ライフサイエンスコーパス: by suppressing

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します

1 ua/CIC maintains peripheral immune tolerance by suppressing aberrant activation of adaptive immunity.

2 their proper development and maturation and by suppressing aberrant proliferation mediated by the T

3 nced the production of full-size transcripts by suppressing abortive loss of short RNAs.

4 evelopment and progression of renal fibrosis by suppressing activation and expression of multiple pro

5 vent neuroinflammation and neurodegeneration by suppressing activation of innate and adaptive immune

6 ough inhibition of renal fibroblasts and EMT by suppressing activation of multiple profibrotic signal

7 gluteofemoral adipose tissue (AT) expansion by suppressing adipogenesis and increasing gluteal adipo

8 BYL719 exerts its therapeutic effect by suppressing AKT and other proliferative signaling mec

9 rosis can potentially be treated effectively by suppressing allele-specific genes using small interfe

10 es both innate and adaptive immune responses by suppressing alpha-SYN-induced microglia and macrophag

11 x vivo, ibuprofen also affected Sertoli cell by suppressing AMH production and mRNA expression of AMH

12 itor may more effectively treat glioblastoma by suppressing an important temozolomide resistance mech

13 A1AR) protects against cisplatin ototoxicity by suppressing an inflammatory response initiated by ROS

14 e (AMPK), which regulates energy homeostasis by suppressing anabolic and activating catabolic process

15 electron transfer reactions, and control it by suppressing and enhancing reaction paths.

16 may hold promise as an anti-cancer strategy by suppressing angiogenesis.

17 hesis that people can attenuate future fears by suppressing anticipatory simulations of dreaded event

18 inhibited the AAA development in AdAPN mice by suppressing aortic inflammatory cell infiltration, me

19 with doxorubicin, paclitaxel, or carboplatin by suppressing apoptosis and upregulating NF-kappaB Over

20 of mouse PGCs and spermatogonial stem cells by suppressing apoptosis.

21 L5/CCR5 may influence proplatelet production by suppressing apoptosis.

22 ng antagonist sgp130Fc ameliorated emphysema by suppressing augmented alveolar type II cell apoptosis

23 y for enhancing responses to EGFR inhibitors by suppressing AURKA-driven residual disease and acquire

24 re moderately effective at reversing disease by suppressing autoimmune inflammation in the skin and p

25 nsumption may exacerbate cerebral I/R injury by suppressing autophagy.

26 the maintenance of immunological homeostasis by suppressing autoreactive T cells in liver and lung.

27 plant hormone auxin, prevents bud outgrowth by suppressing auxin canalization and export from axilla

28 tumor development and resistance to therapy by suppressing BAX and its activators.

29 ophagy flux and increased BAX protein levels by suppressing BAX degradation.

30 selected thymocytes from negative selection by suppressing Bim expression.

31 colysis and cancer stem-like characteristics by suppressing both H19 and PDK1.

32 rs, we show that foretinib protected neurons by suppressing both known degenerative pathways and a ne

33 ting oncogenic and tumor suppressive effects by suppressing both tumor suppressive mRNAs and oncogeni

34 may enhance (or intensify) cryptosporidiosis by suppressing C. parvum-induced cell turnover and caspa

35 ) in tumor cells promotes cancer progression by suppressing cancer immunity.

36 and extent of inflammatory cell infiltration by suppressing canonical BMP signaling, thereby providin

37 om 4T1 tumor-bearing mice, induce metastasis by suppressing CD8(+) T-cells in EMT6-primed mice.

38 which EZH2 controls GC B cell proliferation by suppressing CDKN1A, enabling cell cycle progression w

39 is specifically controls postnatal RV growth by suppressing cell cycle regulatory genes.

40 effectively mitigate the surface degradation by suppressing chemical delithiation.

41 ing allows management as a chronic condition by suppressing circulating viral load to allow for a nea

42 (QDs) enhances triplet energy transfer (TET) by suppressing competitive charge transfer from QDs to m

43 TCF3 sanctions rewiring by suppressing components of the ESC transcription facto

44 This delayed aging is mediated by suppressing constitutive endocytosis, which alters th

45 This is accomplished by suppressing contractions of the detrusor muscle that

46 poietic stem cell transplantation (allo-HCT) by suppressing conventional T cells (Tconvs).

