ライフサイエンスコーパス: callus

1 crophages, predominated in the maturing hard callus.

2 s, stems, flowers, roots and seeds) and from callus.

3 mprovement of angiogenesis in the COX-2(-/-) callus.

4 MMP-9 expression in the COX-2(-/-) fracture callus.

5 was regenerated from independently selected callus.

6 teral branches and a reduced ability to form callus.

7 hondrocytes, and osteoblasts of the fracture callus.

8 n of the transcription factor LEAFY (LFY) in callus.

9 don, whereas the abaxial side evolves into a callus.

10 on, and delayed ossification of the fracture callus.

11 evels of ACT7 protein than did the wild-type callus.

12 nt in the nuclei of cotyledons and endosperm callus.

13 as been transformed into Black Mexican Sweet callus.

14 y and polyamines were measured in transgenic callus.

15 y microprojectile bombardment of embryogenic callus.

16 oot meristems and organ primordia but not in callus.

17 ranging from 7% to 150% of wild-type hNP 588 callus.

18 ium and dynamically expressed in pluripotent callus.

19 neutralization of Th17 cell influx into the callus.

20 ed DNA methylation patterns of the embryonic callus.

21 ion is linearly related to the strain in the callus.

22 t TBI induces the formation of a more robust callus.

23 , and indica rice (Oryza sativa var. indica) callus.

24 stiffness and bony bridging of the fracture callus.

25 e TGF-beta1 protein levels in mouse fracture callus.

26 osteoblasts but no chondrocytes in fracture calluses.

27 lla induced from the (60)Co-gamma-irradiated calluses.

28 r DH sites detected in both the seedling and callus, 31% displayed significantly different levels of

29 ), lesser toe deformities (60.0%), corns and calluses (58.2%), bunions (37.1%), and signs of fungal i

30 In the knot callus, a high density of defects originate within 1mm o

31 al. (2014) show that muscle and the fracture callus actively position fractured neonatal bone fragmen

32 cells were localized throughout the healing callus after fracture.

33 (1.7-3.7 micromol g(-1) fresh weight in the callus and 0.6-2.0 micromol g(-1) fresh weight in the le

34 healing results in reduction of the fracture callus and a delay in conversion of cartilage to bone.

35 ce-associated signatures within the fracture callus and accelerated fracture healing.

36 als may both augment the size of the initial callus and boost its ossification into reparative bone.

37 nges were detected between total embryogenic callus and callus enriched for transition stage somatic

38 inflammatory macrophages within the fracture callus and decreased the level of inflammatory biomarker

39 signaling reduces the size of the cartilage callus and delays its conversion into bone, resulting in

40 erate for a limited time as undifferentiated callus and do not show the massive deposition of ectopic

41 The presence of DC8 activity in carrot callus and endosperm is consistent with the notion that

42 s (SE) requires the induction of embryogenic callus and establishment of proliferation before plant r

43 ilage-to-bone transformation in the fracture callus and for undisturbed bone healing.

44 n microprojectile bombardment of embryogenic callus and hygromycin selection.

45 MARs on transgene expression levels in maize callus and in transformed maize plants.

46 omplex II ratio differs by factor 37 between callus and leaf, indicating drastic differences in elect

47 r, the amount of mannitol accumulated in the callus and mature fifth leaf (1.7-3.7 micromol g(-1) fre

48 ses identify discrete profiles for embryonic callus and other tissues.

49 e over-expressing GFP-CENH3 and CENH3-YFP in callus and plants is not and can be partly deposited nor

50 The aim of this study was to measure callus and plasma amino acid concentrations in patients

51 egrating automated biofoundry engineering of callus and protoplast cells with single-cell matrix-assi

52 sativa, the technique has been initiated in callus and shoots, but has not been optimized ever since

53 senescence and SASP markers in the fracture callus and significantly accelerated the time course of

54 ons in plants, such as regeneration from the callus and the initiation of flowering.

