ライフサイエンスコーパス: cAMP response element

1 ynthetic promoters containing CREB1-specific cAMP response elements.

2 hich contains evolutionarily conserved AP-1, cAMP-response element, and C/EBP half-sites, in synergis

3 ) prevent immunosuppressive transcription of cAMP response element- and hypoxia response element-cont

4 and the subsequent release of ATF3 from its cAMP response element/ATF target site.

5 Here we show that the cAMP response element binding (CREB) coactivator Crtc3 p

6 mRNA expression and secretion by inhibiting cAMP response element binding (CREB) protein and AKT pho

7 n (KID) of the DNA transcriptional activator cAMP response element binding (CREB) protein and the KIX

8 ough the PKA-mediated phosphorylation of the cAMP response element binding (CREB) protein and via the

9 , which decreased two transcription factors, cAMP response element binding and Nurr1, controlling syn

10 The transcription factors CREB (cAMP response element binding factor), SRF (serum respon

11 itutively activates the transcription factor cAMP response element binding protein (CREB) and CREB ta

12 CF included increased phosphorylation of the cAMP response element binding protein (CREB) and elevate

13 s function via increasing phosphorylation of CAMP response element binding protein (CREB) and PSD95 a

14 we found that activated PERK phosphorylates CAMP response element binding protein (CREB) and PSD95 d

15 ecessary role for two transcription factors, cAMP response element binding protein (CREB) and serum r

16 t) and downstream transcription factors, the cAMP response element binding protein (CREB) and signal

17 activating transcription factors such as the cAMP response element binding protein (CREB) and the ser

18 neurons transfected with a dominant-negative cAMP response element binding protein (CREB) and was eli

19 nts for IL-1beta and IFN-gamma were bound by cAMP response element binding protein (CREB) and zinc-fi

20 ssociates with the transcriptional regulator cAMP response element binding protein (CREB) in both mou

21 of cellular cAMP and the phosphorylation of cAMP response element binding protein (CREB) in INS-1 ce

22 ing, and its downstream transcription factor cAMP response element binding protein (CREB) in the cont

23 cAMP response element binding protein (CREB) is a key re

24 In particular, the PKC target protein cAMP response element binding protein (CREB) is a major

25 monophosphate (cAMP)-protein kinase A (PKA)-cAMP response element binding protein (CREB) pathway.

26 anization all impair efficient TSH-dependent cAMP response element binding protein (CREB) phosphoryla

27 Furthermore, TDCA significantly increased cAMP response element binding protein (CREB) phosphoryla

28 us slices through mechanisms associated with cAMP response element binding protein (CREB) signaling.

29 fying the stimulatory effects of the drug on cAMP response element binding protein (CREB) signalling.

30 ootshock) activates the transcription factor cAMP response element binding protein (CREB) within the

31 te (cGMP) leading to increased levels of the cAMP response element binding protein (CREB), a transcri

32 ed binding site for the transcription factor cAMP response element binding protein (CREB), and we dem

33 in striatal MSNs and resultant activation of cAMP response element binding protein (CREB), in rat pri

34 phosphomimetic mutant S386/396E bound to the cAMP response element binding protein (CREB)-binding pro

35 The starvation-inducible coactivator cAMP response element binding protein (CREB)-cAMP-regula

36 /calmodulin kinase-I (CaMKI), which triggers cAMP response element binding protein (CREB)-dependent W

37 This photic resetting involves cAMP response element binding protein (CREB)-mediated up

38 Chronic exposure to addictive drugs enhances cAMP response element binding protein (CREB)-regulated g

39 slocation that generates an unusual chimeric cAMP response element binding protein (CREB)-regulated t

40 n intracellular domain associated-1 protein, cAMP response element binding protein (CREB)-regulated t

41 For example, the cAMP response element binding protein (CREB)-regulated t

42 eptors, adenyl cyclase, protein kinase A and cAMP response element binding protein (CREB).

43 orylation of the transcription factor Ca(2+)/cAMP response element binding protein (CREB).

