ライフサイエンスコーパス: calcium mobilization

1 tion of BCR signaling results in a decreased calcium mobilization.

2 echanism either downstream or independent of calcium mobilization.

3 r signaling networks that critically rely on calcium mobilization.

4 tial for repair, acts downstream of TFF2 and calcium mobilization.

5 ted activation of PLCgamma1 and that inhibit calcium mobilization.

6 red intermediate signaling events, including calcium mobilization.

7 blocked estrogen-induced PI3K activation and calcium mobilization.

8 negative cooperativity for agonist-mediated calcium mobilization.

9 in-induced inositol phosphate production and calcium mobilization.

10 ed kinases, and AKT, and blocked BCR-induced calcium mobilization.

11 , significantly increased carbachol-mediated calcium mobilization.

12 o significantly increased carbachol-mediated calcium mobilization.

13 of mammalian cells and mediate IP(3)-induced calcium mobilization.

14 l adhesion and is inefficient in stimulating calcium mobilization.

15 n a measure of membrane potential and not in calcium mobilization.

16 via the Galphai/phospholipase C pathway for calcium mobilization.

17 eading to activation of protein kinase C and calcium mobilization.

18 PAR4mediated aggregation are independent of calcium mobilization.

19 ptor gamma-chain signaling and intracellular calcium mobilization.

20 t, and its coligation with the BCR inhibited calcium mobilization.

21 d nonhemopoietic compartments via effects on calcium mobilization.

22 , to scavengers of ROS, and to inhibitors of calcium mobilization.

23 tive mutant strongly inhibited BCR-dependent calcium mobilization.

24 g each step leading to Rho activation and/or calcium mobilization.

25 cRgamma phosphorylation, Syk activation, and calcium mobilization.

26 totoxic T lymphocyte antigen-4 (CTLA-4), and calcium mobilization.

27 ated platelet shape change and intracellular calcium mobilization.

28 diated platelet alphaIIbbeta3 activation and calcium mobilization.

29 Ten-fold increases in IP3 caused saturated calcium mobilization.

30 eutrophil respiratory burst, chemotaxis, and calcium mobilization.

31 ly or largely eliminated dopamine-stimulated calcium mobilization.

32 st, SKF83959 appeared to be an antagonist of calcium mobilization.

33 increased elastase release and intracellular calcium mobilization.

34 protein-coupled receptors and leads to rapid calcium mobilization.

35 11 with improved or comparable potencies in calcium mobilization, [(35)S]GTPgammaS binding, and cAMP

36 Inhibitors of calcineurin and calcium mobilization abolished proline oxidase-mediated

37 ists leukotriene D(4) and UDP signal through calcium mobilization, acting on separate receptors, and

38 However, CD22 negative regulation of calcium mobilization after B cell antigen receptor ligat

39 f surface immunoglobulin M and impairment of calcium mobilization after BCR engagement in vitro.

40 Indeed, we investigated the degree of calcium mobilization after FasL stimulation and found th

41 or a kinase-dead mutant (GRK2-K220R) reduced calcium mobilization after receptor activation, whereas

42 addition, overexpression of DGKeta enhanced calcium mobilization after stimulating muscarinic recept

43 However, calcium mobilization alone by ionomycin was insufficient

44 d in a significant increase in intracellular calcium mobilization, an effect that was both delayed an

45 Validation was conducted using both calcium mobilization and [(125)I]-orexin-A competition b

46 tly inhibited CP55,940-induced intracellular calcium mobilization and [(35)S]GTP-gamma-S binding whil

47 antagonist of chemerin induced intracellular calcium mobilization and a much higher potency against t

48 an affect second messenger systems including calcium mobilization and a plethora of kinases to alter

49 an affect second messenger systems including calcium mobilization and a plethora of kinases within ne

50 through a mechanism dependent upon cytosolic-calcium mobilization and a significant decline in GRP78/

51 ; G(q) protein and pertussis toxin-sensitive calcium mobilization and activation of a nonselective ca

52 ed we demonstrate that LPI is able to induce calcium mobilization and activation of Akt and extracell

53 lation and was needed for optimal TCR-driven calcium mobilization and activation of the kinase Erk.

