ライフサイエンスコーパス: calcium ionophore

1 induced by sperm (but twice that achieved by calcium ionophore).

2 oted after treatment with cAMP and A23187 (a calcium ionophore).

3 when both cell populations were activated by calcium ionophore.

4 ssion in mast cells stimulated with PMA plus calcium ionophore.

5 15-EET when the cells were stimulated with a calcium ionophore.

6 enerated by activating washed platelets with calcium ionophore.

7 occurred when sporozoites were treated with calcium ionophore.

8 ls more slowly in response to treatment with calcium ionophore.

9 e nuclear envelope upon stimulation with the calcium ionophore.

10 lon mAb, or stimulated with combined PMA and calcium ionophore.

11 the impaired relaxation to acetylcholine or calcium ionophore.

12 ts and breast cancer cells when treated with calcium ionophore.

13 nduced upon treatment with phorbol ester and calcium ionophore.

14 of polychlorinated biphenyls, and A23187, a calcium ionophore.

15 pid calcium influx induced by 1 and 5 microM calcium ionophore.

16 ither serum deprivation or by treatment with calcium ionophore.

17 th was induced by glucocorticoid (6-48 h) or calcium ionophore.

18 elaxant responses to acetylcholine (ACh) and calcium ionophore.

19 tored after treatment with phorbol ester and calcium ionophore.

20 and phosphorylation decrease in response to calcium ionophore.

21 tosis stimulated by cytokine deprivation and calcium ionophore.

22 n buffer using phorbol myristate actetate or calcium ionophore.

23 n cells in response to interleukin-1beta and calcium ionophore.

24 d by treatment with either phorbol esters or calcium ionophores.

25 als delivered through the FcepsilonRI or via calcium ionophores.

26 gher concentrations, presumably by acting as calcium ionophores.

27 rapidly exit their host cell when exposed to calcium ionophores.

28 necessary for inhibition of NHE3 activity by calcium ionophore 4-Br-A23187.

29 l; 17.8 microm) and then stimulated with the calcium ionophore, (5 microm).

30 flammatory agonist N-formyl peptides (FMLP), calcium ionophore A(23187), or phorbol mirystate acetate

31 This synthesis was enhanced by the calcium ionophore A-23187, by IL-1beta, or by supplying

32 hyl ether)-N-N' -tetraacetic acid (EGTA) and calcium ionophore (A-23187) on phosphorus uptake was stu

33 Additionally, the calcium ionophore, a reagent that promotes nitric oxide

34 upon stimulation with lipopolysaccharide and calcium ionophore A21387, as compared with that in untre

35 topical application of bradykinin (-59%) or calcium ionophore A23187 (-49%) and by systemic hypercap

36 ent with staurosporine (20-100 ng/mL) or the calcium ionophore A23187 (0.5 microM).

37 m the lens is tightly regulated, neither the calcium ionophore A23187 (1 microM) nor depolarization b

38 orophenylhydrazone (CCCP, 10 microM) and the calcium ionophore A23187 (10 microM), abolished TPP+ acc

39 al conditions and after stimulation with the calcium ionophore A23187 (10(-6) M).

40 he receptor-independent endothelial agonist, calcium ionophore A23187 (10(-7) mol/L and 3 x 10(-7) mo

41 te flow (from 0 to 25 microL/min) and to the calcium ionophore A23187 (5 x 10(-8) to 10(-6) mol/L), n

42 Ch, 100+/-2% versus 75+/-2%, P<0.05) and the calcium ionophore A23187 (92+/-2% versus 72+/-3%, P<0.05

43 ther recombinant TNF alpha supernatants from calcium ionophore A23187 (CaI)-stimulated mast cells or

44 rmal livers in response to acetylcholine and calcium ionophore A23187 (P < .0001).

