コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 channel KCNN2 was similarly regulated by the calcium pump.
2 monomers, the active/inhibitory units of the calcium pump.
3 smic reticulum (ER) Ca2+-ATPase (SERCA)-type calcium pump.
4 cilitating recognition and inhibition of the calcium pump.
5 ing a sarco/endoplasmic reticulum (ER)-Golgi calcium pump.
6 form of a human erythrocyte plasma membrane calcium pump.
7 and inhibition of the sarcoplasmic reticulum calcium-pump.
8 s able to rescue a yeast mutant deficient in calcium pumps.
9 regional and cellular distribution of these calcium pumps.
10 spine neck, and (iii) the strength of spine calcium pumps.
11 , which harbors a deletion of its endogenous calcium pumps.
12 iffusion through the spine neck and by spine calcium pumps.
13 lcium sensitivity and sarcoplasmic reticulum calcium pumping.
15 , these results support a model in which the calcium pump activity of ACA12 is primarily regulated by
16 through direct uncoupling of the ATPase and calcium pump activity of SERCA, resulting in the energy
17 ffness may increase light-scattering, reduce calcium pump activity, alter protein-lipid interactions,
19 ty and marked inhibition of the sarcoplasmic calcium pump affinity for calcium, resulting in depresse
20 a previously unknown lipid modulator of the calcium pump, an effect that may predispose kidney tissu
21 t to be found in a mammalian plasma membrane calcium pump and define a new class of deafness genes th
23 ding of Mg(2+) before P(i) implies that both calcium pump and sodium pump form a ternary enzyme.metal
24 of endurance: enforced cycles of presynaptic calcium pumping and, separately, enforced body-wall cont
25 calmodulin (CaM) can affect the activity of calcium pumps and channels to modulate the amplitude and
26 ical model, we find that the contribution of calcium pumps and diffusion varies from spine to spine.
27 and U-73122 are inhibitors of plant type IIA calcium pumps and phospholipase C, respectively, and imp
29 dies have aimed to explain how it influences calcium pumps and to determine whether it acts as an ion
30 rative Ca2+ extrusion by the plasma membrane calcium pump, and chloride efflux by the Jacobs-Steward
31 uxes via the calcium current (I(Ca)), the SR calcium pump, and passive leak from the SR were evaluate
32 r phosphorylation and dephosphorylation, the calcium pump appears to catalyze phosphoryl group transf
34 codes the sarcoplasmic/endoplasmic reticulum calcium-pumping ATPase SERCA2, which has a central role
36 n, an inhibitor of the endoplasmic reticulum calcium pumps, but buffering cytosolic calcium had no ef
37 integral membrane protein that regulates the calcium pump (Ca-ATPase) in cardiac sarcoplasmic reticul
41 TP-sensitive K(+) current, a plasma membrane calcium-pump current and a Na(+) background current.
42 f the circulatory lifespan was determined by calcium pump decay whereas late density reversal was sha
43 ing immunogold cytochemistry and showed this calcium pumping enzyme to be present throughout the subs
45 pproximately 6-fold), and the probability of calcium pump forming phosphoenzyme from bound P(i) (P(c)
48 ulin (ALN) are reported to bind to the SERCA calcium pump in a manner similar to that of known regula
49 Having demonstrated the efficiency of the calcium pump in background electrolyte solutions, a comp
51 ects of the fractions on the plasma membrane calcium pump in human red blood cells were assessed.