44 the GcgR cDNA restored hepatic GcgR, phospho-cAMP response element binding protein (P-CREB), and phos

45 rmal rats, riluzole increased phosphorylated cAMP response element binding protein (pCREB) expressing

46 [Caenorhabditis elegans homolog of mammalian cAMP response element binding protein 1 (CREB1)] isoform

47 Luman/cAMP response element binding protein 3 is an endoplasmi

48 C1A-induced IRS2 expression was dependent on cAMP Response Element Binding Protein activity and that

49 a (~1,200 pg/mL), but hepatic phosphorylated cAMP response element binding protein and phosphoenolpyr

50 signaling, leading to the phosphorylation of cAMP response element binding protein and, consequently,

51 nscription levels of specific members of the cAMP Response Element Binding protein gene family.

52 lin-dependent protein kinase IV (CaMKIV) and cAMP response element binding protein mediate the Ca(2)(

53 providing the first evidence that enhancing cAMP response element binding protein signaling can alle

54 we reveal a previously unappreciated role of cAMP response element binding protein signaling in RTT p

55 4D is hyperactive, leading to a reduction in cAMP response element binding protein signaling, but thi

56 eting human Apolipoprotien C3 (Apoc3), CREB (cAMP Response Element Binding Protein) Regulated Transcr

57 morphine suppressed binding of phospho-CREB (cAMP response element binding protein) to Bdnf promoters

58 cAMP-regulated phosphoprotein-32) and CREB (cAMP response element binding protein), and locomotor ac

59 roaches that boost memories by targeting the cAMP response element binding protein-CCAAT enhancer bin

60 injured DRG through the transcription factor cAMP response element binding protein-triggered transcri

61 se kinase-3, ribosomal S6 kinase, c-Jun, and cAMP response element binding protein.

62 hosphorylated AKT (p-AKT) and phosphorylated cAMP response element binding-factor (p-CREB) than simil

63 ing pathway culminating in activation of the cAMP response element binding.

64 oprecipitation experiments demonstrated that cAMP response elements binding protein regulates the exp

65 fferential regulation of CRH relies upon the cAMP response-element binding protein coactivator CRTC2,

66 gh increased cyclic adenosine monophosphate (cAMP) response element binding (CREB)/CREB binding prote

67 ption factor cyclic adenosine monophosphate (cAMP) response element binding protein (CREB) and the ne

68 F1 to the GH promoter along with cyclic AMP (cAMP) response element binding protein (CREB) binding pr

69 e demonstrated that silencing of cyclic AMP (cAMP) response element binding protein 3-like 1 (CREB3L1

70 gene expression after learning requires the cAMP-response element binding (CREB) interaction domain

71 phosphorylation of the transcription factors cAMP-response element binding protein (CREB) and signal

72 cAMP-response element binding protein (CREB) is a nuclea

73 extracellular signal-regulated kinase (ERK)-cAMP-response element binding protein (CREB) signaling,

74 itical role in memory formation, such as the cAMP-response element binding protein (CREB), have been

75 tes the formation of Abeta by activating the cAMP-response element binding protein (CREB), which in t

76 ed a novel intracellular signaling molecule, cAMP-response element binding protein (CREB), which serv

77 phokinase A (PKA)-mediated activation of the cAMP-response element binding protein (CREB1) to transcr

78 activity toward protein substrates, such as cAMP-response element binding protein and cardiac tropon

79 itute for WT-PKD1 as an in vivo activator of cAMP-response element binding protein and ERK phosphoryl

80 ound an enduring reduction in phosphorylated cAMP-response element binding protein levels in the NAcS

81 e activity of the transcription factor CREB (cAMP-response element binding protein).