55 Analyses of callus and untransformed plants regenerated from callus

56 high in mature shoots, but extremely low in callus and young shoots; in E. arvense strobili, it was

57 ccumulate transiently in the murine fracture callus and, in contrast to the skin, their clearance doe

58 8 in Populus led to significant increases in callusing and formation of both stem-born roots and base

59 e embryos, sugarcane (Saccharum officinarum) callus, and indica rice (Oryza sativa var. indica) callu

60 varieties (in vivo plants, in vitro shoots, callus, and suspension cultures) were investigated for t

61 cular invasion of the hypertrophic cartilage callus, and that Mmp9(-/-) mice have non-unions and dela

62 th patient-derived keratinocytes and patient callus; and (3) demonstrate that repeated siRNA treatmen

63 e use of ultrasound in the evaluation of the callus are rare and this could be a method equivalent to

64 ) newly formed bone density (NFBD); 3) total callus area (TCA); 4) osteoclast number (ON) in the call

65 We report that hyperkeratotic calluses arising in the glabrous skin of individuals wit

66 m which embryos develop and from the abaxial callus at five time points over the course of the 4 week

67 c sequence restored their ability to produce callus at rates similar to those of wild-type plants, co

68 increased amount of trabecular bone in MULTI calluses at 21 days post-injury.

69 sts by differentiating between soft and hard callus based on radiodensity.

70 heir biological activities in the P. lunatus callus bioassay, indicating that there may be similariti

71 er in vitro and ex vitro culture conditions (callus biomass, shoot production, and ex vitro survival)

72 However, under HFD, callus bone volume was significantly reduced exclusively

73 parental rDNA expression in root, flower and callus, but not in leaf where D-genome rDNA dominance wa

74 ng to the formation of shoots, new roots, or callus by transferring to the appropriate organ inductio

75 The RNAi suppression of CsMYBF1 in citrus callus caused a down-regulation of many phenylpropanoid

76 A callus cDNA library from the maize inbred Mp708 was scre

77 This type of callus cell may be critical for bridging large bone inju

78 spensable for the proliferative expansion of callus cells in response to injury.

79 GUS) expression cassette was introduced into callus cells via tungsten microparticles, and stable tra

80 In callus cells, an automated transformation platform was e

81 ALDI-MS with the biofoundry, protoplast, and callus cells, differentiating engineered and unengineere

82 hus despite contributing to only a subset of callus cells, Sox9-positive progenitors play a major rol

83 ian strands were detected in any Arabidopsis callus cells, strands were present in leaf epidermal cel

84 alysis of the DNA damage response network in callus cells.

85 obacco (Nicotiana tabacum) and Ginkgo biloba callus cells.

86 Membranes from one callus clone expressed m1 MAChR at the level of 2.0-2.5

87 in wt mice resulted in significantly larger calluses compared to wt mice without systemic M-CSF trea

88 ssue in response to hormones, and the mutant callus contained at least two to three times lower level

89 The callus culture for stem and leaf explants was initiated

90 Dedifferentiation in callus culture resulted in an increase of the terminal r

91 rentiation, dedifferentiation, and long-term callus culture were consistent among genotypes.

92 putrescine and these metabolites in tobacco callus cultured in vitro.

93 lation in wheat seedlings, crown tissue, and callus cultures after transfer from control (25 degrees

94 ast, no obvious differences could be seen in callus cultures between the transgene and vector control

95 Long-term callus cultures had very long telomeres.

96 f ASA1 mRNA and protein were also similar in callus cultures of mutant and wild type, although the le

97 nts with anticancer properties from in vitro callus cultures of stems and leaves of SM.

98 mbined with the observation that Arabidopsis callus cultures overexpressing CKI1 exhibit a "cytokinin

99 Undifferentiated callus cultures regenerated from the transgenic plants w

100 Hormonal treatments in callus cultures revealed auxin-mediated upregulation of

101 ts, regenerated from immature embryo derived callus cultures were normal, fertile, and transmitted th

102 However, the mutant seedlings and callus cultures will grow in tissue culture in the dark,

103 to a naturally occurring phenomenon whereby callus cultures, upon continued passage, lose their requ

104 eaves were examined with those of respective callus cultures.