52 oplasmic-endoplasmic reticulum Ca(2+)-ATPase calcium pump in mammals and is of industrial importance
54 tructural demonstration of abundant sites of calcium pumps in the SSC supports a role for this struct
55 amino acid protein, regulates the Ca-ATPase (calcium pump) in cardiac sarcoplasmic reticulum (SR) thr
56 l membrane protein, regulates the Ca-ATPase (calcium pump) in cardiac sarcoplasmic reticulum through
57 lear differences between some members of the calcium pumps indicate that evolutionarily conserved clu
58 d (10-30 microM CPA), an inhibitor of the ER calcium pump, inhibited the calcium rise produced by TMA
64 onclude that the reverse flux through the SR calcium pump is an important factor in comprehensive und
65 biochemical analyses revealed that the PMCA2 calcium pump is highly up-regulated in CCs and CCTs in m
66 t hippocampus, we identified plasma membrane calcium pump isoform 1 (PMCA1), a plasma membrane calciu
67 Alternate splicing of human plasma membrane calcium pump isoform 4 (hPMCA4) transcripts causes the e
68 a and b variants of the rat plasma membrane calcium pump, isoform 2 (rPMCA2a and rPMCA2b), were cons
69 lasmic reticulum calcium ATPase] and SERCA2a calcium pump isoforms by phospholamban (PLB), we quantif
70 intact myocytes both forward and reverse SR calcium pump kinetics as well as intra-SR calcium buffer
73 increasing evidence placing the sarcolemmal calcium pump, or plasma membrane calcium/calmodulin ATPa
76 sion at the protein level of plasma membrane calcium pump (PMCA) isoforms in rat brain was detected b
78 f this work was to study the plasma membrane calcium pump (PMCA) reaction cycle by characterizing con
79 ulatory factor 1 (NHERF1) interacts with the calcium pump PMCA2 and the tyrosine kinase receptor ErbB
80 unctional variant (V586M) in plasma-membrane calcium pump PMCA2, which is encoded by ATP2B2, was asso
81 colleagues demonstrate that inhibiting SERCA calcium pumps preferentially impairs the maturation of t
82 ul effects of perturbations in plasma and ER calcium pump rates and the current through the CRAC chan
84 on, sitagliptin preserves function of the ER calcium pump, sarco-endoplasmic reticulum Ca(2+)-ATPase
86 ion with cellular partners, including the ER calcium pump sarcoplasmic/endoplasmic reticulum calcium
87 d molecular dynamics (MD) simulations of the calcium pump (sarcoplasmic reticulum Ca(2+)-ATPase (SERC
88 on (FDA)-approved drugs interacting with the calcium pump (Sarcoplasmic reticulum Ca(2+)-ATPase, SERC
89 -poor ER membrane inhibits the macrophage ER calcium pump, sarcoplasmic-endoplasmic reticulum calcium
90 host Golgi compartment-resident ATP-powered calcium pump (secretory pathway calcium ATPase 1 [SPCA1]
92 nterest such as adenylate kinase, ATP-driven calcium pump SERCA, leucine transporter and glutamate tr
94 acutely stimulate the sarcoplasmic reticulum calcium pump (SERCA) by relieving its inhibition by PLN.
95 ial cells modulates ER store by upregulating calcium pump (SERCA) expression without affecting the re
98 duced activity of the sarcoplasmic reticulum calcium pump (SERCA), contribute to this contractile dys
99 n cytoplasmic calcium through an ER-resident calcium pump, SERCA, and a calcium-activated ion channel
101 h Hif-1alpha-dependent overexpression of the calcium pump SERCA2 (sarcoplasmic/endoplasmic reticulum
103 from mutations in the endoplasmic reticulum calcium pump, SERCA2, and the Golgi calcium/manganese pu
104 , PLN plays a crucial role in regulating the calcium pump SERCA2a (sarco[endo]plasmic reticulum calci
105 nvestigated whether overexpression of the SR calcium pump (SERCA2a) in transgenic mice could reduce t
106 show that the thioredoxin TMX1 inhibits the calcium pump SERCA2b at ER-mitochondria contact sites, t
108 function, our data suggest that PMCA1a is a calcium pump specialized for neurons, where it may contr
109 very strong inhibitor of the red blood cell calcium pump, suggesting that this substance may have re
110 ntained a powerful specific inhibitor of the calcium pump that had little or no effect on the Na+/K+-
111 eplete albumin, secreted an inhibitor of the calcium pump that migrated in the same chromatographic b
112 lasma membrane calcium ATPase 2 (PMCA2) is a calcium pump that plays important roles in neuronal func
114 s correlates with the higher expression of a calcium pump, the gene for which was cloned and sequence
116 latory protein phospholamban can inhibit the calcium pump, we evaluated it as a potential target to i
117 cally activated by Na(+) or K(+); and in the calcium pump, where a niche is integrally involved in th
118 his work is that the coupling of a selective calcium pump with a thin layer element can give rise to
119 h phorbol myristate acetate or poisoning the calcium pump with thapsigargin stimulates transcytosis o
120 Ca2+ release; however, after inhibiting the calcium pump with thapsigargin the same stimulus release