戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 aining (2 mM, EC(50) = 3.49 +/- 0.77 muM) or calcium free (0 mM, EC(50) = 9.5 +/- 1.5 muM) buffers.
2 ibitor-bound furin; and the respective (iii) calcium-free and (iv) calcium-bound forms.
3  a stable, noncovalent homodimer in both its calcium-free and -bound states.
4 iously reported CaM structures and resembles calcium-free apo-calmodulin (apo-CaM), despite the zinc
5                   Structural comparison with calcium-free apo-CaM, calcium-loaded CaM, and a cross-li
6                                         When calcium-free artificial cerebrospinal fluid was used as
7 an alpha7-nAChR subunit-specific antagonist, calcium-free artificial cerebrospinal fluid, or a cockta
8 ited significantly when the concentration of calcium-free Ator exceeded that of Abeta by at least a f
9 on-transfer difference NMR data suggest that calcium-free Ator exerts its effect through interaction
10 e Premier Perspective Database, the use of a calcium-free balanced crystalloid for replacement of flu
11 ation with 0.9% NaCl versus Plasma-Lyte A, a calcium-free balanced crystalloid solution, hypothesizin
12 pplications in calcium containing but not in calcium free buffer.
13       The plateau potential was abolished in calcium-free buffer, as well as by nickel or cadmium.
14                                           In calcium-free buffer, there was no difference in the ATP-
15                  In the UK-BB, serum ionized calcium (free calcium, most active form) was inversely a
16 ation dispersion for the nine methionines in calcium-free calmodulin (apo-CaM).
17                           Under low calcium, calcium-free calmodulin 2 (Apo-CaM2) interacts with CNGC
18  studies from our laboratory have shown that calcium-free calmodulin and calcium bound calmodulin pro
19  Our data suggest that the binding sites for calcium-free calmodulin and calcium-bound calmodulin are
20                 We now demonstrate that both calcium-free calmodulin and calcium-bound calmodulin bin
21 channel, ryanodine receptor, is activated by calcium-free calmodulin and inhibited by calcium-bound c
22  destroys the ability of the peptide to bind calcium-free calmodulin, but not calcium-bound calmoduli
23                Dialysis anticoagulation with calcium-free citrate-containing dialysate and calcium re
24 icoagulation of the dialysis circuit using a calcium-free citrate-containing dialysate, with calcium
25                                 Furthermore, calcium-free conditions in the absence of anti-Fas antib
26 e effect of hypoxia on MKP-1 persisted under calcium-free conditions.
27 ium-sensing dyes under calcium-saturated and calcium-free conditions.
28 from kinetic separation of calcium-bound and calcium-free cross-bridge pools.
29 ctroscopic characterization of GCaMP2 in the calcium-free dark state, and in two calcium-bound bright
30 ated stimulation with chemical agonists in a calcium-free environment and this sensitisation is very
31  XE991 did not enhance mEPSCs frequency in a calcium-free external medium.
32                                    Nominally calcium-free external solution immediately and reversibl
33 gnals could still be triggered by TLC-S in a calcium-free external solution.
34 ted by exposure to thapsigargin/ionomycin in calcium-free external solution.
35 o statistically significant difference among calcium-free external solutions containing different imp
36 ese oscillations were abolished in nominally calcium-free extracellular medium and in 1 muM tetrodoto
37                           Axonal injury in a calcium-free extracellular solution resulted in no chang
38 anisms of spontaneous neuronal bursting in a calcium-free extracellular solution.
39  a higher three-photon cross-section for the calcium-free form of Indo-1 than for the calcium-bound f
40 s the solution NMR structure of the inactive calcium-free form of the protein.
41 s the conformational entropy observed in the calcium-free form.
42 solin by releasing the tail latch that locks calcium-free gelsolin in a conformation unable to bind a
43           The tightly packed architecture of calcium-free gelsolin, seen from both SAXS and x-ray cry
44 rapeutic advances include the development of calcium-free intestinal phosphate binders, calcimimetics
45 incubated on ever-decreasing volumes of cold calcium-free KCl closed on the lowest volume (0.2 cm(3))
46 l-like calcium-binding linker, and an EC9-10 calcium-free linker that alters the linear arrangement o
47     Continued incubation of 5AHSmyc cells in calcium-free media induced substantial apoptotic DNA fra
48 en regulated release was prevented by use of calcium-free media, glucose-stimulated IAPP-LI release w
49     TCR-mediated apoptosis can be induced in calcium-free media, indicating that extracellular calciu
50 on of intracellular calcium did not occur in calcium-free media, indicating that the increase in intr
51 n regulated secretion is prevented by use of calcium-free media, suggesting that IAPP secretion occur
52 ed to prevent spontaneous store depletion in calcium-free media.