45 The calcium ionophore A23187 also induced NOD-dependent sign

46 h phorbol 12,13-dibutyrate together with the calcium ionophore A23187 also promoted ubiquitination an

47 hat increase intracellular calcium (i.e. the calcium ionophore A23187 and thapsigargin) or protein ki

48 Calcium ionophore A23187 and the muscarinic cholinergic

49 ted degranulation of storage granules by the calcium ionophore A23187 caused release of ET into the c

50 Treatment of infected host cells with the calcium ionophore A23187 causes the parasites to undergo

51 The calcium ionophore A23187 destabilized YY1 in cultured my

52 tracellular cyclic AMP or treatment with the calcium ionophore A23187 do not cause SPR phosphorylatio

53 Activation of cPLA(2)alpha by the calcium ionophore A23187 enhanced PPARdelta binding to P

54 Although the calcium ionophore A23187 increased the expression of CD1

55 Interestingly, the calcium ionophore A23187 induced accumulation of a polyu

56 ists such as collagen, ADP, epinephrine, and calcium ionophore A23187 induced RAFTK phosphorylation.

57 inhibited ACTH-induced SAPK activity and the calcium ionophore A23187 induced SAPK activity 3-fold.

58 The calcium ionophore A23187 induced SFK phosphorylation in

59 of intact aortic endothelial cells with the calcium ionophore A23187 leads to the rapid disruption o

60 of HUVECs or isolated mice aortas to either calcium ionophore A23187 or bradykinin significantly inc

61 en Jurkat cells were treated with either the calcium ionophore A23187 or thapsigargin, z-VAD failed t

62 e also show that treatment of LNCaP with the calcium ionophore A23187 or the phorbol ester phorbol 12

63 Addition of calcium ionophore A23187 produced a rapid release of 20-

64 Treatment with the calcium ionophore A23187 recapitulated the force-induced

65 ytes with LPS followed by treatment with the calcium ionophore A23187 resulted in the formation of PG

66 nase C by TPA or increases in [Ca2+]i by the calcium ionophore A23187 stimulated p130(Cas) phosphoryl

67 hed using the chemotherapy drug VP-16 or the calcium ionophore A23187 to induce apoptosis in Burkitt'

68 Adding calcium ionophore A23187 to Mphi inoculated with Mtb fur

69 oethyl ether)-N,N'-tetraacetic acid (EGTA) + calcium ionophore A23187 treatments resulted in reduced

70 to acetylcholine and to a lesser extent the calcium ionophore A23187 were highly variable and correl

71 Basal and stimulated (by the calcium ionophore A23187) secretion of NO and intracellu

72 in, 0.1 U/ml; collagen, 4 microg/ml; and the calcium ionophore A23187, 1 microM) results in shedding

73 Stimulation of these vessels with calcium ionophore A23187, a known secretagogue of Weibel

74 ivated with bacterial lipopolysaccharide and calcium ionophore A23187, and biosynthesis was blocked b

75 l exocytosis by progesterone, but not by the calcium ionophore A23187, and elicited a concomitant red

76 Relaxations to acetylcholine, the calcium ionophore A23187, and nitroglycerin, as well as

77 responsiveness of several cell lines to the calcium ionophore A23187, bacterial lipopolysaccharide (

78 demonstrate that induction of ER stress with calcium ionophore A23187, brefeldin A, or tunicamycin is

79 o induced by thapsigargin, okadaic acid, and calcium ionophore A23187, but not by phenobarbital or th

80 sphorylation of Pyk2 was also induced by the calcium ionophore A23187, by phorbol myristate acetate,

81 re increased or decreased in C cells, by the calcium ionophore A23187, by physiologic concentrations

82 stimulation with histamine, thrombin or the calcium ionophore A23187, cPLA2-alpha relocated to intra

83 ylation of T-753 in beta3 is reversed by the calcium ionophore A23187, demonstrating that these effec

84 e following treatment of thymocytes with the calcium ionophore A23187, ionomycin, or forskolin.

