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1 itonin (hCT), a peptide hormone important in calcium metabolism.
2 ctive pharmacological candidate for managing calcium metabolism.
3 ht, which alters the endocrine regulation of calcium metabolism.
4 tamin D insufficiency and a sign of impaired calcium metabolism.
5 zyme Q (CoQ) biosynthesis, and mitochondrial calcium metabolism.
6 r hypoxic environment, possibly by modifying calcium metabolism.
7  eventual failure, partly through effects on calcium metabolism.
8 protein synthesis, membrane trafficking, and calcium metabolism.
9 differential expression of genes relating to calcium metabolism.
10  the importance of evaluating all aspects of calcium metabolism.
11 Soy isoflavones did not significantly affect calcium metabolism.
12  suggest that CaBP-D28k is not essential for calcium metabolism.
13 peutic agents aimed at treating disorders of calcium metabolism.
14  were also measured as well as parameters of calcium metabolism.
15 rly understood actions of dietary protein on calcium metabolism.
16 pressure is associated with abnormalities in calcium metabolism.
17 ate the role of the CaSR in regulating fetal calcium metabolism.
18  be related to the effects of hypothermia on calcium metabolism.
19 t dietary protein is a powerful regulator of calcium metabolism.
20 ied as CCC1, previously shown to function in calcium metabolism.
21 otein particles in osteoporosis and impaired calcium metabolisms.
22   PHPT is a complex endocrinopathy involving calcium metabolism and a potent hormone made by the para
23         Vitamin D plays an important role in calcium metabolism and affects other metabolic pathways.
24 estigation, the impact of dietary protein on calcium metabolism and bone balance remains controversia
25 eriod can be a time when profound changes in calcium metabolism and bone mineral density (BMD) occur,
26 ometry, and biochemical variables related to calcium metabolism and bone turnover were measured.
27 excitability which may be linked to abnormal calcium metabolism and loss of cholinergic neurons in th
28 s the impact of vitamin D supplementation on calcium metabolism and non-calcemic broad gene expressio
29 ation of renin expression was independent of calcium metabolism and that 1,25(OH)(2)D(3) markedly sup
30 nism involving vulnerability to dysregulated calcium metabolism and the combination of fusing a lipop
31 as candidates involved in regulating phospho-calcium metabolism and valvular angiotensin II synthesis
32 ds-scale spatiotemporal dynamics of neuronal calcium, metabolism and brain blood oxygen can be accura
33 d genes, including many related to lipid and calcium metabolism, and 1292 down-regulated genes, some
34 fusion, hemodynamic stresses, alterations in calcium metabolism, and dysregulation of cell signaling
35 fects ventricular performance and mechanics, calcium metabolism, and electrical properties of myocyte
36                  Parathyroid hormone levels, calcium metabolism, and previous scintigraphy and ultras
37 contact [MERC]), which plays a major role in calcium metabolism, apoptotic processes, and inflammatio
38 ence, we established that abnormal lipid and calcium metabolism are important contributors to hepatic
39 ermediate phenotypes of cardiac function and calcium metabolism are responsible for the difference in
40  myocytes revealed unique characteristics of calcium metabolism as a function of myocyte shape.
41 coupled receptor that has been implicated in calcium metabolism, as the top candidate gene for modula
42      To determine the role of PTHrP in fetal calcium metabolism, blood calcium was measured in mice h
43 are associated with changes in vitamin D and calcium metabolism but the impact of these changes on vi
44 thyroidism is a common endocrine disorder of calcium metabolism characterised by hypercalcaemia and e
45                        Mechanisms regulating calcium metabolism during WL are unclear.
46  PIH are unclear, but several alterations in calcium metabolism have been identified.
47                          The disturbances in calcium metabolism have been shown to contribute signifi
48                                              Calcium metabolism in 15 postmenopausal women was studie
49  neurological deterioration, led us to study calcium metabolism in a cohort of 42 children.
50 tion are critical for controlling whole-body calcium metabolism in growing mice.
51 eficial skeletal effects, but the effects on calcium metabolism in humans are not known.
52  and corrects abnormalities of intracellular calcium metabolism in insulin-sensitive tissues (liver,
53 bean isoflavones, soy protein, or both alter calcium metabolism in postmenopausal women.
54      We analyzed the side effects related to calcium metabolism in RCTs, specifically hypercalcemia,
55 nce of its central role in the regulation of calcium metabolism in the platelet, the plasma membrane
56 mediated control of parathyroid function and calcium metabolism in vivo.
57                      SR proteins involved in calcium metabolism, including the sarcoplasmic/endoplasm
58                      In addition to abnormal calcium metabolism, inflammation, genetic makeup and die
59    Pathologic dysregulation of extracellular calcium metabolism is difficult to correct.
60 ne groups have direct or secondary action on calcium metabolism or transport.
61 criptional and translational regulators, and calcium metabolism-related genes.
62 t the importance of extraparathyroid CaSR in calcium metabolism remains unknown.
63 r export was not inhibited by antagonists of calcium metabolism that block nuclear import.
64 mune-cell infiltration, and deranged phospho-calcium metabolism that collectively perpetuate a pro-in
65 atients often have an underlying disorder in calcium metabolism that results in hypercalcuria and hyp
66 nts had at least one abnormal measurement of calcium metabolism, the commonest being moderately low s
67          Parathyroid hormone (PTH) regulates calcium metabolism through a specific G protein-coupled,
68                                    Stress to calcium metabolism was defined as elevated intact parath
69          Other phenotypes were revealed when calcium metabolism was normalized in KO/TG mice: serum 1