47 ve mechanical resilience of flexible devices by suppressing crack formation upon stretching and bendi

48 that miR-23a prevents ROS-elicited necrosis by suppressing cyclophilin D (PPIF), a regulator of ROS

49 Talpha1 in regulating mitochondrial function by suppressing CYP2E1 expression at multiple levels.

50 differentiation of neural stem cells (NSCs) by suppressing cytoskeletal contractility.

51 ve molecule that promotes beta-cell survival by suppressing DDR signaling and attenuating DNA damage-

52 e excitability of SLC5A11-expressing neurons by suppressing dKCNQ channels, thereby conferring the hu

53 body, dE2F/dDP limits cell-cycle progression by suppressing DNA damage responses.

54 luding DNA repair and inflammatory response, by suppressing downstream targets IKBKB, WEE1, FGF2, RAD

55 Kr-h1 maintains the immature stage by suppressing E93 (early ecdysone response gene) in N4.

56 de oxytocin contributes to metabolic control by suppressing eating behavior.

57 y a convergent strategy of TE load reduction by suppressing element origination in their independent

58 c cells and functions to favor NHEJ over HDR by suppressing end resection, which is the rate-limiting

59 to reduce nanoparticle uptake in macrophages by suppressing endocytosis.

60 signaling in cancer cells promotes survival by suppressing endogenous DNA damage, and may control ce

61 rated the enhancement of dendritic formation by suppressing endogenous TRIO, and similarly decreasing

62 ardiac fibrosis and functional deterioration by suppressing endothelial autophagy and promoting endot

63 hus, TGF-beta induces SG ILC differentiation by suppressing Eomes.

64 tment efficacy against multiple malignancies by suppressing epigenetic hypermethylation in tumor cell

65 and mucous metaplasia following RV infection by suppressing epithelial cell innate cytokine expressio

66 pound 13d alleviates ER stress in beta-cells by suppressing ER stress-mediated activation of all thre

67 onated irradiation and could not be reversed by suppressing excessive end resection.

68 10 also indirectly supports humoral immunity by suppressing excessive IFN-gamma, which induces T-bet

69 T (Treg) cells prevent autoimmune disorders by suppressing excessive lymphocyte activity, but how in

70 vo activity of these cells, at least in part by suppressing excitatory drive from PBN inputs onto CRF

71 ng two-distinct stages of positive selection by suppressing expression levels of pro-apoptotic molecu

72 plored as a novel therapy for CRPC treatment by suppressing expression of AR and AR splice variants t

73 n of GPR30 by G-1 enhanced cholelithogenesis by suppressing expression of cholesterol 7alpha-hydroxyl

74 l self-renewal and reprogram immune response by suppressing expression of immune checkpoint genes, es

75 ion protects against fatty liver development by suppressing expression of peroxisome proliferator-act

76 , promoted apoptosis in BAP1-deficient cells by suppressing expression of the prosurvival genes Bcl2

77 ration, clonogenic ability, and invasiveness by suppressing extracellular signal-regulated kinases 1/

78 itation of hippocampal CA1 pyramidal neurons by suppressing feedforward inhibition.

79 bolished dopamine-induced salivary secretion by suppressing fluid transport in type III acini.

80 glucose production and promotes lipogenesis by suppressing FOXO1-dependent activation of G6pase and

81 of tau in GABAergic interneurons impairs AHN by suppressing GABAergic transmission and disinhibiting

82 ance and lineage determination of stem cells by suppressing genes that regulate cellular differentiat

83 s the predominant glutamate transporter GLT1 by suppressing GLT1-inhibiting miRs.

84 impairs upper airway dilator muscle activity by suppressing glutamatergic input from PH premotoneuron

85 at IL-9 reduces autoimmune neuroinflammation by suppressing GM-CSF production by CD4(+) T cells throu

86 y, a major issue for Sb(2)Se(3), is resolved by suppressing growth kinetics via close space sublimati

87 llee effects can limit species distributions by suppressing growth rates of peripheral populations wh

88 cient and flexible coding in hippocampal CA1 by suppressing heterogeneously tuned excitation.