54 am signaling events, including intracellular calcium mobilization and activation of the Ras-extracell

55 rs did not affect fMLF-induced intracellular calcium mobilization and Akt phosphorylation.

56 her, Pak1(-/-) BMMCs demonstrated diminished calcium mobilization and altered depolymerization of cor

57 s well as reductions in chemokine-stimulated calcium mobilization and altered ERK and Akt activation.

58 ase C but activated phospholipase Cgamma and calcium mobilization and augmented these signals as well

59 Pharmacological evaluation in calcium mobilization and binding assays revealed the imp

60 Ghrelin increased intracellular calcium mobilization and cAMP levels in oral epithelial

61 mately reduced cancer cell functions such as calcium mobilization and cellular chemotaxis.

62 Furthermore, the calcium mobilization and chemotactic responses of LPS-st

63 mor cell line 66.1 resulted in intracellular calcium mobilization and chemotaxis in vitro.

64 nting cell function to T helper cells and DC calcium mobilization and chemotaxis to histamine.

65 ic Src family enzymes, Ly-49D/DAP12-mediated calcium mobilization and cytokine production by CD45 nul

66 This was linked to a similar enhancement in calcium mobilization and cytoskeletal rearrangement attr

67 ary for MC activation, including Ag-mediated calcium mobilization and cytoskeletal reorganization, po

68 y different FcalphaRI-mediated intracellular calcium mobilization and degranulation in rat basophilic

69 mma (PLCgamma) is a ubiquitous gatekeeper of calcium mobilization and diacylglycerol-mediated events

70 PR120S receptors elicited both intracellular calcium mobilization and DMR responses in human embryoni

71 +)CD21(low) subpopulation exhibits decreased calcium mobilization and does not efficiently differenti

72 king of GPR120S and GPR120L receptors, using calcium mobilization and dynamic mass redistribution (DM

73 n epidermal melanocytes (HEMs) to UVA causes calcium mobilization and early melanin synthesis.

74 recise organization is essential for optimal calcium mobilization and efficient and effective muscle

75 n the present study, we have used cell-based calcium mobilization and electrophysiological assays to

76 Stimulation with those CCR6 ligands leads to calcium mobilization and elevated active RhoA, phosphory

77 de corresponding to its V3-like loop induced calcium mobilization and enhanced thrombin-mediated plat

78 onal EP(1) receptor coupled to intracellular calcium mobilization and EP(2) receptor coupled to cAMP

79 This results in impaired TCR-mediated calcium mobilization and Erk activation.

80 inating biased agonism between intracellular calcium mobilization and ERK1/2 phosphorylation.

81 2 and S1P3 receptors activated intracellular calcium mobilization and extracellular signal-regulated

82 as functional tests of H3 receptor-mediated calcium mobilization and GTPgammaS binding.

83 d or toll-like receptor signaling to improve calcium mobilization and improve keratinocyte differenti

84 ation, this chimeric receptor induced robust calcium mobilization and increase of adenylate cyclase a

85 tein coupled signaling pathway that leads to calcium mobilization and increased melanin.

86 Kynurenic acid elicits calcium mobilization and inositol phosphate production i

87 ssessed significant in vivo activity on bone calcium mobilization and intestinal calcium transport.