85 s pathways induced by five stimuli; PMA, the calcium ionophore A23187, nigericin, Candida albicans an

86 PGI2 synthesis stimulated by bradykinin, the calcium ionophore A23187, or arachidonic acid.

87 uch as sphingomyelinase and phospholipase C, calcium ionophore A23187, or heat resulted in the dramat

88 During platelet activation with thrombin, calcium ionophore A23187, or phorbol 12-myristate 13-ace

89 doleyl maleimide, cyclic nucleotide analogs, calcium ionophore A23187, or phorbol 12-myristate 13-ace

90 -19 cells with tunicamycin, brefeldin A, the calcium ionophore A23187, or thapsigargin increased the

91 peptide all induced Rap1 activation, as did calcium ionophore A23187, phorbol ester, forskolin, 8-br

92 stion using cross-adaptation to H2O2 and the calcium ionophore A23187, stable DSCR1 (Adapt78) transfe

93 effect of endothelin-1 was also mimicked by calcium ionophore A23187, suggesting the importance of C

94 Treatment with the calcium ionophore A23187, which increases Ca(2+)-calmodu

95 e approximately 50% of the levels induced by calcium ionophore A23187.

96 mulated by FcepsilonRI cross-linking and the calcium ionophore A23187.

97 lease of superoxide anion in response to the calcium ionophore A23187.

98 %; P = 0.1), as were relaxation responses to calcium ionophore A23187.

99 cked ERK activation evoked by Ang II and the calcium ionophore A23187.

100 ficiently as wt TGF-alpha in response to the calcium ionophore A23187.

101 rences in responses to acetylcholine and the calcium ionophore A23187.

102 regulated within minutes by phorbol ester or calcium ionophore A23187.

103 enhanced endothelium-dependent relaxation to calcium ionophore A23187.

104 pecially following degranulation, induced by calcium ionophore A23187.

105 ther enhanced by IKKgamma in the presence of calcium ionophore A23187.

106 ells upon stimulation of the CESS cells with calcium ionophore A23187.

107 riene B(4) (LTB(4)) when stimulated with the calcium ionophore A23187.

108 of granule exocytosis are induced by PMA and calcium ionophore A23187.

109 Ca(2+) channels, but not that induced by the calcium ionophore A23187.

110 er than that observed after stimulation with calcium ionophore A23187.

111 nd ERK activation, which was reversed by the calcium ionophore A23187.

112 Treatment with calcium ionophores A23187 or thapsigargin markedly inhib

113 es caused by prostaglandin E2, theophylline, calcium-ionophore A23187, 8-bromoadenosine 3':5'-cyclic

114 Pro-BTC shedding was activated by calcium ionophore (A23187) and by the metalloprotease ac

115 ivation by epidermal growth factor (EGF) and calcium ionophore (A23187) on IL-8 and COX-2 reporter ge

116 ximal membrane signaling, and with PMA and a calcium ionophore (A23187) to bypass membrane-mediated s

117 helium-dependent vasorelaxation induced by a calcium ionophore (A23187) was increased in both groups

118 2) activity of CD34(+) cells stimulated with calcium ionophore (A23187) was of similar magnitude to t

119 veolar lavage and stimulated in vitro with a calcium ionophore (A23187), and the concentrations of le

120 However, both spontaneous and calcium ionophore (A23187)-initiated AR were unaffected.

121 -O-tetradecanoylphorbol-13-acetate; TPA) and calcium ionophore (A23187)-mediated induction of PGE2 sy

122 ducts, 12(S)-HETE-LPC and 12(S)-HETE-LPE, in calcium ionophore (A23187)-stimulated murine platelets.

123 H2O+.OH)], interferon-gamma (IFN-gamma), and calcium ionophore (A23187).

124 es induced by not only ethanol but also by a calcium ionophore (A23187).

125 mast cells (MC) on stimulation with PMA and calcium ionophore (A23187).

126 age to C2C12 skeletal muscle cells using the calcium ionophore, A23187 and the mitochondrial uncouple

127 TPA (1 microM) caused 45% and the calcium ionophore, A23187, 11% of maximal tyrosine phosp

128 Interestingly, treatment with the calcium ionophore, A23187, and the PKC activator, PMA, r

129 ) can also inhibit the light response, and a calcium ionophore, A23187, can substitute for light.