89 s endochondral ossification at the epiphysis by suppressing HIF signaling pathways.

90 By suppressing HIF-1alpha and other oncogenic transcript

91 pontaneous and drug-induced latency reversal by suppressing HIV-1 transcriptional elongation.

92 ugs might prove to be an effective treatment by suppressing HK2 infectivity in diseases where these v

93 efficiency from 60.3+/-6.1 % to 71.8+/-6.2 % by suppressing hole transfer.

94 nificantly increased bacterial pathogenicity by suppressing host immune defense.

95 n may, therefore, increase TB susceptibility by suppressing IFN-gamma and IL-17A production during th

96 lopment and onset of SS in NOD mice, in part by suppressing IFN-gamma production.

97 f immune effector molecules, including iNOS, by suppressing IFN-gamma-JAK-STAT1 transcription-factor

98 ly reduced late, but not early, infarct size by suppressing IGF-1 degradation and, consequently, dimi

99 asis-related proinflammatory gene expression by suppressing IkappaBzeta induction in keratinocytes.

100 the differentiation of inflammatory T cells by suppressing IL-10 expression through histone demethyl

101 i-TNF-alpha therapy may act at least in part by suppressing IL-22BP and point toward a more specific

102 pDCs alleviates AHR and airway inflammation by suppressing ILC2 function and survival.

103 eutic approach in treating allergic diseases by suppressing ILC2 function.

104 uate sleep has deleterious effects on health by suppressing immunity and promoting inflammation.

105 mprovements of survival and cardiac function by suppressing infiltration of multiple kinds of inflamm

106 By suppressing inflammasome activation, IRGM/Irgm1 prote

107 e that GCN2 controls intestinal inflammation by suppressing inflammasome activation.

108 tection against IR-induced intestinal injury by suppressing inflammation and nitrosative stress and u

109 They regulate immune response by suppressing inflammation and promoting self-tolerance

110 ated inflammation in the injured brain areas by suppressing inflammatory cytokines expression whereas

111 predisposes to invasive pneumococcal disease by suppressing inflammatory processes of the upper respi

112 with Kv4.2 channel and promotes its function by suppressing inhibitory modulation by cAMP-protein kin

113 hyperresponsiveness and airway inflammation by suppressing innate and adaptive antiviral responses a

114 Ca(2+) influx maintained the quiescent state by suppressing insulin-like growth factor (IGF)-mediated

115 that ATM kinase promotes Alt-EJ-mediated CSR by suppressing interchromosomal translocations independe

116 m changes in bone marrow-derived macrophages by suppressing interleukin 1beta, CD68, and phagocytosis

117 orate the pathological outcomes of infection by suppressing interleukin 6 production in the brain.

118 ysmetabolism and improves muscle performance by suppressing intramuscular fat deposits, Phospho-AKT l

119 environment at the distant organ sites, and by suppressing invasive properties of the cancer cells.

120 pneumoniae responds to exogenous fatty acids by suppressing its de novo biosynthetic pathway and excl

121 nase delta (PI3Kdelta) pathway regulates AID by suppressing its expression in B cells.

122 Inhibiting LIN9 genetically or by suppressing its expression with a global BET inhibito

123 Derepression of wild-type p53 by suppressing its negative inhibitor iASPP (Inhibitor o

124 -colored emissions from upper excited states by "suppressing" Kasha's rule.

125 suppressed pancreatic tumor growth, in part, by suppressing KRAS signaling.

126 at lower visual contrast and was diminished by suppressing L1 activity.

127 relieved when nuclear stiffness is decreased by suppressing lamin A/C expression.

128 C pyramidal neuron responses to MThal inputs by suppressing local feed-forward GABA signaling from pa

129 astatic process of lung cancer cells in vivo by suppressing local invasion and distant colonization.

130 tly attenuate ischemia-induced VA, primarily by suppressing LSG activity.