88 signals that induce changes in intracellular calcium mobilization and maintain tolerance by preventin

89 gnaling pathway in melanocytes that leads to calcium mobilization and melanin synthesis and may under

90 tromal cell-derived factor-1 (SDF-1)-induced calcium mobilization and migration and do not localize t

91 cPLA(2)alpha is regulated by calcium mobilization and mitogen-activated protein kinas

92 , and downstream signaling events, including calcium mobilization and mitogen-activated protein kinas

93 is pathway is dependent on protein kinase C, calcium mobilization and mitogen-activated protein kinas

94 epsilonRI and CD300c increased intracellular calcium mobilization and phosphorylation of signaling in

95 Apical ATP release with subsequent calcium mobilization and PKA activation are involved in

96 atelet responses by inhibiting intracellular calcium mobilization and PKC activation through G(q) pat

97 strate PLCgamma2 to enhance collagen-induced calcium mobilization and platelet activation.

98 roprotection by decreasing glutamate-induced calcium mobilization and preventing apoptotic gene expre

99 signal-regulated kinase activation, to drive calcium mobilization and promote gastric repair.

100 10(8) cells, thrombin), is externalized via calcium mobilization and protease-activated receptors-1

101 e variants on their effects on AT1R-mediated calcium mobilization and provides molecular understandin

102 Likewise, calcium mobilization and radioligand binding assays foun

103 Intracellular calcium mobilization and receptor antagonist studies rev

104 TCR-CD28 linked calcium mobilization and subsequent uptake by mitochondr

105 GPR30 by estrogen resulted in intracellular calcium mobilization and synthesis of phosphatidylinosit

106 CRAMP also induced calcium mobilization and the activation of MAPK in monoc

107 ritical for the CXCR2-mediated intracellular calcium mobilization and the resultant migration and inf

108 y secreted nucleotides plays a major role in calcium mobilization and the subsequent calcium-dependen

109 n cells was found to prolong mGluR5-mediated calcium mobilization and to also potentiate mGluR5-media

110 tdIns(3,4,5)P3, leading to the inhibition of calcium mobilization and to the attenuation of mast cell

111 ormal BCR signal transduction as measured by calcium mobilization and tyrosine phosphorylation.

112 otion of cell proliferation, cell migration, calcium mobilization, and activation of ERK1/2.

113 Effects of the drugs on DC chemotaxis, calcium mobilization, and antigen-presenting cell functi

114 n IL-4-stimulated monocytes, are elevated on calcium mobilization, and are detected at increased leve

115 hosphorylation, adenylyl cyclase inhibition, calcium mobilization, and beta-arrestin recruitment.

116 In human PBMC, RvE1 partially induced calcium mobilization, and blocked subsequent stimulation

117 ns-4,5-P(2) synthesis, PLCgamma2 activation, calcium mobilization, and cell proliferation.

118 hK2 is required for murine MC degranulation, calcium mobilization, and cytokine and leukotriene produ

119 s enhanced platelet activation, monitored by calcium mobilization, and endothelial activation, monito

120 e 5'-O-(3-[(35)S]thio)-triphosphate binding, calcium mobilization, and extracellular signal-regulated

121 f the Syk-Akt-S6kinase signaling pathway and calcium mobilization, and inhibits SHP-1 activity upon B

122 re measured, including platelet aggregation, calcium mobilization, and integrin activation.

123 nd CCK(2)) receptors using receptor binding, calcium mobilization, and internalization studies.

124 RP1 has since been implicated in suppressing calcium mobilization, and its expression is misregulated

125 Cell structural integrity and viability, calcium mobilization, and nitric oxide generation were d

126 rtery endothelial organization and function, calcium mobilization, and nitric oxide generation were w

127 , caspase-1, cathepsin-mediated degradation, calcium mobilization, and potassium efflux but not caspa

128 al-regulated kinase 1/2 (ERK1/2) activation, calcium mobilization, and receptor endocytosis] in the s

129 rotein q, the activation of phospholipase C, calcium mobilization, and the release of the inhibitory

130 ation, C5a- and UTP-stimulated intracellular calcium mobilization, and the stimulation of cAMP format

131 augment phospholipase Cgamma1 activation and calcium mobilization, and to phosphorylate NFkappaB and

132 lling activity, measured by anti-IgM-induced calcium mobilization, and with increased expression of C

133 , including phosphatidylinositol hydrolysis, calcium mobilization, arachidonic acid release, mitogen-

134 (PLD) and protein kinase C (PKC) as well as calcium mobilization are essential signals for degranula

135 ncluding that of phospholipase C-gamma2, and calcium mobilization are impaired in IgA-presenting WIP

136 phospho-phospholipase Cgamma1 induction and calcium mobilization are largely unaffected.

