ライフサイエンスコーパス: callosum

1 e matter of cerebrum, cerebellum, and corpus callosum).

2 l-occipital fasciculus, cingulum, and corpus callosum).

3 pus callosum, and fiber strain of the corpus callosum.

4 s, ponto-cerebellar atrophy, and thin corpus callosum.

5 was associated with hypoplasia of the corpus callosum.

6 vered by 21 d post MCAO in KI mice in corpus callosum.

7 sal cingulum bundle above genu of the corpus callosum.

8 al connectivity in anterior/posterior corpus callosum.

9 ter organization, particularly in the corpus callosum.

10 FA, MD, RD, and AD), primarily in the corpus callosum.

11 appeared progressive, and a prominent corpus callosum.

12 measures of the corticospinal tract and mid-callosum.

13 ic commissural bridges traversing the corpus callosum.

14 ntal white matter and the genu of the corpus callosum.

15 iled to cross the midline to form the corpus callosum.

16 P < .05) in posterior portions of the corpus callosum.

17 ncrease in unmyelinated fibers in the corpus callosum.

18 the superior cerebellar peduncle and corpus callosum.

19 tion of the axonal projections in the corpus callosum.

20 ntation within white matter, e.g. the corpus callosum.

21 hemispheric communication through the corpus callosum.

22 llar vermis and lobules V and VI, and corpus callosum.

23 rizone, and OPCs were sorted from the corpus callosum.

24 EDF was capable of infiltration along corpus callosum.

25 related to myelination and increased corpus callosum.

26 predominantly in the splenium of the corpus callosum.

27 ant axonal bundles within the rostral corpus callosum.

28 ed numbers of myelinated axons in the corpus callosum.

29 ardation (MR) and malformation of the corpus callosum.

30 rhemispheric connecting fibers of the corpus callosum.

31 ental retardation and agenesis of the corpus callosum.

32 us, as well as in the splenium of the corpus callosum.

33 associated with the evolution of the corpus callosum.

34 myelinating injury to the adult mouse corpus callosum.

35 internal capsule, and splenium of the corpus callosum.

36 patient had a cytotoxic lesion of the corpus callosum.

37 lateral eye to this region comes through the callosum.

38 thinning of the myelin sheath in the corpus callosum.

39 xonal projections to the internal capsule or callosum.

40 rain anomalies, including agenesis of corpus callosum.

41 igodendrocyte lineage cells (OLCs) in corpus callosum.

42 s had abnormalities in the cortex and corpus callosum.

43 : 0.05 ug . g(-1) +/- 0.01, P = .002; corpus callosum: 0.05 ug . g(-1) +/- 0.02, P = .001; cranial ne

44 ology and with a predilection for the corpus callosum (23 of 39; 59%; 95% CI: 42, 74) and juxtacortic

45 sistent with cytotoxic lesions of the corpus callosum (4.1%).

46 eved with FA index measurement of the corpus callosum (51%).

47 such as ventriculomegaly and a small corpus callosum (67%).

48 atures on neuroimaging include a thin corpus callosum (90%), ventriculomegaly (65%) often with colpoc

49 pecifies neuronal projections via the corpus callosum, a large axon tract connecting the two neocorti

50 included intracranial calcifications, corpus callosum abnormalities, abnormal cortical formation, cer

51 viduals with isolated agenesis of the corpus callosum (ACC) without intellectual disability.

52 in the structural organization of the corpus callosum accounts for these differences.

53 or protein aggregates in axons of the corpus callosum after traumatic brain injury as compared to Sar

54 25 participants with agenesis of the corpus callosum (AgCC) and 21 matched neurotypical individuals.

55 ance in patients with agenesis of the corpus callosum (AgCC) and found reduced laterality (i.e., grea

56 tigated the effect of agenesis of the corpus callosum (AgCC), one of the most common brain malformati

57 d with brain malformations, including corpus callosum agenesis (ACC) and microcephaly.

58 delay and/or intellectual disability, corpus callosum agenesis or hypoplasia, flexion contractures, b

59 y, variable axon pathfinding defects (corpus callosum agenesis or hypoplasia, mirror movements, Duane

60 s, such as autism spectrum disorders, corpus callosum agenesis, Joubert syndrome, Kallmann syndrome,

61 isorder, intellectual disability, and corpus callosum agenesis.

62 l cases exhibited partial or complete corpus callosum agenesis.