131 AtRAP negatively regulates defense responses by suppressing LSU2 through physical interaction.

132 hown to exacerbate motor symptoms of DA loss by suppressing M(4)-autoreceptor-Galpha (i/o) signaling

133 reatine reprogrammed macrophage polarization by suppressing M(interferon-gamma [IFN-gamma]) yet promo

134 d enhance the anti-neoplasia effect of BPTES by suppressing malignant cells' glucose utilization.

135 nhibited alpha-SYN-induced neuroinflammation by suppressing microglial activation, macrophage and CD4

136 drive this learning-induced memory mechanism by suppressing microRNA-mediated translational silencing

137 a-secretase system decreases neuronal losses by suppressing miR-212 and increasing its target surviva

138 ive stress-induced DNA damage and cell death by suppressing mitochondrial ROS production.

139 oliferation in the forebrain, gut and breast by suppressing mitogenic WNT signaling in mouse.

140 They regulate gene expression by suppressing mRNA translation and reducing mRNA stabil

141 CKD induces loss of muscle proteins partly by suppressing muscle protein synthesis.

142 data establish that MNT synergizes with MYC by suppressing MYC-driven apoptosis, and that it does so

143 n modeled responses to MYCN-directed therapy by suppressing MYCN expression in MYCN-driven retinoblas

144 Intriguingly, Deltex2 inhibits myogenesis by suppressing MyoD transcription, and the Deltex2 knock

145 Traf2 in myocardial survival and homeostasis by suppressing necroptosis.

146 ndependent, prosurvival pathway in the heart by suppressing necroptotic signaling, which may serve as

147 SI-09, phenocopied the effects of Epac1 null by suppressing neointima formation and proliferative VSM

148 Microglia respond to neuronal activation by suppressing neuronal activity, and ablation of microg

149 ve regulation of acute inflammatory response by suppressing NF-kappaB activation and proinflammatory

150 KDM3C demonstrates anti-inflammatory effects by suppressing NF-kappaB signaling and osteoclastogenesi

151 ptor ligands, and oxidized lipids, primarily by suppressing NF-kappaB signaling.

152 (GM-CSF)-induced CD103(+) DC differentiation by suppressing NF-kappaB, STAT1/5, and p38 activation.

153 tor of HIV-1 gene-expression and replication by suppressing NF-kappaB-mediated activation of viral tr

154 a positively regulates inflammatory response by suppressing NKRF production, which might be targeted

155 tPA deficit attenuated functional hyperemia by suppressing NMDAR-dependent nitric oxide production d

156 oting sleep during the night but not the day by suppressing nocturnal arousal and hyperactivity.

157 n detectors improves motion discriminability by suppressing noise generated by the local structure of

158 niche pinwheel structures, at least in part by suppressing Notch activation in radial glial cells, w

159 EZH2 activated the Notch pathway by suppressing Notch repressors Numb and Fbxw7 via trime

160 suggest that the A1AR provides otoprotection by suppressing NOX3 and inflammation in the cochlea and

161 androgen-independent prostate cancer growth by suppressing nuclear receptor-mediated oxidative stres

162 el components in high dynamic range mixtures by suppressing one-bond (13)C satellite signals in (1)H

163 Methadone and buprenorphine act by suppressing opioid withdrawal symptoms and attenuatin

164 with a role in reducing physiological tremor by suppressing oscillations around 10 Hz.

165 Teriparatide increases osteoblast numbers by suppressing osteoblast apoptosis and activating bone-

166 oading regulates subchondral bone remodeling by suppressing osteoclast development, and prevents degr

167 emonstrate that Hdac3 controls bone modeling by suppressing osteoclast lineage cell responsiveness to

168 f OA, knee loading exerts protective effects by suppressing osteoclastogenesis through Wnt signaling,

169 ore, knee loading exerted protective effects by suppressing osteoclastogenesis through Wnt signaling.

170 destruction and alleviating bone cancer pain by suppressing osteoclastogenesis.

171 4E phosphorylation and was not recapitulated by suppressing other cabozantinib targets.

