone reduction to a theoretical value of 100% (S)-2-
161 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone results in accumulation of Bcl2 in the nucleus
162 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone results in accumulation of Bcl2 in the nucleus,
164 lectivity for producing (S)-2-butanol from 2-butanone that was unaffected by modulators that interact
165 onated monomers and proton-bound dimers of 2-butanone, the limits of detection are 1.3 ppt(v) and 57
166 sion of 4-hydroxy-4-(6-methoxy-2-naphthyl)-2-butanone to 6-methoxy-2-naphthaldehyde and acetone.
167 ase the selectivity for the CBS reduction of butanone to 80% enantiomeric excess (ee), the highest po
168 cetone, pinacolone, butanone, and 3-methyl-2-butanone to give fair to good yields of 2H-1,2-benzothia
169 An Oscillospira decrease coupled to a 2-butanone up-regulation and increases in Ruminococcus and
170 The effect of proportions of hexane and butanone was first studied by varying the volume ratio o
174 rcinogen 4-methylnitrosamino-1-(3-pyridyl)-1-butanone were hypermethylated at the p16 gene promoter;
175 nder Dakin-West conditions, forms 4-phenyl-2-butanone when the reaction is catalyzed by 1-methylimida
176 -LPC at 60 degrees C in the presence of 30 % butanone, where the synthesis rate was 100 times higher
177 the Corey-Bakshi-Shibata (CBS) reduction of butanone, which constitutes the classic and prototypical
178 te the partial oxidation of 2-butanol into 2-butanone with near 100% selectivity at low oxygen covera
179 nogen, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, with a catalytic efficiency much greater than