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1 after hyperstimulation with the CCK-8 analog caerulein.
2 lenged with seven more weeks of twice weekly caerulein.
3 B-mediated trypsinogen activation induced by caerulein.
4 r supramaximal secretagogue stimulation with caerulein.
5 but not when they are induced by exposure to caerulein.
6 d by 12 hourly intraperitoneal injections of caerulein.
7 ximally stimulating dose of the secretagogue caerulein.
8 LSL-Kras(G12D/+); Pdx-1-Cre) stimulated with caerulein.
9  supramaximally stimulating concentration of caerulein (10 nM).
10 se 5 weeks before induction of pancreatitis (caerulein, 50 mug/kg).
11                                              Caerulein administration to nontransgenic mice produced
12 ration of supraphysiologic concentrations of caerulein, an ortholog of cholecystokinin.
13 in Sprague-Dawley rats with a combination of caerulein and controlled intraductal infusion.
14 ol and fatty acids was between the extent of caerulein and L-arginine induction, with obvious inflamm
15 at HO-1 is induced in pancreatitis caused by caerulein and more prominently in severe pancreatitis ca
16                                              Caerulein and palmitoleic acid (POA)/ethanol-induced pan
17 he effect of prior water immersion stress on caerulein and tumor necrosis factor-alpha (TNF-alpha)-in
18 ole orally or intravenously for 30 days, and caerulein as a positive control.
19 administration of L-arginine (also in rats), caerulein, bile acid, or an AP-inducing diet.
20 lar effects to water immersion in preventing caerulein but not TNF-alpha-induced NF-kappaB activation
21 TG-AP by poloxamer 407 (P-407) combined with caerulein (Cae).
22 imal (10 microg/kg/h) dose of the CCK analog caerulein (CER).
23  NF-kappaB bioluminescence following 12 h of caerulein compared with baseline luminescence (p < 0.05)
24 ificantly lower in transgenic mice receiving caerulein compared with nontransgenic mice.
25 , in two groups of frog, an identical toxin, caerulein, has arisen repeatedly from unique genes in th
26 ntrations of cholecystokinin or its analogue caerulein have been shown to stimulate the proteolytic a
27  Further, pancreatitis outcomes using a mild caerulein hyperstimulation model were similar between IP
28 episodes of acute pancreatitis (AP) based on caerulein hyperstimulation.
29 rve, in real time, trypsinogen activation by caerulein in the pancreatic cancer cell line, MIA PaCa-2
30                                Specifically, caerulein induced mild edematous pancreatitis accompanie
31       Rgs16::GFP is expressed in response to caerulein-induced acinar cell dedifferentiation, early n
32  to evaluate the progression and response of caerulein-induced acute and chronic pancreatitis.
33 aches aimed at characterising the effects of caerulein-induced acute pancreatitis (AP) on the vagal n
34  Plk1 overexpression significantly inhibited caerulein-induced acute pancreatitis and delayed develop
35 a(Cre/+)LSL-Kras(G12D/+)LSL-p53R172H/(+) and caerulein-induced acute pancreatitis mice models were us
36  for the miR-216a and miR-216b KOs following caerulein-induced acute pancreatitis.
37 duced by repeated episodes (twice weekly) of caerulein-induced AP (AP), we studied the involvement of
38 t hematopoietic cells were protected against caerulein-induced AP.
39 electron microscopy showed that BFA arrested caerulein-induced autophagosomal maturation.
40                                       In the caerulein-induced murine model of CP, administration of
41 Water immersion stress prevents supramaximal caerulein-induced NF-kappaB activation in pancreas in vi
42  of rats for up to 6 h prevents supramaximal caerulein-induced pancreatic IkappaB-alpha degradation a
43                             The reduction in caerulein-induced pancreatic inflammation is dependent u
44 hate supplementation reduced the severity of caerulein-induced pancreatic injury in mice on a low-pho
45 plasticity and pancreatic regeneration after caerulein-induced pancreatitis and in Kras(G12D)-driven
46                     Prior stress ameliorates caerulein-induced pancreatitis in rats.
47             Similarly, hemin pretreatment in caerulein-induced pancreatitis reduces serum amylase and
48 ult mice, we compared regeneration following caerulein-induced pancreatitis to that of normal pancrea
49 ry and impaired regeneration following acute caerulein-induced pancreatitis, leading to more severe d
50 y metaplasia, and persistent ADM after acute caerulein-induced pancreatitis.