172 ductance enhances rate coding in place cells by suppressing out-of-field excitation and by limiting d

173 atory role in lineage-committed tissue cells by suppressing overactivation of multiple signalling pat

174 9 acts as a regulator of cellular metabolism by suppressing oxidation of fatty acids, and thus adapts

175 ion alleviated IRI and improved OLT survival by suppressing p-p38 upregulation, ROS induction, and HM

176 d the capacity of macrophages to proliferate by suppressing p21 expression to halt their differentiat

177 2 promotes breast cancer early dissemination by suppressing p38, but how Her2 downregulates p38 is un

178 reduces GBM cell migration and invasiveness by suppressing PCP signaling.

179 ductive and effector sites of oral tolerance by suppressing peripheral regulatory T cell (pTreg) conv

180 layed in AMPKalpha2 cKO mice were alleviated by suppressing PERK activity pharmacologically or geneti

181 ll proliferation, survival, and tumor growth by suppressing PI3K/mTOR/Akt activities in mouse models

182 phogenesis and modulates thermomorphogenesis by suppressing PIF4 activity, through a reduction in PIF

183 microbes can favor or improve insect fitness by suppressing plant defenses and detoxifying defensive

184 stability among all reported Mg/S batteries by suppressing polysulfide dissolution.

185 tion may protect against cerebral I/R injury by suppressing post-ischemic apoptosis, whereas heavy et

186 may protect the brain against its I/R injury by suppressing post-ischemic inflammation.

187 By suppressing precursors, scaling calculations show air

188 peptides into amyloid fibrils, in particular by suppressing primary nucleation.

189 ficantly attenuated early/acute inflammation by suppressing pro-inflammatory cytokines (TNF-alpha, IL

190 diolipin inhibits resolution of inflammation by suppressing production of anti-inflammatory IL-10 by

191 d the intracellular survival of T. marneffei by suppressing proinflammatory responses.

192 ith maturation and commitment to iNKT1 cells by suppressing proliferation and Opa1-related mitochondr

193 ion of aberrant neovessels into the vitreous by suppressing proliferation of endothelial cells (EC) i

194 was able to modulate HIF-1alpha degradation by suppressing prolyl hydroxylase 3 expression.

195 rmation of a more reactive phenotype enabled by suppressing proteolytic cleavage of sIL-6R from IL-6R

196 whether structural changes shape adaptation by suppressing recombination or by creating new mutation

197 r, vitamin D may also promote renoprotection by suppressing renin transcription through cross-talk be

198 increased spontaneous EPSC (sEPSC) frequency by suppressing RIM1alpha-facilitated CaV2.2 expression i

199 2 has a role in protecting cartilage from OA by suppressing Runx2-induced Adamts5 via Runx2 poly-ubiq

200 MAGE-B2, increases cellular stress tolerance by suppressing SG formation through translational inhibi

201 s, likely enhancing their information coding by suppressing shared spontaneous activity.

202 marrow-derived cells and limits inflammation by suppressing signaling through stimulatory receptors.

203 e foraging of other macronutrients primarily by suppressing simple sugar consumption.

204 Attenuating apprehensiveness by suppressing simulations of feared events may thus be

205 gic advantage to singlet fission solar cells by suppressing singlet dissociation at optimal driving f

206 thora sojae effector PSR1 promotes infection by suppressing small RNA biogenesis in plant hosts.

207 es, which maintains transcriptional fidelity by suppressing spurious transcription.

208 Blocking the nuclear import of GAPDH by suppressing Src signaling or through a GAPDH Tyr41 mu

209 that VP reduces Src kinase phosphorylation: by suppressing Src using the inhibitor dasatinib and siR

210 ulatory protein protects against nephropathy by suppressing STAT-mediated cell responses to diabetic

211 lopment of psoriasis in various mouse models by suppressing STAT3-mediated IkappaBzeta expression.

212 moting environmental condition, acts in part by suppressing stomatal defense and is linked to hormone

213 dings, technology was developed to treat RHT by suppressing sympathetic activity with electrical stim

214 Furthermore, we find that NCA promotes sleep by suppressing synaptic release from a dispersed wake-pr

215 en implicated in immune regulation, not only by suppressing T cell responses but also by regulating a

216 cancer cell stem-like properties as well as by suppressing T-cell activation.