130 Treatment of endothelial cells with a calcium ionophore, A23187, decreased alpha-actinin-4-eNO

131 Incubation of cells with thapsigargin or the calcium ionophore, A23187, had no effect on SOK-1 activi

132 ssion of calpain or exposure of cells to the calcium ionophore, A23187, increased CCTalpha degradatio

133 okine, interleukin-1beta (IL-1beta), and the calcium ionophore, A23187, induce an increase in C-1-P l

134 sside for 7 d or the single application of a calcium ionophore, A23187, mimicked effects of glucose,

135 Stimulation of platelets with the calcium ionophore, A23187, resulted in complete proteoly

136 At physiological pH, treatment with the calcium ionophore, A23187, resulted in increased GFAP-IR

137 nts, induced by exposure of the cells to the calcium ionophore, A23187, were resolved at the level of

138 capable of exocytosis upon activation by the calcium ionophore, A23187.

139 se constructs are strongly responsive to the calcium ionophore, A23187.

140 activator, phorbol myristate acetate, or the calcium ionophore, A23187; such mast cells can rapidly r

141 s from acute coronary syndrome patients, and calcium ionophores abolished the observed differences in

142 03 for AUC; P = .0002 for PT) and similar to calcium-ionophore activated platelets (AUC of 332 +/- 15

143 y platelets and leukotriene B4 released from calcium ionophore-activated blood were markedly reduced,

144 tase did not influence matrix release by the calcium ionophore-activated chondrocytes.

145 In contrast to cytolytic IL-4 release from calcium ionophore-activated eosinophils, eotaxin and RAN

146 on at lipid bodies, and priming for enhanced calcium ionophore-activated LTC(4) release.

147 yte is artificially activated by exposure to calcium ionophore, after which PB2 is biopsied and colle

148 ibited by a combination of phorbol ester and calcium ionophore, agents that bypass proximal signaling

149 can be induced by stimulation of T cells by calcium ionophore alone and requires the calcineurin-dep

150 ly, however, in contrast to the IL-2 gene, a calcium ionophore alone was sufficient to induce IL-21 g

151 xposure to tumor necrosis factor alpha or to calcium ionophore also was prevented by removal of unest

152 erence to fibronectin in response to PMA and calcium ionophore and allows higher saturation densities

153 s in response to inflammatory agonists (e.g. calcium ionophore and ATP).

154 e treated these podocytes temporarily with a Calcium Ionophore and facultatively with Latrunculin A,

155 ggregates was significantly augmented by the calcium ionophore and prevented by the inhibitor cystami

156 The concept is established with lithium and calcium ionophores and accompanied by a response model t

157 Calcium ionophores and ionomycin also activated ERK and

158 y Ni2+ exposure because Cap43 was induced by calcium ionophores and its induction was attenuated by b

159 ostimulation of WEHI-231 and CH31 cells with calcium ionophores and protein kinase C agonists partial

160 Furthermore, ionomycin (calcium ionophore) and TPA augmented the enhancer activi

161 TM cells is regulated by a corticosteroid, a calcium ionophore, and a component of aqueous humor, sug

162 hed carborane, a recently introduced grafted calcium ionophore, and an MMA-DMA polymer matrix.

163 The calcium ionophore, and bradykinin, which are known to ac

164 activated by growth factors, phorbol ester, calcium ionophore, and hypertonic stress.

165 tussis toxin-insensitive, can be mimicked by calcium ionophore, and is inhibited by treatment with cy

166 pertussis toxin treatment, is not induced by calcium ionophore, and is insensitive to cytochalasin D.

167 s significantly enhanced by stimulation with calcium ionophore, and was drastically reduced by cyclos

168 vates a Gs-coupled beta-adrenergic receptor, calcium ionophores, and phorbol 12-myristate 13-acetate,

169 cipitates with NF-ATc in nuclear extracts of calcium ionophore- and Dex-treated cells.

170 nduced by a combination of phorbol ester and calcium ionophore at the highest level among the inducib

171 mpared the response of human erythrocytes to calcium ionophore at various temperatures in the range o

172 mical calcium pump based on a fast diffusive calcium ionophore-based membrane is reported.

173 , gamma irradiation, UV irradiation, and the calcium ionophore beauvericin.