63 provide support for the hypothesis that the callosum aids in functional specialization throughout ne

64 ent in frontal and temporal lobes and corpus callosum (all p values <0.01).

65 However, it is unclear whether corpus callosum alterations are related to the underlying famil

66 Except for the genu of the corpus callosum, an increase in AD values was also found in the

67 ion anisotropy of the splenium of the corpus callosum and adjacent parietal white matter (P < .05).

68 recessive inheritance pattern of thin corpus callosum and axonal Charcot-Marie-Tooth disease in three

69 reditary spastic paraplegia with thin corpus callosum and axonal peripheral neuropathy (SPG7/PGN, SPG

70 d MD in more extended portions of the corpus callosum and beyond (eg, corona radiata and inferior lon

71 eurons, but strongly expressed in the corpus callosum and caudate axon fibers.

72 r FD in numerous tracts including the corpus callosum and corona radiata compared to mature-born adul

73 gest differences were observed in the corpus callosum and corona radiata.

74 , and 3) fractional anisotropy in the corpus callosum and corona radiata.

75 a1 was significantly decreased in the corpus callosum and cortex of Cav1.2 knock-out mice through dem

76 hood onset cognitive impairment, thin corpus callosum and enlarged ventricles.

77 ament NF200, the reduced thickness of corpus callosum and external capsule, and decline of mature oli

78 ts, which were most pronounced in the corpus callosum and external capsule.

79 te matter injury was prominent in the corpus callosum and internal capsule on day 3 and then partiall

80 We examined the corpus callosum and its subregion volumes and their relationshi

81 ced number of myelinated axons in the corpus callosum and optic nerves.

82 amage to the genu and splenium of the corpus callosum and parahippocampal tract bilaterally (P < .05)

83 reditary spastic paraplegia with thin corpus callosum and peripheral axonal neuropathy, and account f

84 attenuated white matter growth of the corpus callosum and pons relative to nondrinkers.

85 isotropy is reduced in Plp-Nf1 (fl/+) corpus callosum and that interhemispheric functional connectivi

86 ion of the graft-derived axons in the corpus callosum and that their terminals form excitatory, gluta

87 r mean diffusivity in the genu of the corpus callosum and the anterior thalamic tracts.

88 at altered WM connectivity within the corpus callosum and the cingulum are strongly associated with B

89 the extensive regions of the CST, the corpus callosum and the right PCG.

90 teins regulating the formation of the corpus callosum and their respective developmental functions.

91 d improved overall myelination in the corpus callosum and white matter tracts of the spinal cord.

92 o white matter tracts, especially the corpus callosum, and at a low level in neurons, while PI5P4Kbet

93 the hippocampus, degeneration of the corpus callosum, and ataxia and seizures.

94 not Nova1-/- mice had agenesis of the corpus callosum, and axonal outgrowth defects specific to ventr

95 sent with spastic paraparesis, a thin corpus callosum, and cognitive impairment.

96 developmental delay, agenesis of the corpus callosum, and enlargement of the third cerebral ventricl

97 (MPS) of the whole brain, MPS of the corpus callosum, and fiber strain of the corpus callosum.

98 rus, cortex of the temporal lobes and corpus callosum, and fractional anisotropy (FA) index measureme

99 lic commissures (anterior commissure, corpus callosum, and hippocampal commissure) along with an enla

100 telencephalic commissures (anterior, corpus callosum, and hippocampal), an enlarged posterior commis

101 stage around the striatum, within the corpus callosum, and in posterior white matter tracts.

102 y higher in the medial cortex, in the corpus callosum, and in the thalamus than in the corresponding

103 onnect auditory and motor structures, corpus callosum, and in tracts interconnecting cortical and sub

104 normalities, cytotoxic lesions of the corpus callosum, and intensive care unit-related complications,

105 s residing in secondary motor cortex, corpus callosum, and primary somatosensory cortex of adult mice

106 es in astrocytes, particularly in the corpus callosum, and provide support for hypotheses that focus

107 utant BTBR mouse brain, which lacks a corpus callosum, and recapitulated its known connectopathies.

108 developmental delay, thinning of the corpus callosum, and seizures.

109 lformation (microcephaly, agenesis of corpus callosum, and simplified gyration), and severe encephalo

110 nodes of Ranvier in the optic nerve, corpus callosum, and spinal cord of young adult mice or rats.