137 actin rearrangement, MTOC polarization, and calcium mobilization are not.

138 he same enhancing effect on carbachol-evoked calcium mobilization as overexpressed DGKeta, and overex

139 ive vesicle-associated membrane proteins and calcium mobilization, as well as PI3K, MAPK, and IkappaB

140 Sequential stimulation in the calcium mobilization assay gave no evidence for desensit

141 sted for OX1 and OX2 receptor activity using calcium mobilization assay in recombinant cell lines.

142 ist (efficacy ca. 50%) in a 5-HT(2)-mediated calcium mobilization assay.

143 ng a cell-based high-throughput fluorescence calcium mobilization assay.

144 ovel D2 dopamine receptor modulators using a calcium mobilization assay.

145 MMPIP acts as a noncompetitive antagonist in calcium mobilization assays in cells coexpressing mGluR7

146 With calcium mobilization assays of FPR3-transfected HEK 293

147 meric receptors were evaluated in functional calcium mobilization assays to compare orthosteric agoni

148 , Brilliant Black BN is commonly used within calcium mobilization assays to quench extracellular fluo

149 ced the Emax of the agonist curve in the CB1 calcium mobilization assays, confirming their negative a

150 and determined their potency in OX1 and OX2 calcium mobilization assays.

151 g GTP-gamma-S accumulation and intracellular calcium mobilization assays.

152 restin-2 recruitment, cAMP accumulation, and calcium mobilization assays.

153 ed them responsive to CXCL17, as measured by calcium-mobilization assays.

154 with Fluo-4 resulted in a rapid increase in calcium mobilization associated cell fluorescence simila

155 s desensitization of a receptor(s) linked to calcium mobilization because a second application of SP-

156 osphorylation was dependent on intracellular calcium mobilization, because BAPTA-AM blocked DRAK2 kin

157 -induced global tyrosine phosphorylation and calcium mobilization but impaired the Gab2-PI3K pathway

158 s required for CXCR3-mediated chemotaxis and calcium mobilization but is not required for ligand bind

159 d found that knockdown of CK1alpha increased calcium mobilization but not ERK activation.

160 internalization, increased PI production and calcium mobilization by LTD4, and significantly attenuat

161 tion assays but marked loss of intracellular calcium mobilization by peptide agonists.

162 this hypothesis, we disrupted intracellular calcium mobilization by treating immortalized mouse M-1

163 control B cell activation was determined by calcium mobilization, by tyrosine phosphorylation of sig

164 Since platelet intracellular calcium mobilization [Ca(t)]i controls granule release,

165 Lgmn activated PAR(2) to induce calcium mobilization, cAMP formation, and activation of

166 ular tyrosine phosphorylation, intracellular calcium mobilization, CD69 surface expression, interleuk

167 d defects in chemokine-induced intracellular calcium mobilization, chemotaxis, and homing, whereas Gn

168 ining the PDZ motif) inhibited intracellular calcium mobilization, chemotaxis, and transepithelial mi

169 edly enhanced CysLT-stimulated intracellular calcium mobilization compared with endogenous expression

170 Calcium mobilization correlated with the secretion of ly

171 luorescent avidin-based degranulation assay, calcium mobilization, cytokine production in supernatant

172 lonRI, evidenced by enhanced Erk activation, calcium mobilization, degranulation, and cytokine produc

173 esponses in leukocytes including chemotaxis, calcium mobilization, degranulation, and gene expression

174 estigate the molecular mechanisms regulating calcium mobilization during Fas-mediated apoptosis, we h

175 onist (EC50, 802 nM) yet potently stimulated calcium mobilization (EC50, 7 nM) in 5-HT2A receptor-exp

176 termined by mRNA knockdown and intracellular calcium mobilization experiments.