174 ith microtubule antagonists, using the caged-calcium ionophore Br-A23187, was capable of inducing the

175 istate 13-acetate plus phytohemagglutinin or calcium ionophore but not to anti-CD3/anti-CD28 costimul

176 We also discovered that BWSV and the calcium ionophore calcimycin stimulate FM1-43 destaining

177 Calcium ionophores, calmodulin antagonists, and tyrosine

178 ast, activation of platelets by thrombin and calcium ionophore caused release of both APP and Abeta i

179 Ionomycin, a calcium ionophore, causes rapid mitochondrial accumulati

180 m entry from intracellular stores or through calcium ionophore channels activated secretion, though n

181 d from endogenous esterified lipid stores by calcium ionophore (CI) calcimycin (A-23187).

182 fied CD33(+) CML cells from 15 patients with calcium ionophore (CI) consistently resulted in de novo

183 blood monocytes or dendritic cells (DC) with calcium ionophore (CI) led to the rapid (18 hour) acquis

184 Cs, additional overnight culture with either calcium ionophore (CI) or interferon gamma (IFN-gamma),

185 In contrast, calcium ionophore (CI) treatment induced rapid and consi

186 We have shown previously that calcium ionophore (CI) treatment of various myeloid orig

187 d DCs on culture with Bryo-1 alone, Bryo-1 + calcium ionophore (CI), but not CI alone exhibited morph

188 in response to bacterial LPS, TNF-alpha, or calcium ionophore (CI).

189 ring of cytoplasmic Ca(2+) blocks egress and calcium ionophores circumvent the need for a reduction o

190 ctivation of endogenous calcineurin with the calcium ionophore decreased p38 phosphorylation and incr

191 d oocytes could be artificially activated by calcium ionophore, demonstrating that the oocytes were f

192 lation of mouse astrocytes with ionomycin, a calcium ionophore, enhanced the production of anandamide

193 1 cells pretreated with thapsigargin but not calcium ionophore exhibited increased Duox-1 mRNA expres

194 Following stimulation with phorbol ester and calcium ionophore, expression of the bovine gene was det

195 Incubation of DCs with A23187 calcium ionophore for 48 h induced an expression of matu

196 Additional treatment of the cells with calcium ionophore further enhanced the hydrolysis rate a

197 Conversely, calcium ionophore greatly enhanced SR-A up-regulation by

198 NFAT activation, whereas and ionomycin, two calcium ionophores, had synergistic effects with vanadiu

199 orate and calcium ionophore IV (ETH 5234) or calcium ionophore I (ETH 1001).

200 bol ester alone, whereas B-2 cells require a calcium ionophore in addition to phorbol ester to trigge

201 , porcine aqueous humor, dexamethasone, or a calcium ionophore in cells pretreated with dexamethasone

202 ways with a combination of phorbol ester and calcium ionophore in clinorotation resulted in full expr

203 modified merocyanine photoacid polymer and a calcium ionophore in plasticized poly(vinyl chloride).

204 Nitrite generation after stimulation with calcium ionophore increased in Ad.CMVeNOS veins (1420.0+

205 atment of nonprimed macrophages with ATP and calcium ionophore induced a rapid release of MV that wer

206 n with EGTA suppressed p38 activation, while calcium ionophore induced p38 activity.

207 Moreover, although Raf activation or calcium ionophores induced little c-Fos expression, we o

208 Activation of cPLA(2) by the calcium ionophore, induced a dose-dependent increase of

209 tact T cells with the BTP compounds prior to calcium ionophore-induced activation of CaN caused NFAT

210 we report that dexamethasone (Dex) inhibits calcium ionophore-induced activation of the human IL-4 p

211 Evaluation of 3-oxa analogues 4 and 5 in calcium ionophore-induced acute skin inflammation model

212 denced by their distinct effects on LPS- and calcium ionophore-induced DC differentiation, on LPS-ind

213 thermore, tamoxifen pretreatment accelerated calcium ionophore-induced death by more than 20 min, sug

214 ponse to LPS, but had little or no effect on calcium ionophore-induced differentiation.

215 e same promoter region was also required for calcium ionophore-induced gene expression.