111 he CNS, including in the optic nerve, corpus callosum, and the spinal cord.

112 entate gyrus, partial agenesis of the corpus callosum, and ventriculomegaly.

113 stem (white matter disturbances, thin corpus callosum, and widened ventricles); global delay with sig

114 individuals included agenesis of the corpus callosum, ano-rectal malformations, seizures, and hearin

115 normally proportioned cerebellum, and corpus callosum anomalies.

116 DystoniaNet identified clusters in corpus callosum, anterior and posterior thalamic radiations, in

117 s included genu, body and splenium of corpus callosum, anterior and superior corona radiata, superior

118 s in the neocortex results in lack of corpus callosum, anterior commissure, and corticospinal tract f

119 and MPF in all anatomical structures (corpus callosum, anterior commissure, internal capsule, thalamu

120 new astrocytes from the SVZ into the corpus callosum appears to be balanced by astroglial apoptosis,

121 Brain malformations involving the corpus callosum are common in children with developmental disab

122 We found significantly increased corpus callosum area and thickness in children with autism spec

123 ctional anisotropy alterations in the corpus callosum) as a shared feature of ASD, ADHD, and OCD.

124 ippocampus hypoplasia and agenesis of corpus callosum, as well as neuromuscular and behavioral altera

125 fate-mapping, we demonstrate that new corpus callosum astrocytes are continuously generated from nest

126 These nestin fate-mapped corpus callosum astrocytes are uniformly postmitotic, express g

127 apoptosis, because overall numbers of corpus callosum astrocytes remain constant during normal adulth

128 e particularly robust in the anterior corpus callosum at the 6 and 12 month time points.

129 d be named ACOG syndrome (agenesis of corpus callosum, axon pathfinding, cardiac, ocular, and genital

130 terminalis, genu and splenium of the corpus callosum, bilateral anterior and posterior limbs of inte

131 d white matter areas (splenium of the corpus callosum, bilateral superior-parietal lobe, bilateral an

132 Corpus callosum bisections demonstrated that premotor cortex he

133 diet induces rapid loss of OL in the corpus callosum by 2 d, accompanied by expression of several ma

134 during the remyelination phase to the corpus callosum (CC) and are capable of forming new oligodendro

135 Lesions in the corpus callosum (CC) are important radiological clues to the di

136 ort white matter abnormalities of the corpus callosum (CC) as an important predictor of neurodevelopm

137 cells essential for interhemispheric corpus callosum (CC) axon navigation.

138 The corpus callosum (cc) contains nitric oxide (NO)-producing neuro

139 esponses to these unseen stimuli, the corpus callosum (CC) dynamically recruited areas in the visual

140 Interhemispheric axons of the corpus callosum (CC) facilitate the higher order functions of t

141 While microstructural alterations in corpus callosum (CC) have been identified as a consistent featu

142 ght cingulum hippocampus and anterior corpus callosum (CC) in aMCI compared to HC.

143 The corpus callosum (CC) is one of the most commonly reported areas

144 The corpus callosum (CC) is the largest fiber tract in the mammalia

145 In this light, the corpus callosum (CC) may represent the main responsible for cro

146 rength from the intact cortex via the corpus callosum (CC) onto deep neurons in deprived primary soma

147 ols the formation of the layer II/III corpus callosum (CC) projections through the developmental tran

148 The corpus callosum (CC) represents a clinically-relevant long-rang

149 been often reported to have a smaller corpus callosum (CC) than control subjects.

150 dths of the frontal horn (FH) and the corpus callosum (CC) were not significantly different between t

151 perior longitudinal fasciculus (SLF), corpus callosum (CC), and corticospinal tract (CST).

152 D) and radial diffusivity (RD) in the corpus callosum (CC), superior longitudinal fasciculus (LF), co

153 the two cerebral hemispheres via the corpus callosum (CC).

154 ion, brain atrophy, dysgenesis of the corpus callosum, cerebellar vermis hypoplasia, and facial dysmo

155 sed volume and abnormal signal), thin corpus callosum, cerebellar vermis hypoplasia, optic nerve hypo

156 s of the brain, such as the amygdala, corpus callosum, cerebellum, and gyrnecephalic index, all indic

157 ant decrease in FA in the genu of the corpus callosum characterized both conditions.

158 wed white matter abnormalities in the corpus callosum, cingulum and external capsule, with differing

159 edium effect sizes, especially in the corpus callosum, cingulum and external capsule.

