ライフサイエンスコーパス: calcium concentration

1 anoconfined solution can be tuned by varying calcium concentration.

2 differentiation by an elevated extracellular calcium concentration.

3 bited persistent elevations in intracellular calcium concentration.

4 ponsible for oscillations in the cytoplasmic calcium concentration.

5 by the substantial elevation of cytoplasmic calcium concentration.

6 displaying an acute effect on free cytosolic calcium concentration.

7 tive contribution dependent on extracellular calcium concentration.

8 pond to the signal of an increased cytosolic calcium concentration.

9 e of generating oscillatory power at a fixed calcium concentration.

10 wn (KD) cells were more sensitive to reduced calcium concentration.

11 ling and prevention of a deleterious rise in calcium concentration.

12 ts function and structure depend strongly on calcium concentration.

13 s to RyR1 at the apo site, regardless of the calcium concentration.

14 65% increase in the mean free intracellular calcium concentration.

15 y of NCAD12 dimers was strongly dependent on calcium concentration.

16 rently affected by increases in the external calcium concentration.

17 ting is regulated by changes in intraciliary calcium concentration.

18 iculin is remarkably thermostable at a given calcium concentration.

19 egulated in part by an optimal intracellular calcium concentration.

20 calcium channels rather than changes in bulk calcium concentration.

21 mps and a low-bone-marrow interstitial fluid calcium concentration.

22 n more sensitive to changes in environmental calcium concentration.

23 II phosphorylation following a surge of high calcium concentration.

24 ntent, and increased the basal intracellular calcium concentration.

25 ion of membrane potential into intracellular calcium concentration.

26 ould be clearly distinguished by varying the calcium concentration.

27 cium-sensing receptor (CaSR) regulates serum calcium concentrations.

28 ogenesis in response to increasing cytosolic calcium concentrations.

29 ns under both physiological and pathological calcium concentrations.

30 perties and its role in maintaining systemic calcium concentrations.

31 ents, or NMDA-induced increases in cytosolic calcium concentrations.

32 itochondria contact points and mitochondrial calcium concentrations.

33 was collected for a wide range of specified calcium concentrations.

34 um permeability and elevated basal cytosolic calcium concentrations.

35 dividual domains differ significantly at low calcium concentrations.

36 can also target Ser-282 at nonphysiological calcium concentrations.

37 ual actin filaments by VLN3 at physiological calcium concentrations.

38 accounted for 0.54% of the variance in serum calcium concentrations.

39 um and phosphorus concentrations or in urine calcium concentrations.

40 mal Ca(2+) buffering characteristics at high calcium concentrations.

41 ffinity for eNOS under resting physiological calcium concentrations.

42 measured over a range of extra mitochondrial calcium concentrations.

43 el activity because of the low extracellular calcium concentrations (0.2-0.5 mM) used typically to as

44 .22, 0.96-1.55; p=0.1010) or raised adjusted calcium concentration (1.08, 0.88-1.34; p=0.4602).

45 2.05-2.60; p<0.0001), and raised total serum calcium concentration (1.43, 1.21-1.69; p<0.0001), but n

46 mulation frequencies (0.1-4 Hz) and external calcium concentrations (1.8-3.6 mm) at 37 degrees C.

47 At high nonphysiological calcium concentration, A8V, E134D, and D145E mutations m

48 mal expansion media combines atypically high calcium concentrations, activation of cAMP, FGF, and R-s

49 VO) and non-DLVO forces as a function of the calcium concentration, also after charge reversal of bot

50 Pf) in response to increases in the external calcium concentration, an effect that is mediated by an

51 P24A1-associated locus correlates with serum calcium concentration and a number of nephrolithiasis ep

52 te (WPI) nanoparticles prepared by different calcium concentration and aggregation pH.

53 ly entangled linear polymers is sensitive to calcium concentration and changes in pH.

54 tely lead to increased intracellular ionized calcium concentration and contraction of pulmonary arter

55 hannel activity depends on the intracellular calcium concentration and is associated with D-serine re

56 uence channel sensitivity to fluctuations in calcium concentration and perhaps even metabolic state.