216 ar concentrations, both LTRAs also inhibited calcium ionophore-induced leukotriene (leukotriene B(4)

217 Addition of the calcium ionophore ionomycin alone induced the translocat

218 The calcium ionophore ionomycin also induced a rapid hyperpo

219 vated by phorbol 12-myristate 13-acetate and calcium ionophore ionomycin and was blocked by the NFAT

220 In this study, using the calcium ionophore ionomycin and/or PMA on Jurkat T cells

221 When the cells were treated with the calcium ionophore ionomycin in the nominal absence of ex

222 calcium signaling, and 2) treatment with the calcium ionophore ionomycin or cotransfection with activ

223 Exposure of human erythrocytes to the calcium ionophore ionomycin rendered them susceptible to

224 Treatment with the calcium ionophore ionomycin resulted in increased GlcAT-

225 ing MARK2 activity blocks the ability of the calcium ionophore ionomycin to promote neurite outgrowth

226 After stimulation with the calcium ionophore ionomycin, 2-AG levels in MGL-silenced

227 plus TNF-alpha, or PMA (with or without the calcium ionophore ionomycin, or TNF-alpha), with differe

228 h elevated extracellular potassium, with the calcium ionophore ionomycin, or with thyrotropin releasi

229 vate the nitric oxide pathway, including the calcium ionophore ionomycin, the nitric oxide donor S-ni

230 nd AAV transduction in vitro Conversely, the calcium ionophore ionomycin, which disrupts calcium grad

231 measured before and after application of the calcium ionophore ionomycin, which promotes the inflow o

232 er this mucin is secreted in response to the calcium ionophore ionomycin.

233 The same is true of the calcium ionophore ionomycin.

234 y-5-methyl-4-isoxalonepropionic acid, or the calcium ionophore ionomycin.

235 by constitutively active calcineurin or the calcium ionophore ionomycin.

236 ecreased upon exposure of the parasites to a calcium ionophore (ionomycin), to an inhibitor of the V-

237 aling pathways using phorbol ester (PMA) and calcium ionophore (ionomycin).

238 The calcium ionophore, ionomycin, induced chondrogenesis thr

239 cium ATPase inhibitor, thapsigargin, and the calcium ionophore, ionomycin, were used to deplete store

240 n SH-SY5Y human neuroblastoma cells with the calcium ionophore, ionomycin.

241 Second, exposure of macrophages to the calcium ionophores, ionomycin or A23187, mimicked recept

242 ar sites where, following stimulation with a calcium ionophore, it can be acted upon by cytosolic pho

243 is[3,5-bis(trifluoromethyl)phenyl]borate and calcium ionophore IV (ETH 5234) or calcium ionophore I (

244 A23187, a calcium ionophore known to block furin maturation, also

245 he endoplasmic reticulum to the Golgi, or, a calcium ionophore known to inhibit the autoactivation of

246 Treatment of cells with the calcium ionophore, leading to inhibition of calcium-depe

247 nt of beta cell lines (betaTC1 and HIT) with calcium ionophore led to a significant elevation in acti

248 t desensitization of NPR-B, and ionomycin, a calcium ionophore, mimics the AVP effect.

249 FK506 overcame the antiadipogenic effect of calcium ionophore on the differentiation of 3T3-L1 pread

250 Addition of a calcium ionophore or 100 microm NMDA decreased phospho-E

251 at observed for typical NO synthase agonists calcium ionophore or acetylcholine.

252 When Prss31-null MCs were activated with a calcium ionophore or by their high affinity IgE receptor

253 ne, without any alteration in sensitivity to calcium ionophore or hydrogen peroxide.

254 ndothelial cells were stimulated by either a calcium ionophore or thapsigargin to produce NO.