82 ria, cytochrome oxidase subunit 4 isoform 1, cAMP-response element binding, p38 MAPK and adenosine mo

83 the induction of a persistent activation of cAMP-response element binding-protein (CREB) and C/EBPbe

84 ults in decreased cAMP signaling and reduced cAMP-response-element binding protein (CREB) activation,

85 observed a significant increase in levels of cAMP response element- binding (CREB) transcripts in the

86 tigated the role of Transducers of Regulated cAMP response element-binding (CREB) protein activity (T

87 IL-10 production via the phosphorylation of cAMP response element-binding (CREB) protein on the IL-1

88 ower, behavioral-stimulant doses enhance the cAMP response element-binding (CREB) signaling system, w

89 e production, at least in part by increasing cAMP response element-binding (CREB) stability.

90 complex acted as a competitive inhibitor of cAMP response element-binding (CREB) transcription facto

91 n actions with histone acetylation enhancing cAMP response element-binding (CREB)-associated transcri

92 h increased NF-kappaB-mediated and decreased cAMP Response Element-Binding (CREB)-mediated Tax activi

93 ption factor 5 (ATF5) is a member of the ATF/cAMP response element-binding family of transcription fa

94 cells and induced Treg expansion through the cAMP response element-binding pathway.

95 PKA-induced cAMP response element-binding phosphorylation is normal

96 odegeneration) in neonatal mice and reversed cAMP response element-binding protein (CREB) activation

97 rophage-derived IL-10 resulted in epithelial cAMP response element-binding protein (CREB) activation

98 to increased Erk1/2 MAP-kinase signaling and cAMP response element-binding protein (CREB) activation

99 ha) activation by free fatty acid (FFA), and cAMP response element-binding protein (CREB) activation

100 kers of antidepressant action: activation of cAMP response element-binding protein (CREB) and inducti

101 cAMP response element-binding protein (CREB) and nuclear

102 TX phosphorylated the cAMP response element-binding protein (CREB) and recruit

103 genic program through the phosphorylation of cAMP response element-binding protein (CREB) and the dep

104 he activated PKA subsequently phosphorylated cAMP response element-binding protein (CREB) at Ser-133

105 For example, deficits in cAMP response element-binding protein (CREB) binding pro

106 R) and the fasting transcriptional activator cAMP response element-binding protein (CREB) coordinatel

107 ar mechanisms of transcriptional control via cAMP response element-binding protein (CREB) during indu

108 The transcription factor cAMP response element-binding protein (CREB) has been im

109 dulin-dependent protein kinase (CaMK) II and cAMP response element-binding protein (CREB) in cultured

110 demonstrated that E2 rapidly phosphorylates cAMP response element-binding protein (CREB) in GnRH neu

111 ssion of FTO delays the dephosphorylation of cAMP response element-binding protein (CREB) in human ne

112 reasing activity of the transcription factor cAMP response element-binding protein (CREB) in young ad

113 The cAMP response element-binding protein (CREB) is a highly

114 The cAMP response element-binding protein (CREB) is a nuclea

115 Phosphorylation of cAMP response element-binding protein (CREB) is also sig

116 The activity-regulated transcription factor cAMP response element-binding protein (CREB) is an essen

117 The transcription factor cAMP response element-binding protein (CREB) is required

118 nd that the p90 ribosomal S6 kinase 2 (RSK2)-cAMP response element-binding protein (CREB) pathway is

119 Low picomolar PregS similarly activates cAMP response element-binding protein (CREB) phosphoryla

120 ic administration of rolipram also increased cAMP response element-binding protein (CREB) phosphoryla

121 -WT, exhibited higher basal PKA activity and cAMP response element-binding protein (CREB) phosphoryla

122 n part via the PKA-mediated induction of the cAMP response element-binding protein (CREB) signaling p

123 ate phase (Day 5) characterized by increased cAMP response element-binding protein (CREB) signalling

124 IP) and whole-genome sequencing) to identify cAMP response element-binding protein (CREB) targets fol

125 t uncovered that HDAC2 is a direct target of cAMP response element-binding protein (CREB) that is act

126 kinases converge on the transcription factor cAMP response element-binding protein (CREB) to enhance

127 mitogen-activated protein kinase (MAPK), and cAMP response element-binding protein (CREB) transcripti