105 the measurements of length and width of the callus demonstrated that the differences between results

106 intermediate pluripotent cell mass known as callus derived from Arabidopsis root cells.

107 we isolated a population of early periosteum-callus-derived mesenchymal stem cells (PCDSCs) from the

108 Cactus callus-derived oils were more lethal to medfly (up to 10

109 m preparation of the plant cell materials to callus development is approximately 5 weeks.

110 During callus development, a significant negative correlation w

111 ologous silencing in in vitro cultured wheat callus differ from that in differentiated organs, given

112 The molecular mechanisms underlying callus embryogenic potential are not well understood.

113 owing and/or differentiating cells including callus, emerging leaves, and meristem regions.

114 etected between total embryogenic callus and callus enriched for transition stage somatic embryos.

115 ful efforts at regenerating plants from seed callus, establishing a transient transformation system,

116 at Mmp9(-/-) mice generate a large cartilage callus even when fractured bones are stabilized, which i

117 ies ranging between 4 and 5.3% of transgenic callus events, in addition to generating a high frequenc

118 ilarly, carotenoid levels in PYGG-expressing callus exceeded that in PSY- or GGPS11-overexpression li

119 In this study, extracts of the mutant callus exhibited higher AS activity than wild-type callu

120 sion data from beta-NGF stimulated cartilage callus explants show a promotion in markers associated w

121 n is impaired in loss-of-function mutants of callus-expressed CLAVATA1 (CLV1) and BARELY ANY MERISTEM

122 When larvae were reared on callus expressing the proteinase, their growth was inhib

123 Stem and leaf callus extracts exerted cytotoxic effects towards CCRF-C

124 tion and genome editing, friable embryogenic callus (FEC).

125 efficacy of ultrasound in the evaluation of callus formation after fractures of long bones in childr

126 of ultrasound with radiographs in imaging of callus formation after fractures of long bones in childr

127 ntributed to transient mineralized bone hard callus formation after transplantation into immunodefici

128 chondrocytes leads to a prolonged cartilage callus formation and a delayed osteogenesis initiation a

129 on form of OI, distinguished by hyperplastic callus formation and calcification of the interosseous m

130 to injury results in a near-complete loss of callus formation and rib bone regeneration.

131 d in a near 50% reduction in periosteal bone callus formation at the cortical bone junction as determ

132 ells to the fracture site and impacting hard callus formation by stimulating osteoclastogenesis.

133 previously described stimulation of tobacco callus formation could not be confirmed.

134 ive actin gene, did not significantly affect callus formation from leaf or root tissue.

135 development program is a common mechanism in callus formation from multiple organs.

136 mental defects of the QK mutant and promoted callus formation in A2QK, but not in A2Wt, after heat tr

137 , this peptide growth factor, which promotes callus formation in culture, is proteolytically cleaved

138 Furthermore, callus formation in roots, cotyledons, and petals is blo

139 sal rib 10 are both fractured with extensive callus formation in the later stages of healing.

140 ans such as petals, which clearly shows that callus formation is not a simple reprogramming process b

141 -) mice, as evidenced by restoration of bony callus formation on day 14, a near complete reversal of

142 Overexpression of ESR1 cannot induce callus formation or root formation, suggesting that its

143 Endogenous callus formation precedes specification of postembryonic

144 Callus formation was completely abolished when macrophag

145 rly anabolic progression during endochondral callus formation were investigated.

146 SC sheet-wrapped allografts showed more bony callus formation when compared to allograft alone groups

147 their osteogenic ability and subsequent bony callus formation, and could be used to induce cartilagin

148 s: the inflammatory phase, the soft and hard callus formation, and the remodeling phase.

149 d repression of a regulator of wound-induced callus formation, suggesting that cells in intact tissue

150 urthermore, the quadruple dof mutant reduced callus formation, tissue attachment, vascular regenerati

151 ad to a nonunion as a result of insufficient callus formation.

152 /-) animals and was accompanied by increased callus formation.

153 erentiation, matrix production, and ultimate callus formation.