255 tosolic calcium with adrenergic stimulation, calcium ionophore, or calcium-containing pipette solutio

256 Activation with LPS, PMA, and calcium ionophore, or ligation of CD40 alone or in combi

257 , cytokines with platelet activating factor, calcium ionophore, or phorbol myristate acetate, develop

258 agents (the chemotherapeutic drug etoposide, calcium ionophore, or UV irradiation) and the withdrawal

259 cers of ER stress, including tunicamycin and calcium ionophore, or when a nonpolymerogenic alpha(1)-a

260 Treatment with calcium ionophores overcame agatoxin inhibition in a cal

261 pulse-chase analysis of tissue explants, to calcium ionophore perturbation of intracellular calcium

262 rine B82L fibroblasts that were treated with calcium ionophore, phorbol ester, or epidermal growth fa

263 Functionally, spermatozoa exposed to calcium ionophore, phorbol ester, or H(2)O(2) exhibited

264 macrophages with diverse agonists including calcium ionophores, phorbol myristate acetate (PMA), oka

265 Stimulation of VSM cells with ionomycin, a calcium ionophore, resulted in activation of CaMKIIdelta

266 treatment resulted in enhanced IFN-gamma and calcium ionophore-stimulated arachidonic acid release su

267 lipid body numbers correlated with increased calcium ionophore-stimulated LTC(4) production; and as d

268 caveolae-enriched membranes (2.23-fold), and calcium ionophore-stimulated NO production (1.56-fold).

269 Calcium ionophore-stimulated platelets also recapitulate

270 2 nM) and good in vivo pharmacodynamics in a calcium ionophore-stimulated rat model after oral dosing

271 to study the effect of the glucocorticoid on calcium-ionophore-stimulated 5-lipoxygenase product and

272 sis and killing of S. pneumoniae and reduced calcium-ionophore-stimulated leukotriene B(4) synthesis

273 ility to generate leukotriene (LT) C(4) upon calcium ionophore stimulation but had little effect on L

274 e in vivo and in humans to phorbol ester and calcium ionophore stimulation in vitro in the face of lo

275 on of the biodetector ring relaxation during calcium ionophore stimulation of Ad.CMVeNOS veins.

276 was duplicated by incubating the cells with calcium ionophores such as ionomycin.

277 ) with Mos and restarted the cell cycle with calcium ionophore; the 30 minute cycle was converted int

278 eficient sperm acrosome-react in response to calcium ionophore, they do not acrosome-react in respons

279 failure of microvesiculation in response to calcium ionophore, thrombin, and collagen stimulation.

280 background levels of thrombin in response to calcium ionophore, thrombin, or combined thrombin plus c

281 cts was observed in U87MG cells treated with calcium ionophore to activate the calcium-dependent calp

282 In human erythrocytes, the ability of the calcium ionophore to cause susceptibility depends on tem

283 Furthermore, the ability of calcium ionophore to induce increased membrane order and

284 d with arachidonic acid were stimulated with calcium ionophore to initiate leukotriene biosynthesis.

285 +)](i) was significantly lower compared with calcium ionophore-treated cells, suggesting that a susta

286 ity to phagocytose apoptotic lymphocytes and calcium ionophore-treated erythrocytes but had no effect

287 semia, we studied the role of PS exposure in calcium ionophore-treated normal RBC adherence to human

288 Calcium ionophore treatment of chondrocytes, in a dose-d

289 Calcium ionophore treatment of eggs did not cause associ

290 We have found that calcium ionophore treatment of Rat-1 Elk-1, MCF-7 Elk-1,

291 ot Ras, protein activation following PMA and calcium ionophore treatment.

292 ts, and the amount of adducts increased with calcium ionophore treatment.

293 uption, TF pathway inhibitor inhibition, and calcium ionophore treatment.

294 Leaf infiltration with calcium ionophore triggered high-level PR gene expressio

295 (P = .0001) and by exposing intact cells to calcium ionophore using the cGMP reporter cell assay (P

296 sms, a simplified model of apoptosis using a calcium ionophore was applied.

297 ulted in equivalent up-regulation, whereas a calcium ionophore was relatively ineffective.

298 .) production evoked by either bradykinin or calcium ionophore was unaffected by PEG-catalase.

299 to bradykinin and acetylcholine but not the calcium ionophore were shifted significantly to the left

300 as well as reactive oxygen species (ROS) and calcium ionophore, whereas knockdown of RIP3 and MLKL bl