128 We previously demonstrated that the cAMP response element-binding protein (CREB) transcripti

129 We found that loss of the cAMP response element-binding protein (CREB) transcripti

130 endent protein kinase II (CaMKII) and Ca(2+)/cAMP response element-binding protein (CREB) transcripti

131 ression of targets of protein kinase A (PKA)-cAMP response element-binding protein (CREB), a pathway

132 Here, we use outbred rats to investigate if cAMP response element-binding protein (CREB), a transcri

133 on of Arc and phosphorylation of cofilin and cAMP response element-binding protein (CREB), a transcri

134 n cortisol production and phosphorylation of cAMP response element-binding protein (CREB), a transcri

135 inhibition did not affect phosphorylation of cAMP response element-binding protein (CREB), a well-est

136 ntisense to mRNA for a transcription factor, cAMP response element-binding protein (CREB), and by an

137 espectively, nuclear factor kappa B (NF-kB), cAMP response element-binding protein (CREB), and glucoc

138 G1 also enhanced cAMP response element-binding protein (CREB), as well as

139 ased mRNA of ERK2 and its downstream targets cAMP response element-binding protein (CREB), BDNF, c-Fo

140 ylation and activity of transcription factor cAMP response element-binding protein (CREB), binding si

141 HCV increased activation of cAMP response element-binding protein (CREB), CCAAT/enha

142 enhanced phosphorylation of a CaN substrate, cAMP response element-binding protein (CREB), in the bra

143 Phosphorylated cAMP response element-binding protein (CREB), nuclear NF

144 racellular cAMP increased phosphorylation of cAMP response element-binding protein (CREB), which, in

145 egulation of prosurvival signaling (i.e. the cAMP response element-binding protein (CREB)-Bdnf cascad

146 Here, we show that high glucose induced cAMP response element-binding protein (CREB)-binding pro

147 ssion restored normal ryanodine receptor and cAMP response element-binding protein (CREB)-dependent g

148 rotein kinase (MAPK) signaling, induction of cAMP response element-binding protein (CREB)-insulin rec

149 ffects of HT-0712 on memory formation and on cAMP response element-binding protein (CREB)-regulated g

150 duction by inducing the dephosphorylation of cAMP response element-binding protein (CREB)-regulated t

151 synthases and reduced phosphorylation of the cAMP response element-binding protein (CREB).

152 SCO rapidly enhanced the phosphorylation of cAMP response element-binding protein (CREB).

153 ription factor mediator of Ca(2+)-signals is cAMP response element-binding protein (CREB).

154 downstream cascade adenylyl cyclase-cAMP-PKA-cAMP response element-binding protein (CREB).

155 sensitivity through GPR160-dependent ERK and cAMP response element-binding protein (CREB).

156 n of ID1 by prostaglandin E2 was mediated by cAMP response element-binding protein (CREB).

157 The transcription factor cAMP response element-binding protein (CREB1) has been s

158 esterase 4D (PDE4D) gene expression, phospho-cAMP response element-binding protein (p-CREB), and cAMP

159 ne by increasing occupancy of phosphorylated cAMP response element-binding protein (pCREB) at two sit

160 ippocampi had lower levels of phosphorylated cAMP response element-binding protein (pCREB), an activi

161 phosphorylation of the transcription factor cAMP response element-binding protein (pCREB).

162 hippocampal neurogenesis and phosphorylated cAMP response element-binding protein (pCREB).

163 suggests that the transcriptional activator cAMP response element-binding protein 1 (CREB1) is impor

164 ate (cAMP) to synthetic promoters containing cAMP response element-binding protein 1(CREB1)-specific

165 y decreasing MCU's transcriptional regulator cAMP response element-binding protein 1.

166 of the downstream transcriptional repressor cAMP response element-binding protein 2 in SNs.