57 MUC5B conformation is affected by changes in calcium concentration and pH, factors important for muci

58 ations of glucose increase the intracellular calcium concentration and the frequency of alpha-cell ca

59 contact elimination depends on extracellular calcium concentration and the level of E-cadherin, sugge

60 lume of the synaptic terminal influences the calcium concentration and the number of available vesicl

61 ciliary calcium channel controlling ciliary calcium concentration and thereby modifying SMO-activate

62 fic synaptic changes through the dynamics of calcium concentration and thresholds implementing in sim

63 aPrm1 mutants was sensitive to extracellular calcium concentration and was associated with an increas

64 Gs that may serve to raise local cytoplasmic calcium concentrations and aid in refilling intracellula

65 a, Bmi-1, K15, and ABCG2), whereas increased calcium concentrations and air-lifting induced terminal

66 athyroid surgery include monitoring of serum calcium concentrations and bone density.

67 to occur fast robustly over a large range of calcium concentrations and hence energetic stabilities.

68 cal stimulation with increased intracellular calcium concentrations and increased inward cation curre

69 is maintained in the inactive state at basal calcium concentrations and is activated via CaM binding

70 strate that VWF binds calcium at physiologic calcium concentrations and that calcium stabilizes VWF A

71 topHluorin signals correlate with high local calcium concentrations and that local, spontaneous calci

72 M-free state, were able to bind CaM at lower calcium concentration, and had lower rates of heme reduc

73 ng plasticity at physiological extracellular calcium concentration, and highlight the role of synapti

74 sigmoidal dependence of key parameters with calcium concentration, and is simpler and more suitable

75 in which IQCG stores CaM at low cytoplasmic calcium concentrations, and releases CaM to activate CaM

76 d found that, at physiological endolymphatic calcium concentrations, approximately half of the mechan

77 As such, cellular and systemic calcium concentrations are tightly regulated.

78 nt signals, such as hypoxia or extracellular calcium concentration, are difficult to reproduce.

79 brane potential and an increase in cytosolic calcium concentrations, are inhibited by low luminal pH

80 ees C evoked a 40% increase in intracellular calcium concentration as determined by live-cell confoca

81 ane to 180 mM makes it possible to determine calcium concentrations as high as 3 mM by chronopotentio

82 of TCRzeta, ZAP70, and LAT and intracellular calcium concentration, as well as IL-2 gene expression.

83 Only at mM calcium concentrations, as found in the extracellular en

84 safety measures of renal function and serum calcium concentration assessed every 3 months.

85 uction is often an increase in intracellular calcium concentration associated with intracellular calc

86 obust molecular switch that is responsive to calcium concentrations associated with both the basal st

87 by CO2-rich brine with discrete increases in calcium concentration at reaction boundaries.

88 The increases in calcium concentration at the AIS evoked by subthreshold

89 determine whether maternal 25(OH)D, PTH and calcium concentrations at 26 weeks gestation are associa

90 brane, a transient increase of intracellular calcium concentration, binding of calcium to troponin in

91 d to those of nicotine on intracellular free calcium concentration but were causally associated with

92 rthermore, the extension rate increases with calcium concentration, but at a given concentration, we

93 ease increased with an increase in the added calcium concentration, but the increase was dependent on

94 inhibits contractility at high intracellular calcium concentration by disrupting the actin-myosin ATP

95 rgoes dynamic polymerization with increasing calcium concentration by front-to-front dimerization and

96 xpressed by parathyroid cells controls blood calcium concentration by regulating parathyroid hormone

97 We find that seawater calcium concentration, by strongly influencing carbonate

98 at membrane voltage (V(m)) and intracellular calcium concentrations (Ca) become dissociated during ve

99 Cytosolic calcium concentration ([Ca(2+) ](cyt) ) and heterotrimer

100 The transient elevation of cytosolic free calcium concentration ([Ca(2+) ](cyt) ) induced by cold

101 e heart to function as a pump, intracellular calcium concentration ([Ca(2+) ]i ) must increase during

102 at cardiomyocytes and the free mitochondrial calcium concentration ([Ca(2+) ]m ) was measured at diff

103 Resting motile cilia calcium concentration ([Ca(2+)] ~170 nM) is only slightl

104 sis that Bid regulates endoplasmic reticulum calcium concentration ([Ca(2+)](ER)) homeostasis to affe

105 ial smooth muscle cell (PASMC) intracellular calcium concentration ([Ca(2+)](i)) and pH.