177 memory T cells, characterized by diminished calcium mobilization, expression of CD25, and IL-10 prod

178 and by blocking tyrosine phosphorylation and calcium mobilization following BCR cross-linking.

179 t not R2.60(190)/E6.53(364) was critical for calcium mobilization for all three peptides.

180 ese phmSG cells also demonstrated functional calcium mobilization, formation of epithelial monolayer

181 e vitamin D [1,25(OH)(2)D], which can elicit calcium mobilization from bone when intestinal calcium a

182 stimulation with ionomycin, and BCR-induced calcium mobilization from internal stores is decreased.

183 through voltage-gated calcium channels or on calcium mobilization from intracellular stores.

184 e in inositol trisphosphate (IP3) to trigger calcium mobilization from stores and elevation of [Ca(2+

185 f type II apoptotic cell death that involves calcium mobilization from the ER and that this step is m

186 lls (U937) manifested by immediate cytosolic-calcium mobilization, GADD153 and GADD34 protein inducti

187 nced expression of Itk leads to increases in calcium mobilization, granule release, and cytotoxicity

188 atelet functions such as fibrinogen binding, calcium mobilization, granule secretion, aggregation, an

189 ssays that we have explored, including cAMP, calcium mobilization, guanosine 5'-[gamma-thio]triphosph

190 Although the role of cADPR in modulating calcium mobilization has been extensively examined, its

191 teracting protein, and molecules upstream of calcium mobilization, i.e., activated ZAP70 and phosphol

192 ncluding PLCgamma1 tyrosine phosphorylation, calcium mobilization, IL-2 secretion, and T-cell prolife

193 ar calcium pumps with thapsigargin increases calcium mobilization in 125Tg B cells.

194 nosine, ADP, ATP, UDP, and UTP all stimulate calcium mobilization in bone marrow-derived mast cells w

195 tion, phosphate absorption in the intestine, calcium mobilization in bone, and calcium reabsorption i

196 the amplitude and duration of injury-induced calcium mobilization in cells at the leading edge.

197 dose-response curves assaying intracellular calcium mobilization in cells heterologously expressing

198 We conclude that disruption of calcium mobilization in cells that are normally growth-i

199 We also show that CD38 and cADPR modulate calcium mobilization in chemokine-stimulated DCs and are

200 itol phosphate accumulation or intracellular calcium mobilization in Chinese hamster ovary (CHO) cell

201 suggesting that distinct mechanisms regulate calcium mobilization in each of the two transitional B c

202 led that ethanol inhibited glutamate-induced calcium mobilization in endothelial cells, affected plas

203 f the compounds was supported by analysis of calcium mobilization in HL-60 cells transfected with hum

204 The reduced calcium mobilization in HPKO platelets is associated wit

205 They elicit robust, FPR-dependent calcium mobilization in human and mouse leukocytes and t

206 These agonists produce robust calcium mobilization in human melanoma cell lines which

207 pounds inhibit PAR-4 induced aggregation and calcium mobilization in human platelets and reduce 12-HE

208 tes TRPA1 expression, which is functional in calcium mobilization in mast cells.

209 the first time that FFA3 activation leads to calcium mobilization in murine dorsal root ganglia.

210 eil dematin as a novel regulator of internal calcium mobilization in platelets affecting multiple sig

211 In BSDL(-/-) mice, calcium mobilization in platelets and thrombus formation

212 llagen, and vWF are known to induce in vitro calcium mobilization in platelets.

213 es for their ability to induce intracellular calcium mobilization in RBL-2H3 cells transfected with h

214 t has been implicated in taste buds, elicits calcium mobilization in Receptor (Type II) taste cells.