51 t vasculature maintenance is observed during caerulein-induced pancreatitis.
52 ponsible for the effects of Tpl2 ablation on caerulein-induced proinflammatory events were evaluated
53  of agents that modulate intracellular pH on caerulein-induced trypsin and chymotrypsin activation we
54                                     Ex vivo, caerulein-induced trypsinogen activation is inhibited by
55  acinar cell injury in TLCS-induced, but not caerulein-induced, pancreatitis.
56  and trypsin activation were imaged in a rat caerulein-injection pancreatitis model.
57    We induced acute pancreatitis by repeated caerulein injections and isolated acinar and bone marrow
58         The mild model involved eight hourly caerulein injections and the severe model partial duct l
59                             After 4 weeks of caerulein injections, mice were administered a control o
60  pancreatitis induced in mice via repetitive caerulein injections.
61 ethods, such as the peritoneal injections of caerulein, L-arginine, the retrograde infusion of sodium
62 inflamed pancreatic tissue in a mouse model (caerulein/LPS model).
63 Combining secretagogues such as secretin and caerulein maximized zinc secretion and MRI signal in the
64                               In response to caerulein-mediated chronic or acute pancreatitis, WT mic
65                       The well-characterized caerulein model of CP was used to assess the therapeutic
66 dependently of trypsinogen activation in the caerulein model.
67 gonists of AhR in mice with AP (induced with caerulein or a choline-deficient diet supplemented with
68 hat were depleted of DCs and challenged with caerulein or L-arginine.
69 reatitis was induced in CD11c.DTR mice using caerulein or L-arginine; DCs were depleted by administra
70 st before induction of acute pancreatitis by caerulein or retrograde bile duct infusion of taurolitho
71           miR-21 deficiency protects against caerulein- or L-arginine-induced acute pancreatitis in m
72                NFIC is down-regulated during caerulein pancreatitis and is required for recovery afte
73 matory transcription factor activated during caerulein pancreatitis.
74       Stimulation with either bethanechol or caerulein resulted in a rapid loss of fluorescence inten
75 e in [Ca(2+)](i) in response to supramaximal caerulein stimulation are reduced markedly in acini prep
76                                              Caerulein stimulation caused a time- and concentration-d
77            Acute pancreatitis was induced by caerulein stimulation in wild-type mice and mice deficie
78                                 Supramaximal caerulein stimulation induced subcellular redistribution
79 e pancreatitis by partial duct ligation with caerulein stimulation or intraperitoneal injection of l-
80  events when they are elicited, in vitro, by caerulein stimulation.
81 atitis induced by supramaximal secretagogue (caerulein) stimulation.
82                            Concentrations of caerulein that induced ex vivo trypsinogen activation do
83          In murine pancreata challenged with caerulein to induce acute pancreatitis, we compared CD73
84                      Also predominate in the caerulein treated miR-216a and miR-216b KO mice was the
85 expression were characterized in control and caerulein-treated adult murine pancreas.
86 or adult and embryonic pancreatic markers in caerulein-treated and control pancreas, we addressed cel
87 rypsinogen activation peptide levels between caerulein-treated transgenic and nontransgenic mice.
88                                           In caerulein-treated transgenic mice, the histologic severi
89                                      Indeed, caerulein treatment increased processing of procathepsin
90                                              Caerulein treatment-induced mCAFs increased cytokeratin-
91 However, the cytoskeletal changes induced by caerulein were not affected by wortmannin.