53 ce of intracellular calcium chelators and in calcium-free media.
54               The reactions also occurred in calcium-free media.
55 ation of calcium to retinal slices bathed in calcium-free media.
56  or if the mild irritant was administered in calcium-free media.
57                    Incubation of arteries in calcium-free medium almost abolished the synergistic eff
58                                              Calcium-free medium blocked not only the increase in AA
59                           Culturing cells in calcium-free medium or with the protein kinase Cdelta in
60 oxia or 1-h pretreatment of enterocytes with calcium-free medium resulted in increased internalizatio
61 in vitro in control medium or in a nominally calcium-free medium with high magnesium, glutamate recep
62  calcium channel blockers and/or a nominally calcium-free medium with high magnesium.
63                       In SMC pretreated with calcium-free medium, baseline [Ca2+]i fell by about 60 n
64  using a dual emission microfluorometry in a calcium-free medium, that the 17beta-estradiol-stimulate
65    Using dual emission microfluorometry in a calcium-free medium, the 17beta-estradiol-stimulated rel
66  activation, we used diluted Sendai virus in calcium-free medium.
67 (52 +/- 6 to 297 +/- 26 nM, P: < 0.001) in a calcium-free medium.
68 eport a helical reconstruction of TMV in its calcium-free, metastable assembling state at 3.3 A resol
69                               Simulations of calcium-free monomeric OMPLA, of the Ca(2+)-bound dimer,
70  Here we report the crystal structure of the calcium-free N-terminal half of adseverin (iA1-A3) and t
71 odimers, suggesting that S100A4 can exist as calcium-free or calcium-bound dimers in vivo.
72               A modified cocktail of CDCs in calcium-free PBS, 100 U/mL of heparin, and 250 microg/mL
73                 Although TTX (1 microm) or a calcium-free perfusate both caused reductions in the pow
74 rhythm was further reduced on combination of calcium-free perfusate with octanol (1 mM) and was aboli
75 DA mitochondria were not dependent upon ATP, calcium, free radical species, JNK, or caspase3/PKC path
76 ough the toxin is applied and washed away in calcium-free saline.
77 f SERCA, known for decades as an assembly of calcium-free SERCA molecules induced by the addition of
78 ter H. capsulatum yeasts were subjected to a calcium-free shock, exogenously added CBP allowed yeasts
79 previous replica-exchange molecular dynamics calcium-free simulations as a control, we reached three
80                         We show that dimeric calcium-free sNUCB1 binds to expressed Galpha(i1) and th
81 the present experiments demonstrated that in calcium-free solution (magnesium replacement) zinc can p
82 mitter release has been reported to occur in calcium-free solution and has been attributed either to
83                    The response persisted in calcium-free solution and when nickel chloride (4 mM) wa
84       Also, after prestimulation by AngII, a calcium-free solution completely reversed the effects of
85 release as assayed by spontaneous release in calcium-free solution or by the application of hyperosmo
86  noradrenaline and blocked by perfusion with calcium-free solution.
87  synaptic transmission globally using TTX or calcium-free solutions led to a 40-120% increase in the
88 show that hemichannels open upon exposure to calcium-free solutions when they are either heterologous
89  reduced by tetrodotoxin and were blocked in calcium-free solutions.
90 east 10 min after washout of the compound in calcium-free solutions.
91  spontaneous bursting behavior of neurons in calcium-free solutions.
92 -dimensional structures are available in the calcium-free state (e.g., S100B and S100A1).
93        Here we present the calcium-bound and calcium-free structures of the N- and C-terminal domains
94 elected mutations increased the half-life of calcium-free subtilisin at elevated temperature by 15,00
95 hich is independently stable (proR9) and two calcium-free subtilisin mutants.
96 ially randomizing 12 amino acid positions in calcium-free subtilisin.
97                                Assuming that calcium-free SYT rings are physiologically relevant, the
98                      Treatment for 10 d with calcium-free TPN restored the nocturnal rise in serum PT
99 when introduced into subtilisin BPN' and two calcium-free versions of subtilisin.