106 The intracellular calcium concentration ([Ca(2+)](i)) has been monitored u

107 e role of a Ca(2+) channel and intracellular calcium concentration ([Ca(2+)](i)) in osmotic stress-in

108 found that activity-dependent intracellular calcium concentration ([Ca(2+)](i)) in the axonal initia

109 iol signaling increases the free cytoplasmic calcium concentration ([Ca(2+)](i)) that stimulates the

110 Intracellular calcium concentration ([Ca(2+)](i)) was examined in rods

111 Intracellular calcium concentration ([Ca(2+)](i)) was measured by micr

112 Cytoplasmic calcium concentration ([Ca(2+)](i)) was measured in cult

113 ent early increase (30 min) in intracellular calcium concentration ([Ca(2+)](i)), following Abeta(1-4

114 The consequent lowering of the cytosolic calcium concentration ([Ca(2+)](i)), if protracted, can

115 arly and transient increase of intracellular calcium concentration ([Ca(2+)](i)), required for AhR-re

116 ed with stereotypic changes in intracellular calcium concentration ([Ca(2+)](i)), yet the target of t

117 Extracellular calcium concentration ([Ca(2+)](o)) regulates Ca(2+) ent

118 FSS rapidly increases the intracellular calcium concentration ([Ca(2+)]) and nitric oxide (NO) s

119 the power required for flight by varying the calcium concentration ([Ca(2+)]).

120 Transient elevations in intracellular calcium concentration ([Ca(2+)]i) and migratory pauses o

121 lpha7 nAChR agonist, increases intracellular calcium concentration ([Ca(2+)]i) mainly released from i

122 s of transmembrane voltage and intracellular calcium concentration ([Ca(2+)]i) that gate the channels

123 e arteries, coupling a rise of intracellular calcium concentration ([Ca(2+)]i) to endothelial cell hy

124 ed by a sustained elevation of intracellular calcium concentration ([Ca(2+)]i) which could not be blo

125 stimuli trigger increases in cytosolic free calcium concentration ([Ca(2+)]i).

126 ted by increases in astrocytic intracellular calcium concentrations ([Ca(2)(+)](i)).

127 ted by membrane voltage (Vm ), intracellular calcium concentrations ([Ca(2+) ]i ) and external permea

128 aneous dynamic fluctuations in intracellular calcium concentrations ([Ca(2+)](i)) in smooth muscle ce

129 estradiol rapidly increased free cytoplasmic calcium concentrations ([Ca(2+)](i)) that facilitate pro

130 led simultaneous monitoring of intracellular calcium concentrations ([Ca(2+)]i) in multiple cells and

131 Ionized calcium concentration [Ca(2+)] was significantly lower i

132 membrane potential and decreased cytoplasmic calcium concentration [Ca(2+)](c) and glucose-stimulated

133 205+/-34 seconds, by increased intracellular calcium concentration, [Ca(2+)](i).

134 ar to be in response to changes in cytosolic calcium concentration, [Ca(2+)](i).

135 probability, Ca2+ sparks, and the myoplasmic calcium concentration ([Ca2+]i) during excitation-contra

136 Resting cytoplasmic calcium concentration ([Ca2+]i) in cultured preCGG hippo

137 Non-negativity constraints on the calcium concentration can also be incorporated using a l

138 fects of the imaging procedure, we show that calcium concentration can be estimated up to an affine t

139 retion of misfolded proteins or depletion in calcium concentration causes stress in the ER, which lea

140 fusion induced an increase in intracellular calcium concentration, causing premature oocyte activati

141 ary to expectation, does not affect the peak calcium concentration close to the source but sharpens t

142 d ROS levels and reduced basal intracellular calcium concentration compared with mock cells.

143 r PTH (median 7.7 vs 3.3 pmol/L) and similar calcium concentrations compared to White British women.