167 Remarkably, ceramide-induced RIP of cAMP response element-binding protein 3-like 1 (CREB3L1)

168 inhibits proteolytic processing of CREB3L1 (cAMP response element-binding protein 3-like 1), a membr

169 ing pathway and required for PACAP-dependent cAMP response element-binding protein activation and neu

170 ZIP12 knockdown reduces cAMP response element-binding protein activation and pho

171 Constitutive cAMP response element-binding protein activation restore

172 also increased the nuclear translocation of cAMP response element-binding protein and CCAAT/enhancer

173 ells in part by inhibiting the activation of cAMP response element-binding protein and expression of

174 cer-binding protein beta was required, while cAMP response element-binding protein and signal transdu

175 PKG1 and cAMP response element-binding protein are involved in th

176 The protein acetyltransferases p300 and cAMP response element-binding protein binding protein (C

177 33 (miR-433) inhibition of expression of the cAMP response element-binding protein CREB1 represses he

178 They also showed reduced phosphorylated cAMP response element-binding protein expression within

179 sing a repressor of the transcription factor cAMP response element-binding protein or a calcium/calmo

180 ndent of the canonical cAMP/Protein Kinase A/cAMP response element-binding protein pathway downstream

181 s --> adenylate cyclase --> cAMP --> PKA --> cAMP response element-binding protein pathway mediating

182 pe, we administered rolipram to activate the cAMP response element-binding protein pathway, which led

183 n of the cAMP-dependent protein kinase (PKA)/cAMP response element-binding protein pathway.

184 Consistently, leptin promotes cAMP response element-binding protein phosphorylation in

185 ments of hippocampal synaptic plasticity and cAMP response element-binding protein phosphorylation.

186 and pEC50 of ACEA-induced Galphas-dependent cAMP response element-binding protein phosphorylation.

187 Consequently, the cAMP-->PKA-->cAMP response element-binding protein signaling axis is

188 phosphorylation of the transcription factor "cAMP response element-binding protein" (CREB) are also a

189 cient neuronal cell lines have reduced CREB (cAMP response element-binding protein) expression and in

190 eam targets, such as ATF-2, c-Jun, and CREB (cAMP response element-binding protein), was disrupted.

191 tream target of ERK1/2, pCREB [phospho-CREB (cAMP response element-binding protein)].

192 oxygen species production, and activation of cAMP response element-binding protein, a critical transc

193 activation was followed by downregulation of cAMP response element-binding protein, and LTP impairmen

194 n factors, including activator protein 1 and cAMP response element-binding protein, both of which wer

195 ciated with a reduction in the activation of cAMP response element-binding protein, but not the activ

196 sponse element-binding protein (ChREBP), and cAMP response element-binding protein, hepatocyte specif

197 rylation status of their respective targets, cAMP response element-binding protein, p38, and extracel

198 ecretase activates the transcription factor, cAMP response element-binding protein, regulating miR-21

199 ads to induction of the transcription factor cAMP response element-binding protein-3-like-2 (CREB3L2)

200 RORalpha recruited cAMP response element-binding protein-binding protein (C

201 ylation is diminished, thereby enabling p300/cAMP response element-binding protein-binding protein to

202 acid receptor alpha, and HATs (p300 and p300/cAMP response element-binding protein-binding protein-as

203 ediated ERalpha protein methylation and p300/cAMP response element-binding protein-binding protein-de

204 llular outputs: p38-dependent growth arrest, cAMP response element-binding protein-dependent cell sur

205 Inhibition of cAMP response element-binding protein-dependent signalin

206 kinase 2 (SIK2) is an important regulator of cAMP response element-binding protein-mediated gene expr

207 of a panel of proteins including the ERK and cAMP response element-binding protein.

208 active oxygen species, and the activation of cAMP response element-binding protein.

209 shown by reduction in phosphorylation of the cAMP response element-binding protein.

210 ivation of a pathway involving Akt1/Akt2 and cAMP response element-binding protein.

211 phosphorylation of the transcription factor cAMP response element-binding protein.

212 ated by the transcription factors CREB/CREM (cAMP response element-binding protein/modulator) is link

213 that synaptopodin functions independently of cAMP response element-binding.

214 m channels couple membrane depolarization to cAMP response-element-binding protein (CREB)-dependent t

215 factor (ATF)-adenosine 3',5'-monophosphate (cAMP) response element-binding protein (CREB) family tra

216 l-regulating kinase 1 (ASK1) and cyclic AMP (cAMP) response element-binding protein (CREB).

