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1 ing dialysis have been reported to attenuate calcium receptor activation and modify parathyroid hormo
2 t the idea that hyperphosphatemia attenuates calcium receptor activation by calcium ions and by calci
3 y of single oral doses of 4 to 160 mg of the calcium-receptor agonist drug R-568 in 20 postmenopausal
4 ingle agent to rats, the previously reported calcium receptor antagonist 3 elicited a sustained eleva
6 We then demonstrated the existence of the calcium receptor (CaR) in keratinocytes and determined t
7 nd we have used in situ hybridization to map calcium receptor (CaR) mRNA expression in the adult rat
9 eecham compound collection against the human calcium receptor (CaR) resulted in the identification of
10 may be modulated by local expression of the calcium receptor (CaR), the vitamin D receptor (VDR), an
11 ynaptic scaling is mediated by GluA2-lacking calcium receptors (CP-AMPARs), whereas others have found
13 the extracellular domain (ECD) of the human calcium receptor (hCaR) from the medium of HEK-293 cells
15 (1075) in the carboxyl terminus of the human calcium receptor (hCaR), which causes autosomal dominant
16 residue cytoplasmic carboxyl terminus of the calcium receptor in cell-surface expression and signal t
17 osteoblastic niche, such as quiescence, and calcium receptor, N-cadherin, Notch1, and p21 are higher
19 f the interactions between the intracellular calcium receptor protein calmodulin (CaM) and one of its