144 n of variation in CASR that influences serum calcium concentration confirms the results of earlier ca

145 ion was dependent on a rise in intracellular calcium concentration derived from extracellular sources

146 alcium store, the evolution of intracellular calcium concentration during a train of long-lasting dep

147 The kinetics of solubility and ionic calcium concentration during in vitro digestion were stu

148 e role of GluN2 subunit differences on spine calcium concentration during several STDP protocols in a

149 with nitrite had a lower force and cytosolic calcium concentration during single non-fatiguing contra

150 odel the effects become apparent at elevated calcium concentrations, e.g., at [Ca(2+)] = 25 muM, taua

151 at: calcium signals in the form of cytosolic calcium concentration elevations are nonlinearly amplifi

152 mental in generating sustained intracellular calcium concentration elevations that are necessary for

153 ent cation environment, with the ER range of calcium concentrations enhancing stability, and calcium-

154 At the same calcium concentration, excimer emission increased also,

155 activity-related increases in intracellular calcium concentration (FLiCRE).

156 l agents as measured by changes in cytosolic calcium concentration for the rapid classification of ne

157 h mu-calpain (calpain1) requiring micromolar calcium concentrations for activation and m-calpain (cal

158 to measure a marked heterogeneity in average calcium concentrations from cell to cell in the basal ke

159 d active vitamin D while maintaining a serum calcium concentration greater than or the same as baseli

160 utoprocessing and activity were dependent on calcium concentrations >1 mm, consistent with the protei

161 f those protocols, increases in postsynaptic calcium concentration have been shown to play a crucial

162 egions could be modulated by controlling the calcium concentration: (i) at a low calcium concentratio

163 ere the dissolution of calcite increases the calcium concentration in a thin boundary layer in contac

164 ich leads to a greater rise in intracellular calcium concentration in aging than that in young neuron

165 CaSR inhibition increased blood calcium concentration in animals pretreated with a bisph

166 Additionally, the patient showed an elevated calcium concentration in blood and urine as well as neph

167 rization-activated current and intracellular calcium concentration in both normal control (NC) rats a

168 an increase in baseline and spike-triggered calcium concentration in both the AIS and nearby synapti

169 inetics of the changes in free mitochondrial calcium concentration in cardiac myocytes are largely un

170 inetics of the changes in free mitochondrial calcium concentration in cardiomyocytes.

171 uces a dose-dependent elevation in cytosolic calcium concentration in ET(B)-transfected cells and end

172 lours, and it is related to the increases in calcium concentration in germ and the formation of amylo

173 hermore, by measuring changes of cytoplasmic calcium concentration in hASCs during EFS, our findings

174 how that a chronic increase of the cytosolic calcium concentration in hepatocytes during obesity and

175 ffinity of the exhaustive nanosensors, total calcium concentration in human blood plasma was successf

176 The calcium concentration in mineral water was successfully

177 ve been observed as spikes of the whole-cell calcium concentration in numerous cell types and are ess

178 osinophils showed an increased intracellular calcium concentration in response to Alternaria that was

179 the observations that STIM-1, the sensor of calcium concentration in stores, and Orai-1, the calcium

180 primary hippocampal neurons does not affect calcium concentration in the endoplasmic reticulum.

181 scale can be extended: (i) the extracellular calcium concentration in the experiments used to fit the

182 Importantly, with an increase of the calcium concentration in the growth medium, these phmSG

183 This, in turn, caused secondary reduction of calcium concentration in the intracellular compartments

184 ry to the stratum corneum alterations in the calcium concentration in the outer epidermis are the pri

185 stress but are independent of intracellular calcium concentration in the physiological range.

186 on potentials generated at the soma increase calcium concentration in the somatic cytosol and nucleus

187 ocked response to sinusoidal stimuli, at the calcium concentration in the surrounding fluid near the

188 e Nox5 activity was also observed with fixed calcium concentrations in an isolated enzyme activity as

189 We show silicon and calcium concentrations in bedrock are strongly correlate

190 bnormal synaptic ribbons, and higher resting calcium concentrations in hair cells.

191 These features enable NMR measurements of calcium concentrations in human serum in the presence of

192 Calcium concentrations in the dispersed phase increased

193 ical mapping of transmembrane potentials and calcium concentrations in the zebrafish heart.

