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1 ium buffer, but is randomly oriented in high calcium buffer.
2 m that is sensitive to the level of internal calcium buffer.
3 e the concentration of the mobile endogenous calcium buffer.
4 ers representing fixed and mobile endogenous calcium buffers.
5 f EF-hand proteins as calcium sensors versus calcium buffers.
6 i) decline using rate constants for cellular calcium buffers.
7 g action potential duration, with or without calcium buffering.
8 ighly sensitive to the amount of cytoplasmic calcium buffering.
9 ange is countered by increased intracellular calcium buffering.
10  cell that have a high demand for ATP and/or calcium buffering.
11 e depends on mNCE activity and mitochondrial calcium buffering.
12 are affected by dysfunction of mitochondrial calcium buffering.
13 espiratory dysfunction as well as diminished calcium buffering.
14 signals by a mechanism independent of simple calcium buffering.
15 oupled with temporally precise intracellular calcium buffering.
16  prestimulation and blocked by intracellular calcium buffering.
17 hanges in APD75 are altered by intracellular calcium buffering.
18 s revealed that, in the absence of exogenous calcium buffers, a single action potential evokes transi
19 ady-state intracellular calcium and enhanced calcium-buffering ability.
20 efense responses triggered by its C-terminal calcium-buffering activity in response to pathogen invas
21 owing nerve injury: a depletion of cytosolic calcium buffer allows for the rapid accumulation of intr
22 28k is unique in that it functions as both a calcium buffer and a sensor protein.
23 nsport, functioning both as an intracellular calcium buffer and as a shuttle.
24 ransducer current at physiological levels of calcium buffer and external Ca2+ suggest that transducer
25               Control experiments with a non-calcium buffer and with domain mutants confirm that the
26 ondria influence synaptic plasticity through calcium buffering and are crucial for providing the ener
27 ent anion channels in mitochondrial synaptic calcium buffering and in hippocampal synaptic plasticity
28  oxygen species (ROS) generation, as well as calcium buffering and protease activation.
29 es to the cell, including energy production, calcium buffering and regulation of apoptosis.
30 SR calcium pump kinetics as well as intra-SR calcium buffering and SR calcium leak.
31 y an intracellular solution lacking any fast calcium buffer, and was restored by the addition of 1.2
32 ynaptic introduction of "fast" high-affinity calcium buffers, and the decay of facilitation was accel
33 r enzymatic digestion, to fast intracellular calcium buffers, and to intracellular pressure.
34 forated-patch recordings gave the endogenous calcium buffer as equivalent to 0.21 mM BAPTA in low-fre
35                                          The calcium buffer BAPTA attenuated the voltage effects of A
36 ization was abolished by the addition of the calcium buffers BAPTA and EGTA and could be induced by m
37 uscle cells by lowering the abundance of the calcium buffering/binding protein calsequestrin1 which i
38 ated the effect of HERG on calsequestrin1, a calcium buffering/binding protein known to modulate RYR1
39 of calbindin-D(28k) may result not only from calcium buffering but also from the ability of the prote
40 osition of active microtubule sliding in low calcium buffer, but is randomly oriented in high calcium
41             Increasing the levels of nuclear calcium buffers by means of expression of a nuclearly ta
42 o be efficiently controlled by a native fast calcium buffer, calbindin-D28k, maintaining a lower vesi
43 drial transport in axon are increased axonal calcium buffer capability, diminished reactive oxygen sp
44 T mitochondria exhibit reduced mitochondrial calcium buffering capacity and are highly sensitive to m
45         Additionally, they have an increased calcium buffering capacity and generate fewer mitochondr
46 sults establish a close link between nuclear calcium buffering capacity and the transcription of gene
47 aling using CaMBP4 or increasing the nuclear calcium buffering capacity by means of expression of a n
48 st-mortem human brain that suggested loss of calcium buffering capacity in neurons correlated with ar
49                        Here we show that the calcium buffering capacity of the cell nucleus in mouse
50 mbrane potential that related to an improved calcium buffering capacity.
51 radicals and that it increases mitochondrial calcium-buffering capacity.
52                                     Intra-SR calcium buffering characteristics were also deduced.
53 he range of 2-5 sec) was observed under weak calcium buffering conditions.
54 nt was 77 +/- 3 (n = 14), and the endogenous calcium buffering constant component is likely to be bet
55                        The average cytosolic calcium buffering constant was 77 +/- 3 (n = 14), and th
56 ain whether parvalbumin (Parv), a myoplasmic calcium buffer, could correct the diastolic dysfunction
57 her reports of genetic disruption of EF-hand calcium buffers, deletion of oncomodulin (Ocm), which is
58        Interfering with facilitation via the calcium buffer EGTA or interfering with the presynaptic
59 s, and were uncoupled from each other by the calcium buffer EGTA.