154 onfirming that the ACT7 gene is required for callus formation.

155 H1 depletion also impairs pluripotent callus formation.

156 n, and could be used to induce cartilaginous callus formation.

157 cells with rooting competence that resembles callus formation.

158 ulating factor-1 significantly enhanced soft callus formation.

159 oted anabolic mechanisms during endochondral callus formation.

160 genous cytokinin in both root-elongation and callus-formation assays.

161 tion of MSC sheets, results showed more bony callus formed between allograft and host bone ends in bo

162 xhibit optimal strain behaviour conducive to callus fracture healing.

163 een RNA isolated from intact bone to that of callus from post-fracture (PF) days 3, 5, 7, and 10 as a

164 nducing the formation of friable embryogenic callus from which highly totipotent embryogenic suspensi

165 ean polar moment of inertia when compared to calluses from FX mice at 21 days post-injury.

166 muCT analysis revealed calluses from MULTI mice had a greater bone and total ti

167 hin granulation tissue at the expanding soft callus front.

168 ties and flat feet, as well as for corns and calluses, fungal signs, edema, ankle joint pain, tendern

169 ere, we show that fractures rapidly expanded callus gammadelta T cells, which led to increased gut pe

170 In callus grown on high (11.5 micromolar) alpha-naphthalene

171 re the main putrescine derivatives, while in callus grown on low (1.5 micromolar) alpha-naphthalene a

172 te that RepA can stimulate cell division and callus growth in culture, and improve maize transformati

173 s production of embryogenic calli and longer callus growth periods were required to generate these la

174 RepA increased transformation frequency and callus growth rate of high type II maize germplasm.

175 nse analysis, but sequence changes caused by callus growth, Agrobacterium incubation medium, virulenc

176 ates and stiffness, reflecting physiological callus growth, and offers a method to simulate the heali

177 Scx-mutant mice demonstrated disrupted callus healing and asymmetry.

178 localization and gene expression, as well as callus healing response.

179 ment led to altered long bone properties and callus healing.

180 ctions showed that the area of the chondriod callus in the aged P10 MSC sheet groups was significantl

181 by dystrophic nails, painful hyperkeratotic calluses in glabrous skin, and lesions involving other e

182 t the chondro-osseous border in the fracture callus, in a region we define as the transition zone (TZ

183 us and untransformed plants regenerated from callus indicated that loss of methylation is stochastica

184 on root elongation, lateral root formation, callus induction and greening, and induction of cytokini

185 A similar DC8 activity time-course during callus induction and seed development suggests that expl

186 However, although callus induction from dgt hypocotyl explants required au

187 Reentry into cell cycle after callus induction from differentiated root segments repro

188 15 Days After Pollination (DAP) and for the callus induction from this explant, as compared to 23 DA

189 preincubating root explants on an auxin-rich callus induction medium (CIM) and by transferring explan

190 ss involving pre-incubation on an auxin-rich callus induction medium (CIM) during which time root exp

191 ess requiring preincubation on an auxin-rich callus induction medium.

192 Neither callus induction nor root formation was affected by ESR1

193 r hints as to why this stage is relevant for callus induction with successful proliferation and plant

194 CLE1-CLE7 are induced by callus-induction medium and dynamically expressed in plu

195 k, a 33 kDa cysteine proteinase was found in callus initiated from maize (Zea mays L.) resistant to f

196 ling accelerates conversion of the cartilage callus into bone, improving bone healing.

197 ration, where a pluripotent cell mass termed callus is induced.

198 sults revealed that over-expressing CENH3 in callus is lethal while over-expressing GFP-CENH3 and CEN

199 1.27+/-0.38%; stem Sal B: 0.87+/-0.20%) than callus leaf did (leaf RA: 0.28+/-0.02%; leaf Sal B: 0.07

200 d exhibited developmental defects, including callus-like floral organs and fasciated shoot apical mer

201 does not inhibit cell division, resulting in callus-like sporelings with many rhizoids, whereas pharm

202 notype resulting in widespread production of callus-like structures in the mutant.