194 posing murine or human T cells to equivalent calcium concentrations in vitro enhanced its influx, tri

195 Calcium concentrations increase considerably in gypsum-a

196 y for a calcium sensor, we found that higher calcium concentrations increased the lifetimes of the mi

197 s of embryonic cell division discovered that calcium concentration increases transiently at the divis

198 d renal tubular calcium absorption and blood calcium concentration independent of PTH secretion chang

199 e that CaSR is a direct determinant of blood calcium concentration, independent of PTH, and modulates

200 The subsequent increase in intracellular calcium concentration induces proteolytic processing and

201 lting transient increase in cytoplasmic free calcium concentration is a critical trigger for the init

202 Robust elevation of the cytosolic calcium concentration is a crucial early step for T cell

203 We show that peak calcium concentration is highly correlated with soma-syn

204 study was to test the hypothesis that serum calcium concentration is positively and independently as

205 The evolution of calcium concentration is represented through a smaller s

206 Uncorrected serum calcium concentration is the first mineral metabolism me

207 oidism has been described in which the serum calcium concentration is within normal range but parathy

208 ed by a transient rise in intracellular free calcium concentration linked to a change in the structur

209 At pCa levels above approximately 6.0 (i.e., calcium concentrations <1 microM), CK-2066260 increased

210 Here, we demonstrate that low calcium concentrations (<1.5 mg/L) that are found in man

211 This cellular process of calcium concentration may represent a widespread pathway

212 chlear hair cell stereocilium where local mm calcium concentrations may exist.

213 isms: calcitropes, which alter intracellular calcium concentrations; myotropes, which affect the mole

214 nsient, localized increases in intracellular calcium concentration near the calcium-conducting pores

215 The discovery that transient elevations of calcium concentration occur in astrocytes, and release '

216 e cytoplasm, we show that changes in ciliary calcium concentration occur without substantially alteri

217 inase activity depends on high intracellular calcium concentrations occurring in dying cells.

218 At an external calcium concentration of 1 mM, and a membrane potential

219 and a temporal matrix that characterizes the calcium concentration of each neuron over time.

220 Furthermore, the intracellular calcium concentration of isolated neuroepithelial cells

221 Caco-2 cells treated with the chelate at calcium concentrations of 0-10 mM exhibited enhanced abs

222 Necrosis was induced using calcium concentrations of 100-500 mmol/L and injection v

223 s in stimulation frequency and extracellular calcium concentration on the simulated Ca(2+) transient

224 estigate the effects of fluid flow rates and calcium concentrations on the mass and distribution of M

225 re highly cooperative with respect to either calcium concentration or extent of cRLC phosphorylation.

226 rial complex I, an increase in intracellular calcium concentration, or formation of reactive oxygen s

227 d to E(2)(#) is lower at lower pH, at higher calcium concentrations, or with an inhibitor bound to th

228 At low luminal calcium concentrations, ouf8 had little detectable effec

229 s carriers showed significantly higher serum calcium concentration (P=0.01) and a trend for higher pl

230 ulin remained stably attached independent of calcium concentration (pCa 3-7).

231 nsensitive to both salt (25-1000 mm KCl) and calcium concentrations (pCa 3-7).

232 at individual sites is low at physiological calcium concentration, PF-PC synapses release one or mor

233 imental studies suggest that intrapancreatic calcium concentrations play an important role in the ini

234 asynchronous release increases with external calcium concentration, possibly suggesting that the mode

235 nd that the dominant folding pathway at high calcium concentrations proceeds via a transition state c

236 Increasing calcium concentrations progressively shift this equilibr

237 ve model, we show how membrane potential and calcium concentration provide a fast feedback that can e

238 Upon intense illumination, rhabdomeric calcium concentration reaches millimolar levels that wou

239 pic and metabotropic) that alter cytoplasmic calcium concentration (receptor-agonist challenges) and

240 allow us to propose a mechanistic model for calcium concentration regulated outer shell assembly.

241 cal positive-gating modulator and shifts the calcium-concentration response curve of KCa3.1 to the le

242 hich both lead to an elevated intraendosomal calcium concentration, resulted in the accumulation of i

243 The increase in baseline calcium concentration rose with depolarization and fell

244 el can fit all three data sets with the same calcium-concentration-sensitive parameters.