217 of TCF4, we created an in vivo model of PTHS by suppressing Tcf4 expression in rat prefrontal neurons

218 al growth factor (VEGF) secretion in Mos/Mps by suppressing TGFbeta1 signalling and subsequently inhi

219 We show that LOX regulates EGFR by suppressing TGFbeta1 signalling through the secreted

220 ge performance could potentially be obtained by suppressing the (La,Sr)O termination and stabilizing

221 hat MHC-matching reduces the immune response by suppressing the accumulation of microglia (Iba-1+) an

222 ith improvement in progression-free survival by suppressing the activating mutation and preventing th

223 demonstrate that Notch maintains type II NBs by suppressing the activation of earmuff (erm) by Pointe

224 on pathway, promotes plasma cell development by suppressing the activation of PERK.

225 gm1 negatively regulates IL-1beta maturation by suppressing the activation of the NLRP3 inflammasome.

226 ctivation of transcription of the BIC genes, by suppressing the activity of CONSTITUTIVE PHOTOMORPHOG

227 -124-3p on neuronal inflammation was exerted by suppressing the activity of mTOR signaling.

228 he inhibitory effect of the spacing was lost by suppressing the activity of Ras or mitogen-activated

229 ated macrophages (TAMs) support tumor growth by suppressing the activity of tumor-infiltrating T cell

230 pancreatitis in EtOH-sensitized acinar cells by suppressing the adaptive unfolded protein response si

231 affects the RNA remodeling ability of LAF-1 by suppressing the affinity, dynamics, and annealing act

232 latory network controlling tumor progression by suppressing the Akt pathway.

233 y shaping the architecture of the tumour and by suppressing the antitumour activities of immune cells

234 exposure may be harmful to our immune system by suppressing the antiviral gene program.

235 tion, regulates miRNA abundance and function by suppressing the biogenesis of ribosomal RNA-derived s

236 viate pain and reduce fever and inflammation by suppressing the biosynthesis of prostacyclin (PGI2) a

237 othesized that PIN1 relieves CDK2 inhibition by suppressing the CDK inhibitory activity of p27.

238 ecular strategy to adapt to hot environments by suppressing the efficiency of TRPV1-mediated heat det

239 novel class of NRF2 activators that function by suppressing the enzymatic activity of an epigenetic r

240 not Tyr(701)) and STAT1 luciferase activity by suppressing the ERK1/2, p38, and JNK mitogen-activate

241 pectral resolution, which can be implemented by suppressing the experimental noises.

242 inhibition in an experience-dependent manner by suppressing the expression of EphB4; PV cells hemizyg

243 0 protected LRP5/6(DeltaCD11c) mice from CAC by suppressing the expression of inflammatory factors.

244 miR-126-3p enhanced angiogenesis by suppressing the expression of its known target, SPRED

245 beta inhibits macrophage foam cell formation by suppressing the expression of key genes implicated in

246 reover, BCL11A promotes cancer cell invasion by suppressing the expression of muscleblind-like splici

247 ed against renal ischemia-reperfusion injury by suppressing the expression of proinflammatory genes a

248 er engagement of the B cell antigen receptor by suppressing the expression of the autoimmune suppress

249 node that switches off antimigratory miR-198 by suppressing the expression of the regulatory factor,

250 d that IDR-1002 reduced sterile inflammation by suppressing the expression of transmembrane G protein

251 gnetoreception in honey bees can be explored by suppressing the formation of iron granules.

252 The coating results in superior performance by suppressing the formation of Li dendrites and inactiv

253 ify chromatin and to support DNA replication by suppressing the formation of R-loops (DNA/RNA-hybrids

254 The compounds likely enhanced cell viability by suppressing the formation of reactive oxygen species

255 increase in the yield of expected runoff RNA by suppressing the formation of undesired longer RNA byp

256 IFN-gamma repressed genes by suppressing the function of enhancers enriched for bi

257 spinal mechanism whereby pain inhibits itch by suppressing the function of GRPR neurons.