160 imaging abnormalities observed in the corpus callosum, cingulum, and temporal lobe likely constitute

161 opy (FA) in the tapetum region of the corpus callosum (Cohen's d = -0.11, p = 0.0055).

162 ite-matter abnormalities, hypoplastic corpus callosum, congenital heart defects, and central hypovent

163 ident in the anterior portions of the corpus callosum connecting left and right frontal lobes.

164 creased white matter integrity in the corpus callosum connecting these regions, suggesting an involve

165 restricted diffusion foci within the corpus callosum consistent with cytotoxic lesions of the corpus

166 acrostructural variability within the corpus callosum, consistent with differential effects on cross-

167 tions in fractional anisotropy in the corpus callosum, corona radiata and external capsule, and incre

168 M tracts, respectively, including the corpus callosum, corona radiata, superior longitudinal fascicul

169 veral brain structures, including the corpus callosum, cortex, and striatum, and the corpus callosum

170 sALS group, encompassing parts of the corpus callosum, corticospinal tracts and superior longitudinal

171 anterior corona radiata (d=0.40) and corpus callosum (d=0.39), specifically its body (d=0.39) and ge

172 Remarkably, all seven genes showed corpus callosum defects, including thicker (Atg16l1, Coro1c, Dm

173 poral lobe epilepsy, microcephaly and corpus callosum deficiency, and by postnatal Day 21, microcepha

174 ing of an axonal subpopulation of the corpus callosum derived from the anterior cingulate cortex.

175 The role that corpus callosum development has on the hemispheric specializati

176 identifying new proteins integral to corpus callosum development that will provide new insights into

177 sly-identified proteins in aspects of corpus callosum development, and identifies new candidates in u

178 y hypomyelination, microcephaly, thin corpus callosum, developmental delay, intellectual disability,

179 , but not ALSFRS-R, correlated with anterior callosum DTI measures.

180 lateral aspect of the truncus of the corpus callosum, due to greater increase in FA (standardized ef

181 ne the translatome of OLCs in vivo in corpus callosum during the remyelination phase of a chronic cup

182 be3b (-/-) mice had hypoplasia of the corpus callosum, enlarged ventricles, and decreased thickness o

183 The human corpus callosum exhibits substantial atrophy in old age, which

184 ructural integrity of the body of the corpus callosum (FA, beta = 0.01 [P = .01]; RD, beta = -0.02 [P

185 al atrophy of the dorsal cingulum and corpus callosum fibers, which we interpret as a consequence of

186 mispheric connectivity by controlling corpus callosum formation remains unclear.

187 damage that involved the body of the corpus callosum, fornix, and main anterior-posterior pathways (

188 Thus, the accentuated decline of the corpus callosum found in aging humans is not a universal charac

189 In slices of corpus callosum from mice subjected to a demyelination protocol

190 al area and regional thickness of the corpus callosum from T1-weighted MRI data from 213 chimpanzees,

191 s suggest novel dual mechanism of the corpus callosum function in spatial attention and have broader

192 First, whereas FA in anterior corpus callosum (genu) correlated with word-matching BPA, FA in

193 The corpus callosum has been implicated in the pathogenesis of schi

194 le (dorsomedial) lying closest to the corpus callosum has the most complete set of commissural connec

195 These findings suggest that the corpus callosum helps to drive language lateralization.

196 al abnormalities and the absence of a corpus callosum; his immune deficit was fully corrected by hema

197 howed subtle abnormalities, including corpus callosum hypoplasia and ventriculomegaly.

198 simplification of cerebral gyration, corpus callosum hypoplasia, and dysmorphic facial features, we

199 ovement disorders, behavior problems, corpus callosum hypoplasia, and epilepsy.

200 s, respectively; abnormalities of the corpus callosum in 16 of 17 (94%) and 22 of 28 (78%) infections

201 ore and after surgical section of the corpus callosum in 22 patients with medically refractory epilep

202 ttention scores were related to AD in corpus callosum in a cluster sized 716 voxels.

203 ndings suggest a notable role for the corpus callosum in maintaining stable functional communication

204 structure, supporting a role for the corpus callosum in mediating functional asymmetry.

205 posterior thalamic radiation and left corpus callosum in patients (all p < 0.05).

206 t brain injury in the hippocampus and corpus callosum in rats with vascular dementia.

