戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 s ZINC58368839, brilliant blue G, KN-62, and calmidazolium.
2 did the calmodulin (CaM) antagonists W-7 and calmidazolium.
3 n is attenuated by the calmodulin antagonist calmidazolium.
4  calmodulin antagonists, trifluoperazine and calmidazolium.
5 ibitor, trifluoperazine, but not by another, calmidazolium.
6 h necessary and sufficient for inhibition by calmidazolium.
7                                              Calmidazolium (10 microM), a potent calmodulin inhibitor
8                                              Calmidazolium (100 nM), a calmodulin inhibitor, inhibite
9 tion of the Egr-1 induction by H89 (48%) and calmidazolium (35%), but not by mitogen-activated protei
10  or the calmodulin inhibitors mastoparan and calmidazolium (5 microM), did not alter the action of Ca
11                           Preincubation with calmidazolium, a calmodulin antagonist, produced modest
12 ct was blocked by pretreating the cells with calmidazolium, a calmodulin inhibitor.
13                           In the presence of calmidazolium, a CaM inhibitor, S235D showed high P(f) a
14 brane patches, Trp4 is activated strongly by calmidazolium, an antagonist of CaM, and a high (50 micr
15  that prevents CaM from binding to Trp3, and calmidazolium, an inactivator of Ca(2+)/CaM.
16 ucturally distinct inhibitors (fluphenazine, calmidazolium and a W-7 analogue) of the Ca2+-binding re
17 be blocked by calmodulin antagonists W-7 and calmidazolium and CaM kinase inhibitor KN-93.
18 ) with Smart-3SEQ and examine the effects of calmidazolium and fludrocortisone-induced perturbation o
19                                              Calmidazolium and W-7 also activated a persistent inward
20 ongly activated by the calmodulin inhibitors calmidazolium and W-7 in on-cell and excised patches.
21                 While calmodulin inhibitors (calmidazolium and W-7; each n = 5) decreased baseline CB
22 ructurally distinct CaM antagonists, W-7 and calmidazolium, and by CaM-dependent protein kinase II in
23                    The calmodulin inhibitor, calmidazolium, and the tyrosine kinase inhibitor, genist
24 e derivatives W-7/W-13, trifluoperazine, and calmidazolium, are used widely to investigate the role o
25 s of calmodulin activity, compound 48/80 and calmidazolium, blocked both curvature and gravity-induce
26  this was inhibited by genistein, TMB-8, and calmidazolium but not by pertussis toxin or GF109203X.
27                        Moreover, infusion of calmidazolium (calmodulin inhibitor) with GR-73632 preve
28  force production, we studied the effects of calmidazolium (CDZ) on steady-state force and the rate o
29                  Exposure of myofilaments to calmidazolium (CDZ), which binds to cTnC and increases i
30                      One of these compounds, calmidazolium chloride (CLC), rapidly and efficiently co
31  by use of the protofibril-stabilizing agent calmidazolium chloride.
32 th KN-62, or inclusion of the CaM inhibitor, calmidazolium, did not prevent agonist-induced inhibitio
33 retreated with calmodulin inhibitors (W-7 or calmidazolium) exhibited an attenuated ERK response to i
34  Moreover, channel activation was blocked by calmidazolium (IC(50) = 5 microm), suggesting a role for
35 The ACV-ACII fusion protein was inhibited by calmidazolium (IC(50), approximately 20 microM) as well
36 odulin antagonists, trifluoperazine, W7, and calmidazolium, impaired this cleavage, indicating comple
37                                              Calmidazolium inhibited the activities of both full-leng
38 is insensitive to adenosine analogs and that calmidazolium inhibits AC activity by a novel, noncompet
39                    Blockade of calmodulin by calmidazolium or blockade of CaM kinase II with either K
40 ogical salt solution (PSS) containing either calmidazolium or W-7, both known antagonists of CaM.
41 er antagonists, brilliant blue G, KN-62, and calmidazolium, our data imply an overlapping but distinc
42                          Both nifedipine and calmidazolium partially blocked nocturnal increases in m
43  of the (Ca(2+)+Mg(2+))-ATPase, 10(-5) mol/L calmidazolium (R24571) was added to the isolated plasma
44                                         This calmidazolium-reduced activity could then be stimulated
45 dulin antagonists (W-7, trifluoperazine, and calmidazolium) resulted in the robust release of arachid
46 aM), pretreatment of islets with CaM blocker calmidazolium showed effects very similar to those of Sy
47 y alphaBgTx and by the calmodulin antagonist calmidazolium, suggesting that Ca2+ entry through alpha7
48  The application of the calmodulin inhibitor calmidazolium via the intracellular pipette solution did
49                        Inhibition of ACIX by calmidazolium was mediated by direct interaction with th
50 almodulin-dependent kinase II, melittin, and calmidazolium were effective inhibitors of CLNMT and eac
51 pH (pH(o)), and by the calmodulin inhibitor, calmidazolium, whereas it is acutely activated by NH(4)(
52 modulin antagonists, trifluoperazine and the calmidazolium, with IC50 values of 0.25-1.5 microM.