203 level of sucrose stimulated the formation of callus-like tissue in place of the gland under N-replete

204 an Arabidopsis mutant, tso1, which develops callus-like tissues in place of floral organs.

205 suspension cultures of the maize (Zea mays) callus line.

206 te functional transposase activity in barley callus lines stably transformed with an Ac transposase g

207 , we report that adult Krt9(-/-)mice develop calluses marked by hyperpigmentation that are exclusivel

208 taining amplifiable mRNA from human skin and callus material; (2) quantitatively distinguish the sing

209 ytes may play an essential role in cartilage callus maturation at an early stage of fracture healing.

210 ped using Arabidopsis (Arabidopsis thaliana) callus membranes.

211 t in the stumps and then all over the tendon callus, merging into large, calcified structures, which

212 stromal progenitor (f-BCSP) in the fracture callus of adult mice.

213 MYB11 was responsive to the red spots in the callus of the lip, and PeMYB12 participated in the full

214 resent observations of whirled grain in knot calluses of Populus deltoides (eastern cottonwood), and

215 ajor bioactive components detected in cactus callus oil while (E)-9-Octadecenoic acid, ethyl ester an

216 id were the predominant compounds in moringa callus oil.

217 s showed metal particles and a bone fracture callus on the osseous defect.

218 l lesions as well as PPK-like hyperkeratotic calluses on Krt16(-/-) front and hind paws, which severe

219 PPK, OMIM:613000), which each entail painful calluses on palmar and plantar skin.

220 A1 in transgenic tobacco (Nicotiana tabacum) callus or somatic soybean (Glycine max) embryos resulted

221 O1 DNA binding activity in diabetic fracture calluses (P < 0.05).

222 by hypertrophic chondrocytes during the soft callus phase of endochondral fracture repair.

223 Callus production in this mutant requires the cytokinin

224 L Furthermore, plastid stress-induced apical callus production requires elevated plastidic reactive o

225 ed protein has high homology with an alfalfa callus protein or translationally controlled human or mo

226 ts and with the subjective assessment of the callus quality.

227 <0.01), area of NFC (P <0.01), and ON in the callus region (P <0.01).

228 area (TCA); 4) osteoclast number (ON) in the callus region; and 5) newly formed dental cementum-like

229 penetrated hyperkeratotic PC skin and normal callused regions compared with nonaffected areas, and (2

230 We demonstrate that callus resembles the tip of a root meristem, even if it

231 s campestris pv campestris)-infected plants, callus, roots, and young seedlings.

232 tion of GUS activity in light- or dark-grown callus, roots, silk, developing or mature kernels, the s

233 Bone fracture callus samples were collected and analyzed by X-ray, mic

234 ged bone defects without forming the massive callus seen with rhBMP-2.

235 d to investigate muscle unloading effects on callus shape.

236 compared to allograft alone groups, the bony callus size in aged P10 MSC sheet groups was significant

237 Regarding callus size, the application of M-CSF in wt mice resulte

238 p (P < 0.05), which was reflected by smaller callus size.

239 Specifically, mouse bone fracture callus specimens were extracted into a single solution t

240 se chain reaction analysis for both sites at callus stage, and one DD43 homology-directed recombinati

241 Quantitative analyses revealed that callus stem extracts produced higher amount of RA and Sa

242 t pin displacement decreases as the fracture callus stiffens and that pin motion is linearly related

243 erent phenotype of pnt1 cells in embryos and callus suggest a differential requirement for GPI-anchor

244 We found 58% more DH sites in the callus than in the seedling.

245 ntly higher degree of vascularization of the callus than of the healthy periosteum.

246 uently walk barefoot have thicker and harder calluses than those who typically use footwear.

247 ngs indicate that closed head TBI results in calluses that are larger in size and have an increased b

248 Similar to the regenerating callus, the artificial tissue undergoes intramembranous

249 Because calluses-thickened and hardened areas of the epidermal l

250 Additionally, unlike cushioned footwear, callus thickness does not affect how hard the feet strik

251 However, in contrast to shoes, callus thickness does not trade-off protection, measured

252 e intestine and enhanced their homing to the callus through a CCL20-mediated mechanism.