207 here we investigated the role of the corpus callosum in the cortical spreading of NREM slow waves th

208 ults demonstrate a causal role of the corpus callosum in the cross-hemispheric traveling of sleep slo

209 terhemispheric connections within the corpus callosum, in particular parieto-parietal connections.

210 nect the cerebral hemispheres via the corpus callosum, integrating cortical information and playing k

211 ite matter (subcortical white matter, corpus callosum, internal capsule, anterior commissure), gray m

212 malities of the cerebellum, cingulum, corpus callosum, internal capsule, thalamus, basal forebrain, o

213 alities with sparing of the U fibers, corpus callosum involvement with sparing of the outer blades, a

214 ain imaging studies indicate that the corpus callosum is smaller in older children and adults with au

215 al tract is unique to mammals and the corpus callosum is unique to placental mammals (eutherians).

216 e even though it is richly innervated by the callosum (Laing, Turecek, Takahata, & Olavarria, 2015).

217 esults in abnormal development of the corpus callosum, lateral ventricles, and hippocampus.

218 FA values, including the splenium of corpus callosum, left posterior corona radiate/posterior thalam

219 The corpus callosum, longer WM tracts and areas that are more dense

220 interactions of glucose x FA in left corpus callosum, longitudinal fasciculus and corona radiata wer

221 Section of the corpus callosum markedly reduced interhemispheric functional co

222 adults, our findings suggest that the corpus callosum may be larger in infants who go on to develop a

223 gether, our findings suggest that the corpus callosum may have a dual inhibitory and excitatory funct

224 y comparing age-related difference in corpus callosum morphology of chimpanzees and humans.

225 n these hippocampal subfields and the corpus callosum, novel findings that would have been difficult

226 ly within the basal ganglia, thalami, corpus callosum, occipital, temporal, parietal and frontal lobe

227 py data from the anterior body of the corpus callosum of 13 patients with systemic lupus erythematosu

228 ages of white matter samples from the corpus callosum of a monkey brain reveal that blood vessels, ce

229 cal uptake was found in the posterior corpus callosum of a TBI subject.

230 of myelin, were also detected in the corpus callosum of adult HdhQ250 mice.

231 elination phase of the CPZ model, the corpus callosum of Cav1.2(KO) animals presented a significant d

232 llosum, cortex, and striatum, and the corpus callosum of Cav1.2(KO) animals showed an important decre

233 Furthermore, the corpus callosum of Fth KO animals presented a significant decre

234 ate nucleus, subthalamic nucleus, and corpus callosum of multiple system atrophy, and in all regions

235 differentiation were impaired in the corpus callosum of Olig1-null mice, resulting in hypomyelinatio

236 ng of astrocytes, particularly in the corpus callosum of the Gnptab Ser321Gly homozygote mice compare

237 imaging also detected deficits in the corpus callosum of the Gnptab Ser321Gly mice.

238 t loss of OLIG2-positive cells in the corpus callosum of Tmem106b-/- mice, which was present already

239 l gyri, corticospinal tracts, and the corpus callosum) of participants with ALS (two-sample t test, P

240 athy, cognitive impairment and a thin corpus callosum on brain MRI.

241 rounding the anterior crossing of the corpus callosum on E18 as well as the persistence of large numb

242 e interaction of glucose x FA in left corpus callosum, or in longitudinal fasciculus was associated w

243 evant to limited brain areas like the corpus callosum, or multiple orientations but without the abili

244 al ganglia (caudate nucleus, putamen, corpus callosum, posterior limb of internal capsule), level of

245 regions, and lower FA in the body of corpus callosum, posterior superior longitudinal fasciculus and

246 ional connectivity in relation to the corpus callosum presents a case in point.

247 d action potentials in the myelinated corpus callosum projections of Msh2-null mice were smaller than

248 The volumetric assessment of the corpus callosum proved to be a useful tool in discriminating be

249 me subcortical regions, including the corpus callosum, putamen, and cerebellum.

250 hest effect sizes observed within the corpus callosum (R(2) = 0.041, P(corr) < 0.001) and cingulum (r

251 eurons of the corticospinal tract and corpus callosum, respectively.

252 .4% and 203.0% in the hippocampus and corpus callosum, respectively.

253 IFOF), genu (GCC) and splenium of the corpus callosum (SCC), posterior limbs of the internal capsules

254 ctural differences in the body of the corpus callosum; schizophrenia and bipolar disorder featured co

255 pathy with or without agenesis of the corpus callosum (SLC12A6).

