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1 comes susceptible following focal changes in calcified cartilage.
2 rs of hypertrophic chondrocytes in articular calcified cartilage.
3 , confirming a decrease in resorption of the calcified cartilage.
4  at the boundary of the subchondral bone and calcified cartilage.
5 focused on the intervening tissue, articular calcified cartilage (ACC) and its role in the initiation
6 re present in resorption lacunae in areas of calcified cartilage and in subchondral bone immediately
7  and spectra were successfully obtained from calcified cartilage and subchondral bone for the first t
8 ng of the subchondral bone plate, comprising calcified cartilage and underlying subchondral bone.
9  in subchondral bone immediately adjacent to calcified cartilage, as previously described.
10                                     Bone and calcified cartilage can be fossilized and preserved for
11 f tissues (uncalcified cartilage [UCC] only, calcified cartilage [CC] and subchondral bone [bone] [CC
12  to upward invasion by vascular canals or to calcified cartilage erosion may contribute to biomechani
13 ed to the massive accumulation of unresorbed calcified cartilage in a large area below the growth pla
14 LN also inhibited vascular invasion into the calcified cartilage in rats with OA and blocked osteocla
15           There were large discrete areas of calcified cartilage in the hypertrophic zone, few signs
16                                          The calcified cartilage layer was thicker (1.5-fold) in part
17 at the osteochondral interface do not have a calcified cartilage layer, displaying only a thin, highl
18 e is aggressive resorption of the underlying calcified cartilage leading to an extraordinary phenotyp
19 % of these atherosclerotic lesions developed calcified cartilage-like metaplasia.
20 ineralized tissues suggests that bone and/or calcified cartilage provide signals that are critical fo
21                                              Calcified cartilage remodeling due to upward invasion by
22 act loaded joints and to explore the role of calcified-cartilage stiffness on the biomechanics of hea
23 were analyzed by 3-dimensional histology for calcified cartilage, subchondral bone, and subchondral b
24 artilage and decreased hyaline and increased calcified cartilage thickness.
25 tic mice also exhibited an elevated ratio of calcified cartilage to total articular cartilage (CC/TAC
26 gnificantly reduced OARSI scores and CC/TAC (calcified cartilage to total articular cartilage), but i
27 subchondral bone turnover and hypertrophy in calcified cartilage, yet additional mechanical or metabo