217 we found that PKA-induced phosphorylation of cAMP-response element-binding protein ((P)CREB) and EPAC

218 The transcription factors, cAMP-response element-binding protein (CREB) and heat-sh

219 phosphorylation of the transcription factor cAMP-response element-binding protein (CREB) as well as

220 he murine RGS2 promoter that is critical for cAMP-response element-binding protein (CREB) binding and

221 A cAMP-response element-binding protein (CREB) binding seq

222 AT/enhancer-binding protein (C/EBPbeta), and cAMP-response element-binding protein (CREB) have been i

223 inase A activity, thus reducing both phospho-cAMP-response element-binding protein (CREB) levels and

224 to G transition under basal/nicotine-induced/cAMP-response element-binding protein (CREB) overexpress

225 -3 expression via the protein kinase C (PKC)-cAMP-response element-binding protein (CREB) pathway.

226 tracellular cAMP production was impaired and cAMP-response element-binding protein (CREB) phosphoryla

227 hrough a cAMP-dependent binding of FGFR1 and cAMP-response element-binding protein (CREB) to a conser

228 nstrate that RNS60 induced the activation of cAMP-response element-binding protein (CREB) via the PI

229 The activation of cAMP-response element-binding protein (CREB), but not NF

230 attenuating the expression of phosphorylated cAMP-response element-binding protein (CREB), c-Fos, and

231 e endogenous Galphas promoter is occupied by cAMP-response element-binding protein (CREB), Egr-1, and

232 However, phosphorylation of cAMP-response element-binding protein (CREB), the cAMP-r

233 cAMP-response element-binding protein 1 (CREB1) has been

234 nalysis reveals C1q-activated phosphorylated cAMP-response element-binding protein and AP-1, two tran

235 eir key transcription factors phosphorylated cAMP-response element-binding protein and forkhead box O

236 rgets with functions in adipogenesis such as cAMP-response element-binding protein and FOXO1; however

237 regulates hepatic glucose production through cAMP-response element-binding protein co-activators, we

238 protein, which induced the expression of the cAMP-response element-binding protein family repressor c

239 n neurons, and in the efficient signaling to cAMP-response element-binding protein in neurons.

240 ed inactivation of the cAMP/protein kinase A/cAMP-response element-binding protein signaling pathway,

241 was regulated by transcription factor CREB (cAMP-response element-binding protein) and silencing of

242 ta-mediated increases in PKA activity, CREB (cAMP-response element-binding protein) phosphorylation,

243 activity and elevated STMN Ser-63 and CREB (cAMP-response element-binding protein) Ser-133 phosphory

244 ceptor-gamma accompanied with ATF2 and CREB (cAMP-response element-binding protein) was enhanced acro

245 nal activators, Tax and phosphorylated CREB (cAMP-response element-binding protein), recruited the p3

246 Furthermore, cAMP-response element-binding protein, as a downstream t

247 Silencing of cAMP-response element-binding protein, LRP1B or GPR6 exp

248 phosphorylation of the transcription factor cAMP-response element-binding protein, which induced the

249 Overexpression of the p300/cAMP-response element-binding protein-binding protein (C

250 monstrate that the lysine acetyltransferases cAMP-response element-binding protein-binding protein (C

251 H promoter recruited the epigenetic modifier cAMP-response element-binding protein-binding protein/p3

252 n and mouse islet alpha cells by GS/cAMP/PKA/cAMP-response element-binding protein-dependent activati

253 t not PDE4B ablation significantly prolonged cAMP-response element-binding protein-mediated transcrip

254 e classical nuclear calcium-CaMKIV-CREB/CBP (cAMP-response element-binding protein/CREB-binding prote

255 racellular cAMP levels and the activation of cAMP-response element-binding, a protein kinase A downst

256 ad determined that nuclear sAC activates the cAMP-response-element-binding (CREB) transcription facto

257 lin-dependent protein kinase type (CAMK)-IV, cAMP-response-element-binding protein (CREB) and brain-d

258 ed CREB with consequent augmented binding on cAMP response element consensus sequence on peroxisome p

259 ng PPARgamma target genes and ERK-regulated, cAMP response element-containing target genes.