256 iori regions of interest: splenium of corpus callosum (SPCC) and posterior limb of internal capsule (

257 th word-matching BPA, FA in posterior corpus callosum (splenium-occipital) correlated with face-match

258 radiata, right tapetum, and bilateral corpus callosum, statistically moderates whether sleep spindles

259 cellular degeneration between cortex, corpus callosum, striatum, globus pallidus, and thalamus after

260 (by measuring the volume of thalamus, corpus callosum, subcortical nuclei, hippocampus) as parameters

261 brain areas, including left and right corpus callosum, superior longitudinal fasciculus, posterior th

262 -thalamic fibers: the corona radiata, corpus callosum, superior longitudinal fasciculus, posterior th

263 trogliosis significantly increased in corpus callosum (TBI = 6.7 +/- 0.69, Sham = 2.5 +/- 0.38; P = 0

264 hite matter tracts of the genu of the corpus callosum that connect the two hemispheres of the prefron

265 nnectivity in individuals in whom the corpus callosum (the major commissure between the hemispheres)

266 ith the volumetric measurement of the corpus callosum - the values were 73% and 71%, respectively, an

267 repair deficiency is agenesis of the corpus callosum, the cause of which has not been established.

268 axons than in wild-type mice and, in corpus callosum, the myelin is thinner than in controls.

269 (RLIC), the body and splenium of the corpus callosum, the superior and posterior corona radiata, and

270 taxon of eutherians but do not have a corpus callosum; their intercortical commissural neurons instea

271 Reduced corpus callosum thickness confirmed trend-level observations fr

272 Glial cells in the cortex and corpus callosum underwent delayed FJB staining from d7 to d28,

273 sotropy (FA) index measurement of the corpus callosum using diffusion tensor imaging.

274 well as the areas and volumes of the corpus callosum, ventricular system, hippocampus, amygdala, cer

275 ncy may contribute to agenesis of the corpus callosum via reduction in CUX1(+) neurons.

276 e; a QTL on chromosome 1p influencing corpus callosum volume and a region on chromosome 7p linked to

277 th alcohol use behavior and posterior corpus callosum volume, both in a subset of COGA and in the UK

278 average, but lower right caudate and corpus callosum volume, relative to 22q-del carriers.

279 ric intrinsic connectivity and larger corpus callosum volume.

280 anisotropy within the splenium of the corpus callosum was found in each NDD group, compared with the

281 nal corticospinal tract and bilateral corpus callosum was increased but sensorimotor CBF was decrease

282 come were significantly improved, the corpus callosum was intact, whereas this was not the case in cT

283 toms and structure of the genu of the corpus callosum was negative in youths with TBI and positive in

284 Absence of the corpus callosum was noted at screening prenatal head ultrasonog

285 Luxol fast blue staining of the corpus callosum was significantly greater in the BCCAO rats tre

286 thinner (Kif21b and Wdr89), or absent corpus callosum (Wdr47), revealing a common role for WDR genes

287 bution volume (VT), determined in the corpus callosum, we calculated the binding potential (receptor

288 bellar atrophy, and hypoplasia of the corpus callosum were consistent among individuals harboring rec

289 al pattern of the cortex and abnormal corpus callosum were noted on MRI of three individuals, and the

290 ipsilateral cortical regions and the corpus callosum were significantly heritable, ranging from tota

291 ssociations were found in the genu of corpus callosum which accounted for short-term memory binding i

292 ter tracts, including the body of the corpus callosum, which are most commonly affected by diffuse ax

293 ral palsy and partial agenesis of the corpus callosum, while histochemical and biochemical analyses o

294 nt with a left splenium lesion of the corpus callosum who perceives the right side of faces as 'melte

295 ularly significant with regard to the corpus callosum, whose development undergoes several dynamic st

296 In WT mice, microglia expanded in the corpus callosum with age, whereas aged Trem2(-/-) mice had fewe

297 Dysplasia of corpus callosum with focal thinning of the posterior part and c

298 imaging ranging from agenesis of the corpus callosum with hydrocephalus to cystic formations, abnorm

299 ter with sparing of the U fibers, the corpus callosum with sparing of the outer blades, the basis pon

300 ontal cortex to 0.46 mL cm(-3) in the corpus callosum, with intermediate VT values in subcortical str