215 from the BLT2(-/-) mice showed intracellular calcium mobilization in response to 12(S)-hydroxyheptade

216 h that initiates and maintains intracellular calcium mobilization in response to B cell antigen recep

217 tion, reduced Erk activation, and attenuated calcium mobilization in response to BCR engagement.

218 LTE(4) enhanced calcium mobilization in response to PGD(2) in Th2 cells

219 s that disrupted dimer formation had reduced calcium mobilization in response to the PAR4 agonist pep

220 ignaling molecules, cytokine production, and calcium mobilization in response to this toxin.

221 e BCR signalosome is pivotal to BCR-mediated calcium mobilization in the cytosol.

222 n of DGKeta produced no additional effect on calcium mobilization in the presence of BIM I.

223 ctivation threshold by targeting TCR-induced calcium mobilization in thymocytes and peripheral T cell

224 s inhibited trout C3a-mediated intracellular calcium mobilization in trout leukocytes.

225 ), both of which have been shown to modulate calcium mobilization in vitro.

226 t responses that require BDNF expression and calcium mobilization in vmPFC.

227 reduced phospholipase Cgamma activation and calcium mobilization, indicating a defect in calcium sig

228 Tyrosine phosphorylation and calcium mobilization induced by BCR engagement is dimini

229 Platelet aggregation and calcium mobilization induced by the PAR4 thrombin recept

230 PACE4/PC5A decreased calcium mobilization induced by thrombin stimulation.

231 new insight into the molecular basis of bone calcium mobilization induced by vitamin D.

232 Medium-chain FFAs elicit calcium mobilization, inhibit 3',5'-cyclic AMP productio

233 B cell receptor (BCR) inhibited BCR-mediated calcium mobilization, intracellular tyrosine phosphoryla

234 We hypothesize that calcium mobilization is a potential key molecular initia

235 In addition, calcium mobilization is enhanced in RAW264.7 macrophages

236 trant KD, and we identify that regulation of calcium mobilization is fundamental to the underlying im

237 Similarly, calcium mobilization is Galpha(q)-mediated and independe

238 n this article, we confirm that BCR-mediated calcium mobilization is reduced in Cd20(-)/(-) murine B

239 diated activation of RhoGTPase signaling and calcium mobilization, leading to the regulation of kerat

240 to G(o)- but not G(q)-mediated intracellular calcium mobilization, leading us to investigate the role

241 echanism through which reduced intracellular calcium mobilization may be altering cellular proliferat

242 The thrombin-induced calcium mobilization may play an important permissive ro

243 ated ERK signaling and altered intracellular calcium mobilization mediated by these receptors.

244 Cyclic ADP-ribose (cADPR) is a calcium mobilization messenger important for mediating a

245 cle progression, PLC-gamma2 phosphorylation, calcium mobilization, NF-ATp dephosphorylation, and Erk

246 signals downstream of this enzyme including calcium mobilization, NFAT, NF-kappaB, and Ras-ERK-AP-1

247 Intracellular calcium mobilization occurs within milliseconds of pulse

248 chanistically, loss of Cbl-b rescues reduced calcium mobilization of anergic T cells, which was attri

249 f PAR-1 or PAR-2 by agonist peptides induced calcium mobilization, only PAR-1 activation affected bar

250 ta had no detectable effect on intracellular calcium mobilization or endothelial cell viability.

251 pter protein, despite the inability to cause calcium mobilization or TCR polarization.

252 in intracellular signaling pathways, such as calcium mobilization, phosphatidylinositol 3-kinase-AKT

253 IM of FcgammaRIIb and suppressed BCR-induced calcium mobilization, proliferation, and costimulatory m

254 lls resulted in an increase in intracellular calcium mobilization, protein kinase C activation, actin

255 se Cgamma2 activity leading to activation of calcium mobilization, Rap1 and protein kinase C activati

256 of phospholipase Cgamma2, and intracellular calcium mobilization, reflecting competency of BCR signa

257 wound healing regulatory pathway, along with calcium mobilization, regulates CCR6-directed epithelial

258 Forced calcium mobilization rescued the TNFalpha secretion defe

259 cement in the magnitude of the intracellular calcium mobilization response to the group I metabotropi

260 as observed in dose-dependent chemotaxis and calcium mobilization responses.