260 lement-binding protein (CREB) to a conserved cAMP response element (CRE) contiguous with the NFAT bin

261 tergic selection gene tlx3 through a variant cAMP response element (CRE) in its promoter.

262 d transcriptional activation mediated by the cAMP response element (CRE) in reporter gene studies (10

263 differentially expressed genes containing a cAMP response element (CRE) in their proximal promoter,

264 ly through the methylation of a CpG within a cAMP response element (CRE) motif defined by its promote

265 promoter region contains putative conserved cAMP response element (CRE) regions, which we found can

266 t AC inhibitors in HEK293 cells expressing a cAMP response element (CRE) reporter gene, and MDL-12,33

267 ER stress or ATF4 coexpression: the -267 ATF/cAMP response element (CRE) site and a novel -248 ATF/CR

268 A newly identified cAMP response element (CRE) site on the SYK promoter was

269 found that the synapsin promoter contained a cAMP response element (CRE), raising the possibility tha

270 The cAMP response element (CRE)-binding protein (CREB) is a

271 oter of human GLUT3 contains three potential cAMP response element (CRE)-like elements, CRE1, CRE2 an

272 sponse element-binding protein (p-CREB), and cAMP response element (CRE)-Luc, or PDGF-induced cyclin

273 activated T cells (NFAT) and the cyclic AMP (cAMP) response element (CRE) modulate KSHV-mediated tran

274 Moreover, we identified a conserved cAMP-response element (CRE) in the murine RGS2 promoter

275 or the function of a proximal renin promoter cAMP-response element (CRE).

276 eceptor binding elements [RARE], cyclic AMP [cAMP] response elements [CRE], NF-kappaB binding sites [

277 ack binding sites for RA-response element or cAMP-response element (CREB) in their promoter regions.

278 ivating function of DHX9, which acts through cAMP response elements (CREs), suggesting that SM may al

279 ffinity on palindromic and variant half-site cAMP response elements (CREs).

280 Two functional cAMP-response elements (CREs) were identified in the nmn

281 in DEL homozygotes treated with PDE3i was a cAMP response element enhancer 61 nt downstream from the

282 transcriptional control by the c-Jun-binding cAMP response element in the GADD34 gene promoter and po

283 (PGC-1alpha) and involved activation of the cAMP response element in the PEPCK promoter.

284 porter gene constructs revealed a functional cAMP response element in the proximal promoter of Lrrtm2

285 19 gene transcription through binding to the cAMP-response element in the p19 promoter.

286 ockdown of CREB, the presence of a consensus cAMP-response element in the promoter of CNTF, and the r

287 gical function in reproduction, we used Cre (cAMP response element)/LoxP technology to generate GnRH

288 The transcriptional repressor cAMP response element modulator (CREM) alpha has importa

289 eased expression of the transcription factor cAMP response element modulator (CREM) alpha promotes al

290 EB knock-outs (KOs)] with and without global cAMP response element modulator (CREM) deletion (global-

291 s a transcriptional repressor isoform of the cAMP response element modulator (CREM).

292 Transcription factor cAMP response element modulator (CREM)alpha contributes

293 cription through decreased activation of the cAMP response element modulator alpha (CREM-alpha) and r

294 Upregulation of the CREB-family protein cAMP response-element modulator (CREM) was observed afte

295 report the critical role of ventral striatal cAMP-response element modulator (CREM) in mediating impu

296 In turn, cAMP-response element modulator was found to associate w

297 nse element-binding protein family repressor cAMP-response element modulator.

298 r (DAF) promoter activity via binding to the cAMP response element, mutation of which attenuated prom

299 ene expression through the binding to tandem cAMP-response element sites in the proximal promoter reg

300 f3 encode transcription factors that contain cAMP response elements, suggesting that the methylation