60 human eyes from 17 patients were fixed in 5% calcium-buffered formalin.
61 e, we estimated that the dominant endogenous calcium buffer in dendrites is relatively immobile (diff
62                                    The major calcium buffer in these cells, calbindin D28K, is presen
63  have investigated the role of mitochondrial calcium buffering in excitotoxic cell death.
64 two orders of magnitude in the efficiency of calcium buffering in the cytosol and the ER lumen.
65 sm of the InsP3-receptor, InsP3 degradation, calcium buffering in the cytosol, and refilling of the E
66  These results demonstrate conclusively that calcium buffering in the mitochondrial matrix in live ce
67 onal role to immobile or fixed intracellular calcium buffers in central neurons because the amount of
68                      Finally, the endogenous calcium buffers in spines remain unknown.
69 aptation to compare the effects of exogenous calcium buffers in the patch electrode solution with tho
70 mate transmission, mitochondrial fusion, and calcium buffering, is complex and was differentially reg
71 that persistent alterations in intracellular calcium buffering may be associated with opiate toleranc
72                    In the inner ear, EF-hand calcium buffers may play a significant role in hair cell
73 e the effects that two commonly used "caged" calcium buffers (NP-EGTA and nitr-5) have on the amplitu
74          In this process, the effect of this calcium buffer on the intracellular calcium signaling an
75                 The effects of intracellular calcium buffering on electrical tuning were studied in h
76                                The impact of calcium buffering on the initiation and propagation of m
77                 In the presence of a nominal calcium buffer or EGTA, amphotericin B-induced IL-1beta
78 d mice can be partially rescued by improving calcium buffering, or decreasing action potential-evoked
79                                Mitochondrial calcium buffering plays a major role after this type of
80       Back-extrapolation of the increases in calcium buffering power allowed us to calculate the calc
81 three experiments we modelled the additional calcium buffering power produced by multiple pressure in
82                                          The calcium buffering power, binding capacity and non-linear
83       Similarly, inhibition of the RyR and a calcium buffer prevented induction of priming by PKCepsi
84            Overexpression of calreticulin, a calcium buffer protein, blocked p12(I)-mediated NFAT act
85 ed the location, neurotransmitter phenotype, calcium-buffering protein expression, and axon distribut
86 ventral interneurons identified according to calcium-buffering protein expression, two groups (1 and
87 oviral gene transfer of parvalbumin, a small calcium-buffering protein found exclusively in skeletal
88                      We expressed vertebrate calcium-buffer proteins in groups of cells in the networ
89 o, as evidenced by immunostaining intensity, calcium-buffering proteins were significantly elevated i
90 tion of the stimulus and sensitivity to slow calcium buffers reported for asynchronous release.
91 ria is crucial for precise ATP provision and calcium buffering required to support neuronal signaling
92 tribution to localized energy production and calcium buffering requirements.
93 r unique architecture and special energy and calcium-buffering requirements at the synapse.
94                                    Exogenous calcium buffers slow endocytosis but have no additional
95 oncentration with important implications for calcium buffering, synaptic plasticity, and protein-memb
96 , driven most significantly by the cytosolic calcium buffering system and changes in diastolic Ca(2+)
97 such as parvalbumin are part of the cellular calcium buffering system that determines intracellular c
98 , enabled by (2) impairment in intracellular calcium buffering systems.
99 r muscarinic cholinergic receptors or by low calcium buffer, tetrodotoxin or vesamicol.
100  has heretofore been considered a diffusible calcium buffer that is dispersed uniformly throughout th
101 cilitation mechanism involving an endogenous calcium buffer that is more efficiently saturated with l
102 e find that there is a time lag equal to the calcium buffering time constant between the instantaneou
103 ndicate, and (iii) is confined by endogenous calcium buffers to local dendritic regions even when syn
104 tration, can serve as a diffusionally mobile calcium buffer/transporter capable of regulating calcium
105 oltages and under conditions of low and high calcium buffering: tripling contrast reduced gain by app
106 nofluorescence in phagolysosome membranes in calcium-buffered vs. control macrophages.
107                     Increasing intraterminal calcium buffering with EGTA-AM or decreasing calcium inf
108           Calsequestrin (CSQ) is the primary calcium buffer within the sarcoplasmic reticulum (SR) of

 
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