261 s, dual inhibition of the P2Y12 receptor and calcium mobilization result in a complete inhibition of

262 ng both phosphoinositide (PI) production and calcium mobilization stimulated by LTD4 yet had almost n

263 500 microM) had no significant effect on the calcium mobilization stimulated by the nonselective aden

264 Importantly, Galphai immunoprecipitation and calcium mobilization studies indicated that nuclear CXCR

265 Intracellular calcium mobilization studies using a human epithelial ce

266 Calcium mobilization studies were done utilizing a fluor

267 tion, a detailed analysis of the dynamics of calcium mobilization suggests a possible inhibitory role

268 sphorylation but does not alter C5a-mediated calcium mobilization, supporting its role in modulation

269 taTCR dimerization also alter ligand-induced calcium mobilization, TCR accumulation at the site of pM

270 wed a preference of AEME for the M2 subtype; calcium mobilization tests revealed partial agonist effe

271 ls had lower glutamate-induced intracellular calcium mobilization than did normal RGC-5 cells, and th

272 t cancer cells or isogenic cancer cells, via calcium mobilization through the activation of ryanodine

273 Like other PAR1 activators, C4a induced calcium mobilization through the PAR1/Galphaq/PLCbeta si

274 We monitored calcium mobilization to assess activation of the laser-t

275 during pregnancy may cause maternal skeletal calcium mobilization to meet fetal needs.

276 WT neutrophils in vitro along with increased calcium mobilization upon activation and expression of e

277 ly assessed mAChR-A to monitor intracellular calcium mobilization upon receptor activation.

278 ative responses to anti-CD40, and diminished calcium mobilization upon stimulation with BCR-dependent

279 d the Fc region of chicken IgY and to induce calcium mobilization via association with the common gam

280 utamate receptor subtype 5 (mGlu5) activates calcium mobilization via binding of glutamate, the major

281 ival promotion by OXB required intracellular calcium mobilization via inositol-1,4,5-triphosphate and

282 Calcium mobilization was activated when a mouse fibrobla

283 Cis-UCA-induced calcium mobilization was blocked with a selective 5-HT2A

284 In contrast, bone calcium mobilization was equal to that produced by 1alph

285 BCR-induced calcium mobilization was intact after Mef2c deletion, bu

286 Glutamate-induced intracellular calcium mobilization was measured by using ratiometric c

287 image unstimulated and stimulated platelets, calcium mobilization was monitored as a reporter of plat

288 dicating that P2Y2R-stimulated intracellular calcium mobilization was not involved.

289 Intracellular calcium mobilization was noted at early time points usin

290 Increased intracellular calcium mobilization was observed in B cell lines from h

291 XCR4 signaling in HEK293 cells revealed that calcium mobilization was primarily negatively regulated

292 eness, respiratory burst, degranulation, and calcium mobilization were conducted in human peripheral

293 The mechanisms involved in this calcium mobilization were investigated in the present st

294 iptional changes indicative of intracellular calcium mobilization were significantly overrepresented

295 Agonists that elicit distinct modes of calcium mobilization were used.

296 ed [3H]inositol phosphate (IP) formation and calcium mobilization with EC50 values of 8.5 nM and 0.8

297 19 and CCL21 induce G protein activation and calcium mobilization with equal potency.

298 er-1177, whereas inhibition of intracellular calcium mobilization with TMB-8 blunted Thr-495 dephosph

299 ibit platelet accumulation resulted in rapid calcium mobilization within the endothelium.

300 its IP(3) (inositol-1,4,5-trisphosphate) and calcium mobilization, without inhibiting Ang II binding