253 ctor for 81.4 % of the baits screened for in callus tissue and T1 seedlings.

254 shoot apical meristem, and develop a mass of callus tissue at the shoot apex.

255 CUC1 was generally expressed in callus tissue during early incubation on SIM, but later

256 overexpressed, BdbZIP10 protects plants and callus tissue from oxidative stress insults, most likely

257 However, we also find that in vitro callus tissue has a higher mutation rate (per unit time)

258 us act7-1 mutant plants were slow to produce callus tissue in response to hormones, and the mutant ca

259 asis, were both significantly upregulated in callus tissue isolated at the fracture site of metformin

260 Homozygous mutant seedlings and callus tissue produced from rescued seeds appeared norma

261 we observed no silencing in any of the wheat callus tissue tested.

262 AG was produced from plant callus tissue under sterile conditions to avoid the infl

263 Immunolabeling of callus tissue with actin subclass-specific antibodies re

264 ch lower amounts in non-green organs (roots, callus tissue).

265 itivity and enhances shoot regeneration from callus tissue, correlating with enhanced stability of th

266 Proliferation of undifferentiated callus tissue, greening, and the formation of shoot stru

267 st level of AtEBP expression was detected in callus tissue, where ocs elements are very active.

268 on of stably transformed roots directly from callus tissue.

269 LeMir is also expressed in tomato callus tissue.

270 atory and resident cells within the fracture callus tissue.

271 rsensitive (DH) sites from both seedling and callus tissues of rice (Oryza sativa).

272 ree genes causes the intermediate cell mass, callus, to be incompetent to form shoot progenitors, whe

273 Black Mexican Sweetcorn callus transformed with mir1, the gene encoding the 33-k

274 sociated with key events during soft-to-hard callus transition.

275 idization with pTACR7 probe from seedling or callus treated with ABA, salt, dehydration, or heat stre

276 In callus, two of the MAR elements (Adh1 5' MAR and ARS1) r

277 nstructed from leaf, and cold stress treated callus, using high-throughput sequencing technology.

278 Using the Power Doppler callus vascularity was visualized and vascular resistanc

279 dels have shown that EtOH decreases fracture callus volume, diameter, and biomechanical strength.

280 iameter and a short-term benefit in reducing callus volume.

281 Indeterminate callus was generated and maintained from the sporophytes

282 Asymmetry of Scx-mutant callus was not due to muscle unloading.

283 wth of caterpillars reared on the transgenic callus was reduced 60-80%.

284 in vivo using xanthophyll-accumulating rice callus, we propose that disintegration of the cellular s

285 differentially expressed in the seedling and callus were frequently associated with DH sites in both

286 mutant and wild type, although the levels in callus were higher than in leaf tissue.

287 Amino acid concentrations in plasma and callus were measured with HPLC in atrophic nonunions (n

288 g ultrasound examination measurements of the callus were performed.

289 5 compared with 149 mumol/mg; P < 0.0001) in callus were significantly lower in atrophic-nonunion pat

290 Fracture calluses were harvested 7 days, 14 days, 21 days, and 28

291 e common in men, while bunions and corns and calluses were more common in women (p < 0.001).

292 exhibited higher AS activity than wild-type callus when assayed with either glutamine or ammonium su

293 nalysis of microRNAs in M. oleifera leaf and callus which represents an important addition to the exi

294 d in E. fluviatile intercalary meristems and callus (which lacked MLG).

295 epair by forming a bridging structure called callus, which begins as soft tissue and gradually ossifi

296 AtTERT mRNA is 10-20 times more abundant in callus, which has high levels of telomerase activity, ve

297 tes ubiquitous urease activity in leaves and callus while retaining normal embryo-specific urease act

298 most abundant conserved microRNA in leaf and callus, while microRNA393 was most abundantly expressed

299 RI) and the degree of vascularization of the callus with a subjective radiological assessment of the

300 ractured bone because of a larger periosteal callus with newly formed bone without changing the plasm