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1  of the molecular and cellular mechanisms of calcium pyrophosphate dihydrate and apatite crystal form
2 the pathologic matrix mineralization seen in calcium pyrophosphate dihydrate and basic calcium phosph
3  the cellular responses to monosodium urate, calcium pyrophosphate dihydrate and basic calcium phosph
4 The pathologic matrix mineralization seen in calcium pyrophosphate dihydrate and basic calcium phosph
5                  Calcium crystals, including calcium pyrophosphate dihydrate and basic calcium phosph
6                                              Calcium pyrophosphate dihydrate and basic calcium phosph
7 concerning clinical and etiologic aspects of calcium pyrophosphate dihydrate and basic calcium phosph
8 ystals of calcium oxalate, monosodium urate, calcium pyrophosphate dihydrate and cystine trigger casp
9 ved in the pathogenesis of monosodium urate, calcium pyrophosphate dihydrate and hydroxyapatite cryst
10             In addition to monosodium urate, calcium pyrophosphate dihydrate, and apatite crystals, a
11 als of calcium oxalate, monosodium urate, or calcium pyrophosphate dihydrate, as well as silica micro
12                                              Calcium pyrophosphate dihydrate (CPPD) and basic calcium
13        Calcium deposition diseases caused by calcium pyrophosphate dihydrate (CPPD) and basic calcium
14                             Microcrystals of calcium pyrophosphate dihydrate (CPPD) and monosodium ur
15                  Familial autosomal dominant calcium pyrophosphate dihydrate (CPPD) chondrocalcinosis
16 tion, and increased concentrations promoting calcium pyrophosphate dihydrate (CPPD) crystal depositio
17                                              Calcium pyrophosphate dihydrate (CPPD) crystal depositio
18  characterization of the role of NTPPHase in calcium pyrophosphate dihydrate (CPPD) crystal depositio
19 he pathologic mineralization that results in calcium pyrophosphate dihydrate (CPPD) crystal formation
20 In this study, following our earlier work on calcium pyrophosphate dihydrate (CPPD) crystal-induced m
21       Monosodium urate monohydrate (MSU) and calcium pyrophosphate dihydrate (CPPD) crystals cause ac
22                             The formation of calcium pyrophosphate dihydrate (CPPD) crystals in artic
23 luding nanoscale silicon dioxide (NanoSiO2), calcium pyrophosphate dihydrate (CPPD) crystals, and mur
24  Articular calcium-containing crystals cause calcium pyrophosphate dihydrate (CPPD) deposition diseas
25 of fluid containing synthetic or native BCP, calcium pyrophosphate dihydrate (CPPD), or monosodium ur
26 ed spectroscopy revealed crystals resembling calcium pyrophosphate dihydrate (CPPD).
27 of ePPi while excess levels of ePPi leads to calcium pyrophosphate dihydrate crystal deposition (CPPD
28  load is a likely source of pyrophosphate in calcium pyrophosphate dihydrate crystal deposition disea
29                                  In familial calcium pyrophosphate dihydrate crystal deposition disea
30 hic arthropathy of the atlantoaxial joint in calcium pyrophosphate dihydrate crystal deposition disea
31 c pyrophosphate in cartilage matrix leads to calcium pyrophosphate dihydrate crystal deposits.
32                            Current models of calcium pyrophosphate dihydrate crystal formation are le
33 view, the author discusses various models of calcium pyrophosphate dihydrate crystal formation from e
34 l for studying the major factors involved in calcium pyrophosphate dihydrate crystal formation.
35                                              Calcium pyrophosphate dihydrate crystals are common comp
36        Progress in understanding why and how calcium pyrophosphate dihydrate crystals form in articul
37 young animals might promote the formation of calcium pyrophosphate dihydrate crystals in aged cartila
38                               Elimination of calcium pyrophosphate dihydrate crystals may occur extra
39                        Tumoral deposition of calcium pyrophosphate dihydrate crystals may occur in si
40                                The effect of calcium pyrophosphate dihydrate crystals on the progress
41                                  Addition of calcium pyrophosphate dihydrate crystals to a lapine men
42 osition of either basic calcium phosphate or calcium pyrophosphate dihydrate crystals, remains unclea
43 sphate (PPi) that promotes the deposition of calcium pyrophosphate dihydrate crystals.
44         A discussion of ANKH as the familial calcium pyrophosphate dihydrate deposition disease gene
45  radiographic techniques to the diagnosis of calcium pyrophosphate dihydrate deposition disease holds
46 nt as a definitive rheumatic disease such as calcium pyrophosphate dihydrate deposition disease or as
47 nts were identified that segregated with the calcium pyrophosphate dihydrate deposition disease pheno
48 t literature reminds us of the propensity of calcium pyrophosphate dihydrate deposition disease to mi
49               In genetic studies of familial calcium pyrophosphate dihydrate deposition disease, a re
50 as a potential positional candidate gene for calcium pyrophosphate dihydrate deposition disease, and
51 rs: craniometaphyseal dysplasia and familial calcium pyrophosphate dihydrate deposition disease.
52 evalence and significance of extra-articular calcium pyrophosphate dihydrate deposits, and demonstrat
53 play radiographically detectable crystals of calcium pyrophosphate dihydrate in their joint spaces.
54 lysis supports the role of ANKH mutations in calcium pyrophosphate dihydrate-induced arthritis.
55 ATD5 cells were basic calcium phosphate, not calcium pyrophosphate dihydrate, underlying the signific