258 ally, mir156 mutations enhance seed dormancy by suppressing the gibberellin (GA) pathway through de-r

259 , simvastatin improved restenosis indicators by suppressing the HIF-1alpha/calpains/MMP2/TGF-beta1 pa

260 essential for facilitating biotrophic growth by suppressing the host oxidative burst-the first line o

261 thelialization, and collagen deposition, and by suppressing the inflammatory response and apoptosis.

262 otes CSR and aberrant genomic rearrangements by suppressing the insertional DNA repair pathway.

263 Mechanistically, e18 induces HDR by suppressing the localization of the NHEJ-promoting fa

264 stricts the EHT of lymphoid-myeloid lineages by suppressing the mannosyltransferase alg2 and sialyltr

265 it blocks MDA5-mediated antiviral signaling by suppressing the MDA5-dsRNA interaction.

266 hieve more control over the mode selectivity by suppressing the mode that has the highest gain (i.e.

267 onstrates that Ubx stabilizes lineage choice by suppressing the multipotency encoded in the genome vi

268 r stress counteracts endothelial dysfunction by suppressing the pro-inflammatory non-canonical TGF-be

269 ally expanded CD8(+) T cells inhibit disease by suppressing the proliferation of MOG-specific CD4(+)

270 When the population is further increased by suppressing the radiative recombination rate with the

271 s strong evidence that the brain compensates by suppressing the retinal motion due to self-motion, ho

272 yclosporine into the aqueous phase, probably by suppressing the solubilization capability of the mice

273 utes to female germline stem cell unipotency by suppressing the somatic program and is potentially in

274 l monitoring of genetic and protein circuits by suppressing the spectral cross-talk among multiple fl

275 ominent loss-of-function mechanism in cancer by suppressing the steady-state levels of PKC.

276 y regulates hepatic cancer stem cells (CSCs) by suppressing the tumor suppressor p53.

277 miR-200 family promotes the epithelial state by suppressing the Zeb1/Zeb2 epithelial gene transcripti

278 INB1) limited the activity of those caspases by suppressing their caspase-recruitment domain (CARD) o

279 n by removing unused or unwanted memories or by suppressing their expression.

280 People often respond to intrusions by suppressing their retrieval, excluding them from awar

281 represents a promising therapeutic strategy by suppressing therapy-induced senescence-associated CSC

282 AAL(S) reduced the severity of acute AAD by suppressing tissue eosinophils and inflammation, mucu

283 hibit experimental fungal keratitis in mouse by suppressing TLR4 and inflammatory cytokines such as T

284 dendritic cell (DC) responses, specifically by suppressing TLR4 signalling to inhibit Th1/Th17-drive

285 ack and alters macrophage metabolic pathways by suppressing TLR4-mediated gene activation.

286 naive CD8(+) T cells required for activation by suppressing TMEM20, a negative regulator of Ca(2+) ex

287 meostasis, it blocked osteoclast development by suppressing transcription factors c-Fos and JunB, but

288 ATR protects the genome from R loops by suppressing transcription-replication collisions, pro

289 overed that OCTR-1 regulates innate immunity by suppressing translation and the unfolded protein resp

290 nt role in modulating disease-risk in humans by suppressing tumour progression, decreasing inflammati

291 s expressing a LIF transgene reduce fibrosis by suppressing type 2 immunity and highlight a novel app

292 he FGF19-SHP-LSD1 axis maintains homeostasis by suppressing unnecessary autophagic breakdown of cellu

293 h revealed that Daam2 promotes tumorigenesis by suppressing VHL expression.

294 ox protein O1 (FoxO1) maintains KSHV latency by suppressing viral lytic replication.

295 ferentiation-related gene expression profile by suppressing Wnt pathway activity and reducing mesench

296 the emergence of an abnormal stem cell state by suppressing Wnt signaling in stem cells.

297 umor suppressor, which inhibits tumor growth by suppressing Wnt/beta-catenin signaling in lung adenoc

298 ppears to modify the specificity of the MAPK by suppressing Y kinase and enhancing S/T kinase activit

299 ine is able to inhibit survival of GBM cells by suppressing YAP/TAZ activities.

300 n epithelial to mesenchymal transition (EMT) by suppressing Zeb1 and Zeb2 expression.