245 of a high prevalence of hyperparathyroidism, calcium concentrations should be checked before and duri

246 diffraction but only for its dormant or high-calcium-concentration state, not its low-calcium-concent

247 igh-calcium-concentration state, not its low-calcium-concentration state, which is relevant to viral

248 dc73(L/L)/PTH-Cre mice had higher mean serum calcium concentrations than wild-type littermates, and C

249 )O(2) caused a 25% increase in mitochondrial calcium concentration that was associated with a 50% dec

250 lls caused rapid elevations in intracellular calcium concentration that were independent of phospholi

251 ling the calcium concentration: (i) at a low calcium concentration the droplets were evenly distribut

252 m influx is changed by altering the external calcium concentration, the calcium cooperativity of p is

253 Finally, above a threshold cadherin and calcium concentration, the cis and trans protein interac

254 we show that with increased bulk cytoplasmic calcium concentration, the CRU model exhibits determinis

255 In contrast, at lower physiological calcium concentration, the D145E mutation led to an appr

256 ation and illustration refer to a monitor of calcium concentration, the method is applicable to any s

257 evenly distributed; (ii) at an intermediate calcium concentration they formed a layer around the sta

258 around the starch granules; (iii) at a high calcium concentration they formed a network of aggregate

259 are impermeant and unable to bind PS at low calcium concentration, they are unsuitable for intracell

260 Glucagon increased cytosolic calcium concentration through the PKA-mediated phosphory

261 ular switch that ties shifting intracellular calcium concentration to association and dissociation of

262 Lowering extracellular calcium concentration to in vivo levels leads to an incr

263 s, and ultimately elevates the intracellular calcium concentration to increase the release of glutama

264 r data suggest that changes in intracellular calcium concentrations triggered by nAChR activation can

265 in the transgenic mouse retinas at the free calcium concentrations typical for dark-adapted rods.

266 Finally, we analyze the AP and calcium concentration under healthy and LBBB conditions.

267 Notably, the rise of intracellular calcium concentration upon immunoglobulin E (IgE)-mediat

268 The abnormally high calcium concentration used in in vitro studies of STDP s

269 rements of cell length changes and cytosolic calcium concentration using confocal line scanning at a

270 n White British women, each 1 SD increase in calcium concentration was associated with a 34% increase

271 he H(2)O(2)-stimulated rise in mitochondrial calcium concentration was attenuated by 40%.

272 the lysoPC-induced increase in intracellular calcium concentration was inhibited in ECs transiently t

273 Free mitochondrial calcium concentration was measured in adult rat ventricu

274 Calcium concentration was the only variable that influen

275 GWAS of serum calcium concentrations was performed in 20 611 individua

276 lyzing single-channel activities at limiting calcium concentrations, we find that temperature alters

277 Changes of intracellular calcium concentration were involved not only in high glu

278 phorylation of Cx36 or in intracellular free calcium concentration were not involved in the observed

279 atively slow decreases in free mitochondrial calcium concentration were observed in rat cardiac myocy

280 caffeine, similar increases in intracellular calcium concentration were observed in Stac3-deleted and

281 Serum and urinary calcium concentrations were also measured.

282 Cytosolic calcium concentrations were assessed under the same expe

283 re media in which essential trace metals and calcium concentrations were manipulated.

284 Adverse events and plasma calcium concentrations were similar between groups.

285 g on CB(1) receptors increases intracellular calcium concentration when administered intracellularly

286 ARI photoswitching kinetics are modulated by calcium concentration when illuminating with blue light,

287 (Q(10) >100) to heating particularly at low-calcium concentrations whereas channels lacking the calc

288 dimer disassembles rapidly regardless of the calcium concentration, whereas the disassembly of NCAD12

289 ensing receptors to changes in extracellular calcium concentrations, whereas autosomal dominant hypoc

290 ny synapses by increasing the intra-terminal calcium concentration, which may increase the quantal co

291 control is exerted by affecting the internal calcium concentration, which sets the resting open proba

292 The extracellular calcium concentration, which triggers activation of the

293 hyperexcited states cause high intracellular calcium concentrations, which could trigger transcriptio

294 II model system is never bistable at resting calcium concentrations, which suggests that CaMKII activ

295 ssessed the association of uncorrected serum calcium concentration with clinical outcomes.

296 Significantly, the binding mode depends on calcium concentration with important implications for ca

297 asymmetry caused a rise in the mitochondrial calcium concentration with stimulation frequency.

298 and cultured in low to medium (0.03-0.4 mM) calcium concentrations with proper serum levels (10% FCS

299 Local control of calcium concentration within neurons is critical for sig

300 However, moderate variations in calcium concentrations within the physiological range ca